Mitochondrial ATP-sensitive 'K POT. +' channels avoid oxidative stress during ischemia/reperfusion (2005)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: MITOCÔNDRIAS; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher place: Águas de Lindóia
- Date published: 2005
- Source:
- Título do periódico: Abstratcs
- Conference titles: Meeting of the South American Group of the Society for Free Radical Biology and Medicine
-
ABNT
FACUNDO, Hebert Di Tarso Fernandes e PAULA, Juliane Gomes de e KOWALTOWSKI, Alicia Juliana. Mitochondrial ATP-sensitive 'K POT. +' channels avoid oxidative stress during ischemia/reperfusion. 2005, Anais.. Águas de Lindóia: Instituto de Química, Universidade de São Paulo, 2005. . Acesso em: 19 set. 2024. -
APA
Facundo, H. D. T. F., Paula, J. G. de, & Kowaltowski, A. J. (2005). Mitochondrial ATP-sensitive 'K POT. +' channels avoid oxidative stress during ischemia/reperfusion. In Abstratcs. Águas de Lindóia: Instituto de Química, Universidade de São Paulo. -
NLM
Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT. +' channels avoid oxidative stress during ischemia/reperfusion. Abstratcs. 2005 ;[citado 2024 set. 19 ] -
Vancouver
Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT. +' channels avoid oxidative stress during ischemia/reperfusion. Abstratcs. 2005 ;[citado 2024 set. 19 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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