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Filtros : "ULRICH, ALEXANDER HENNING" Limpar

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  • Artigo de periodico

    Effects of nanoflubendazole and purinergic signaling modulation in overcoming neuroblastoma chemoresistance (2025)

    Mello, Renata Siqueira de ; Bento, Carolina Adriane ; Faria, Rafael de Oliveira ; Sampaio, Vanessa Fernandes Arnaud ; Ulrich, Henning ; Miyagi, Mariana Yasue Saito; Araujo, Gabriel Lima Barros de ; Lameu, Claudiana

    Fonte: Purinergic Signalling. Unidades: IQ, FCF

    Assuntos: NEUROBLASTOMA, QUIMIOTERAPIA

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      MELLO, Renata Siqueira de et al. Effects of nanoflubendazole and purinergic signaling modulation in overcoming neuroblastoma chemoresistance. Purinergic Signalling, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s11302-025-10078-7. Acesso em: 01 dez. 2025.

    • APA

      Mello, R. S. de, Bento, C. A., Faria, R. de O., Sampaio, V. F. A., Ulrich, H., Miyagi, M. Y. S., et al. (2025). Effects of nanoflubendazole and purinergic signaling modulation in overcoming neuroblastoma chemoresistance. Purinergic Signalling. doi:10.1007/s11302-025-10078-7

    • NLM

      Mello RS de, Bento CA, Faria R de O, Sampaio VFA, Ulrich H, Miyagi MYS, Araujo GLB de, Lameu C. Effects of nanoflubendazole and purinergic signaling modulation in overcoming neuroblastoma chemoresistance [Internet]. Purinergic Signalling. 2025 ;[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-025-10078-7

    • Vancouver

      Mello RS de, Bento CA, Faria R de O, Sampaio VFA, Ulrich H, Miyagi MYS, Araujo GLB de, Lameu C. Effects of nanoflubendazole and purinergic signaling modulation in overcoming neuroblastoma chemoresistance [Internet]. Purinergic Signalling. 2025 ;[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-025-10078-7

  • Artigo de periodico

    Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology (2025)

    Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Mori, Yasuo; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de

    Fonte: Molecular Neurobiology. Unidades: IQ, ICB

    Assuntos: DELEÇÃO DE GENES, DOENÇA DE PARKINSON, RATOS

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      FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, v. 62, p. 5333-5346, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s12035-024-04611-9. Acesso em: 01 dez. 2025.

    • APA

      Ferreira, A. F. F., Ulrich, H., Mori, Y., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, 62, 5333-5346. doi:10.1007/s12035-024-04611-9

    • NLM

      Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9

    • Vancouver

      Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9

  • Artigo de periodico

    Self assembly and 3D bioprinting of neurospheres and evaluation of caffeine and photobiomodulation effects in an alzheimer’s disease in vitro model (2025)

    Salles, Geisa Rodrigues; Granato, Alessandro Eustaquio Campos ; Viero, Fernanda Tibolla ; Soares, Cristina Pacheco ; Ferreira, Sergio T ; Porcionatto, Marimelia; Ulrich, Henning

    Fonte: Stem Cell Reviews and Reports. Unidade: IQ

    Assuntos: IMPRESSÃO 3-D, CAFEÍNA, DOENÇA DE ALZHEIMER, CÉLULAS-TRONCO, DOENÇA DE PARKINSON

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      SALLES, Geisa Rodrigues et al. Self assembly and 3D bioprinting of neurospheres and evaluation of caffeine and photobiomodulation effects in an alzheimer’s disease in vitro model. Stem Cell Reviews and Reports, v. 21, p. 988–1000, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s12015-025-10850-7. Acesso em: 01 dez. 2025.

    • APA

      Salles, G. R., Granato, A. E. C., Viero, F. T., Soares, C. P., Ferreira, S. T., Porcionatto, M., & Ulrich, H. (2025). Self assembly and 3D bioprinting of neurospheres and evaluation of caffeine and photobiomodulation effects in an alzheimer’s disease in vitro model. Stem Cell Reviews and Reports, 21, 988–1000. doi:10.1007/s12015-025-10850-7

    • NLM

      Salles GR, Granato AEC, Viero FT, Soares CP, Ferreira ST, Porcionatto M, Ulrich H. Self assembly and 3D bioprinting of neurospheres and evaluation of caffeine and photobiomodulation effects in an alzheimer’s disease in vitro model [Internet]. Stem Cell Reviews and Reports. 2025 ; 21 988–1000.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s12015-025-10850-7

    • Vancouver

      Salles GR, Granato AEC, Viero FT, Soares CP, Ferreira ST, Porcionatto M, Ulrich H. Self assembly and 3D bioprinting of neurospheres and evaluation of caffeine and photobiomodulation effects in an alzheimer’s disease in vitro model [Internet]. Stem Cell Reviews and Reports. 2025 ; 21 988–1000.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s12015-025-10850-7

  • Artigo de periodico

    Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology (2025)

    Ferreira, Ana Flávia Fernandes ; Mori, Yasuo ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de

    Fonte: Molecular Neurobiology. Unidades: ICB, IQ

    Assuntos: FISIOLOGIA, DOENÇA DE PARKINSON, PROTEÍNAS DA MEMBRANA, APOPTOSE, ATIVIDADE MOTORA, RECEPTORES DE DOPAMINA, ESTRESSE OXIDATIVO, CAMUNDONGOS

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    • ABNT

      FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, v. 62, n. 4, p. 5333-5346, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-024-04611-9. Acesso em: 01 dez. 2025.

    • APA

      Ferreira, A. F. F., Mori, Y., Feng, Z. -P., Sun, H. -S., Ulrich, H., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, 62( 4), 5333-5346. doi:10.1007/s12035-024-04611-9

    • NLM

      Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1007/s12035-024-04611-9

    • Vancouver

      Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1007/s12035-024-04611-9

  • Artigo de periodico

    Partners in health and disease: pineal gland and purinergic signalling (2025)

    Markus, Regina P ; Sousa, Kassiano S ; Ulrich, Henning ; Ferreira, Zulma S

    Fonte: Purinergic Signalling. Unidades: IB, IQ

    Assuntos: IMUNOLOGIA CELULAR, MELATONINA, GLÂNDULA PINEAL

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      MARKUS, Regina P et al. Partners in health and disease: pineal gland and purinergic signalling. Purinergic Signalling, v. 21, p. 99–112, 2025Tradução . . Disponível em: https://doi.org/10.1007/s11302-024-10037-8. Acesso em: 01 dez. 2025.

    • APA

      Markus, R. P., Sousa, K. S., Ulrich, H., & Ferreira, Z. S. (2025). Partners in health and disease: pineal gland and purinergic signalling. Purinergic Signalling, 21, 99–112. doi:10.1007/s11302-024-10037-8

    • NLM

      Markus RP, Sousa KS, Ulrich H, Ferreira ZS. Partners in health and disease: pineal gland and purinergic signalling [Internet]. Purinergic Signalling. 2025 ; 21 99–112.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1007/s11302-024-10037-8

    • Vancouver

      Markus RP, Sousa KS, Ulrich H, Ferreira ZS. Partners in health and disease: pineal gland and purinergic signalling [Internet]. Purinergic Signalling. 2025 ; 21 99–112.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1007/s11302-024-10037-8

  • Artigo de periodico

    Neurobiology of L-proline: from molecules to behavior (2025)

    Carvalho, Gustavo Almeida de ; Cavalcante, Daniel Pereira; Parreira, Ricardo C ; Chiareli, Raphaela A ; Leoncini, Giovanni Ortiz ; Gomez, Renato Santiago; Ulrich, Henning ; Caixeta, Leonardo Ferreira; Lima, Onésia Cristina Oliveira; Pinto, Mauro Cunha Xavier

    Fonte: Neuroscience. Unidade: IQ

    Assuntos: NEUROPSIQUIATRIA, AMINOÁCIDOS, SISTEMA NERVOSO CENTRAL

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      CARVALHO, Gustavo Almeida de et al. Neurobiology of L-proline: from molecules to behavior. Neuroscience, v. 568, p. 116–129, 2025Tradução . . Disponível em: https://dx.doi.org/10.1016/j.neuroscience.2025.01.036. Acesso em: 01 dez. 2025.

    • APA

      Carvalho, G. A. de, Cavalcante, D. P., Parreira, R. C., Chiareli, R. A., Leoncini, G. O., Gomez, R. S., et al. (2025). Neurobiology of L-proline: from molecules to behavior. Neuroscience, 568, 116–129. doi:10.1016/j.neuroscience.2025.01.036

    • NLM

      Carvalho GA de, Cavalcante DP, Parreira RC, Chiareli RA, Leoncini GO, Gomez RS, Ulrich H, Caixeta LF, Lima OCO, Pinto MCX. Neurobiology of L-proline: from molecules to behavior [Internet]. Neuroscience. 2025 ; 568 116–129.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1016/j.neuroscience.2025.01.036

    • Vancouver

      Carvalho GA de, Cavalcante DP, Parreira RC, Chiareli RA, Leoncini GO, Gomez RS, Ulrich H, Caixeta LF, Lima OCO, Pinto MCX. Neurobiology of L-proline: from molecules to behavior [Internet]. Neuroscience. 2025 ; 568 116–129.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1016/j.neuroscience.2025.01.036

  • Artigo de periodico

    GlyT1 inhibition promotes neuroprotection in the middle cerebral artery occlusion model through the activation of GluN2A-containing NMDAR (2025)

    Cavalcante, Daniel Pereira; Nunes, Antonio Ítalo dos Santos; Silva, Eduardo Rosa da; Carvalho, Gustavo Almeida de; Chiareli, Raphaela Almeida; Lima, Onésia Cristina Oliveira; Leoncini, Giovanni Ortiz; Ulrich, Henning ; Gomez, Renato Santiago; Pinto, Mauro Cunha Xavier

    Fonte: Experimental Neurology. Unidade: IQ

    Assuntos: ARTÉRIA CEREBRAL MÉDIA, PROTEÍNAS QUINASES

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      CAVALCANTE, Daniel Pereira et al. GlyT1 inhibition promotes neuroprotection in the middle cerebral artery occlusion model through the activation of GluN2A-containing NMDAR. Experimental Neurology, v. 383, p. 1-13 art. 115006, 2025Tradução . . Disponível em: https://dx.doi.org/10.1016/j.expneurol.2024.115006. Acesso em: 01 dez. 2025.

    • APA

      Cavalcante, D. P., Nunes, A. Í. dos S., Silva, E. R. da, Carvalho, G. A. de, Chiareli, R. A., Lima, O. C. O., et al. (2025). GlyT1 inhibition promotes neuroprotection in the middle cerebral artery occlusion model through the activation of GluN2A-containing NMDAR. Experimental Neurology, 383, 1-13 art. 115006. doi:10.1016/j.expneurol.2024.115006

    • NLM

      Cavalcante DP, Nunes AÍ dos S, Silva ER da, Carvalho GA de, Chiareli RA, Lima OCO, Leoncini GO, Ulrich H, Gomez RS, Pinto MCX. GlyT1 inhibition promotes neuroprotection in the middle cerebral artery occlusion model through the activation of GluN2A-containing NMDAR [Internet]. Experimental Neurology. 2025 ; 383 1-13 art. 115006.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1016/j.expneurol.2024.115006

    • Vancouver

      Cavalcante DP, Nunes AÍ dos S, Silva ER da, Carvalho GA de, Chiareli RA, Lima OCO, Leoncini GO, Ulrich H, Gomez RS, Pinto MCX. GlyT1 inhibition promotes neuroprotection in the middle cerebral artery occlusion model through the activation of GluN2A-containing NMDAR [Internet]. Experimental Neurology. 2025 ; 383 1-13 art. 115006.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1016/j.expneurol.2024.115006

  • Artigo de periodico

    Artemisinin exerts antidepressant-like effects via activation of AKT and ERK signaling pathways (2025)

    Lin, Ruohong; Zhou, Zhiwei; Jiang, Yizhou; Liu, Song; Jinfeng Xie; Wang, Haitao; Ulrich, Henning ; Zheng, Wenhua

    Fonte: Frontiers in Pharmacology. Unidade: IQ

    Assunto: ANTIDEPRESSIVOS

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      LIN, Ruohong et al. Artemisinin exerts antidepressant-like effects via activation of AKT and ERK signaling pathways. Frontiers in Pharmacology, v. 16, p. 1-15 art. 1642167, 2025Tradução . . Disponível em: https://dx.doi.org/10.3389/fphar.2025.1642167. Acesso em: 01 dez. 2025.

    • APA

      Lin, R., Zhou, Z., Jiang, Y., Liu, S., Jinfeng Xie,, Wang, H., et al. (2025). Artemisinin exerts antidepressant-like effects via activation of AKT and ERK signaling pathways. Frontiers in Pharmacology, 16, 1-15 art. 1642167. doi:10.3389/fphar.2025.1642167

    • NLM

      Lin R, Zhou Z, Jiang Y, Liu S, Jinfeng Xie, Wang H, Ulrich H, Zheng W. Artemisinin exerts antidepressant-like effects via activation of AKT and ERK signaling pathways [Internet]. Frontiers in Pharmacology. 2025 ; 16 1-15 art. 1642167.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.3389/fphar.2025.1642167

    • Vancouver

      Lin R, Zhou Z, Jiang Y, Liu S, Jinfeng Xie, Wang H, Ulrich H, Zheng W. Artemisinin exerts antidepressant-like effects via activation of AKT and ERK signaling pathways [Internet]. Frontiers in Pharmacology. 2025 ; 16 1-15 art. 1642167.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.3389/fphar.2025.1642167

  • Artigo de periodico

    P2X7 receptor in macrophage polarization and its implications in neuroblastoma tumor behavior (2025)

    Bento, Carolina Adriane; Sampaio, Vanessa Fernandes Arnaud; Glaser, Talita ; Adinolfi, Elena; Silva, Robson Coutinho ; Ulrich, Henning ; Lameu, Claudiana

    Fonte: Purinergic Signalling. Unidade: IQ

    Assuntos: NEUROBLASTOMA, MACRÓFAGOS

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      BENTO, Carolina Adriane et al. P2X7 receptor in macrophage polarization and its implications in neuroblastoma tumor behavior. Purinergic Signalling, v. 21, p. 51–68, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s11302-024-10051-w. Acesso em: 01 dez. 2025.

    • APA

      Bento, C. A., Sampaio, V. F. A., Glaser, T., Adinolfi, E., Silva, R. C., Ulrich, H., & Lameu, C. (2025). P2X7 receptor in macrophage polarization and its implications in neuroblastoma tumor behavior. Purinergic Signalling, 21, 51–68. doi:10.1007/s11302-024-10051-w

    • NLM

      Bento CA, Sampaio VFA, Glaser T, Adinolfi E, Silva RC, Ulrich H, Lameu C. P2X7 receptor in macrophage polarization and its implications in neuroblastoma tumor behavior [Internet]. Purinergic Signalling. 2025 ; 21 51–68.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-024-10051-w

    • Vancouver

      Bento CA, Sampaio VFA, Glaser T, Adinolfi E, Silva RC, Ulrich H, Lameu C. P2X7 receptor in macrophage polarization and its implications in neuroblastoma tumor behavior [Internet]. Purinergic Signalling. 2025 ; 21 51–68.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-024-10051-w

  • Artigo de periodico

    Naringenin exhibits antiglioma activity related to aryl hydrocarbon receptor activity and IL-6, CCL2, and TNF-α expression (2025)

    Santana, Monique Reis de ; Argolo, Deivison Silva; Lima, Irlã Santos ; Santos, Cleonice Creusa dos; Victor, Maurício Moraes ; Ramos, Gabriel dos Santos; Nascimento, Ravena Pereira do ; Ulrich, Henning ; Costa, Silvia Lima

    Fonte: Brain Sciences. Unidade: IQ

    Assuntos: FLAVONOIDES, HIDROCARBONETOS

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      SANTANA, Monique Reis de et al. Naringenin exhibits antiglioma activity related to aryl hydrocarbon receptor activity and IL-6, CCL2, and TNF-α expression. Brain Sciences, v. 15, n. 3, p. 1-13 art. 325, 2025Tradução . . Disponível em: https://dx.doi.org/10.3390/brainsci15030325. Acesso em: 01 dez. 2025.

    • APA

      Santana, M. R. de, Argolo, D. S., Lima, I. S., Santos, C. C. dos, Victor, M. M., Ramos, G. dos S., et al. (2025). Naringenin exhibits antiglioma activity related to aryl hydrocarbon receptor activity and IL-6, CCL2, and TNF-α expression. Brain Sciences, 15( 3), 1-13 art. 325. doi:10.3390/brainsci15030325

    • NLM

      Santana MR de, Argolo DS, Lima IS, Santos CC dos, Victor MM, Ramos G dos S, Nascimento RP do, Ulrich H, Costa SL. Naringenin exhibits antiglioma activity related to aryl hydrocarbon receptor activity and IL-6, CCL2, and TNF-α expression [Internet]. Brain Sciences. 2025 ; 15( 3): 1-13 art. 325.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.3390/brainsci15030325

    • Vancouver

      Santana MR de, Argolo DS, Lima IS, Santos CC dos, Victor MM, Ramos G dos S, Nascimento RP do, Ulrich H, Costa SL. Naringenin exhibits antiglioma activity related to aryl hydrocarbon receptor activity and IL-6, CCL2, and TNF-α expression [Internet]. Brain Sciences. 2025 ; 15( 3): 1-13 art. 325.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.3390/brainsci15030325

  • Artigo de periodico

    Purinergic signaling in liver disease: calcium signaling and induction of inflammation (2025)

    Ulrich, Henning ; Glaser, Talita ; Thomas, Andrew P

    Fonte: Purinergic Signalling. Unidade: IQ

    Assuntos: HEPATOPATIAS, RECEPTORES, INFLAMAÇÃO, RECEPTORES

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    • ABNT

      ULRICH, Henning e GLASER, Talita e THOMAS, Andrew P. Purinergic signaling in liver disease: calcium signaling and induction of inflammation. Purinergic Signalling, v. 21, p. 69–81, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s11302-024-10044-9. Acesso em: 01 dez. 2025.

    • APA

      Ulrich, H., Glaser, T., & Thomas, A. P. (2025). Purinergic signaling in liver disease: calcium signaling and induction of inflammation. Purinergic Signalling, 21, 69–81. doi:10.1007/s11302-024-10044-9

    • NLM

      Ulrich H, Glaser T, Thomas AP. Purinergic signaling in liver disease: calcium signaling and induction of inflammation [Internet]. Purinergic Signalling. 2025 ; 21 69–81.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-024-10044-9

    • Vancouver

      Ulrich H, Glaser T, Thomas AP. Purinergic signaling in liver disease: calcium signaling and induction of inflammation [Internet]. Purinergic Signalling. 2025 ; 21 69–81.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-024-10044-9

  • Artigo de periodico

    Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease (2025)

    Juvenal, Guilherme Araújo ; Higa, Guilherme Shigueto Vilar ; Marques, Lucas Bonfim; Zampieri, Thais Tessari; Viana, Felipe José Costa ; Britto, Luiz Roberto Giorgetti de ; Tang, Yong; Illes, Peter ; Virgilio, Francesco Di ; Ulrich, Henning ; Pasquale, Roberto de

    Fonte: Purinergic Signalling. Unidades: ICB, IQ

    Assuntos: NEUROTRANSMISSORES, RECEPTORES

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      JUVENAL, Guilherme Araújo et al. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease. Purinergic Signalling, v. 21, p. 149-177, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s11302-024-10034-x. Acesso em: 01 dez. 2025.

    • APA

      Juvenal, G. A., Higa, G. S. V., Marques, L. B., Zampieri, T. T., Viana, F. J. C., Britto, L. R. G. de, et al. (2025). Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease. Purinergic Signalling, 21, 149-177. doi:10.1007/s11302-024-10034-x

    • NLM

      Juvenal GA, Higa GSV, Marques LB, Zampieri TT, Viana FJC, Britto LRG de, Tang Y, Illes P, Virgilio FD, Ulrich H, Pasquale R de. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease [Internet]. Purinergic Signalling. 2025 ;21 149-177.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-024-10034-x

    • Vancouver

      Juvenal GA, Higa GSV, Marques LB, Zampieri TT, Viana FJC, Britto LRG de, Tang Y, Illes P, Virgilio FD, Ulrich H, Pasquale R de. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease [Internet]. Purinergic Signalling. 2025 ;21 149-177.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-024-10034-x

  • Artigo de periodico

    Hematopoietic stem cells on the crossroad between purinergic signaling and innate immunity (2025)

    Franczak, Stephanie; Ulrich, Henning ; Ratajczak, Mariusz Z

    Fonte: Purinergic Signalling. Unidade: IQ

    Assuntos: METABOLISMO, CÉLULAS-TRONCO

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      FRANCZAK, Stephanie e ULRICH, Henning e RATAJCZAK, Mariusz Z. Hematopoietic stem cells on the crossroad between purinergic signaling and innate immunity. Purinergic Signalling, v. 21, p. 3-9, 2025Tradução . . Disponível em: https://doi.org/10.1007/s11302-023-09943-0. Acesso em: 01 dez. 2025.

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      Franczak, S., Ulrich, H., & Ratajczak, M. Z. (2025). Hematopoietic stem cells on the crossroad between purinergic signaling and innate immunity. Purinergic Signalling, 21, 3-9. doi:10.1007/s11302-023-09943-0

    • NLM

      Franczak S, Ulrich H, Ratajczak MZ. Hematopoietic stem cells on the crossroad between purinergic signaling and innate immunity [Internet]. Purinergic Signalling. 2025 ; 21 3-9.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1007/s11302-023-09943-0

    • Vancouver

      Franczak S, Ulrich H, Ratajczak MZ. Hematopoietic stem cells on the crossroad between purinergic signaling and innate immunity [Internet]. Purinergic Signalling. 2025 ; 21 3-9.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1007/s11302-023-09943-0

  • Artigo de periodico

    Photobiomodulation by LED 660 nm and Taurine against H2O2 oxidative stress in SH‑SY5Y cells (2025)

    Rossato, Rafaella Carvalho ; Salles, Geisa Rodrigues ; Albuquerque, Amanda Lira; Porcionatto, Marimélia Aparecida ; Granato, Alessandro Eustaquio Campos ; Ulrich, Henning ; Santos, Mariela Inês Batista dos ; Soares, Cristina Pacheco

    Fonte: Lasers in Medical Science. Unidade: IQ

    Assuntos: ESTRESSE OXIDATIVO, DOENÇA DE ALZHEIMER, PEROXIDASE, FOTOBIOLOGIA

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    • ABNT

      ROSSATO, Rafaella Carvalho et al. Photobiomodulation by LED 660 nm and Taurine against H2O2 oxidative stress in SH‑SY5Y cells. Lasers in Medical Science, v. 40, p. 1-11 art. 211, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s10103-025-04467-y. Acesso em: 01 dez. 2025.

    • APA

      Rossato, R. C., Salles, G. R., Albuquerque, A. L., Porcionatto, M. A., Granato, A. E. C., Ulrich, H., et al. (2025). Photobiomodulation by LED 660 nm and Taurine against H2O2 oxidative stress in SH‑SY5Y cells. Lasers in Medical Science, 40, 1-11 art. 211. doi:10.1007/s10103-025-04467-y

    • NLM

      Rossato RC, Salles GR, Albuquerque AL, Porcionatto MA, Granato AEC, Ulrich H, Santos MIB dos, Soares CP. Photobiomodulation by LED 660 nm and Taurine against H2O2 oxidative stress in SH‑SY5Y cells [Internet]. Lasers in Medical Science. 2025 ; 40 1-11 art. 211.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s10103-025-04467-y

    • Vancouver

      Rossato RC, Salles GR, Albuquerque AL, Porcionatto MA, Granato AEC, Ulrich H, Santos MIB dos, Soares CP. Photobiomodulation by LED 660 nm and Taurine against H2O2 oxidative stress in SH‑SY5Y cells [Internet]. Lasers in Medical Science. 2025 ; 40 1-11 art. 211.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s10103-025-04467-y

  • Artigo de periodico

    Purinergic system in cancer stem cells (2025)

    Rios, J. D. Nuñez; Ulrich, Henning ; Muñoz, M. Díaz; Lameu, Claudiana ; Cuevas, F. G. Vázquez

    Fonte: Purinergic Signalling. Unidade: IQ

    Assuntos: CÉLULAS-TRONCO, NEOPLASIAS

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    • ABNT

      RIOS, J. D. Nuñez et al. Purinergic system in cancer stem cells. Purinergic Signalling, v. 21, p. 23-38, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s11302-023-09976-5. Acesso em: 01 dez. 2025.

    • APA

      Rios, J. D. N., Ulrich, H., Muñoz, M. D., Lameu, C., & Cuevas, F. G. V. (2025). Purinergic system in cancer stem cells. Purinergic Signalling, 21, 23-38. doi:10.1007/s11302-023-09976-5

    • NLM

      Rios JDN, Ulrich H, Muñoz MD, Lameu C, Cuevas FGV. Purinergic system in cancer stem cells [Internet]. Purinergic Signalling. 2025 ; 21 23-38.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-023-09976-5

    • Vancouver

      Rios JDN, Ulrich H, Muñoz MD, Lameu C, Cuevas FGV. Purinergic system in cancer stem cells [Internet]. Purinergic Signalling. 2025 ; 21 23-38.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-023-09976-5

  • Artigo de periodico-carta/editorial

    Purinergic signaling in health: special issue of purines 2022 in Brazil [Editorial] (2025)

    Ulrich, Henning ; Tasca, Carla I; Lameu, Claudiana

    Fonte: Purinergic Signalling. Unidade: IQ

    Assuntos: PURINAS, HEMATOPOESE

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    • ABNT

      ULRICH, Henning e TASCA, Carla I e LAMEU, Claudiana. Purinergic signaling in health: special issue of purines 2022 in Brazil [Editorial]. Purinergic Signalling. Dordrecht: Instituto de Química, Universidade de São Paulo. Disponível em: https://dx.doi.org/10.1007/s11302-025-10067-w. Acesso em: 01 dez. 2025. , 2025

    • APA

      Ulrich, H., Tasca, C. I., & Lameu, C. (2025). Purinergic signaling in health: special issue of purines 2022 in Brazil [Editorial]. Purinergic Signalling. Dordrecht: Instituto de Química, Universidade de São Paulo. doi:10.1007/s11302-025-10067-w

    • NLM

      Ulrich H, Tasca CI, Lameu C. Purinergic signaling in health: special issue of purines 2022 in Brazil [Editorial] [Internet]. Purinergic Signalling. 2025 ;[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-025-10067-w

    • Vancouver

      Ulrich H, Tasca CI, Lameu C. Purinergic signaling in health: special issue of purines 2022 in Brazil [Editorial] [Internet]. Purinergic Signalling. 2025 ;[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1007/s11302-025-10067-w

  • Artigo de periodico

    The role of interleukin-17 in neurological disorders (2025)

    Parka, Seung Eun ; Ferreira, Ana Flávia Fernandes ; Kwon, Hyeokbin ; Yu, Jeongwoo ; Diniz, Leila; Kim, Eunha ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de

    Fonte: Animal Cells and Systems. Unidades: IQ, ICB

    Assuntos: FISIOLOGIA, INTERLEUCINAS, TRANSTORNOS MENTAIS, DOENÇAS NEURODEGENERATIVAS, CITOCINAS, DOENÇAS DO SISTEMA NERVOSO CENTRAL

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    • ABNT

      PARKA, Seung Eun et al. The role of interleukin-17 in neurological disorders. Animal Cells and Systems, v. 29, n. 1, p. 372-386, 2025Tradução . . Disponível em: https://doi.org/10.1080/19768354.2025.2510994. Acesso em: 01 dez. 2025.

    • APA

      Parka, S. E., Ferreira, A. F. F., Kwon, H., Yu, J., Diniz, L., Kim, E., et al. (2025). The role of interleukin-17 in neurological disorders. Animal Cells and Systems, 29( 1), 372-386. doi:10.1080/19768354.2025.2510994

    • NLM

      Parka SE, Ferreira AFF, Kwon H, Yu J, Diniz L, Kim E, Ulrich H, Britto LRG de. The role of interleukin-17 in neurological disorders [Internet]. Animal Cells and Systems. 2025 ; 29( 1): 372-386.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1080/19768354.2025.2510994

    • Vancouver

      Parka SE, Ferreira AFF, Kwon H, Yu J, Diniz L, Kim E, Ulrich H, Britto LRG de. The role of interleukin-17 in neurological disorders [Internet]. Animal Cells and Systems. 2025 ; 29( 1): 372-386.[citado 2025 dez. 01 ] Available from: https://doi.org/10.1080/19768354.2025.2510994

  • Artigo de periodico

    The complosome - a key regulator of hematopoietic stem cells and innate immune responses (2025)

    Ulrich, Henning

    Fonte: Leukemia. Unidade: IQ

    Assuntos: CÉLULAS-TRONCO, PROTEÍNAS

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    • ABNT

      ULRICH, Henning. The complosome - a key regulator of hematopoietic stem cells and innate immune responses. Leukemia, v. 39, p. 2297–2298, 2025Tradução . . Disponível em: https://dx.doi.org/10.1038/s41375-025-02728-x. Acesso em: 01 dez. 2025.

    • APA

      Ulrich, H. (2025). The complosome - a key regulator of hematopoietic stem cells and innate immune responses. Leukemia, 39, 2297–2298. doi:10.1038/s41375-025-02728-x

    • NLM

      Ulrich H. The complosome - a key regulator of hematopoietic stem cells and innate immune responses [Internet]. Leukemia. 2025 ; 39 2297–2298.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1038/s41375-025-02728-x

    • Vancouver

      Ulrich H. The complosome - a key regulator of hematopoietic stem cells and innate immune responses [Internet]. Leukemia. 2025 ; 39 2297–2298.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1038/s41375-025-02728-x

  • Artigo de periodico

    Agathisflavone inhibits viability and modulates the expression of miR-125b, miR-155, IL-6, and arginase in glioblastoma cells and microglia/macrophage activation (2025)

    Silva, Karina Costa da; Lima, Irlã Santos ; Santos, Cleonice Creusa dos; Nonaka, Carolina Kymie Vasques ; Souza, Bruno Solano de Freitas ; David, Jorge Mauricio ; Ulrich, Henning ; Nascimento, Ravena Pereira do ; Costa, Maria de Fátima Dias ; Santos, Balbino Lino dos ; Costa, Silvia Lima

    Fonte: Molecules. Unidade: IQ

    Assuntos: MACRÓFAGOS, QUIMIOTERAPIA

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    • ABNT

      SILVA, Karina Costa da et al. Agathisflavone inhibits viability and modulates the expression of miR-125b, miR-155, IL-6, and arginase in glioblastoma cells and microglia/macrophage activation. Molecules, v. 30, n. 1, p. 1-15 art. 158, 2025Tradução . . Disponível em: https://dx.doi.org/10.3390/molecules30010158. Acesso em: 01 dez. 2025.

    • APA

      Silva, K. C. da, Lima, I. S., Santos, C. C. dos, Nonaka, C. K. V., Souza, B. S. de F., David, J. M., et al. (2025). Agathisflavone inhibits viability and modulates the expression of miR-125b, miR-155, IL-6, and arginase in glioblastoma cells and microglia/macrophage activation. Molecules, 30( 1), 1-15 art. 158. doi:10.3390/molecules30010158

    • NLM

      Silva KC da, Lima IS, Santos CC dos, Nonaka CKV, Souza BS de F, David JM, Ulrich H, Nascimento RP do, Costa M de FD, Santos BL dos, Costa SL. Agathisflavone inhibits viability and modulates the expression of miR-125b, miR-155, IL-6, and arginase in glioblastoma cells and microglia/macrophage activation [Internet]. Molecules. 2025 ; 30( 1): 1-15 art. 158.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.3390/molecules30010158

    • Vancouver

      Silva KC da, Lima IS, Santos CC dos, Nonaka CKV, Souza BS de F, David JM, Ulrich H, Nascimento RP do, Costa M de FD, Santos BL dos, Costa SL. Agathisflavone inhibits viability and modulates the expression of miR-125b, miR-155, IL-6, and arginase in glioblastoma cells and microglia/macrophage activation [Internet]. Molecules. 2025 ; 30( 1): 1-15 art. 158.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.3390/molecules30010158

  • Artigo de periodico

    Dysregulation of astrocytic ATP/adenosine release in the hippocampus cause cognitive and affective disorders: molecular mechanisms, diagnosis, and therapy (2025)

    Illes, Peter; Rubini, Patrizia; Ulrich, Henning ; Yin, Hai-Yan; Tang, Yong

    Fonte: MedComm. Unidade: IQ

    Assuntos: RECEPTORES, ADENOSINA, TRANSTORNOS COGNITIVOS

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    • ABNT

      ILLES, Peter et al. Dysregulation of astrocytic ATP/adenosine release in the hippocampus cause cognitive and affective disorders: molecular mechanisms, diagnosis, and therapy. MedComm, v. 6, p. 1-23 art. e70177, 2025Tradução . . Disponível em: https://dx.doi.org/10.1002/mco2.70177. Acesso em: 01 dez. 2025.

    • APA

      Illes, P., Rubini, P., Ulrich, H., Yin, H. -Y., & Tang, Y. (2025). Dysregulation of astrocytic ATP/adenosine release in the hippocampus cause cognitive and affective disorders: molecular mechanisms, diagnosis, and therapy. MedComm, 6, 1-23 art. e70177. doi:10.1002/mco2.70177

    • NLM

      Illes P, Rubini P, Ulrich H, Yin H-Y, Tang Y. Dysregulation of astrocytic ATP/adenosine release in the hippocampus cause cognitive and affective disorders: molecular mechanisms, diagnosis, and therapy [Internet]. MedComm. 2025 ; 6 1-23 art. e70177.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1002/mco2.70177

    • Vancouver

      Illes P, Rubini P, Ulrich H, Yin H-Y, Tang Y. Dysregulation of astrocytic ATP/adenosine release in the hippocampus cause cognitive and affective disorders: molecular mechanisms, diagnosis, and therapy [Internet]. MedComm. 2025 ; 6 1-23 art. e70177.[citado 2025 dez. 01 ] Available from: https://dx.doi.org/10.1002/mco2.70177

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