Enhanced oxidative stress as a potential mechanism underlyng the programming of hypertension in utero (2002)
- Authors:
- USP affiliated authors: SCAVONE, CRISTOFORO - ICB ; CARVALHO, MARIA HELENA CATELLI DE - ICB ; NIGRO, DOROTHY - ICB ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - ICB ; FORTES, ZULEICA BRUNO - ICB
- Unidade: ICB
- Assunto: FARMACOLOGIA
- Language: Português
- Imprenta:
- Publisher: A sociedade
- Publisher place: Águas de Lindóia
- Date published: 2002
- Source:
- Título do periódico: Anais
- Conference titles: Congresso Brasileiro de Farmacologia e Terapêutica Experimental
-
ABNT
FRANCO, M C P. et al. Enhanced oxidative stress as a potential mechanism underlyng the programming of hypertension in utero. 2002, Anais.. Águas de Lindóia: A sociedade, 2002. . Acesso em: 24 abr. 2024. -
APA
Franco, M. C. P., Dantas, A. P. V., Akamine, E. H., Oliveira, M. A., Kawamoto, E. M., Scavone, C., et al. (2002). Enhanced oxidative stress as a potential mechanism underlyng the programming of hypertension in utero. In Anais. Águas de Lindóia: A sociedade. -
NLM
Franco MCP, Dantas APV, Akamine EH, Oliveira MA, Kawamoto EM, Scavone C, Carvalho MHC de, Nigro D, Fortes ZB. Enhanced oxidative stress as a potential mechanism underlyng the programming of hypertension in utero. Anais. 2002 ;[citado 2024 abr. 24 ] -
Vancouver
Franco MCP, Dantas APV, Akamine EH, Oliveira MA, Kawamoto EM, Scavone C, Carvalho MHC de, Nigro D, Fortes ZB. Enhanced oxidative stress as a potential mechanism underlyng the programming of hypertension in utero. Anais. 2002 ;[citado 2024 abr. 24 ] - In vivo evidence of oxidative stress in intrauterine programming of hypertensive in wistar rats
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- Nitric oxide and membrane hyperpolarization are involved in the restoring effect of insulin on angiotensin-(1-7) and bradykinin interaction in type 2 diabetes
- Metformin treatment restores the altered microvascular reactivity in neonatal streptozotocin-induced diabetic rats increasing NOS activity, but not NOS expression
- Nitric oxide synthase, angiotensin AT1- and AT2- receptors and bradykinin B2-receptor in male and female spontaneously hypertensive rats treated with losartan
- Differential effect of losartan in female and male spontaneously hypertensive rats
- Nitric oxide synthase, angiotensin AT1- and AT2- receptors and bradykinin B2-receptor in male and female spontaneously hypertensive rats treated with losartan
- Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero
- In vivo evidence of oxidative stress in intrauterine programming of hypertensive in Wistar rats
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