Initial characterization of polyductin, the PKHD1 gene product (2003)
- Authors:
- Autor USP: ONUCHIC, LUIZ FERNANDO - FM
- Unidade: FM
- Subjects: NEFROLOGIA; GENES
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2003
- Source:
- Título: Journal of the American Society of Nephrology
- Volume/Número/Paginação/Ano: v. 14
- Conference titles: Accredited Sponsor: The American Society of Nephrology
-
ABNT
MENEZES, L. F. et al. Initial characterization of polyductin, the PKHD1 gene product. Journal of the American Society of Nephrology. Philadelphia: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 28 dez. 2025. , 2003 -
APA
Menezes, L. F., Cai, Y., Nagasawa, Y., Almeida, F., Somlo, S., Guay Woodford, L. M., et al. (2003). Initial characterization of polyductin, the PKHD1 gene product. Journal of the American Society of Nephrology. Philadelphia: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Menezes LF, Cai Y, Nagasawa Y, Almeida F, Somlo S, Guay Woodford LM, Germino GG, Onuchic LF. Initial characterization of polyductin, the PKHD1 gene product. Journal of the American Society of Nephrology. 2003 ; 14[citado 2025 dez. 28 ] -
Vancouver
Menezes LF, Cai Y, Nagasawa Y, Almeida F, Somlo S, Guay Woodford LM, Germino GG, Onuchic LF. Initial characterization of polyductin, the PKHD1 gene product. Journal of the American Society of Nephrology. 2003 ; 14[citado 2025 dez. 28 ] - Somatic mutation in individual liver cysts supports a two-hit model of cystogenesis in autosomal dominat polycystic kidney disease
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- Polycystin-1, the gene product of PKD1, induces resistance to apoptosis and spontaneous tubulogenesis in MDCK cells
- Pkd1 haploinsufficiency increases functional and morphological damage following renal ischemia /repersufion in mice: extend analysis
- Ischemia/reperfusion injury determines increased early tubular dysfunction in Pkd1 haploinsufficient mice compared to wil types
- Biliary and Pancreatic Dysgenesis in Mice Harboring a Mutation in Pkhd1
- Genomic organization of the KIAA0057 gene that encodes a TRAM-like protein and its exclusion as a polycystic kidney and hepatic disease 1 (PKHD1) candidate gene
- Bases moleculares e celulares da doença renal policística autossômica recessiva
- Clinical aspects of autosomal recessive polycystic kidney disease
- Intragenic motifs regulate the transcriptional complexity of Pkhd1/PKHD1
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