Source: The Journal of Clinical Endocrinology & Metabolism. Unidade: FM
Subjects: ENDOCRINOLOGIA, ANATOMIA PATOLÓGICA
ABNT
MELO, Karla F. S. et al. An unusual phenotype of fraiser syndrome due to IVS9 +4C>T mutation in the WT1 gene: predominantly male ambiguous genitalia and absence of gonadal dysgenesis. The Journal of Clinical Endocrinology & Metabolism, v. 87, n. 6, p. 2500-2505, 2002Tradução . . Acesso em: 14 out. 2024.APA
Melo, K. F. S., Martin, R. M., Costa, E. M. F., Carvalho, F., Jorge, A. A., Arnhold, I. J. P., & Mendonça, B. B. (2002). An unusual phenotype of fraiser syndrome due to IVS9 +4C>T mutation in the WT1 gene: predominantly male ambiguous genitalia and absence of gonadal dysgenesis. The Journal of Clinical Endocrinology & Metabolism, 87( 6), 2500-2505.NLM
Melo KFS, Martin RM, Costa EMF, Carvalho F, Jorge AA, Arnhold IJP, Mendonça BB. An unusual phenotype of fraiser syndrome due to IVS9 +4C>T mutation in the WT1 gene: predominantly male ambiguous genitalia and absence of gonadal dysgenesis. The Journal of Clinical Endocrinology & Metabolism. 2002 ; 87( 6): 2500-2505.[citado 2024 out. 14 ]Vancouver
Melo KFS, Martin RM, Costa EMF, Carvalho F, Jorge AA, Arnhold IJP, Mendonça BB. An unusual phenotype of fraiser syndrome due to IVS9 +4C>T mutation in the WT1 gene: predominantly male ambiguous genitalia and absence of gonadal dysgenesis. The Journal of Clinical Endocrinology & Metabolism. 2002 ; 87( 6): 2500-2505.[citado 2024 out. 14 ]