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Artigo de periodico
Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni (2020)
Mota, Ester Alves; Patrocínio, Andressa Barban do; Rodrigues, Vanderlei ; Silva, João Santana da ; Pereira, Vanessa Carregaro; Guerra-Sá, Renata
Source: PLOS Neglected Tropical Diseases. Unidade: FMRP
Subjects: SCHISTOSOMA MANSONI, METILAÇÃO DE DNA, ANIMAIS GENETICAMENTE MODIFICADOS, DOENÇAS PARASITÁRIAS, GRANULOMA, EXPRESSÃO GÊNICA, MICRORNAS
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MOTA, Ester Alves et al. Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni. PLOS Neglected Tropical Diseases, v. 14, n. 2, p. 1-24, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0008080. Acesso em: 10 nov. 2024.
APA
Mota, E. A., Patrocínio, A. B. do, Rodrigues, V., Silva, J. S. da, Pereira, V. C., & Guerra-Sá, R. (2020). Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni. PLOS Neglected Tropical Diseases, 14( 2), 1-24. doi:10.1371/journal.pntd.0008080
NLM
Mota EA, Patrocínio AB do, Rodrigues V, Silva JS da, Pereira VC, Guerra-Sá R. Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 2): 1-24.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pntd.0008080
Vancouver
Mota EA, Patrocínio AB do, Rodrigues V, Silva JS da, Pereira VC, Guerra-Sá R. Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 2): 1-24.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pntd.0008080
Artigo de periodico
The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway (2020)
Leite, Jefferson Antonio; Pessenda, Gabriela ; Gomes, Isabel Cristina Guerra ; Santana, Alynne Karen Mendonça de; Pereira, Camila André; Costa, Frederico Ribeiro Campos; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Faria, Ana Maria Caetano ; Almeida, Danilo Candido de ; Câmara, Niels Olsen Saraiva ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Cells. Unidades: FMRP, ICB
Assunto: IMUNOLOGIA
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LEITE, Jefferson Antonio et al. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, v. 9, n. 4, p. 21 , 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9040959. Acesso em: 10 nov. 2024.
APA
Leite, J. A., Pessenda, G., Gomes, I. C. G., Santana, A. K. M. de, Pereira, C. A., Costa, F. R. C., et al. (2020). The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, 9( 4), 21 . doi:10.3390/cells9040959
NLM
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/cells9040959
Vancouver
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/cells9040959
Artigo de periodico
Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication (2020)
Mota, Caroline M.; Lima Júnior, Djalma de Souza; França, Flávia Batista Ferreira; Torres, Jhoan David Aguillón; Barros, Patrício da Silva Cardoso; Santiago, Fernanda Maria; Silva, João Santana da ; Mineo, José Roberto; Zamboni, Dario Simões; Mineo, Tiago Wilson Patriarca
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FMRP
Subjects: INTERLEUCINA 1, NEOSPORA CANINUM, ESPÉCIES REATIVAS DE OXIGÊNIO
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MOTA, Caroline M. et al. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2020.00243. Acesso em: 10 nov. 2024.
APA
Mota, C. M., Lima Júnior, D. de S., França, F. B. F., Torres, J. D. A., Barros, P. da S. C., Santiago, F. M., et al. (2020). Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, 10. doi:10.3389/fcimb.2020.00243
NLM
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fcimb.2020.00243
Vancouver
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fcimb.2020.00243
Artigo de periodico
Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment (2020)
Albuquerque, Sérgio de ; Cianni, Lorenzo ; Vita, Daniela de ; Lopes, Carla Duque ; Gomes, Ana S. M.; Gomes, Paula; Laughton, Charles; Leitão, Andrei ; Montanari, Carlos Alberto ; Montanari, Raphael ; Ribeiro, Jean Francisco Rosa ; Silva, João Santana da ; Teixeira, Cátia
Source: Chemical Biology & Drug Design. Unidades: FCFRP, IQSC, FMRP, EESC, ICMC
Subjects: DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI
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ALBUQUERQUE, Sérgio de et al. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment. Chemical Biology & Drug Design, v. 96, p. 948–960, 2020Tradução . . Disponível em: https://doi.org/10.1111/cbdd.13663. Acesso em: 10 nov. 2024.
APA
Albuquerque, S. de, Cianni, L., Vita, D. de, Lopes, C. D., Gomes, A. S. M., Gomes, P., et al. (2020). Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment. Chemical Biology & Drug Design, 96, 948–960. doi:10.1111/cbdd.13663
NLM
Albuquerque S de, Cianni L, Vita D de, Lopes CD, Gomes ASM, Gomes P, Laughton C, Leitão A, Montanari CA, Montanari R, Ribeiro JFR, Silva JS da, Teixeira C. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment [Internet]. Chemical Biology & Drug Design. 2020 ; 96 948–960.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/cbdd.13663
Vancouver
Albuquerque S de, Cianni L, Vita D de, Lopes CD, Gomes ASM, Gomes P, Laughton C, Leitão A, Montanari CA, Montanari R, Ribeiro JFR, Silva JS da, Teixeira C. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment [Internet]. Chemical Biology & Drug Design. 2020 ; 96 948–960.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/cbdd.13663
Artigo de periodico
Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole (2020)
Silva, Maria Cláudia da; Carbone, Débora Cardozo Bonfim; Diniz, Pedro Fernando; Fernandes, Fabrício Freitas; Fuzo, Carlos Alessandro; Trindade, Carolina Santos Pereira Cardoso; Chang, Marilene Rodrigues; Silva, João Santana da
Source: Mycopathologia. Unidades: FMRP, FCFRP
Subjects: CANDIDA, EXPRESSÃO GÊNICA, RESISTÊNCIA MICROBIANA ÀS DROGAS, CANDIDOSE, ANTIFÚNGICOS
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SILVA, Maria Cláudia da et al. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole. Mycopathologia, v. 185, n. 4, p. 675-684, 2020Tradução . . Disponível em: https://doi.org/10.1007/s11046-020-00465-6. Acesso em: 10 nov. 2024.
APA
Silva, M. C. da, Carbone, D. C. B., Diniz, P. F., Fernandes, F. F., Fuzo, C. A., Trindade, C. S. P. C., et al. (2020). Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole. Mycopathologia, 185( 4), 675-684. doi:10.1007/s11046-020-00465-6
NLM
Silva MC da, Carbone DCB, Diniz PF, Fernandes FF, Fuzo CA, Trindade CSPC, Chang MR, Silva JS da. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole [Internet]. Mycopathologia. 2020 ; 185( 4): 675-684.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s11046-020-00465-6
Vancouver
Silva MC da, Carbone DCB, Diniz PF, Fernandes FF, Fuzo CA, Trindade CSPC, Chang MR, Silva JS da. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole [Internet]. Mycopathologia. 2020 ; 185( 4): 675-684.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s11046-020-00465-6
Artigo de periodico
Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes (2020)
Pereira, Camila André; Carlos, Daniela; Ferreira, Nathanne dos Santos; Silva, Josiane F. da; Zanotto, Camila Ziliotto; Zamboni, Dario Simões ; Garcia, Valéria Duarte; Ventura, Dora Selma Fix ; Silva, João Santana da ; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidades: FMRP, IP
Subjects: INFLAMAÇÃO, DNA MITOCONDRIAL, ESPÉCIES REATIVAS DE OXIGÊNIO
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PEREIRA, Camila André et al. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes. Frontiers in Physiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.01557. Acesso em: 10 nov. 2024.
APA
Pereira, C. A., Carlos, D., Ferreira, N. dos S., Silva, J. F. da, Zanotto, C. Z., Zamboni, D. S., et al. (2020). Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes. Frontiers in Physiology, 10. doi:10.3389/fphys.2019.01557
NLM
Pereira CA, Carlos D, Ferreira N dos S, Silva JF da, Zanotto CZ, Zamboni DS, Garcia VD, Ventura DSF, Silva JS da, Tostes R de CA. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fphys.2019.01557
Vancouver
Pereira CA, Carlos D, Ferreira N dos S, Silva JF da, Zanotto CZ, Zamboni DS, Garcia VD, Ventura DSF, Silva JS da, Tostes R de CA. Mitochondrial DNA promotes NLRP3 inflammasome activation and contributes to endothelial dysfunction and inflammation in type 1 diabetes [Internet]. Frontiers in Physiology. 2020 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fphys.2019.01557
Artigo de periodico
NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? (2020)
Oliveira, Jefferson Elias ; Leite, Jefferson Antônio; Pereira, Ítalo Sousa; Guimarães, Jhefferson Barbosa; Manso, Gabriel Martins da Costa; Silva, João Santana da ; Passaglia, Rita de Cassia Aleixo Tostes ; Sartori, Daniela Carlos
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: ENTEROPATIAS INFLAMATÓRIAS, DIABETES MELLITUS, HOMEOSTASE, INTESTINOS, BACTÉRIAS, RECEPTORES IMUNOLÓGICOS
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OLIVEIRA, Jefferson Elias et al. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers?. Frontiers in Immunology, v. 11, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01810. Acesso em: 10 nov. 2024.
APA
Oliveira, J. E., Leite, J. A., Pereira, Í. S., Guimarães, J. B., Manso, G. M. da C., Silva, J. S. da, et al. (2020). NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? Frontiers in Immunology, 11, 1-9. doi:10.3389/fimmu.2020.01810
NLM
Oliveira JE, Leite JA, Pereira ÍS, Guimarães JB, Manso GM da C, Silva JS da, Passaglia R de CAT, Sartori DC. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? [Internet]. Frontiers in Immunology. 2020 ; 11 1-9.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fimmu.2020.01810
Vancouver
Oliveira JE, Leite JA, Pereira ÍS, Guimarães JB, Manso GM da C, Silva JS da, Passaglia R de CAT, Sartori DC. NLR and intestinal dysbiosis-associated inflammatory illness: drivers or dampers? [Internet]. Frontiers in Immunology. 2020 ; 11 1-9.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fimmu.2020.01810
Artigo de periodico
Arginase and its mechanisms in Leishmania persistence (2020)
Pessenda, Gabriela; Silva, João Santana da
Source: Parasite Immunology. Unidade: FMRP
Subjects: LEISHMANIA, ÓXIDO NÍTRICO, MACRÓFAGOS, SISTEMA IMUNE, ARGININA
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PESSENDA, Gabriela e SILVA, João Santana da. Arginase and its mechanisms in Leishmania persistence. Parasite Immunology, v. 42, n. 7, p. 1-12, 2020Tradução . . Disponível em: https://doi.org/10.1111/pim.12722. Acesso em: 10 nov. 2024.
APA
Pessenda, G., & Silva, J. S. da. (2020). Arginase and its mechanisms in Leishmania persistence. Parasite Immunology, 42( 7), 1-12. doi:10.1111/pim.12722
NLM
Pessenda G, Silva JS da. Arginase and its mechanisms in Leishmania persistence [Internet]. Parasite Immunology. 2020 ; 42( 7): 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/pim.12722
Vancouver
Pessenda G, Silva JS da. Arginase and its mechanisms in Leishmania persistence [Internet]. Parasite Immunology. 2020 ; 42( 7): 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/pim.12722
Artigo de periodico
TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection (2020)
Sacramento, Laís Amorim ; Benevides, Luciana; Maruyama, Sandra Regina Costa; Tavares, Lucas Alves ; Fukutani, Kiyoshi Ferreira; Françozo, Marcela; Sparwasser, Tim; Cunha, Fernando de Queiroz ; Almeida, Roque Pacheco de; Silva, João Santana da ; Carregaro, Vanessa
Source: PLOS Pathogens. Unidade: FMRP
Subjects: INFLAMAÇÃO, IMUNOLOGIA, INTERFERONS, LEISHMANIA INFANTUM, LEISHMANIOSE VISCERAL
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SACRAMENTO, Laís Amorim et al. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection. PLOS Pathogens, v. 16, n. 3, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1008435. Acesso em: 10 nov. 2024.
APA
Sacramento, L. A., Benevides, L., Maruyama, S. R. C., Tavares, L. A., Fukutani, K. F., Françozo, M., et al. (2020). TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection. PLOS Pathogens, 16( 3). doi:10.1371/journal.ppat.1008435
NLM
Sacramento LA, Benevides L, Maruyama SRC, Tavares LA, Fukutani KF, Françozo M, Sparwasser T, Cunha F de Q, Almeida RP de, Silva JS da, Carregaro V. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection [Internet]. PLOS Pathogens. 2020 ; 16( 3):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.ppat.1008435
Vancouver
Sacramento LA, Benevides L, Maruyama SRC, Tavares LA, Fukutani KF, Françozo M, Sparwasser T, Cunha F de Q, Almeida RP de, Silva JS da, Carregaro V. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection [Internet]. PLOS Pathogens. 2020 ; 16( 3):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.ppat.1008435
Artigo de periodico
Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain (2020)
Oliveira, Maykon Tavares de; Sulleiro, Elena; Gimenez, Aroa Silgado; Lana, Marta de; Zingales, Bianca ; Silva, João Santana da ; Marin Neto, José Antonio; MolinaI, Israel
Source: PLOS Neglected Tropical Diseases. Unidades: IQ, FMRP
Subjects: TRYPANOSOMA CRUZI, IMIGRANTES, LATINO-AMERICANOS, REAÇÃO EM CADEIA POR POLIMERASE, DOENÇAS PARASITÁRIAS
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OLIVEIRA, Maykon Tavares de et al. Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain. PLOS Neglected Tropical Diseases, v. 14, n. 6, p. 1-14 art. e0008311, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0008311. Acesso em: 10 nov. 2024.
APA
Oliveira, M. T. de, Sulleiro, E., Gimenez, A. S., Lana, M. de, Zingales, B., Silva, J. S. da, et al. (2020). Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain. PLOS Neglected Tropical Diseases, 14( 6), 1-14 art. e0008311. doi:10.1371/journal.pntd.0008311
NLM
Oliveira MT de, Sulleiro E, Gimenez AS, Lana M de, Zingales B, Silva JS da, Marin Neto JA, MolinaI I. Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 6): 1-14 art. e0008311.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pntd.0008311
Vancouver
Oliveira MT de, Sulleiro E, Gimenez AS, Lana M de, Zingales B, Silva JS da, Marin Neto JA, MolinaI I. Quantification of parasite burden of Trypanosoma cruzi and identification of Discrete Typing Units (DTUs) in blood samples of Latin American immigrants residing in Barcelona, Spain [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 6): 1-14 art. e0008311.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pntd.0008311
Artigo de periodico
NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model (2020)
Sartori, Daniela Carlos ; Pérez, Malena Martínez ; Leite, Jefferson Antonio; Rocha, Fernanda Agostini; Martins, Larissa Marfori Sampaio; Pereira, Camila André; Silva, Thais Fernanda de Campos Fraga da ; Pucci, Taís Andrade; Ramos, Simone Gusmão ; Câmara, Niels Olsen Saraiva ; Bonato, Vânia Luiza Deperon ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB, FCFRP
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIABETES MELLITUS, INFLAMAÇÃO, OBESIDADE, ADIPOSIDADE, CAMUNDONGOS, RESISTÊNCIA À INSULINA, HIPERGLICEMIA, LINFONODOS DE ANIMAL, EXPRESSÃO GÊNICA, BACTÉRIAS GRAM-NEGATIVAS, BACTEROIDES
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ABNT
SARTORI, Daniela Carlos et al. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, v. 11, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01265. Acesso em: 10 nov. 2024.
APA
Sartori, D. C., Pérez, M. M., Leite, J. A., Rocha, F. A., Martins, L. M. S., Pereira, C. A., et al. (2020). NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, 11, 16 . doi:10.3389/fimmu.2020.01265
NLM
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Vancouver
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fimmu.2020.01265

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