Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client (2017)
- Authors:
- Autor USP: CAMARGO, MARISTELA MARTINS DE - ICB
- Unidade: ICB
- DOI: 10.1016/j.bbrc.2017.04.069
- Subjects: IMUNOLOGIA; REPLICAÇÃO DO DNA; IMUNOGENÉTICA; IMUNOGLOBULINAS; REGULAÇÃO GÊNICA
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2017
- Source:
- Título do periódico: Biochemical and Biophysical Research Communications
- ISSN: 1090-2104
- Volume/Número/Paginação/Ano: v. 487, n. 2, p. 375-380, 2017
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
STAN, Razvan C. e SILVA, Raissa Leal e CAMARGO, Maristela Martins de. Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client. Biochemical and Biophysical Research Communications, v. 487, n. 2, p. 375-380, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2017.04.069. Acesso em: 24 abr. 2024. -
APA
Stan, R. C., Silva, R. L., & Camargo, M. M. de. (2017). Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client. Biochemical and Biophysical Research Communications, 487( 2), 375-380. doi:10.1016/j.bbrc.2017.04.069 -
NLM
Stan RC, Silva RL, Camargo MM de. Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client [Internet]. Biochemical and Biophysical Research Communications. 2017 ; 487( 2): 375-380.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1016/j.bbrc.2017.04.069 -
Vancouver
Stan RC, Silva RL, Camargo MM de. Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client [Internet]. Biochemical and Biophysical Research Communications. 2017 ; 487( 2): 375-380.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1016/j.bbrc.2017.04.069 - Existe vida após o doutorado? Parte II
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Informações sobre o DOI: 10.1016/j.bbrc.2017.04.069 (Fonte: oaDOI API)
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