Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero (2002)
- Authors:
- USP affiliated authors: PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - ICB ; FORTES, ZULEICA BRUNO - ICB ; SCAVONE, CRISTOFORO - ICB ; CARVALHO, MARIA HELENA CATELLI DE - ICB ; NIGRO, DOROTHY - ICB
- Unidade: ICB
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2002
- Source:
- Título: Journal of Cardiovascular Pharmacology
- ISSN: 0160-2446
- Volume/Número/Paginação/Ano: v. 40, n. 4, p. 501-509, 2002
-
ABNT
FRANCOS, Maria do Carmo P. et al. Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero. Journal of Cardiovascular Pharmacology, v. 40, n. 4, p. 501-509, 2002Tradução . . Acesso em: 26 jan. 2026. -
APA
Francos, M. do C. P., Dantas, A. P. V., Akamine, E. H., Kawamoto, E. M., Fortes, Z. B., Scavone, C., et al. (2002). Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero. Journal of Cardiovascular Pharmacology, 40( 4), 501-509. -
NLM
Francos M do CP, Dantas APV, Akamine EH, Kawamoto EM, Fortes ZB, Scavone C, Passaglia R de CAT, Carvalho MHC de, Nigro D. Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero. Journal of Cardiovascular Pharmacology. 2002 ; 40( 4): 501-509.[citado 2026 jan. 26 ] -
Vancouver
Francos M do CP, Dantas APV, Akamine EH, Kawamoto EM, Fortes ZB, Scavone C, Passaglia R de CAT, Carvalho MHC de, Nigro D. Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero. Journal of Cardiovascular Pharmacology. 2002 ; 40( 4): 501-509.[citado 2026 jan. 26 ] - In vivo evidence of oxidative stress in intrauterine programming of hypertensive in wistar rats
- Nitric oxide synthase, angiotensin AT1- and AT2- receptors and bradykinin B2-receptor in male and female spontaneously hypertensive rats treated with losartan
- Differential effect of losartan in female and male spontaneously hypertensive rats
- Nitric oxide synthase, angiotensin AT1- and AT2- receptors and bradykinin B2-receptor in male and female spontaneously hypertensive rats treated with losartan
- Intrauterine undernutrition expression and activity of the endothelial nitric oxide synthase in male and female adult offspring
- Insulin corrects the altered vascular reactivity without restoring the reduced nitric oxide synthase activity in diabetic female rats
- Metformin treatment restores the altered microvascular reactivity in neonatal streptozotocin-induced diabetic rats increasing NOS activity, but not NOS expression
- Enhanced oxidative stress as a potential mechanism underlyng the programming of hypertension in utero
- Role of reactive oxygen species, nitric oxide synthase enzyme and angiotensin AT1 receptor in male and female spontaneously hypertensive rats treated with losartan
- Insulin and nitric oxide synthase in diabetic female rats
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