Insulin and nitric oxide synthase in diabetic female rats (2004)
- Authors:
- USP affiliated authors: CARVALHO, MARIA HELENA CATELLI DE - ICB ; NIGRO, DOROTHY - ICB ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - ICB ; FORTES, ZULEICA BRUNO - ICB ; SCAVONE, CRISTOFORO - ICB
- Unidade: ICB
- Assunto: FARMACOLOGIA
- Language: Português
- Imprenta:
- Conference titles: Jornada de Farmacologia Qualidade Científica
-
ABNT
AKAMINE, Eliana Hiromi et al. Insulin and nitric oxide synthase in diabetic female rats. 2004, Anais.. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2004. . Acesso em: 26 jan. 2026. -
APA
Akamine, E. H., Kawamoto, E. M., Scavone, C., Nigro, D., Carvalho, M. H. C. de, Tostes, R. de C. A., & Fortes, Z. B. (2004). Insulin and nitric oxide synthase in diabetic female rats. In . São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Akamine EH, Kawamoto EM, Scavone C, Nigro D, Carvalho MHC de, Tostes R de CA, Fortes ZB. Insulin and nitric oxide synthase in diabetic female rats. 2004 ;[citado 2026 jan. 26 ] -
Vancouver
Akamine EH, Kawamoto EM, Scavone C, Nigro D, Carvalho MHC de, Tostes R de CA, Fortes ZB. Insulin and nitric oxide synthase in diabetic female rats. 2004 ;[citado 2026 jan. 26 ] - In vivo evidence of oxidative stress in intrauterine programming of hypertensive in wistar rats
- Nitric oxide synthase, angiotensin AT1- and AT2- receptors and bradykinin B2-receptor in male and female spontaneously hypertensive rats treated with losartan
- Differential effect of losartan in female and male spontaneously hypertensive rats
- Nitric oxide synthase, angiotensin AT1- and AT2- receptors and bradykinin B2-receptor in male and female spontaneously hypertensive rats treated with losartan
- Intrauterine undernutrition expression and activity of the endothelial nitric oxide synthase in male and female adult offspring
- Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero
- Insulin corrects the altered vascular reactivity without restoring the reduced nitric oxide synthase activity in diabetic female rats
- Metformin treatment restores the altered microvascular reactivity in neonatal streptozotocin-induced diabetic rats increasing NOS activity, but not NOS expression
- Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero
- Enhanced oxidative stress as a potential mechanism underlyng the programming of hypertension in utero
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