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  • Source: Scientific Reports. Unidade: IME

    Assunto: CIÊNCIA DA COMPUTAÇÃO

    Versão PublicadaAcesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      GUPTA, Shantanu et al. Dynamical modeling of miR‑34a, miR‑449a, and miR‑16 reveals numerous DDR signaling pathways regulating senescence, autophagy, and apoptosis in HeLa cells. Scientific Reports, v. 12, n. artigo 4911, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41598-022-08900-y. Acesso em: 08 out. 2025.
    • APA

      Gupta, S., Panda, P. K., Hashimoto, R. F., Samal, S. K., Mishra, S., Verma, S. K., et al. (2022). Dynamical modeling of miR‑34a, miR‑449a, and miR‑16 reveals numerous DDR signaling pathways regulating senescence, autophagy, and apoptosis in HeLa cells. Scientific Reports, 12( artigo 4911), 1-13. doi:10.1038/s41598-022-08900-y
    • NLM

      Gupta S, Panda PK, Hashimoto RF, Samal SK, Mishra S, Verma SK, Mishra YK, Ahuja R. Dynamical modeling of miR‑34a, miR‑449a, and miR‑16 reveals numerous DDR signaling pathways regulating senescence, autophagy, and apoptosis in HeLa cells [Internet]. Scientific Reports. 2022 ; 12( artigo 4911): 1-13.[citado 2025 out. 08 ] Available from: https://doi.org/10.1038/s41598-022-08900-y
    • Vancouver

      Gupta S, Panda PK, Hashimoto RF, Samal SK, Mishra S, Verma SK, Mishra YK, Ahuja R. Dynamical modeling of miR‑34a, miR‑449a, and miR‑16 reveals numerous DDR signaling pathways regulating senescence, autophagy, and apoptosis in HeLa cells [Internet]. Scientific Reports. 2022 ; 12( artigo 4911): 1-13.[citado 2025 out. 08 ] Available from: https://doi.org/10.1038/s41598-022-08900-y
  • Source: Scientific Reports. Unidade: FCF

    Subjects: NEOPLASIAS CEREBRAIS, QUIMIOTERAPIA, RADIOTERAPIA

    Versão PublicadaAcesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      RODRIGUES JUNIOR, Dorival Mendes et al. Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity. Scientific Reports, v. 12, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41598-022-25534-2. Acesso em: 08 out. 2025.
    • APA

      Rodrigues Junior, D. M., Raminelli, C., Hassanie, H., Trossini, G. H. G., Perecim, G. P., Puigsubira, L. C., et al. (2022). Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity. Scientific Reports, 12, 1-17. doi:10.1038/s41598-022-25534-2
    • NLM

      Rodrigues Junior DM, Raminelli C, Hassanie H, Trossini GHG, Perecim GP, Puigsubira LC, Moustakas A, Vettore AL. Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity [Internet]. Scientific Reports. 2022 ; 12 1-17.[citado 2025 out. 08 ] Available from: https://doi.org/10.1038/s41598-022-25534-2
    • Vancouver

      Rodrigues Junior DM, Raminelli C, Hassanie H, Trossini GHG, Perecim GP, Puigsubira LC, Moustakas A, Vettore AL. Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity [Internet]. Scientific Reports. 2022 ; 12 1-17.[citado 2025 out. 08 ] Available from: https://doi.org/10.1038/s41598-022-25534-2

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