Filtros : "Neuroscience" "IQ" Removido: "2006" Limpar

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  • Source: Neuroscience. Unidades: IQ, ICB

    Subjects: DOENÇA DE PARKINSON, NEURÔNIOS

    Acesso à fonteDOIHow to cite
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    • ABNT

      DATI, L. M et al. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, v. 356, p. 176-181, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.05.013. Acesso em: 17 set. 2024.
    • APA

      Dati, L. M., Ulrich, H., Real, C. C., Feng, Z. P., Sun, H. S., & Britto, L. R. G. de. (2017). Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model. Neuroscience, 356, 176-181. doi:10.1016/j.neuroscience.2017.05.013
    • NLM

      Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013
    • Vancouver

      Dati LM, Ulrich H, Real CC, Feng ZP, Sun HS, Britto LRG de. Carvacrol promotes neuroprotection in the mouse hemiparkinsonian model [Internet]. Neuroscience. 2017 ; 356 176-181.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.05.013
  • Source: Neuroscience. Unidade: IQ

    Subjects: BRADICININA, COMPOSTOS DE FÓSFORO

    Acesso à fonteDOIHow to cite
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    • ABNT

      TORRES-RIVERA, Wilmarie et al. Kinin-B2 receptor exerted neuroprotection after diisopropylfluorophosphate-induced neuronal damage. Neuroscience, v. 247, p. 273-279, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.05.054. Acesso em: 17 set. 2024.
    • APA

      Torres-Rivera, W., Pérez, D., Park, K. Y., Carrasco, M., Platt, M. O., Eterovic, V. A., et al. (2013). Kinin-B2 receptor exerted neuroprotection after diisopropylfluorophosphate-induced neuronal damage. Neuroscience, 247, 273-279. doi:10.1016/j.neuroscience.2013.05.054
    • NLM

      Torres-Rivera W, Pérez D, Park KY, Carrasco M, Platt MO, Eterovic VA, Ferchmin PA, Ulrich H, Martins AH. Kinin-B2 receptor exerted neuroprotection after diisopropylfluorophosphate-induced neuronal damage [Internet]. Neuroscience. 2013 ; 247 273-279.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.05.054
    • Vancouver

      Torres-Rivera W, Pérez D, Park KY, Carrasco M, Platt MO, Eterovic VA, Ferchmin PA, Ulrich H, Martins AH. Kinin-B2 receptor exerted neuroprotection after diisopropylfluorophosphate-induced neuronal damage [Internet]. Neuroscience. 2013 ; 247 273-279.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.05.054
  • Source: Neuroscience. Unidade: IQ

    Subjects: RECEPTORES, GLUTAMATOS

    Acesso à fonteDOIHow to cite
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    • ABNT

      BICCA, M. A et al. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice. Neuroscience, v. 192, n. 29, p. 631-641, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2011.06.038. Acesso em: 17 set. 2024.
    • APA

      Bicca, M. A., Figueiredo, C. P., Piermartiri, T. C., Meotti, F. C., Bouzon, Z. L., Tasca, C. I., et al. (2011). The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice. Neuroscience, 192( 29), 631-641. doi:10.1016/j.neuroscience.2011.06.038
    • NLM

      Bicca MA, Figueiredo CP, Piermartiri TC, Meotti FC, Bouzon ZL, Tasca CI, Medeiros R, Calixto JB. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice [Internet]. Neuroscience. 2011 ; 192( 29): 631-641.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.06.038
    • Vancouver

      Bicca MA, Figueiredo CP, Piermartiri TC, Meotti FC, Bouzon ZL, Tasca CI, Medeiros R, Calixto JB. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice [Internet]. Neuroscience. 2011 ; 192( 29): 631-641.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.06.038
  • Source: Neuroscience. Unidades: ICB, IQ

    Subjects: FISIOLOGIA, BIOQUÍMICA, CARCINOMA

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    • ABNT

      RESENDE, Rodrigo Ribeiro et al. P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation. Neuroscience, v. 146, n. 3, p. 1169-1181, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2007.02.041. Acesso em: 17 set. 2024.
    • APA

      Resende, R. R., Majumder, P., Gomes, K. N., Britto, L. R. G. de, & Ulrich, H. (2007). P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation. Neuroscience, 146( 3), 1169-1181. doi:10.1016/j.neuroscience.2007.02.041
    • NLM

      Resende RR, Majumder P, Gomes KN, Britto LRG de, Ulrich H. P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation [Internet]. Neuroscience. 2007 ; 146( 3): 1169-1181.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2007.02.041
    • Vancouver

      Resende RR, Majumder P, Gomes KN, Britto LRG de, Ulrich H. P19 embryonal carcinoma cells as in vitro model for studying purinergic receptor expression and modulation of N-methyl-D-aspartate-glutamate and acetylcholine receptors during neuronal differentiation [Internet]. Neuroscience. 2007 ; 146( 3): 1169-1181.[citado 2024 set. 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2007.02.041

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