ATP production in HnRNP K-overexpressing HEK293 cells (2012)
- Autores:
- Autores USP: LEOPOLDINO, ANDRÉIA MACHADO - FCFRP ; CURTI, CARLOS - FCFRP
- Unidade: FCFRP
- Assuntos: PROTEÍNAS; BIOQUÍMICA CELULAR; NEOPLASIAS; MITOCÔNDRIAS
- Idioma: Inglês
- Imprenta:
- Local: Foz do Iguaçu
- Data de publicação: 2012
- Nome do evento: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
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ABNT
GARCIA, C. B. et al. ATP production in HnRNP K-overexpressing HEK293 cells. 2012, Anais.. Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 26 abr. 2024. -
APA
Garcia, C. B., Reis, F. H. Z., Alberici, L. C., Curti, C., & Leopoldino, A. M. (2012). ATP production in HnRNP K-overexpressing HEK293 cells. In . Foz do Iguaçu: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Garcia CB, Reis FHZ, Alberici LC, Curti C, Leopoldino AM. ATP production in HnRNP K-overexpressing HEK293 cells. 2012 ;[citado 2024 abr. 26 ] -
Vancouver
Garcia CB, Reis FHZ, Alberici LC, Curti C, Leopoldino AM. ATP production in HnRNP K-overexpressing HEK293 cells. 2012 ;[citado 2024 abr. 26 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
- Potential modulation of PTEN phosphatase by PP2A and oxidative stress
- SET regulates levels of HnRNP K and its binding to nucleic acid in HNSCC
- The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
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