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Filtros : "REGULAÇÃO GÊNICA" "Biochemical and Biophysical Research Communications" Removido: "Abstracts" Limpar

Filtros

USP affiliated authors
- camargo, maristela martins de (~1)
- curti, carlos (~1)
- garcia, cristiana bernadelli (~1)
- leopoldino, andréia machado (~1)
Date published
- 2017 (~1)
- 2014 (~1)
Subjects
- regulação gênica (2)
- carcinoma de células escamosas (1)
- imunogenética (1)
- imunoglobulinas (1)
- imunologia (1)

Artigo de periodico
Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client (2017)
Stan, Razvan C.; Silva, Raissa Leal; Camargo, Maristela Martins de
Source: Biochemical and Biophysical Research Communications. Unidade: ICB
Subjects: IMUNOLOGIA, REPLICAÇÃO DO DNA, IMUNOGENÉTICA, IMUNOGLOBULINAS, REGULAÇÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
STAN, Razvan C. e SILVA, Raissa Leal e CAMARGO, Maristela Martins de. Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client. Biochemical and Biophysical Research Communications, v. 487, n. 2, p. 375-380, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2017.04.069. Acesso em: 27 nov. 2025.
APA
Stan, R. C., Silva, R. L., & Camargo, M. M. de. (2017). Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client. Biochemical and Biophysical Research Communications, 487( 2), 375-380. doi:10.1016/j.bbrc.2017.04.069
NLM
Stan RC, Silva RL, Camargo MM de. Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client [Internet]. Biochemical and Biophysical Research Communications. 2017 ; 487( 2): 375-380.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.bbrc.2017.04.069
Vancouver
Stan RC, Silva RL, Camargo MM de. Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client [Internet]. Biochemical and Biophysical Research Communications. 2017 ; 487( 2): 375-380.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.bbrc.2017.04.069
Artigo de periodico
Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation (2014)
Almeida, Luciana O.; Garcia, Cristiana Bernadelli; Silva, Flávia Amoroso Matos e; Curti, Carlos; Leopoldino, Andréia Machado
Source: Biochemical and Biophysical Research Communications. Unidade: FCFRP
Subjects: CARCINOMA DE CÉLULAS ESCAMOSAS, LINHAGEM CELULAR, PROLIFERAÇÃO CELULAR, REGULAÇÃO GÊNICA, NEOPLASIAS DE CABEÇA E PESCOÇO, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Luciana O. et al. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, v. 445, n. 1, p. 196\2013202, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2014.01.175. Acesso em: 27 nov. 2025.
APA
Almeida, L. O., Garcia, C. B., Silva, F. A. M. e, Curti, C., & Leopoldino, A. M. (2014). Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, 445( 1), 196\2013202. doi:10.1016/j.bbrc.2014.01.175
NLM
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175
Vancouver
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175

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