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Dissertação (Mestrado)
Investigação do papel de Alquiladenina DNA glicosilase (AAG) no reparo de bases alquiladas no DNA mitocondrial de camundongos (2021)
Alves, Rebeca Bueno ; Souza-Pinto, Nadja Cristhina de (Orientador)
Unidade: IQ
Subjects: MITOCÔNDRIAS, DNA, ALQUILAÇÃO
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ABNT
ALVES, Rebeca Bueno. Investigação do papel de Alquiladenina DNA glicosilase (AAG) no reparo de bases alquiladas no DNA mitocondrial de camundongos. 2021. Dissertação (Mestrado) – Universidade de São Paulo, São Paulo, 2021. Disponível em: https://www.teses.usp.br/teses/disponiveis/46/46131/tde-17012022-172119/. Acesso em: 07 ago. 2024.
APA
Alves, R. B. (2021). Investigação do papel de Alquiladenina DNA glicosilase (AAG) no reparo de bases alquiladas no DNA mitocondrial de camundongos (Dissertação (Mestrado). Universidade de São Paulo, São Paulo. Recuperado de https://www.teses.usp.br/teses/disponiveis/46/46131/tde-17012022-172119/
NLM
Alves RB. Investigação do papel de Alquiladenina DNA glicosilase (AAG) no reparo de bases alquiladas no DNA mitocondrial de camundongos [Internet]. 2021 ;[citado 2024 ago. 07 ] Available from: https://www.teses.usp.br/teses/disponiveis/46/46131/tde-17012022-172119/
Vancouver
Alves RB. Investigação do papel de Alquiladenina DNA glicosilase (AAG) no reparo de bases alquiladas no DNA mitocondrial de camundongos [Internet]. 2021 ;[citado 2024 ago. 07 ] Available from: https://www.teses.usp.br/teses/disponiveis/46/46131/tde-17012022-172119/
Trabalho de evento-resumo
Tumor heterogeneity and resistance to cancer therapies: peroxiredoxin 1 as a contributor in melanoma (2021)
Carvalho, Larissa Anastácio da Costa ; Maria-Engler, Silvya Stuchi ; Uehara, Adriana Hiromi; Siena, Ádamo Davi Diogenes; Mori, Matheus Prates; Souza-Pinto, Nadja Cristhina de ; Silva Junior, Wilson Araújo da ; Smalley, Keiran
Source: Anais. Conference titles: Congress of the Brazilian Society for Cell Biology. Unidades: FCF, IQ, FMRP
Subjects: HETEROGENEIDADE, NEOPLASIAS, MELANOMA, BIOMARCADORES
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ABNT
CARVALHO, Larissa Anastácio da Costa et al. Tumor heterogeneity and resistance to cancer therapies: peroxiredoxin 1 as a contributor in melanoma. 2021, Anais.. São Paulo: SBBC, 2021. Disponível em: http://www.sbbc.org.br/wp-content/uploads/2014/08/SCIENTIFIC-PROGRAM-CONGRESS_2021.pdf. Acesso em: 07 ago. 2024.
APA
Carvalho, L. A. da C., Maria-Engler, S. S., Uehara, A. H., Siena, Á. D. D., Mori, M. P., Souza-Pinto, N. C. de, et al. (2021). Tumor heterogeneity and resistance to cancer therapies: peroxiredoxin 1 as a contributor in melanoma. In Anais. São Paulo: SBBC. Recuperado de http://www.sbbc.org.br/wp-content/uploads/2014/08/SCIENTIFIC-PROGRAM-CONGRESS_2021.pdf
NLM
Carvalho LA da C, Maria-Engler SS, Uehara AH, Siena ÁDD, Mori MP, Souza-Pinto NC de, Silva Junior WA da, Smalley K. Tumor heterogeneity and resistance to cancer therapies: peroxiredoxin 1 as a contributor in melanoma [Internet]. Anais. 2021 ;[citado 2024 ago. 07 ] Available from: http://www.sbbc.org.br/wp-content/uploads/2014/08/SCIENTIFIC-PROGRAM-CONGRESS_2021.pdf
Vancouver
Carvalho LA da C, Maria-Engler SS, Uehara AH, Siena ÁDD, Mori MP, Souza-Pinto NC de, Silva Junior WA da, Smalley K. Tumor heterogeneity and resistance to cancer therapies: peroxiredoxin 1 as a contributor in melanoma [Internet]. Anais. 2021 ;[citado 2024 ago. 07 ] Available from: http://www.sbbc.org.br/wp-content/uploads/2014/08/SCIENTIFIC-PROGRAM-CONGRESS_2021.pdf
Artigo de periodico
Manganese-induced neurotoxicity through impairment of cross-talk pathways in human neuroblastoma cell line SH-SY5Y differentiated with retinoic acid (2021)
Hernández, Raúl Bonne ; Souza-Pinto, Nadja Cristhina de ; Kleinjans, Jos; Herwijnen, Marjan van; Piepers, Jolanda; Moteshareie, Houman; Burnside, Daniel; Golshani, Ashkan
Source: Toxics. Unidade: IQ
Subjects: METABOLISMO DE PROTEÍNA, DOENÇAS NEURODEGENERATIVAS, MANGANÊS
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ABNT
HERNÁNDEZ, Raúl Bonne et al. Manganese-induced neurotoxicity through impairment of cross-talk pathways in human neuroblastoma cell line SH-SY5Y differentiated with retinoic acid. Toxics, v. 9, n. 12, p. 1-21, 2021Tradução . . Disponível em: https://doi.org/10.3390/toxics9120348. Acesso em: 07 ago. 2024.
APA
Hernández, R. B., Souza-Pinto, N. C. de, Kleinjans, J., Herwijnen, M. van, Piepers, J., Moteshareie, H., et al. (2021). Manganese-induced neurotoxicity through impairment of cross-talk pathways in human neuroblastoma cell line SH-SY5Y differentiated with retinoic acid. Toxics, 9( 12), 1-21. doi:10.3390/toxics9120348
NLM
Hernández RB, Souza-Pinto NC de, Kleinjans J, Herwijnen M van, Piepers J, Moteshareie H, Burnside D, Golshani A. Manganese-induced neurotoxicity through impairment of cross-talk pathways in human neuroblastoma cell line SH-SY5Y differentiated with retinoic acid [Internet]. Toxics. 2021 ; 9( 12): 1-21.[citado 2024 ago. 07 ] Available from: https://doi.org/10.3390/toxics9120348
Vancouver
Hernández RB, Souza-Pinto NC de, Kleinjans J, Herwijnen M van, Piepers J, Moteshareie H, Burnside D, Golshani A. Manganese-induced neurotoxicity through impairment of cross-talk pathways in human neuroblastoma cell line SH-SY5Y differentiated with retinoic acid [Internet]. Toxics. 2021 ; 9( 12): 1-21.[citado 2024 ago. 07 ] Available from: https://doi.org/10.3390/toxics9120348
Artigo de periodico
NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance (2021)
Ferezin, Camila de Castro ; Basei, Fernanda Luisa; Hanchuk, Talita D. Melo ; Oliveira, Ana Luisa de; Oliveira, Andressa Peres de ; Mori, Mateus P; Souza-Pinto, Nadja Cristhina de
Source: FEBS Open Bio. Unidade: IQ
Subjects: MITOCÔNDRIAS, CICLO CELULAR
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ABNT
FEREZIN, Camila de Castro et al. NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance. FEBS Open Bio, v. 11, p. 546–563, 2021Tradução . . Disponível em: https://doi.org/10.1002/2211-5463.13108. Acesso em: 07 ago. 2024.
APA
Ferezin, C. de C., Basei, F. L., Hanchuk, T. D. M., Oliveira, A. L. de, Oliveira, A. P. de, Mori, M. P., & Souza-Pinto, N. C. de. (2021). NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance. FEBS Open Bio, 11, 546–563. doi:10.1002/2211-5463.13108
NLM
Ferezin C de C, Basei FL, Hanchuk TDM, Oliveira AL de, Oliveira AP de, Mori MP, Souza-Pinto NC de. NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance [Internet]. FEBS Open Bio. 2021 ; 11 546–563.[citado 2024 ago. 07 ] Available from: https://doi.org/10.1002/2211-5463.13108
Vancouver
Ferezin C de C, Basei FL, Hanchuk TDM, Oliveira AL de, Oliveira AP de, Mori MP, Souza-Pinto NC de. NEK5 interacts with LonP1 and its kinase activity is essential for the regulation of mitochondrial functions and mtDNA maintenance [Internet]. FEBS Open Bio. 2021 ; 11 546–563.[citado 2024 ago. 07 ] Available from: https://doi.org/10.1002/2211-5463.13108
Artigo de periodico
Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells (2021)
Freire, Thiago de Souza; Mori, M.P; Miranda, J. N. F. A; Muta, Lais Yoshie Morikawa; Machado, F. T; Moreno, N. C; Souza-Pinto, Nadja Cristhina de
Source: Carcinogenesis. Unidade: IQ
Subjects: MITOCÔNDRIAS, CÉLULAS, PERÓXIDO DE HIDROGÊNIO
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ABNT
FREIRE, Thiago de Souza et al. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells. Carcinogenesis, v. 42, n. 11, p. 1380-1389, 2021Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgab079. Acesso em: 07 ago. 2024.
APA
Freire, T. de S., Mori, M. P., Miranda, J. N. F. A., Muta, L. Y. M., Machado, F. T., Moreno, N. C., & Souza-Pinto, N. C. de. (2021). Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells. Carcinogenesis, 42( 11), 1380-1389. doi:10.1093/carcin/bgab079
NLM
Freire T de S, Mori MP, Miranda JNFA, Muta LYM, Machado FT, Moreno NC, Souza-Pinto NC de. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells [Internet]. Carcinogenesis. 2021 ; 42( 11): 1380-1389.[citado 2024 ago. 07 ] Available from: https://doi.org/10.1093/carcin/bgab079
Vancouver
Freire T de S, Mori MP, Miranda JNFA, Muta LYM, Machado FT, Moreno NC, Souza-Pinto NC de. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells [Internet]. Carcinogenesis. 2021 ; 42( 11): 1380-1389.[citado 2024 ago. 07 ] Available from: https://doi.org/10.1093/carcin/bgab079

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