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Artigo de periodico
Immunometabolic and vascular health responses among high endurance trained subjects (2024)
Fonseca, Henrique Andrade Rodrigues da ; Bittencourt, Celia Regina; Monteiro, Andrea Moreira; Fonseca, Francisco Antonio; Sanches, Lívia Rentas ; Ferreira, Carlos Eduardo ; Figueiredo Neto, Antonio Martins ; Gidlund, Magnus Ake
Source: International Journal of Sports Medicine. Unidades: IF, ICB
Subjects: IMUNOLOGIA, SISTEMA CARDIOVASCULAR, ARTÉRIAS CARÓTIDAS, INTERLEUCINA 10, METABOLISMO, ESPECTROMETRIA DE MASSAS, HORMÔNIOS GLICOCORTICOIDES, LIPOPROTEÍNAS, EXERCÍCIO FÍSICO, ARTERIOSCLEROSE, ENDOTÉLIO VASCULAR
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FONSECA, Henrique Andrade Rodrigues da et al. Immunometabolic and vascular health responses among high endurance trained subjects. International Journal of Sports Medicine, v. 45, n. 3, p. 245-252, 2024Tradução . . Disponível em: https://doi.org/10.1055/a-2186-2717. Acesso em: 16 nov. 2024.
APA
Fonseca, H. A. R. da, Bittencourt, C. R., Monteiro, A. M., Fonseca, F. A., Sanches, L. R., Ferreira, C. E., et al. (2024). Immunometabolic and vascular health responses among high endurance trained subjects. International Journal of Sports Medicine, 45( 3), 245-252. doi:10.1055/a-2186-2717
NLM
Fonseca HAR da, Bittencourt CR, Monteiro AM, Fonseca FA, Sanches LR, Ferreira CE, Figueiredo Neto AM, Gidlund MA. Immunometabolic and vascular health responses among high endurance trained subjects [Internet]. International Journal of Sports Medicine. 2024 ; 45( 3): 245-252.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1055/a-2186-2717
Vancouver
Fonseca HAR da, Bittencourt CR, Monteiro AM, Fonseca FA, Sanches LR, Ferreira CE, Figueiredo Neto AM, Gidlund MA. Immunometabolic and vascular health responses among high endurance trained subjects [Internet]. International Journal of Sports Medicine. 2024 ; 45( 3): 245-252.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1055/a-2186-2717
Trabalho de evento-resumo periodico
C3 deficiency modulates the phenotypic profile of murine macrophages (2023)
Silva, Tiago Francisco da ; Amamura, Thaís; Valadão, Iuri Cordeiro ; Carneiro, Milena Carvalho ; Freitas, Vanessa Morais ; Lepique, Ana Paula ; Isaac, Lourdes
Source: Immunobiology. Conference titles: International Complement Workshop. Unidade: ICB
Subjects: IMUNOLOGIA, BIOLOGIA CELULAR, MICROTUBOS, IMUNOPROTEÍNAS, MEDULA ÓSSEA DE ANIMAL, CAMUNDONGOS, MACRÓFAGOS, FENÓTIPOS
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SILVA, Tiago Francisco da et al. C3 deficiency modulates the phenotypic profile of murine macrophages. Immunobiology. Muenchen: Elsevier BV. Disponível em: https://doi.org/10.1016/j.imbio.2023.152637. Acesso em: 16 nov. 2024. , 2023
APA
Silva, T. F. da, Amamura, T., Valadão, I. C., Carneiro, M. C., Freitas, V. M., Lepique, A. P., & Isaac, L. (2023). C3 deficiency modulates the phenotypic profile of murine macrophages. Immunobiology. Muenchen: Elsevier BV. doi:10.1016/j.imbio.2023.152637
NLM
Silva TF da, Amamura T, Valadão IC, Carneiro MC, Freitas VM, Lepique AP, Isaac L. C3 deficiency modulates the phenotypic profile of murine macrophages [Internet]. Immunobiology. 2023 ; 228( 5):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2023.152637
Vancouver
Silva TF da, Amamura T, Valadão IC, Carneiro MC, Freitas VM, Lepique AP, Isaac L. C3 deficiency modulates the phenotypic profile of murine macrophages [Internet]. Immunobiology. 2023 ; 228( 5):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2023.152637
Artigo de periodico
Direct and indirect impact of SARS-CoV-2 on the brain (2023)
Peron, Jean Pierre Schatzmann
Source: Human Genetics. Unidade: ICB
Subjects: IMUNOLOGIA, COVID-19, SISTEMA NERVOSO CENTRAL
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PERON, Jean Pierre Schatzmann. Direct and indirect impact of SARS-CoV-2 on the brain. Human Genetics, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00439-023-02549-x. Acesso em: 16 nov. 2024.
APA
Peron, J. P. S. (2023). Direct and indirect impact of SARS-CoV-2 on the brain. Human Genetics, 1-10. doi:10.1007/s00439-023-02549-x
NLM
Peron JPS. Direct and indirect impact of SARS-CoV-2 on the brain [Internet]. Human Genetics. 2023 ; 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00439-023-02549-x
Vancouver
Peron JPS. Direct and indirect impact of SARS-CoV-2 on the brain [Internet]. Human Genetics. 2023 ; 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00439-023-02549-x
Artigo de periodico
Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease (2023)
Cipelli, Marcella ; Silva, Eloisa Martins da ; Câmara, Niels Olsen Saraiva
Source: Current Clinical Microbiology Reports. Unidade: ICB
Subjects: IMUNOLOGIA, ENTEROPATIAS INFLAMATÓRIAS, BIOTA INTESTINAL, SISTEMA GASTROINTESTINAL, INTESTINOS
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CIPELLI, Marcella e SILVA, Eloisa Martins da e CÂMARA, Niels Olsen Saraiva. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease. Current Clinical Microbiology Reports, p. 11 , 2023Tradução . . Disponível em: https://doi.org/10.1007/s40588-023-00207-4. Acesso em: 16 nov. 2024.
APA
Cipelli, M., Silva, E. M. da, & Câmara, N. O. S. (2023). Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease. Current Clinical Microbiology Reports, 11 . doi:10.1007/s40588-023-00207-4
NLM
Cipelli M, Silva EM da, Câmara NOS. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease [Internet]. Current Clinical Microbiology Reports. 2023 ;11 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s40588-023-00207-4
Vancouver
Cipelli M, Silva EM da, Câmara NOS. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease [Internet]. Current Clinical Microbiology Reports. 2023 ;11 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s40588-023-00207-4
Artigo de periodico
The inflammasomes: crosstalk between innate immunity and hematology (2022)
Dutra, Valéria de Freitas ; Leal, Vinicius Nunes Cordeiro ; Pontillo, Alessandra
Source: Inflammation Research. Unidade: ICB
Subjects: IMUNOLOGIA, CITOCINAS, CÉLULAS MORTAS, IMUNIDADE, IMUNOSSUPRESSÃO, DOENÇAS HEMATOLÓGICAS, CÉLULAS CULTIVADAS DE TUMOR, CÉLULAS SANGUÍNEAS, NEOPLASIAS, PROLIFERAÇÃO CELULAR
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DUTRA, Valéria de Freitas e LEAL, Vinicius Nunes Cordeiro e PONTILLO, Alessandra. The inflammasomes: crosstalk between innate immunity and hematology. Inflammation Research, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01646-3. Acesso em: 16 nov. 2024.
APA
Dutra, V. de F., Leal, V. N. C., & Pontillo, A. (2022). The inflammasomes: crosstalk between innate immunity and hematology. Inflammation Research, 1-14. doi:10.1007/s00011-022-01646-3
NLM
Dutra V de F, Leal VNC, Pontillo A. The inflammasomes: crosstalk between innate immunity and hematology [Internet]. Inflammation Research. 2022 ; 1-14.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00011-022-01646-3
Vancouver
Dutra V de F, Leal VNC, Pontillo A. The inflammasomes: crosstalk between innate immunity and hematology [Internet]. Inflammation Research. 2022 ; 1-14.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00011-022-01646-3
Artigo de periodico
A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19 (2022)
Leal, Vinicius Nunes Cordeiro ; Cambui, Raylane Adrielle Gonçalves ; Zupelli, Thiago Gameiro ; Yamada, Suemy Melim ; Oliveira, Leonardo Antonio Teixeira de ; Andrade, Milena Mary de Souza ; Teixeira, Franciane Mouradian Emílio ; Custódio, Ricardo Wesley Alberca ; Silva, Sarah Cristina Gozzi e ; Yendo, Tatiana Mina ; Netto, Lucas Chaves ; Duarte, Alberto José da Silva; Sato, Maria Notomi ; Pontillo, Alessandra
Source: Inflammation Research. Unidades: FM, ICB
Subjects: IMUNOLOGIA, ESTUDOS DE COORTES, VARIAÇÃO GENÉTICA, COVID-19, FATORES DE RISCO, NUCLEOTÍDEOS, CITOCINAS, INFLAMAÇÃO, ENZIMAS PROTEOLÍTICAS
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LEAL, Vinicius Nunes Cordeiro et al. A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19. Inflammation Research, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01670-3. Acesso em: 16 nov. 2024.
APA
Leal, V. N. C., Cambui, R. A. G., Zupelli, T. G., Yamada, S. M., Oliveira, L. A. T. de, Andrade, M. M. de S., et al. (2022). A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19. Inflammation Research, 1-8. doi:10.1007/s00011-022-01670-3
NLM
Leal VNC, Cambui RAG, Zupelli TG, Yamada SM, Oliveira LAT de, Andrade MM de S, Teixeira FME, Custódio RWA, Silva SCG e, Yendo TM, Netto LC, Duarte AJ da S, Sato MN, Pontillo A. A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19 [Internet]. Inflammation Research. 2022 ; 1-8.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00011-022-01670-3
Vancouver
Leal VNC, Cambui RAG, Zupelli TG, Yamada SM, Oliveira LAT de, Andrade MM de S, Teixeira FME, Custódio RWA, Silva SCG e, Yendo TM, Netto LC, Duarte AJ da S, Sato MN, Pontillo A. A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19 [Internet]. Inflammation Research. 2022 ; 1-8.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00011-022-01670-3
Artigo de periodico
The Ala134Thr variant in TMEM176B exerts a beneficial role in colorectal cancer prognosis by increasing NLRP3 inflammasome activation (2022)
Cambui, Raylane Adrielle Gonçalves ; Fernandes, Fernanda Pereira; Leal, Vinicius Nunes Cordeiro ; Reis, Edione Cristina dos ; Lima, Dhemerson Souza de ; Santo, Gilmar Ferreira do Espírito ; Elias, Rosa Maria ; Pontillo, Alessandra
Source: Journal of Cancer Research and Clinical Oncology. Unidade: ICB
Subjects: IMUNOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS CULTIVADAS DE TUMOR, VARIAÇÃO GENÉTICA, COMPLEXOS MULTIENZIMÁTICOS, NUCLEOTÍDEOS, NEOPLASIAS DO COLON, ESTUDOS DE COORTES, GENÓTIPOS
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CAMBUI, Raylane Adrielle Gonçalves et al. The Ala134Thr variant in TMEM176B exerts a beneficial role in colorectal cancer prognosis by increasing NLRP3 inflammasome activation. Journal of Cancer Research and Clinical Oncology, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00432-022-04284-8. Acesso em: 16 nov. 2024.
APA
Cambui, R. A. G., Fernandes, F. P., Leal, V. N. C., Reis, E. C. dos, Lima, D. S. de, Santo, G. F. do E., et al. (2022). The Ala134Thr variant in TMEM176B exerts a beneficial role in colorectal cancer prognosis by increasing NLRP3 inflammasome activation. Journal of Cancer Research and Clinical Oncology, 1-10. doi:10.1007/s00432-022-04284-8
NLM
Cambui RAG, Fernandes FP, Leal VNC, Reis EC dos, Lima DS de, Santo GF do E, Elias RM, Pontillo A. The Ala134Thr variant in TMEM176B exerts a beneficial role in colorectal cancer prognosis by increasing NLRP3 inflammasome activation [Internet]. Journal of Cancer Research and Clinical Oncology. 2022 ; 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00432-022-04284-8
Vancouver
Cambui RAG, Fernandes FP, Leal VNC, Reis EC dos, Lima DS de, Santo GF do E, Elias RM, Pontillo A. The Ala134Thr variant in TMEM176B exerts a beneficial role in colorectal cancer prognosis by increasing NLRP3 inflammasome activation [Internet]. Journal of Cancer Research and Clinical Oncology. 2022 ; 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00432-022-04284-8
Artigo de periodico
Fever as an evolutionary agent to select immune complexes interfaces (2022)
Tofan, Vlad ; Camargo, Maristela Martins de ; Stan, Razvan Costin
Source: Immunogenetics. Unidade: ICB
Subjects: IMUNOLOGIA, FEBRE, ANTÍGENOS, EPITOPOS, ANTICORPOS MONOCLONAIS, NEOPLASIAS, IMUNOPROTEÍNAS, IMUNOGENÉTICA
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TOFAN, Vlad e CAMARGO, Maristela Martins de e STAN, Razvan Costin. Fever as an evolutionary agent to select immune complexes interfaces. Immunogenetics, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00251-022-01263-8. Acesso em: 16 nov. 2024.
APA
Tofan, V., Camargo, M. M. de, & Stan, R. C. (2022). Fever as an evolutionary agent to select immune complexes interfaces. Immunogenetics, 1-10. doi:10.1007/s00251-022-01263-8
NLM
Tofan V, Camargo MM de, Stan RC. Fever as an evolutionary agent to select immune complexes interfaces [Internet]. Immunogenetics. 2022 ; 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00251-022-01263-8
Vancouver
Tofan V, Camargo MM de, Stan RC. Fever as an evolutionary agent to select immune complexes interfaces [Internet]. Immunogenetics. 2022 ; 1-10.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00251-022-01263-8
Artigo de periodico
Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock (2021)
Rodrigues, Pedro Mendes de Azambuja; Valente, Richard Hemmi ; Brunoro, Giselle Villa Flor; Nakaya, Helder Takashi Imoto ; Pereira, Mariana Araujo; Bozza, Patricia Torres; Bozza, Fernando Augusto ; Trugilho, Monique Ramos de Oliveira
Source: Scientifc Reports. Unidade: FCF
Subjects: SEPSE, MONÓCITOS, METABOLISMO ENERGÉTICO, IMUNOLOGIA, INFLAMAÇÃO, PROTEÔMICA
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RODRIGUES, Pedro Mendes de Azambuja et al. Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock. Scientifc Reports, v. 11, p. 1-10 art. 15149, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41598-021-94474-0. Acesso em: 16 nov. 2024.
APA
Rodrigues, P. M. de A., Valente, R. H., Brunoro, G. V. F., Nakaya, H. T. I., Pereira, M. A., Bozza, P. T., et al. (2021). Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock. Scientifc Reports, 11, 1-10 art. 15149. doi:10.1038/s41598-021-94474-0
NLM
Rodrigues PM de A, Valente RH, Brunoro GVF, Nakaya HTI, Pereira MA, Bozza PT, Bozza FA, Trugilho MR de O. Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock [Internet]. Scientifc Reports. 2021 ; 11 1-10 art. 15149.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-021-94474-0
Vancouver
Rodrigues PM de A, Valente RH, Brunoro GVF, Nakaya HTI, Pereira MA, Bozza PT, Bozza FA, Trugilho MR de O. Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock [Internet]. Scientifc Reports. 2021 ; 11 1-10 art. 15149.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-021-94474-0
Artigo de periodico
Sympathetic nerve-adipocyte interactions in response to acute stress (2021)
Santos, Gabryella S. P.; Costa, Alinne C. ; Picoli, Caroline C.; Rocha, Beatriz G. S.; Sulaiman, Sheu O.; Radicchi, Debora C.; Pinto, Mauro C. X.; Batista Junior, Miguel L. ; Amorim, Jaime Henrique ; Azevedo, Vasco A. C.; Resende, Rodrigo Ribeiro ; Câmara, Niels Olsen Saraiva ; Mintz, Akiva ; Birbrair, Alexander
Source: Journal of Molecular Medicine. Unidade: ICB
Subjects: IMUNOLOGIA, ESTRESSE PSICOLÓGICO, HEPATOPATIAS, FÍGADO GORDUROSO, SISTEMA NERVOSO SIMPÁTICO, CITOCINAS, INFLAMAÇÃO, GLICOGENOSE, GLICOSE
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SANTOS, Gabryella S. P. et al. Sympathetic nerve-adipocyte interactions in response to acute stress. Journal of Molecular Medicine, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1007/s00109-021-02157-0. Acesso em: 16 nov. 2024.
APA
Santos, G. S. P., Costa, A. C., Picoli, C. C., Rocha, B. G. S., Sulaiman, S. O., Radicchi, D. C., et al. (2021). Sympathetic nerve-adipocyte interactions in response to acute stress. Journal of Molecular Medicine, 1-15. doi:10.1007/s00109-021-02157-0
NLM
Santos GSP, Costa AC, Picoli CC, Rocha BGS, Sulaiman SO, Radicchi DC, Pinto MCX, Batista Junior ML, Amorim JH, Azevedo VAC, Resende RR, Câmara NOS, Mintz A, Birbrair A. Sympathetic nerve-adipocyte interactions in response to acute stress [Internet]. Journal of Molecular Medicine. 2021 ; 1-15.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00109-021-02157-0
Vancouver
Santos GSP, Costa AC, Picoli CC, Rocha BGS, Sulaiman SO, Radicchi DC, Pinto MCX, Batista Junior ML, Amorim JH, Azevedo VAC, Resende RR, Câmara NOS, Mintz A, Birbrair A. Sympathetic nerve-adipocyte interactions in response to acute stress [Internet]. Journal of Molecular Medicine. 2021 ; 1-15.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00109-021-02157-0
Artigo de periodico
Macrophage immunophenotype but not anti-inflammatory profile is modulated by peroxisome proliferator-activated receptor gamma (PPARγ) in exercised obese mice (2020)
Silveira, Loreana Sanches ; Biondo, Luana Amorim ; Teixeira, Alexandre Abilio de Souza ; Lima Junior, Edson Alves de ; Castoldi, Angela ; Lira, Fábio Santos ; Câmara, Niels Olsen Saraiva ; Festuccia, William Tadeu Lara ; Rosa Neto, José Cesar
Source: Exercise Immunology Review. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, IMUNOLOGIA, TECIDO ADIPOSO, FENÓTIPOS, SISTEMA IMUNE, MACRÓFAGOS, CONDICIONAMENTO FÍSICO, INTERLEUCINAS, EXERCÍCIO FÍSICO, OBESIDADE, CAMUNDONGOS
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SILVEIRA, Loreana Sanches et al. Macrophage immunophenotype but not anti-inflammatory profile is modulated by peroxisome proliferator-activated receptor gamma (PPARγ) in exercised obese mice. Exercise Immunology Review, v. 26, p. 10-22, 2020Tradução . . Disponível em: http://eir-isei.de/2020/eir-2020-010-article.pdf. Acesso em: 16 nov. 2024.
APA
Silveira, L. S., Biondo, L. A., Teixeira, A. A. de S., Lima Junior, E. A. de, Castoldi, A., Lira, F. S., et al. (2020). Macrophage immunophenotype but not anti-inflammatory profile is modulated by peroxisome proliferator-activated receptor gamma (PPARγ) in exercised obese mice. Exercise Immunology Review, 26, 10-22. Recuperado de http://eir-isei.de/2020/eir-2020-010-article.pdf
NLM
Silveira LS, Biondo LA, Teixeira AA de S, Lima Junior EA de, Castoldi A, Lira FS, Câmara NOS, Festuccia WTL, Rosa Neto JC. Macrophage immunophenotype but not anti-inflammatory profile is modulated by peroxisome proliferator-activated receptor gamma (PPARγ) in exercised obese mice [Internet]. Exercise Immunology Review. 2020 ; 26 10-22.[citado 2024 nov. 16 ] Available from: http://eir-isei.de/2020/eir-2020-010-article.pdf
Vancouver
Silveira LS, Biondo LA, Teixeira AA de S, Lima Junior EA de, Castoldi A, Lira FS, Câmara NOS, Festuccia WTL, Rosa Neto JC. Macrophage immunophenotype but not anti-inflammatory profile is modulated by peroxisome proliferator-activated receptor gamma (PPARγ) in exercised obese mice [Internet]. Exercise Immunology Review. 2020 ; 26 10-22.[citado 2024 nov. 16 ] Available from: http://eir-isei.de/2020/eir-2020-010-article.pdf
Artigo de periodico
Rhipicephalus microplus serpins interfere with host immune responses by specifically modulating mast cells and lymphocytes (2020)
Coutinho, Mariana Loner; Bizarro, Bruna; Tirloni, Lucas ; Berger, Markus ; Oliveira, Carlo Jose Freire ; Nunes, Anderson de Sá ; Vaz Júnior, Itabajara Silva
Source: Ticks and Tick-borne Diseases. Unidade: ICB
Subjects: IMUNOLOGIA, CARRAPATOS, SALIVA, PROTEÍNAS, MASTÓCITOS, MACRÓFAGOS, CITOCINAS, LINFÓCITOS, ADJUVANTES IMUNOLÓGICOS
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COUTINHO, Mariana Loner et al. Rhipicephalus microplus serpins interfere with host immune responses by specifically modulating mast cells and lymphocytes. Ticks and Tick-borne Diseases, p. 9 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.ttbdis.2020.101425. Acesso em: 16 nov. 2024.
APA
Coutinho, M. L., Bizarro, B., Tirloni, L., Berger, M., Oliveira, C. J. F., Nunes, A. de S., & Vaz Júnior, I. S. (2020). Rhipicephalus microplus serpins interfere with host immune responses by specifically modulating mast cells and lymphocytes. Ticks and Tick-borne Diseases, 9 . doi:10.1016/j.ttbdis.2020.101425
NLM
Coutinho ML, Bizarro B, Tirloni L, Berger M, Oliveira CJF, Nunes A de S, Vaz Júnior IS. Rhipicephalus microplus serpins interfere with host immune responses by specifically modulating mast cells and lymphocytes [Internet]. Ticks and Tick-borne Diseases. 2020 ; 9 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.ttbdis.2020.101425
Vancouver
Coutinho ML, Bizarro B, Tirloni L, Berger M, Oliveira CJF, Nunes A de S, Vaz Júnior IS. Rhipicephalus microplus serpins interfere with host immune responses by specifically modulating mast cells and lymphocytes [Internet]. Ticks and Tick-borne Diseases. 2020 ; 9 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.ttbdis.2020.101425
Artigo de periodico
Differential expression of the inflammasome complex genes in systemic lupus erythematosus (2020)
Cruz, Heide Lacerda Alves da; Cavalcanti, Catarina Addobbati Jordão; Silva, Jaqueline de Azêvedo ; Lima, Camila Albertina Dantas de; Fragoso, Thiago Sotero; Barbosa, Alexandre Domingues; Dantas, Andréa Tavares; Mariz, Henrique de Ataíde; Duarte, Angela Luzia Branco Pingo; Pontillo, Alessandra ; Crovella, Sergio; Garcia, Paula Sandrin
Source: Immunogenetics. Unidade: ICB
Subjects: IMUNOLOGIA, LÚPUS ERITEMATOSO SISTÊMICO, DOENÇAS AUTOIMUNES, POLIMORFISMO, EXPRESSÃO GÊNICA, NEFRITE, MONÓCITOS, IMUNOGENÉTICA
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ABNT
CRUZ, Heide Lacerda Alves da et al. Differential expression of the inflammasome complex genes in systemic lupus erythematosus. Immunogenetics, p. 8 , 2020Tradução . . Disponível em: https://doi.org/10.1007/s00251-020-01158-6. Acesso em: 16 nov. 2024.
APA
Cruz, H. L. A. da, Cavalcanti, C. A. J., Silva, J. de A., Lima, C. A. D. de, Fragoso, T. S., Barbosa, A. D., et al. (2020). Differential expression of the inflammasome complex genes in systemic lupus erythematosus. Immunogenetics, 8 . doi:10.1007/s00251-020-01158-6
NLM
Cruz HLA da, Cavalcanti CAJ, Silva J de A, Lima CAD de, Fragoso TS, Barbosa AD, Dantas AT, Mariz H de A, Duarte ALBP, Pontillo A, Crovella S, Garcia PS. Differential expression of the inflammasome complex genes in systemic lupus erythematosus [Internet]. Immunogenetics. 2020 ;8 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00251-020-01158-6
Vancouver
Cruz HLA da, Cavalcanti CAJ, Silva J de A, Lima CAD de, Fragoso TS, Barbosa AD, Dantas AT, Mariz H de A, Duarte ALBP, Pontillo A, Crovella S, Garcia PS. Differential expression of the inflammasome complex genes in systemic lupus erythematosus [Internet]. Immunogenetics. 2020 ;8 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00251-020-01158-6
Trabalho de evento-resumo periodico
In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites (2019)
Alvarez, José Maria ; Santana D. Y.; Silva do Nascimento, R.; Fevereiro, M.; Borges da Silva, H.; Barreto-Chaves, Maria Luiza Morais ; Marinho, Claudio Romero Farias ; Epiphanio, Sabrina ; Câmara, Niels Olsen Saraiva ; Lima, Maria Regina D'Império
Source: European Journal of Immunology. Unidades: ICB, FCF
Subjects: DOENÇA DE CHAGAS EM ANIMAL, TRYPANOSOMA CRUZI, CARDIOPATIAS, CAMUNDONGOS, ANIMAIS PARASITOS, IMUNOLOGIA, IMUNOPARASITOLOGIA, ANTIPARASITÁRIOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALVAREZ, José Maria et al. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. Disponível em: https://doi.org/10.1002/eji.201970400. Acesso em: 16 nov. 2024. , 2019
APA
Alvarez, J. M., Santana D. Y.,, Silva do Nascimento, R., Fevereiro, M., Borges da Silva, H., Barreto-Chaves, M. L. M., et al. (2019). In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. doi:10.1002/eji.201970400
NLM
Alvarez JM, Santana D. Y., Silva do Nascimento R, Fevereiro M, Borges da Silva H, Barreto-Chaves MLM, Marinho CRF, Epiphanio S, Câmara NOS, Lima MRD'I. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites [Internet]. European Journal of Immunology. 2019 ; 49 1196-1197.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/eji.201970400
Vancouver
Alvarez JM, Santana D. Y., Silva do Nascimento R, Fevereiro M, Borges da Silva H, Barreto-Chaves MLM, Marinho CRF, Epiphanio S, Câmara NOS, Lima MRD'I. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites [Internet]. European Journal of Immunology. 2019 ; 49 1196-1197.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/eji.201970400
Artigo de periodico
Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients (2019)
Pedroso, Juliana de Fatima ; Lotfollahi, Zahra ; Albattarni, Ghadeer; Schulz, Maiara Arruda; Monteiro, Andrea Moreira; Sehnem, André Luiz; Gidlund, Magnus Ake ; Figueiredo Neto, Antonio Martins ; Jardini, Maria Aparecida Neves
Source: Scientific Reports. Unidades: IF, ICB
Subjects: PERIODONTITE, DIABETES MELLITUS, DOENÇAS CARDIOVASCULARES, HIPERGLICEMIA, INFLAMAÇÃO, COLESTEROL, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEDROSO, Juliana de Fatima et al. Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients. Scientific Reports, v. 9, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-52498-7. Acesso em: 16 nov. 2024.
APA
Pedroso, J. de F., Lotfollahi, Z., Albattarni, G., Schulz, M. A., Monteiro, A. M., Sehnem, A. L., et al. (2019). Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients. Scientific Reports, 9. doi:10.1038/s41598-019-52498-7
NLM
Pedroso J de F, Lotfollahi Z, Albattarni G, Schulz MA, Monteiro AM, Sehnem AL, Gidlund MA, Figueiredo Neto AM, Jardini MAN. Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients [Internet]. Scientific Reports. 2019 ; 9[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-019-52498-7
Vancouver
Pedroso J de F, Lotfollahi Z, Albattarni G, Schulz MA, Monteiro AM, Sehnem AL, Gidlund MA, Figueiredo Neto AM, Jardini MAN. Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients [Internet]. Scientific Reports. 2019 ; 9[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-019-52498-7
Trabalho de evento-resumo periodico
Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway (2019)
Bomfim, Caio César Barbosa ; Amaral, Eduardo Pinheiro ; Mittereder, Lara ; McCann, K.; Costa, Diego Luís ; Namasivayam, Sivaranjani ; Swamydas, M.; Moayeri, Mahtab ; Lima, Maria Regina D'Império ; Sher, Alan
Source: European Journal of Immunology. Conference titles: International Congress of Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, IMUNOLOGIA, MYCOBACTERIUM TUBERCULOSIS, ENZIMAS, METABÓLITOS, ATIVAÇÃO ENZIMÁTICA, MACRÓFAGOS, FAGOCITOSE, INTERFERONS, MEMBRANA PLASMÁTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOMFIM, Caio César Barbosa et al. Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. Disponível em: https://doi.org/10.1002/eji.201970400. Acesso em: 16 nov. 2024. , 2019
APA
Bomfim, C. C. B., Amaral, E. P., Mittereder, L., McCann, K., Costa, D. L., Namasivayam, S., et al. (2019). Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. doi:10.1002/eji.201970400
NLM
Bomfim CCB, Amaral EP, Mittereder L, McCann K, Costa DL, Namasivayam S, Swamydas M, Moayeri M, Lima MRD'I, Sher A. Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway [Internet]. European Journal of Immunology. 2019 ; 49 1129.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/eji.201970400
Vancouver
Bomfim CCB, Amaral EP, Mittereder L, McCann K, Costa DL, Namasivayam S, Swamydas M, Moayeri M, Lima MRD'I, Sher A. Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway [Internet]. European Journal of Immunology. 2019 ; 49 1129.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/eji.201970400
Parte de monografia/livro
Systems Immunology (2018)
Lever, Melissa; Hirata, Thiago Dominguez Crespo; Russo, Pedro de Sá Tavares; Nakaya, Helder Takashi Imoto
Source: Theoretical and Applied Aspects of Systems Biology. Unidade: FCF
Subjects: IMUNOLOGIA, BIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEVER, Melissa et al. Systems Immunology. Theoretical and Applied Aspects of Systems Biology. Tradução . Berlin: Springer, 2018. . . Acesso em: 16 nov. 2024.
APA
Lever, M., Hirata, T. D. C., Russo, P. de S. T., & Nakaya, H. T. I. (2018). Systems Immunology. In Theoretical and Applied Aspects of Systems Biology. Berlin: Springer.
NLM
Lever M, Hirata TDC, Russo P de ST, Nakaya HTI. Systems Immunology. In: Theoretical and Applied Aspects of Systems Biology. Berlin: Springer; 2018. [citado 2024 nov. 16 ]
Vancouver
Lever M, Hirata TDC, Russo P de ST, Nakaya HTI. Systems Immunology. In: Theoretical and Applied Aspects of Systems Biology. Berlin: Springer; 2018. [citado 2024 nov. 16 ]
Artigo de periodico
mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function (2017)
Paschoal, Vivian Almeida; Amano, Mariane Tami; Oliveira,Thiago Belchior de; Magdalon, Juliana; Perandini, Patricia Chimin; Andrade, Maynara Lucca; Ortiz-Silva, Milene; Castro, Érique; Yamashita, Alex Shimura; Rosa Neto, José Cesar ; Câmara, Niels Olsen Saraiva ; Festuccia, William Tadeu Lara
Source: Immunobiology. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA, IMUNOLOGIA, MACRÓFAGOS, TECIDO ADIPOSO, INFLAMAÇÃO, FENÓTIPOS, CITOCINAS, OBESIDADE, CAMUNDONGOS
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ABNT
PASCHOAL, Vivian Almeida et al. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, v. 222, n. 2, p. 261-271, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2016.09.014. Acesso em: 16 nov. 2024.
APA
Paschoal, V. A., Amano, M. T., Oliveira, T. B. de, Magdalon, J., Perandini, P. C., Andrade, M. L., et al. (2017). mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, 222( 2), 261-271. doi:10.1016/j.imbio.2016.09.014
NLM
Paschoal VA, Amano MT, Oliveira TB de, Magdalon J, Perandini PC, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Vancouver
Paschoal VA, Amano MT, Oliveira TB de, Magdalon J, Perandini PC, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Artigo de periodico
Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency (2016)
Silva, Karina Ribeiro da; Fraga, Tatiana Rodrigues; Lucatelli, Juliana Faggion; Grumach, Anete Sevciovic; Isaac, Lourdes
Source: Immunobiology. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
SILVA, Karina Ribeiro da et al. Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency. Immunobiology, v. 221, n. 5, p. 641-649, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2016.01.005. Acesso em: 16 nov. 2024.
APA
Silva, K. R. da, Fraga, T. R., Lucatelli, J. F., Grumach, A. S., & Isaac, L. (2016). Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency. Immunobiology, 221( 5), 641-649. doi:10.1016/j.imbio.2016.01.005
NLM
Silva KR da, Fraga TR, Lucatelli JF, Grumach AS, Isaac L. Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency [Internet]. Immunobiology. 2016 ; 221( 5): 641-649.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2016.01.005
Vancouver
Silva KR da, Fraga TR, Lucatelli JF, Grumach AS, Isaac L. Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency [Internet]. Immunobiology. 2016 ; 221( 5): 641-649.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2016.01.005
Artigo de periodico
Do HLA class II genes protect against pulmonary tuberculosis? A systematic review and meta-analysis (2016)
Oliveira-Cortez, A.; Melo, A. C.; Chaves, V. E.; Condino-Neto, A.; Camargos, P.
Source: European Journal of Clinical Microbiology & Infectious Diseases. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA-CORTEZ, A. et al. Do HLA class II genes protect against pulmonary tuberculosis? A systematic review and meta-analysis. European Journal of Clinical Microbiology & Infectious Diseases, v. 35, n. 10, p. 1567-1580, 2016Tradução . . Disponível em: https://doi.org/10.1007/s10096-016-2713-x. Acesso em: 16 nov. 2024.
APA
Oliveira-Cortez, A., Melo, A. C., Chaves, V. E., Condino-Neto, A., & Camargos, P. (2016). Do HLA class II genes protect against pulmonary tuberculosis? A systematic review and meta-analysis. European Journal of Clinical Microbiology & Infectious Diseases, 35( 10), 1567-1580. doi:10.1007/s10096-016-2713-x
NLM
Oliveira-Cortez A, Melo AC, Chaves VE, Condino-Neto A, Camargos P. Do HLA class II genes protect against pulmonary tuberculosis? A systematic review and meta-analysis [Internet]. European Journal of Clinical Microbiology & Infectious Diseases. 2016 ; 35( 10): 1567-1580.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s10096-016-2713-x
Vancouver
Oliveira-Cortez A, Melo AC, Chaves VE, Condino-Neto A, Camargos P. Do HLA class II genes protect against pulmonary tuberculosis? A systematic review and meta-analysis [Internet]. European Journal of Clinical Microbiology & Infectious Diseases. 2016 ; 35( 10): 1567-1580.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s10096-016-2713-x

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