Caloric restriction increases mitochondrial calcium uptake rate and retention capacity in mouse livers (2016)
- Autores:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Assuntos: MITOCÔNDRIAS; FÍGADO; CÁLCIO
- Idioma: Inglês
- Imprenta:
- Editora: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
- Local: Natal
- Data de publicação: 2016
- Fonte:
- Título do periódico: Abstracts
- Nome do evento: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology (SBBq)
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ABNT
MENEZES FILHO, Sergio Luiz e AMIGO, Ignacio e KOWALTOWSKI, Alicia Juliana. Caloric restriction increases mitochondrial calcium uptake rate and retention capacity in mouse livers. 2016, Anais.. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2016. . Acesso em: 25 set. 2024. -
APA
Menezes Filho, S. L., Amigo, I., & Kowaltowski, A. J. (2016). Caloric restriction increases mitochondrial calcium uptake rate and retention capacity in mouse livers. In Abstracts. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). -
NLM
Menezes Filho SL, Amigo I, Kowaltowski AJ. Caloric restriction increases mitochondrial calcium uptake rate and retention capacity in mouse livers. Abstracts. 2016 ;[citado 2024 set. 25 ] -
Vancouver
Menezes Filho SL, Amigo I, Kowaltowski AJ. Caloric restriction increases mitochondrial calcium uptake rate and retention capacity in mouse livers. Abstracts. 2016 ;[citado 2024 set. 25 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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