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ASSONI, Amanda Faria e FOIJER, Floris e ZATZ, Mayana. Amyotrophic Lateral Sclerosis, FUS and Protein Synthesis Defects. Stem Cell Reviews and Reports, v. 19, p. 625–638, 2023Tradução . . Disponível em: https://doi.org/10.1007/s12015-022-10489-8. Acesso em: 17 jul. 2024.
APA
Assoni, A. F., Foijer, F., & Zatz, M. (2023). Amyotrophic Lateral Sclerosis, FUS and Protein Synthesis Defects. Stem Cell Reviews and Reports, 19, 625–638. doi:10.1007/s12015-022-10489-8
NLM
Assoni AF, Foijer F, Zatz M. Amyotrophic Lateral Sclerosis, FUS and Protein Synthesis Defects [Internet]. Stem Cell Reviews and Reports. 2023 ; 19 625–638.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12015-022-10489-8
Vancouver
Assoni AF, Foijer F, Zatz M. Amyotrophic Lateral Sclerosis, FUS and Protein Synthesis Defects [Internet]. Stem Cell Reviews and Reports. 2023 ; 19 625–638.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12015-022-10489-8
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KOWALTOWSKI, Alicia Juliana e NASLAVSKY, Michel e ZATZ, Mayana. Brazilian scientists denied waivers and discounts [Carta]. Nature. Berlin: Instituto de Química, Universidade de São Paulo. . Acesso em: 17 jul. 2024. , 2022
APA
Kowaltowski, A. J., Naslavsky, M., & Zatz, M. (2022). Brazilian scientists denied waivers and discounts [Carta]. Nature. Berlin: Instituto de Química, Universidade de São Paulo.
NLM
Kowaltowski AJ, Naslavsky M, Zatz M. Brazilian scientists denied waivers and discounts [Carta]. Nature. 2022 ; 603 793.[citado 2024 jul. 17 ]
Vancouver
Kowaltowski AJ, Naslavsky M, Zatz M. Brazilian scientists denied waivers and discounts [Carta]. Nature. 2022 ; 603 793.[citado 2024 jul. 17 ]
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ARAÚJO, Bruno H. S et al. Down Syndrome iPSC-Derived Astrocytes Impair Neuronal Synaptogenesis and the mTOR Pathway In Vitro. Molecular Neurobiology, v. 55, n. 7, p. 5962-5975, 2018Tradução . . Disponível em: https://doi.org/10.1007/s12035-017-0818-6. Acesso em: 17 jul. 2024.
APA
Araújo, B. H. S., Kaid, C., Souza, J. S. D., Silva, S. G. da, Goulart, E., Caires, L. C. J., et al. (2018). Down Syndrome iPSC-Derived Astrocytes Impair Neuronal Synaptogenesis and the mTOR Pathway In Vitro. Molecular Neurobiology, 55( 7), 5962-5975. doi:10.1007/s12035-017-0818-6
NLM
Araújo BHS, Kaid C, Souza JSD, Silva SG da, Goulart E, Caires LCJ, Musso CM, Torres LB, Ferrasa A, Herai R, Zatz M, Okamoto OK, Cavalheiro EA. Down Syndrome iPSC-Derived Astrocytes Impair Neuronal Synaptogenesis and the mTOR Pathway In Vitro [Internet]. Molecular Neurobiology. 2018 ; 55( 7): 5962-5975.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12035-017-0818-6
Vancouver
Araújo BHS, Kaid C, Souza JSD, Silva SG da, Goulart E, Caires LCJ, Musso CM, Torres LB, Ferrasa A, Herai R, Zatz M, Okamoto OK, Cavalheiro EA. Down Syndrome iPSC-Derived Astrocytes Impair Neuronal Synaptogenesis and the mTOR Pathway In Vitro [Internet]. Molecular Neurobiology. 2018 ; 55( 7): 5962-5975.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12035-017-0818-6
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COATTI, Giuliana Castello et al. Pericytes Extend Survival of ALS SOD1 Mice and Induce the Expression of Antioxidant Enzymes in the Murine Model and in IPSCs Derived Neuronal Cells from an ALS Patient. Stem Cell Reviews and Reports, v. 13, n. 5, p. 686–698, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12015-017-9752-2. Acesso em: 17 jul. 2024.
APA
Coatti, G. C., Frangini, M., Valadares, M. C., Gomes, J. P., Lima, N. O., Cavaçana, N., et al. (2017). Pericytes Extend Survival of ALS SOD1 Mice and Induce the Expression of Antioxidant Enzymes in the Murine Model and in IPSCs Derived Neuronal Cells from an ALS Patient. Stem Cell Reviews and Reports, 13( 5), 686–698. doi:10.1007/s12015-017-9752-2
NLM
Coatti GC, Frangini M, Valadares MC, Gomes JP, Lima NO, Cavaçana N, Assoni AF, Pelatti MV, Birbrair A, Lima ACP de, Singer JM, Rocha FMM, Da Silva GL, Mantovani MS, Macedo-Souza LI, Ferrari M de FR, Zatz M. Pericytes Extend Survival of ALS SOD1 Mice and Induce the Expression of Antioxidant Enzymes in the Murine Model and in IPSCs Derived Neuronal Cells from an ALS Patient [Internet]. Stem Cell Reviews and Reports. 2017 ; 13( 5): 686–698.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12015-017-9752-2
Vancouver
Coatti GC, Frangini M, Valadares MC, Gomes JP, Lima NO, Cavaçana N, Assoni AF, Pelatti MV, Birbrair A, Lima ACP de, Singer JM, Rocha FMM, Da Silva GL, Mantovani MS, Macedo-Souza LI, Ferrari M de FR, Zatz M. Pericytes Extend Survival of ALS SOD1 Mice and Induce the Expression of Antioxidant Enzymes in the Murine Model and in IPSCs Derived Neuronal Cells from an ALS Patient [Internet]. Stem Cell Reviews and Reports. 2017 ; 13( 5): 686–698.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12015-017-9752-2
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PELATTI, M. V et al. Transplantation of Human Adipose Mesenchymal Stem Cells in Non-Immunosuppressed GRMD Dogs is a Safe Procedure. Stem Cell Reviews and Reports, v. 12, p. 448-453, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12015-016-9659-3. Acesso em: 17 jul. 2024.
APA
Pelatti, M. V., Gomes, J. P. A., Vieira, N. M. S., Cangussu, E., Landini, V., Andrade, T., et al. (2016). Transplantation of Human Adipose Mesenchymal Stem Cells in Non-Immunosuppressed GRMD Dogs is a Safe Procedure. Stem Cell Reviews and Reports, 12, 448-453. doi:10.1007/s12015-016-9659-3
NLM
Pelatti MV, Gomes JPA, Vieira NMS, Cangussu E, Landini V, Andrade T, Sartori M, Petrus L, Zatz M. Transplantation of Human Adipose Mesenchymal Stem Cells in Non-Immunosuppressed GRMD Dogs is a Safe Procedure [Internet]. Stem Cell Reviews and Reports. 2016 ; 12 448-453.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12015-016-9659-3
Vancouver
Pelatti MV, Gomes JPA, Vieira NMS, Cangussu E, Landini V, Andrade T, Sartori M, Petrus L, Zatz M. Transplantation of Human Adipose Mesenchymal Stem Cells in Non-Immunosuppressed GRMD Dogs is a Safe Procedure [Internet]. Stem Cell Reviews and Reports. 2016 ; 12 448-453.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s12015-016-9659-3
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RICCI, G. e ZATZ, Mayana e TUPLER, R. Facioscapulohumeral muscular dystrophy: more complex than it appears. Current Molecular Medicine, v. 14, n. 8, p. 1052-1068, 2014Tradução . . Disponível em: https://doi.org/10.2174/1566524014666141010155054. Acesso em: 17 jul. 2024.
APA
Ricci, G., Zatz, M., & Tupler, R. (2014). Facioscapulohumeral muscular dystrophy: more complex than it appears. Current Molecular Medicine, 14( 8), 1052-1068. doi:10.2174/1566524014666141010155054
NLM
Ricci G, Zatz M, Tupler R. Facioscapulohumeral muscular dystrophy: more complex than it appears [Internet]. Current Molecular Medicine. 2014 ; 14( 8): 1052-1068.[citado 2024 jul. 17 ] Available from: https://doi.org/10.2174/1566524014666141010155054
Vancouver
Ricci G, Zatz M, Tupler R. Facioscapulohumeral muscular dystrophy: more complex than it appears [Internet]. Current Molecular Medicine. 2014 ; 14( 8): 1052-1068.[citado 2024 jul. 17 ] Available from: https://doi.org/10.2174/1566524014666141010155054
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HSU, Sandy Chan et al. Mutations in SLC20A2 are a major cause of familial idiopathic basal ganglia calcification. Neurogenetics, v. 14, p. online, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10048-012-0349-2. Acesso em: 17 jul. 2024.
APA
Hsu, S. C., Sears, R. L., Lemos, R. R., & Zatz, M. (2013). Mutations in SLC20A2 are a major cause of familial idiopathic basal ganglia calcification. Neurogenetics, 14, online. doi:10.1007/s10048-012-0349-2
NLM
Hsu SC, Sears RL, Lemos RR, Zatz M. Mutations in SLC20A2 are a major cause of familial idiopathic basal ganglia calcification [Internet]. Neurogenetics. 2013 ; 14 online.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s10048-012-0349-2
Vancouver
Hsu SC, Sears RL, Lemos RR, Zatz M. Mutations in SLC20A2 are a major cause of familial idiopathic basal ganglia calcification [Internet]. Neurogenetics. 2013 ; 14 online.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s10048-012-0349-2
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MACEDO-SOUZA, Lúcia Inês et al. Reevaluation of a large family defines a new locus for x-linked recessive pure spastic paraplegia (SPG34) on chromosome Xq25. Neurogenetics, v. 9, n. 3, p. 225-226, 2008Tradução . . Disponível em: https://doi.org/10.1007/s10048-008-0130-8. Acesso em: 17 jul. 2024.
APA
Macedo-Souza, L. I., Kok, F., Santos, S., Licinio, L., Lezirovitz, K., Nascimento, R. M. P., et al. (2008). Reevaluation of a large family defines a new locus for x-linked recessive pure spastic paraplegia (SPG34) on chromosome Xq25. Neurogenetics, 9( 3), 225-226. doi:10.1007/s10048-008-0130-8
NLM
Macedo-Souza LI, Kok F, Santos S, Licinio L, Lezirovitz K, Nascimento RMP, Bueno C, Martyn M, Leão EKE de A, Zatz M. Reevaluation of a large family defines a new locus for x-linked recessive pure spastic paraplegia (SPG34) on chromosome Xq25 [Internet]. Neurogenetics. 2008 ; 9( 3): 225-226.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s10048-008-0130-8
Vancouver
Macedo-Souza LI, Kok F, Santos S, Licinio L, Lezirovitz K, Nascimento RMP, Bueno C, Martyn M, Leão EKE de A, Zatz M. Reevaluation of a large family defines a new locus for x-linked recessive pure spastic paraplegia (SPG34) on chromosome Xq25 [Internet]. Neurogenetics. 2008 ; 9( 3): 225-226.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s10048-008-0130-8
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GOUVEIA, Telma Luciana Furtado et al. A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein. Journal of Molecular Medicine, v. 85, n. 4, p. 415-420, 2007Tradução . . Disponível em: https://doi.org/10.1007/s00109-007-0163-8. Acesso em: 17 jul. 2024.
APA
Gouveia, T. L. F., Kossugue, P. M., Paim, J. F., Zatz, M., Anderson, L. V. B., Nigro, V., & Vainzof, M. (2007). A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein. Journal of Molecular Medicine, 85( 4), 415-420. doi:10.1007/s00109-007-0163-8
NLM
Gouveia TLF, Kossugue PM, Paim JF, Zatz M, Anderson LVB, Nigro V, Vainzof M. A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein [Internet]. Journal of Molecular Medicine. 2007 ; 85( 4): 415-420.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s00109-007-0163-8
Vancouver
Gouveia TLF, Kossugue PM, Paim JF, Zatz M, Anderson LVB, Nigro V, Vainzof M. A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein [Internet]. Journal of Molecular Medicine. 2007 ; 85( 4): 415-420.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s00109-007-0163-8
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TONINI, Maria Manuela O et al. Equal proportions of affected cells in muscle and blood of a mosaic carrier of facioscapulohumeral muscular dystrophy. Human Genetics, v. 119, p. 23-28, 2006Tradução . . Disponível em: https://doi.org/10.1007/s00439-005-0100-2. Acesso em: 17 jul. 2024.
APA
Tonini, M. M. O., Lemmers, R. J. L. F., Pavanello, R. C. M., Cerqueira, A. M. P., Frants, R. R., Maarel, S. M. van der, & Zatz, M. (2006). Equal proportions of affected cells in muscle and blood of a mosaic carrier of facioscapulohumeral muscular dystrophy. Human Genetics, 119, 23-28. doi:10.1007/s00439-005-0100-2
NLM
Tonini MMO, Lemmers RJLF, Pavanello RCM, Cerqueira AMP, Frants RR, Maarel SM van der, Zatz M. Equal proportions of affected cells in muscle and blood of a mosaic carrier of facioscapulohumeral muscular dystrophy [Internet]. Human Genetics. 2006 ; 119 23-28.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s00439-005-0100-2
Vancouver
Tonini MMO, Lemmers RJLF, Pavanello RCM, Cerqueira AMP, Frants RR, Maarel SM van der, Zatz M. Equal proportions of affected cells in muscle and blood of a mosaic carrier of facioscapulohumeral muscular dystrophy [Internet]. Human Genetics. 2006 ; 119 23-28.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s00439-005-0100-2
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NISHIMURA, Agnes L e AL-CHALABI, Ammar e ZATZ, Mayana. A common founder for amyotrophic lateral sclerosis type 8 (ALS8) in the Brazilian population. Hum Genetics, v. 118, n. 3-4, p. 499-500, 2005Tradução . . Disponível em: https://doi.org/10.1007/s00439-005-0031-y. Acesso em: 17 jul. 2024.
APA
Nishimura, A. L., Al-Chalabi, A., & Zatz, M. (2005). A common founder for amyotrophic lateral sclerosis type 8 (ALS8) in the Brazilian population. Hum Genetics, 118( 3-4), 499-500. doi:10.1007/s00439-005-0031-y
NLM
Nishimura AL, Al-Chalabi A, Zatz M. A common founder for amyotrophic lateral sclerosis type 8 (ALS8) in the Brazilian population [Internet]. Hum Genetics. 2005 ; 118( 3-4): 499-500.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s00439-005-0031-y
Vancouver
Nishimura AL, Al-Chalabi A, Zatz M. A common founder for amyotrophic lateral sclerosis type 8 (ALS8) in the Brazilian population [Internet]. Hum Genetics. 2005 ; 118( 3-4): 499-500.[citado 2024 jul. 17 ] Available from: https://doi.org/10.1007/s00439-005-0031-y
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VAINZOF, Mariz et al. Deficiency of 'alfa'-actinin-3 (ACTN3) Occurs in different forms of muscular dystrophy. Neuropediatrics, v. 28, p. 223-228, 1997Tradução . . Acesso em: 17 jul. 2024.
APA
Vainzof, M., Costa, C. S., Marie, S. K. N., Moreira, E. S., Reed, U. C., Passos-Bueno, M. R., et al. (1997). Deficiency of 'alfa'-actinin-3 (ACTN3) Occurs in different forms of muscular dystrophy. Neuropediatrics, 28, 223-228.
NLM
Vainzof M, Costa CS, Marie SKN, Moreira ES, Reed UC, Passos-Bueno MR, Beggs A. H., Zatz M. Deficiency of 'alfa'-actinin-3 (ACTN3) Occurs in different forms of muscular dystrophy. Neuropediatrics. 1997 ; 28 223-228.[citado 2024 jul. 17 ]
Vancouver
Vainzof M, Costa CS, Marie SKN, Moreira ES, Reed UC, Passos-Bueno MR, Beggs A. H., Zatz M. Deficiency of 'alfa'-actinin-3 (ACTN3) Occurs in different forms of muscular dystrophy. Neuropediatrics. 1997 ; 28 223-228.[citado 2024 jul. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
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VAINZOF, Mariz et al. Deficiency of merosin (laminin m or 'ALFA'2) in congenital muscular dystrophy associated with cerebral white matter alterations. Neuropediatrics, v. 26, p. 293-7, 1995Tradução . . Acesso em: 17 jul. 2024.
APA
Vainzof, M., Marie, S. K. N., Reed, U. C., Schwartzman, J. S., Pavanello, R. C. M., Passos-Bueno, M. R., & Zatz, M. (1995). Deficiency of merosin (laminin m or 'ALFA'2) in congenital muscular dystrophy associated with cerebral white matter alterations. Neuropediatrics, 26, 293-7.
NLM
Vainzof M, Marie SKN, Reed UC, Schwartzman JS, Pavanello RCM, Passos-Bueno MR, Zatz M. Deficiency of merosin (laminin m or 'ALFA'2) in congenital muscular dystrophy associated with cerebral white matter alterations. Neuropediatrics. 1995 ;26 293-7.[citado 2024 jul. 17 ]
Vancouver
Vainzof M, Marie SKN, Reed UC, Schwartzman JS, Pavanello RCM, Passos-Bueno MR, Zatz M. Deficiency of merosin (laminin m or 'ALFA'2) in congenital muscular dystrophy associated with cerebral white matter alterations. Neuropediatrics. 1995 ;26 293-7.[citado 2024 jul. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
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VAINZOF, Mariz et al. Immunohistochemical dystrophin study of duchenne dystrophy with the concomitant use of the n-terminal and c-terminal antibodies. 1990, Anais.. Munich: Instituto de Biociências, Universidade de São Paulo, 1990. . Acesso em: 17 jul. 2024.
APA
Vainzof, M., Pavanello, R. C. M., Rapaport, D., Passos-Bueno, M. R., Zubrzycka-Gaarn, E. E., Hoffman, E., & Zatz, M. (1990). Immunohistochemical dystrophin study of duchenne dystrophy with the concomitant use of the n-terminal and c-terminal antibodies. In Abstracts. Munich: Instituto de Biociências, Universidade de São Paulo.
NLM
Vainzof M, Pavanello RCM, Rapaport D, Passos-Bueno MR, Zubrzycka-Gaarn EE, Hoffman E, Zatz M. Immunohistochemical dystrophin study of duchenne dystrophy with the concomitant use of the n-terminal and c-terminal antibodies. Abstracts. 1990 ;[citado 2024 jul. 17 ]
Vancouver
Vainzof M, Pavanello RCM, Rapaport D, Passos-Bueno MR, Zubrzycka-Gaarn EE, Hoffman E, Zatz M. Immunohistochemical dystrophin study of duchenne dystrophy with the concomitant use of the n-terminal and c-terminal antibodies. Abstracts. 1990 ;[citado 2024 jul. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
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RAPAPORT, D et al. Screening of dna deletions in duchenne dystrophy (dmd) through mental retardation. 1990, Anais.. Munich: Instituto de Biociências, Universidade de São Paulo, 1990. . Acesso em: 17 jul. 2024.
APA
Rapaport, D., Passos-Bueno, M. R., Brandao, L., Vainzof, M., Lima, M. A. B. O., & Zatz, M. (1990). Screening of dna deletions in duchenne dystrophy (dmd) through mental retardation. In Abstracts. Munich: Instituto de Biociências, Universidade de São Paulo.
NLM
Rapaport D, Passos-Bueno MR, Brandao L, Vainzof M, Lima MABO, Zatz M. Screening of dna deletions in duchenne dystrophy (dmd) through mental retardation. Abstracts. 1990 ;[citado 2024 jul. 17 ]
Vancouver
Rapaport D, Passos-Bueno MR, Brandao L, Vainzof M, Lima MABO, Zatz M. Screening of dna deletions in duchenne dystrophy (dmd) through mental retardation. Abstracts. 1990 ;[citado 2024 jul. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
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PASSOS-BUENO, Maria Rita et al. Linkage analysis using 6q probes in facioscapulohumeral dystrophy (fsh). 1990, Anais.. Munich: Instituto de Biociências, Universidade de São Paulo, 1990. . Acesso em: 17 jul. 2024.
APA
Passos-Bueno, M. R., Love, D., Byth, B., Terwillinger, J., Rapaport, D., Davies, K. E., & Zatz, M. (1990). Linkage analysis using 6q probes in facioscapulohumeral dystrophy (fsh). In Abstracts. Munich: Instituto de Biociências, Universidade de São Paulo.
NLM
Passos-Bueno MR, Love D, Byth B, Terwillinger J, Rapaport D, Davies KE, Zatz M. Linkage analysis using 6q probes in facioscapulohumeral dystrophy (fsh). Abstracts. 1990 ;[citado 2024 jul. 17 ]
Vancouver
Passos-Bueno MR, Love D, Byth B, Terwillinger J, Rapaport D, Davies KE, Zatz M. Linkage analysis using 6q probes in facioscapulohumeral dystrophy (fsh). Abstracts. 1990 ;[citado 2024 jul. 17 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
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ZATZ, Mayana et al. Recurrence of duchenne dystrophy through non-maternal lines in four genealogies. 1990, Anais.. Munich: Instituto de Biociências, Universidade de São Paulo, 1990. . Acesso em: 17 jul. 2024.
APA
Zatz, M., Passos-Bueno, M. R., Rapaport, D., & Vainzof, M. (1990). Recurrence of duchenne dystrophy through non-maternal lines in four genealogies. In Abstracts. Munich: Instituto de Biociências, Universidade de São Paulo.
NLM
Zatz M, Passos-Bueno MR, Rapaport D, Vainzof M. Recurrence of duchenne dystrophy through non-maternal lines in four genealogies. Abstracts. 1990 ;[citado 2024 jul. 17 ]
Vancouver
Zatz M, Passos-Bueno MR, Rapaport D, Vainzof M. Recurrence of duchenne dystrophy through non-maternal lines in four genealogies. Abstracts. 1990 ;[citado 2024 jul. 17 ]