Induction of fos and jun genes and ap-i activation by acth, phorbol ester and dcamp in mouse adrenocortical cells (1994)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; KIMURA, EICO - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquimica e Biologia Molecular
- Publisher place: São Paulo
- Date published: 1994
- Source:
- Título: Programa e Resumos
- Conference titles: Reuniao Anual da Sociedade Brasileira de Bioquimica e Biologia Molecular
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ABNT
LEPIQUE, Ana Paula e KIMURA, Eico e ARMELIN, Hugo Aguirre. Induction of fos and jun genes and ap-i activation by acth, phorbol ester and dcamp in mouse adrenocortical cells. 1994, Anais.. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular, 1994. . Acesso em: 18 abr. 2026. -
APA
Lepique, A. P., Kimura, E., & Armelin, H. A. (1994). Induction of fos and jun genes and ap-i activation by acth, phorbol ester and dcamp in mouse adrenocortical cells. In Programa e Resumos. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular. -
NLM
Lepique AP, Kimura E, Armelin HA. Induction of fos and jun genes and ap-i activation by acth, phorbol ester and dcamp in mouse adrenocortical cells. Programa e Resumos. 1994 ;[citado 2026 abr. 18 ] -
Vancouver
Lepique AP, Kimura E, Armelin HA. Induction of fos and jun genes and ap-i activation by acth, phorbol ester and dcamp in mouse adrenocortical cells. Programa e Resumos. 1994 ;[citado 2026 abr. 18 ] - Dna synthesis inhibition by adrenocorticotropic hormone (acth) and a phorbol ester in y-1 corticoadrenal cells
- Dual action of acth on adrenal cell cycle: stimulation (early g1) and inhibition (late g1)
- Control of 'G IND.0' -> 'G IND.1' -> s in adrenocortical cell cycle by acth and phorbol ester
- C-fos oncogene transcription pattern in mouse malignant adrenocortical cell cultures
- Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells
- Role of proto-oncogene c-ki-ras amplification and overexpression in the malignancy of y-1 adrenocortical tumor cells
- Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells
- Growth control in mouse y-1 adrenocortical tumor cells: role of c-ki-ras amplification and effects of alth
- Phorbol ester mimics acth action in corticoadrenal cells stimulating steroidogenesis blocking cell cycle, changing cell shape, and inducing c-fos proto-oncogene expression
- Transcriptional pattern of c-fos oncogene in mouse malignant adrenocortical cell cultures
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