Paradoxical roles of the neutrophil in sepsis: protective and deleterious (2016)
- Autores:
- Autores USP: ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.3389/fimmu.2016.00155
- Assuntos: SEPSE; NEUTRÓFILOS; RECEPTORES
- Palavras-chave do autor: Sepsis; Neutrophil migration; Organ dysfunction; Toll-like receptors; Chemotactic receptors
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v.7, art. 155, 2016
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
SÔNEGO, Fabiane et al. Paradoxical roles of the neutrophil in sepsis: protective and deleterious. Frontiers in Immunology, v. 7, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00155. Acesso em: 18 set. 2024. -
APA
Sônego, F., Castanheira, F. V. e S., Ferreira, R. G., Kanashiro, A., Leite, C. A. V. G., Nascimento, D. C., et al. (2016). Paradoxical roles of the neutrophil in sepsis: protective and deleterious. Frontiers in Immunology, 7. doi:10.3389/fimmu.2016.00155 -
NLM
Sônego F, Castanheira FV e S, Ferreira RG, Kanashiro A, Leite CAVG, Nascimento DC, Colón DF, Borges V de F, Alves Filho JCF, Cunha F de Q. Paradoxical roles of the neutrophil in sepsis: protective and deleterious [Internet]. Frontiers in Immunology. 2016 ;7[citado 2024 set. 18 ] Available from: https://doi.org/10.3389/fimmu.2016.00155 -
Vancouver
Sônego F, Castanheira FV e S, Ferreira RG, Kanashiro A, Leite CAVG, Nascimento DC, Colón DF, Borges V de F, Alves Filho JCF, Cunha F de Q. Paradoxical roles of the neutrophil in sepsis: protective and deleterious [Internet]. Frontiers in Immunology. 2016 ;7[citado 2024 set. 18 ] Available from: https://doi.org/10.3389/fimmu.2016.00155 - Failure of neutrophil migration to infection focus correlates with sepsis outcome: critical role of TLR, nitric-cGMP-PKG pathway and chemokines receptors
- NADPH phagocyte oxidase knockout mice control Trypanosoma cruzi proliferation, but develop circulatory collapse and succumb to infection
- CCR4 controls the suppressive effects of regulatory T cells on early and late events during severe sepsis
- Regulation of type 17 helper T-cell function by nitric oxide during inflammation
- Interleukin-10 rs2227307 and CXCR2 rs1126579 polymorphisms modulate the predisposition to septic shock
- IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system
- PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis
- Targeting neutrophils in sepsis
- Blockage of eosinopoiesis by IL-17A is prevented by cytokine and lipid mediators of allergic inflammation
- Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis
Informações sobre o DOI: 10.3389/fimmu.2016.00155 (Fonte: oaDOI API)
Como citar
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
