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Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 16 nov. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico-carta/editorial
Thermal and chronological stability of monocyte distribution width (MDW), the new biomarker for sepsis. [Carta] (2022)
Bordignon, Jardel Cristiano; Gardona, Rodrigo Galvao Bueno; Vasconcellos, Leonardo de Souza; Iachinski, Joslaine Schuartz; Dambros, Robbson Haugusto; Arruda, Gisele; Reis, Beatriz Castro; Abdala, Edson
Source: Clinical chemistry and laboratory medicine. Unidade: FM
Subjects: BIOMARCADORES, SINAIS E SINTOMAS, SEPSE
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ABNT
BORDIGNON, Jardel Cristiano et al. Thermal and chronological stability of monocyte distribution width (MDW), the new biomarker for sepsis. [Carta]. Clinical chemistry and laboratory medicine. Berlin: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1515/cclm-2022-0485. Acesso em: 16 nov. 2024. , 2022
APA
Bordignon, J. C., Gardona, R. G. B., Vasconcellos, L. de S., Iachinski, J. S., Dambros, R. H., Arruda, G., et al. (2022). Thermal and chronological stability of monocyte distribution width (MDW), the new biomarker for sepsis. [Carta]. Clinical chemistry and laboratory medicine. Berlin: Faculdade de Medicina, Universidade de São Paulo. doi:10.1515/cclm-2022-0485
NLM
Bordignon JC, Gardona RGB, Vasconcellos L de S, Iachinski JS, Dambros RH, Arruda G, Reis BC, Abdala E. Thermal and chronological stability of monocyte distribution width (MDW), the new biomarker for sepsis. [Carta] [Internet]. Clinical chemistry and laboratory medicine. 2022 ; 60( 10): E232-E234.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1515/cclm-2022-0485
Vancouver
Bordignon JC, Gardona RGB, Vasconcellos L de S, Iachinski JS, Dambros RH, Arruda G, Reis BC, Abdala E. Thermal and chronological stability of monocyte distribution width (MDW), the new biomarker for sepsis. [Carta] [Internet]. Clinical chemistry and laboratory medicine. 2022 ; 60( 10): E232-E234.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1515/cclm-2022-0485
Artigo de periodico
Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock (2021)
Rodrigues, Pedro Mendes de Azambuja; Valente, Richard Hemmi ; Brunoro, Giselle Villa Flor; Nakaya, Helder Takashi Imoto ; Pereira, Mariana Araujo; Bozza, Patricia Torres; Bozza, Fernando Augusto ; Trugilho, Monique Ramos de Oliveira
Source: Scientifc Reports. Unidade: FCF
Subjects: SEPSE, MONÓCITOS, METABOLISMO ENERGÉTICO, IMUNOLOGIA, INFLAMAÇÃO, PROTEÔMICA
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ABNT
RODRIGUES, Pedro Mendes de Azambuja et al. Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock. Scientifc Reports, v. 11, p. 1-10 art. 15149, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41598-021-94474-0. Acesso em: 16 nov. 2024.
APA
Rodrigues, P. M. de A., Valente, R. H., Brunoro, G. V. F., Nakaya, H. T. I., Pereira, M. A., Bozza, P. T., et al. (2021). Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock. Scientifc Reports, 11, 1-10 art. 15149. doi:10.1038/s41598-021-94474-0
NLM
Rodrigues PM de A, Valente RH, Brunoro GVF, Nakaya HTI, Pereira MA, Bozza PT, Bozza FA, Trugilho MR de O. Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock [Internet]. Scientifc Reports. 2021 ; 11 1-10 art. 15149.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-021-94474-0
Vancouver
Rodrigues PM de A, Valente RH, Brunoro GVF, Nakaya HTI, Pereira MA, Bozza PT, Bozza FA, Trugilho MR de O. Proteomics reveals disturbances in the immune response and energy metabolism of monocytes from patients with septic shock [Internet]. Scientifc Reports. 2021 ; 11 1-10 art. 15149.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-021-94474-0
Artigo de periodico
Microglial activation modulates neuroendocrine secretion during experimental sepsis (2021)
Costa, Luís Henrique Angenendt da ; Santos Junior, Nilton Nascimento dos ; Catalão, Carlos Henrique Rocha ; Rocha, Maria José Alves da
Source: Molecular Neurobiology. Unidades: FMRP, FORP
Subjects: SISTEMA NERVOSO CENTRAL, RATOS WISTAR, SEPSE, INFLAMAÇÃO, INIBIDORES DE ENZIMAS
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ABNT
COSTA, Luís Henrique Angenendt da et al. Microglial activation modulates neuroendocrine secretion during experimental sepsis. Molecular Neurobiology, v. 58, n. 5, p. 2133-2144, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-020-02241-5. Acesso em: 16 nov. 2024.
APA
Costa, L. H. A. da, Santos Junior, N. N. dos, Catalão, C. H. R., & Rocha, M. J. A. da. (2021). Microglial activation modulates neuroendocrine secretion during experimental sepsis. Molecular Neurobiology, 58( 5), 2133-2144. doi:10.1007/s12035-020-02241-5
NLM
Costa LHA da, Santos Junior NN dos, Catalão CHR, Rocha MJA da. Microglial activation modulates neuroendocrine secretion during experimental sepsis [Internet]. Molecular Neurobiology. 2021 ; 58( 5): 2133-2144.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s12035-020-02241-5
Vancouver
Costa LHA da, Santos Junior NN dos, Catalão CHR, Rocha MJA da. Microglial activation modulates neuroendocrine secretion during experimental sepsis [Internet]. Molecular Neurobiology. 2021 ; 58( 5): 2133-2144.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s12035-020-02241-5
Artigo de periodico-carta/editorial
Fluid overload in the PICU [Carta]: still a Challenge (2018)
Colleti Junior, Jose; Carvalho, Werther Brunow de
Source: Journal of pediatric intensive care. Unidade: FM
Subjects: CRIANÇAS, RESSUSCITAÇÃO, MORTALIDADE, SEPSE
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ABNT
COLLETI JUNIOR, Jose e CARVALHO, Werther Brunow de. Fluid overload in the PICU [Carta]: still a Challenge. Journal of pediatric intensive care. Stuttgart: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1055/s-0037-1608953. Acesso em: 16 nov. 2024. , 2018
APA
Colleti Junior, J., & Carvalho, W. B. de. (2018). Fluid overload in the PICU [Carta]: still a Challenge. Journal of pediatric intensive care. Stuttgart: Faculdade de Medicina, Universidade de São Paulo. doi:10.1055/s-0037-1608953
NLM
Colleti Junior J, Carvalho WB de. Fluid overload in the PICU [Carta]: still a Challenge [Internet]. Journal of pediatric intensive care. 2018 ; 7( 1): 67-67.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1055/s-0037-1608953
Vancouver
Colleti Junior J, Carvalho WB de. Fluid overload in the PICU [Carta]: still a Challenge [Internet]. Journal of pediatric intensive care. 2018 ; 7( 1): 67-67.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1055/s-0037-1608953
Artigo de periodico
Cathelicidin-deficient mice exhibit increased survival and upregulation of key inflammatory response genes following cecal ligation and puncture (2017)
Severino, Patricia; Ariga, Suely Kubo; Barbeiro, Hermes Vieira; Lima, Thais Martins de; Silva, Elisangela de Paula; Ribeiro, Denise Frediani; Machado, Marcel Cerqueira César; Nizet, Victor; Silva, Fabiano Pinheiro da
Source: Journal of molecular medicine (Berlin, Germany). Unidade: FM
Subjects: SEPSE, INFLAMAÇÃO, PEPTÍDEOS, EXPRESSÃO GÊNICA
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ABNT
SEVERINO, Patricia et al. Cathelicidin-deficient mice exhibit increased survival and upregulation of key inflammatory response genes following cecal ligation and puncture. Journal of molecular medicine (Berlin, Germany), v. 95, p. 995-1003, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00109-017-1555-z. Acesso em: 16 nov. 2024.
APA
Severino, P., Ariga, S. K., Barbeiro, H. V., Lima, T. M. de, Silva, E. de P., Ribeiro, D. F., et al. (2017). Cathelicidin-deficient mice exhibit increased survival and upregulation of key inflammatory response genes following cecal ligation and puncture. Journal of molecular medicine (Berlin, Germany), 95, 995-1003. doi:10.1007/s00109-017-1555-z
NLM
Severino P, Ariga SK, Barbeiro HV, Lima TM de, Silva E de P, Ribeiro DF, Machado MCC, Nizet V, Silva FP da. Cathelicidin-deficient mice exhibit increased survival and upregulation of key inflammatory response genes following cecal ligation and puncture [Internet]. Journal of molecular medicine (Berlin, Germany). 2017 ; 95 995-1003.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00109-017-1555-z
Vancouver
Severino P, Ariga SK, Barbeiro HV, Lima TM de, Silva E de P, Ribeiro DF, Machado MCC, Nizet V, Silva FP da. Cathelicidin-deficient mice exhibit increased survival and upregulation of key inflammatory response genes following cecal ligation and puncture [Internet]. Journal of molecular medicine (Berlin, Germany). 2017 ; 95 995-1003.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00109-017-1555-z
Artigo de periodico
Brain oxidative stress during experimental sepsis is attenuated by simvastatin administration (2016)
Catalão, Carlos Henrique Rocha; Santos Júnior, Nilton Nascimento; Costa, Luís Henrique Angenendt da; Souza, Anderson Oliveira; Alberici, Luciane Carla; Rocha, Maria José Alves
Source: Molecular Neurobiology. Unidades: FCFRP, FORP
Subjects: ANTIOXIDANTES, DOENÇAS CEREBRAIS, ESTRESSE OXIDATIVO, FÁRMACOS, SEPSE
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ABNT
CATALÃO, Carlos Henrique Rocha et al. Brain oxidative stress during experimental sepsis is attenuated by simvastatin administration. Molecular Neurobiology, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12035-016-0218-3. Acesso em: 16 nov. 2024.
APA
Catalão, C. H. R., Santos Júnior, N. N., Costa, L. H. A. da, Souza, A. O., Alberici, L. C., & Rocha, M. J. A. (2016). Brain oxidative stress during experimental sepsis is attenuated by simvastatin administration. Molecular Neurobiology. doi:10.1007/s12035-016-0218-3
NLM
Catalão CHR, Santos Júnior NN, Costa LHA da, Souza AO, Alberici LC, Rocha MJA. Brain oxidative stress during experimental sepsis is attenuated by simvastatin administration [Internet]. Molecular Neurobiology. 2016 ;[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s12035-016-0218-3
Vancouver
Catalão CHR, Santos Júnior NN, Costa LHA da, Souza AO, Alberici LC, Rocha MJA. Brain oxidative stress during experimental sepsis is attenuated by simvastatin administration [Internet]. Molecular Neurobiology. 2016 ;[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s12035-016-0218-3
Trabalho de evento-anais periodico
Effects of sildenafil in a porcine model of endotoxemia (2014)
Kemper, D. A; Otsuki, Denise Aya; Maia, D. R; Queiroz-Hazarbassanov, N. G; Gomes, Cristina de Oliveira Massoco-Salles ; Auler Júnior, José Otávio Costa; Fantoni, Denise Tabacchi
Source: Intensive Care Medicine Experimental. Conference titles: ESICM LIVES. Unidades: FMVZ, FM
Subjects: SUÍNOS, PNEUMOPATIAS (TRATAMENTO), SEPSE, MODELOS ANIMAIS DE DOENÇAS
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ABNT
KEMPER, D. A et al. Effects of sildenafil in a porcine model of endotoxemia. Intensive Care Medicine Experimental. Heidelberg: SpringerOpen. Disponível em: https://doi.org/10.1186/2197-425X-2-S1-P53. Acesso em: 16 nov. 2024. , 2014
APA
Kemper, D. A., Otsuki, D. A., Maia, D. R., Queiroz-Hazarbassanov, N. G., Gomes, C. de O. M. -S., Auler Júnior, J. O. C., & Fantoni, D. T. (2014). Effects of sildenafil in a porcine model of endotoxemia. Intensive Care Medicine Experimental. Heidelberg: SpringerOpen. doi:10.1186/2197-425X-2-S1-P53
NLM
Kemper DA, Otsuki DA, Maia DR, Queiroz-Hazarbassanov NG, Gomes C de OM-S, Auler Júnior JOC, Fantoni DT. Effects of sildenafil in a porcine model of endotoxemia [Internet]. Intensive Care Medicine Experimental. 2014 ; 2 2 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1186/2197-425X-2-S1-P53
Vancouver
Kemper DA, Otsuki DA, Maia DR, Queiroz-Hazarbassanov NG, Gomes C de OM-S, Auler Júnior JOC, Fantoni DT. Effects of sildenafil in a porcine model of endotoxemia [Internet]. Intensive Care Medicine Experimental. 2014 ; 2 2 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1186/2197-425X-2-S1-P53
Artigo de periodico
Diagnostic value of interleukin-6 and C-reactive protein on early onset bacterial infection in preterm neonates with respiratory distress (2010)
Rego, Maria Aparecida C.; Martinez, Francisco Eulógio; Elias Júnior, Jorge; Mussi-Pinhata, Marisa Márcia
Source: Journal of Perinatal Medicine. Unidade: FMRP
Subjects: SEPSE, BEBÊ PREMATURO, INTERLEUCINA 6, DOENÇAS RESPIRATÓRIAS, CITOCINAS
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ABNT
REGO, Maria Aparecida C. et al. Diagnostic value of interleukin-6 and C-reactive protein on early onset bacterial infection in preterm neonates with respiratory distress. Journal of Perinatal Medicine, v. 38, n. 5, p. 527-533, 2010Tradução . . Disponível em: https://doi.org/10.1515/jpm.2010.071. Acesso em: 16 nov. 2024.
APA
Rego, M. A. C., Martinez, F. E., Elias Júnior, J., & Mussi-Pinhata, M. M. (2010). Diagnostic value of interleukin-6 and C-reactive protein on early onset bacterial infection in preterm neonates with respiratory distress. Journal of Perinatal Medicine, 38( 5), 527-533. doi:10.1515/jpm.2010.071
NLM
Rego MAC, Martinez FE, Elias Júnior J, Mussi-Pinhata MM. Diagnostic value of interleukin-6 and C-reactive protein on early onset bacterial infection in preterm neonates with respiratory distress [Internet]. Journal of Perinatal Medicine. 2010 ; 38( 5): 527-533.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1515/jpm.2010.071
Vancouver
Rego MAC, Martinez FE, Elias Júnior J, Mussi-Pinhata MM. Diagnostic value of interleukin-6 and C-reactive protein on early onset bacterial infection in preterm neonates with respiratory distress [Internet]. Journal of Perinatal Medicine. 2010 ; 38( 5): 527-533.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1515/jpm.2010.071
Artigo de periodico
Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death (2010)
Melo, Edielle S.; Goloubkova, Tatiana; Barbeiro, Denise F.; Gorjão, Renata; Vasconcelos, Dewton; Szabo, Csaba; Curi, Rui; Salgado, Thais Martins de Lima; Velasco, Irineu Tadeu; Soriano, Francisco Garcia
Source: Immunobiology. Unidades: FM, ICB, FMRP
Subjects: ENDOTOXINAS, TOLERÂNCIA, EXPRESSÃO GÊNICA, LINFÓCITOS, APOPTOSE, CITOCINAS, SEPSE
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ABNT
MELO, Edielle S. et al. Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death. Immunobiology, v. 215, n. 6, p. 435-442, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2009.09.002. Acesso em: 16 nov. 2024.
APA
Melo, E. S., Goloubkova, T., Barbeiro, D. F., Gorjão, R., Vasconcelos, D., Szabo, C., et al. (2010). Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death. Immunobiology, 215( 6), 435-442. doi:10.1016/j.imbio.2009.09.002
NLM
Melo ES, Goloubkova T, Barbeiro DF, Gorjão R, Vasconcelos D, Szabo C, Curi R, Salgado TM de L, Velasco IT, Soriano FG. Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death [Internet]. Immunobiology. 2010 ; 215( 6): 435-442.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2009.09.002
Vancouver
Melo ES, Goloubkova T, Barbeiro DF, Gorjão R, Vasconcelos D, Szabo C, Curi R, Salgado TM de L, Velasco IT, Soriano FG. Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death [Internet]. Immunobiology. 2010 ; 215( 6): 435-442.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2009.09.002
Monografia/livro
Pro-inflammatory and anti-inflammatory cytokines: homeostasis or/and an imbalance in neonatal sepsis (2010)
Cianciarullo, Marco Antonio; Ceccon, Maria Esther Jurfest
Unidade: HU
Subjects: SEPSE, RECÉM-NASCIDO, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CIANCIARULLO, Marco Antonio e CECCON, Maria Esther Jurfest. Pro-inflammatory and anti-inflammatory cytokines: homeostasis or/and an imbalance in neonatal sepsis. . Saarbrücken: VDM Verlag. . Acesso em: 16 nov. 2024. , 2010
APA
Cianciarullo, M. A., & Ceccon, M. E. J. (2010). Pro-inflammatory and anti-inflammatory cytokines: homeostasis or/and an imbalance in neonatal sepsis. Saarbrücken: VDM Verlag.
NLM
Cianciarullo MA, Ceccon MEJ. Pro-inflammatory and anti-inflammatory cytokines: homeostasis or/and an imbalance in neonatal sepsis. 2010 ;[citado 2024 nov. 16 ]
Vancouver
Cianciarullo MA, Ceccon MEJ. Pro-inflammatory and anti-inflammatory cytokines: homeostasis or/and an imbalance in neonatal sepsis. 2010 ;[citado 2024 nov. 16 ]
Artigo de periodico
Sepsis-related microvascular myocardial damage with giant cell inflammation and calcification (2003)
Rossi, Marcos Antonio; Santos, Cláudio S.
Source: Virchows Archiv. Unidade: FMRP
Subjects: SEPSE, CORAÇÃO, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSSI, Marcos Antonio e SANTOS, Cláudio S. Sepsis-related microvascular myocardial damage with giant cell inflammation and calcification. Virchows Archiv, v. 443, p. 87-92, 2003Tradução . . Disponível em: https://doi.org/10.1007/s00428-003-0816-0. Acesso em: 16 nov. 2024.
APA
Rossi, M. A., & Santos, C. S. (2003). Sepsis-related microvascular myocardial damage with giant cell inflammation and calcification. Virchows Archiv, 443, 87-92. doi:10.1007/s00428-003-0816-0
NLM
Rossi MA, Santos CS. Sepsis-related microvascular myocardial damage with giant cell inflammation and calcification [Internet]. Virchows Archiv. 2003 ; 443 87-92.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00428-003-0816-0
Vancouver
Rossi MA, Santos CS. Sepsis-related microvascular myocardial damage with giant cell inflammation and calcification [Internet]. Virchows Archiv. 2003 ; 443 87-92.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00428-003-0816-0

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