Elevation of resting mitochondrial membrane potential by cyclosporin A, BAPTA-AM and Bcl-2 (2001)
- Autores:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Biophysical Journal
- ISSN: 0006-3495
- Volume/Número/Paginação/Ano: v. 80, n. 1, part 2, p. 2137, 2001
- Nome do evento: Annual Meeting of the Biophysical Society
-
ABNT
SMAILI, Soraya S. et al. Elevation of resting mitochondrial membrane potential by cyclosporin A, BAPTA-AM and Bcl-2. Biophysical Journal. Bethesda: Instituto de Química, Universidade de São Paulo. . Acesso em: 11 maio 2024. , 2001 -
APA
Smaili, S. S., Kowaltowski, A. J., Russell, J. T., & Fiskum, G. (2001). Elevation of resting mitochondrial membrane potential by cyclosporin A, BAPTA-AM and Bcl-2. Biophysical Journal. Bethesda: Instituto de Química, Universidade de São Paulo. -
NLM
Smaili SS, Kowaltowski AJ, Russell JT, Fiskum G. Elevation of resting mitochondrial membrane potential by cyclosporin A, BAPTA-AM and Bcl-2. Biophysical Journal. 2001 ; 80( 1): 2137.[citado 2024 maio 11 ] -
Vancouver
Smaili SS, Kowaltowski AJ, Russell JT, Fiskum G. Elevation of resting mitochondrial membrane potential by cyclosporin A, BAPTA-AM and Bcl-2. Biophysical Journal. 2001 ; 80( 1): 2137.[citado 2024 maio 11 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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