Filtros : "Ferreira, Sérgio Henrique" "2006" Removido: "IMUNOLOGIA" Limpar

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  • Source: British Journal of Pharmacology. Unidade: FMRP

    Subjects: ÓXIDO NÍTRICO, DOR, ANALGÉSICOS, ANTI-INFLAMATÓRIOS

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    • ABNT

      SANTODOMINGO-GARZÓN, T. et al. Atorvastatin inhibits inflammatory hypernociception. British Journal of Pharmacology, v. 149, p. 14-22, 2006Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0706836. Acesso em: 15 out. 2024.
    • APA

      Santodomingo-Garzón, T., Cunha, T. M., Verri Júnior, W. A., Valério, D. A. R., Parada, C. A., Poole, S., et al. (2006). Atorvastatin inhibits inflammatory hypernociception. British Journal of Pharmacology, 149, 14-22. doi:10.1038/sj.bjp.0706836
    • NLM

      Santodomingo-Garzón T, Cunha TM, Verri Júnior WA, Valério DAR, Parada CA, Poole S, Ferreira SH, Cunha F de Q. Atorvastatin inhibits inflammatory hypernociception [Internet]. British Journal of Pharmacology. 2006 ; 149 14-22.[citado 2024 out. 15 ] Available from: https://doi.org/10.1038/sj.bjp.0706836
    • Vancouver

      Santodomingo-Garzón T, Cunha TM, Verri Júnior WA, Valério DAR, Parada CA, Poole S, Ferreira SH, Cunha F de Q. Atorvastatin inhibits inflammatory hypernociception [Internet]. British Journal of Pharmacology. 2006 ; 149 14-22.[citado 2024 out. 15 ] Available from: https://doi.org/10.1038/sj.bjp.0706836
  • Source: Pharmacology, Biochemistry and Behavior. Unidade: FMRP

    Subjects: DOR, ARTRITE, ARTRALGIA, FARMACOLOGIA

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    • ABNT

      GUERRERO, Ana T. G. et al. Hypernociception elicited by tibio-tarsal joint in mice: a novel experimental arthritis model for pharmacological screening. Pharmacology, Biochemistry and Behavior, v. 84, p. 244-251, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2006.05.008. Acesso em: 15 out. 2024.
    • APA

      Guerrero, A. T. G., Verri Júnior, W. A., Cunha, T. M., Silva, T. A., Rocha, F. A. C., Ferreira, S. H., et al. (2006). Hypernociception elicited by tibio-tarsal joint in mice: a novel experimental arthritis model for pharmacological screening. Pharmacology, Biochemistry and Behavior, 84, 244-251. doi:10.1016/j.pbb.2006.05.008
    • NLM

      Guerrero ATG, Verri Júnior WA, Cunha TM, Silva TA, Rocha FAC, Ferreira SH, Cunha F de Q, Parada CA. Hypernociception elicited by tibio-tarsal joint in mice: a novel experimental arthritis model for pharmacological screening [Internet]. Pharmacology, Biochemistry and Behavior. 2006 ; 84 244-251.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.pbb.2006.05.008
    • Vancouver

      Guerrero ATG, Verri Júnior WA, Cunha TM, Silva TA, Rocha FAC, Ferreira SH, Cunha F de Q, Parada CA. Hypernociception elicited by tibio-tarsal joint in mice: a novel experimental arthritis model for pharmacological screening [Internet]. Pharmacology, Biochemistry and Behavior. 2006 ; 84 244-251.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.pbb.2006.05.008
  • Source: Pharmacology & Therapeutics. Unidade: FMRP

    Subjects: DOR, INFLAMAÇÃO, QUIMIOTERÁPICOS

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    • ABNT

      VERRI JÚNIOR, Waldiceu A. et al. Hypernociceptive role of cytokines and chemokines: targets for analgesic drug development?. Pharmacology & Therapeutics, v. 112, p. 116-138, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.pharmthera.2006.04.001. Acesso em: 15 out. 2024.
    • APA

      Verri Júnior, W. A., Cunha, T. M., Parada, C. A., Poole, S., Cunha, F. de Q., & Ferreira, S. H. (2006). Hypernociceptive role of cytokines and chemokines: targets for analgesic drug development? Pharmacology & Therapeutics, 112, 116-138. doi:10.1016/j.pharmthera.2006.04.001
    • NLM

      Verri Júnior WA, Cunha TM, Parada CA, Poole S, Cunha F de Q, Ferreira SH. Hypernociceptive role of cytokines and chemokines: targets for analgesic drug development? [Internet]. Pharmacology & Therapeutics. 2006 ; 112 116-138.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.pharmthera.2006.04.001
    • Vancouver

      Verri Júnior WA, Cunha TM, Parada CA, Poole S, Cunha F de Q, Ferreira SH. Hypernociceptive role of cytokines and chemokines: targets for analgesic drug development? [Internet]. Pharmacology & Therapeutics. 2006 ; 112 116-138.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.pharmthera.2006.04.001
  • Source: British Journal Pharmacology. Unidade: FMRP

    Subjects: FARMACOLOGIA, INFLAMAÇÃO, ÓXIDO NÍTRICO

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      FREITAS, A. et al. Heme oxygenase/carbon monoxide-biliverdin pathway down regulates neutrophil rolling, adhesion and migration in acute inflammation. British Journal Pharmacology, v. 149, n. 4, p. 345-354, 2006Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0706882. Acesso em: 15 out. 2024.
    • APA

      Freitas, A., Alves-Filho, J. C., Secco, D. D., Neto, A. F., Ferreira, S. H., Barja-Fidalgo, C., & Cunha, F. de Q. (2006). Heme oxygenase/carbon monoxide-biliverdin pathway down regulates neutrophil rolling, adhesion and migration in acute inflammation. British Journal Pharmacology, 149( 4), 345-354. doi:10.1038/sj.bjp.0706882
    • NLM

      Freitas A, Alves-Filho JC, Secco DD, Neto AF, Ferreira SH, Barja-Fidalgo C, Cunha F de Q. Heme oxygenase/carbon monoxide-biliverdin pathway down regulates neutrophil rolling, adhesion and migration in acute inflammation [Internet]. British Journal Pharmacology. 2006 ; 149( 4): 345-354.[citado 2024 out. 15 ] Available from: https://doi.org/10.1038/sj.bjp.0706882
    • Vancouver

      Freitas A, Alves-Filho JC, Secco DD, Neto AF, Ferreira SH, Barja-Fidalgo C, Cunha F de Q. Heme oxygenase/carbon monoxide-biliverdin pathway down regulates neutrophil rolling, adhesion and migration in acute inflammation [Internet]. British Journal Pharmacology. 2006 ; 149( 4): 345-354.[citado 2024 out. 15 ] Available from: https://doi.org/10.1038/sj.bjp.0706882
  • Source: European Journal of Neuroscience. Unidade: FMRP

    Subjects: ÓXIDO NÍTRICO, PROTEÍNAS

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    • ABNT

      BRITO, Gerly A. C. et al. Peripheral antinociceptive effect of pertussis toxin: activation of the arginine /NO/cGMP/PKG/ ATP-sensitive K+ channel pathway. European Journal of Neuroscience, v. 24, p. 1175-1181, 2006Tradução . . Disponível em: https://doi.org/10.1111/j.1460-9568.2006.04991.x. Acesso em: 15 out. 2024.
    • APA

      Brito, G. A. C., Sachs, D., Cunha, F. de Q., Vale, M. L., Lotufo, C. M. da C., Ferreira, S. H., & Ribeiro, R. de A. (2006). Peripheral antinociceptive effect of pertussis toxin: activation of the arginine /NO/cGMP/PKG/ ATP-sensitive K+ channel pathway. European Journal of Neuroscience, 24, 1175-1181. doi:10.1111/j.1460-9568.2006.04991.x
    • NLM

      Brito GAC, Sachs D, Cunha F de Q, Vale ML, Lotufo CM da C, Ferreira SH, Ribeiro R de A. Peripheral antinociceptive effect of pertussis toxin: activation of the arginine /NO/cGMP/PKG/ ATP-sensitive K+ channel pathway [Internet]. European Journal of Neuroscience. 2006 ; 24 1175-1181.[citado 2024 out. 15 ] Available from: https://doi.org/10.1111/j.1460-9568.2006.04991.x
    • Vancouver

      Brito GAC, Sachs D, Cunha F de Q, Vale ML, Lotufo CM da C, Ferreira SH, Ribeiro R de A. Peripheral antinociceptive effect of pertussis toxin: activation of the arginine /NO/cGMP/PKG/ ATP-sensitive K+ channel pathway [Internet]. European Journal of Neuroscience. 2006 ; 24 1175-1181.[citado 2024 out. 15 ] Available from: https://doi.org/10.1111/j.1460-9568.2006.04991.x
  • Source: British Journal of Pharmacology. Unidade: FMRP

    Subjects: ÓXIDO NÍTRICO, NEUTRÓFILOS, INTERLEUCINA 2

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      MORENO, Susana Elisa et al. Systemic administration of interleukin-2 inhibits inflammatory neutrophil migration: role of nitric oxide. British Journal of Pharmacology, v. 148, p. 1060-1066, 2006Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0706835. Acesso em: 15 out. 2024.
    • APA

      Moreno, S. E., Alves-Filho, J. C., Bertozi, G., Alfaya, T. M., Thèze, J., Ferreira, S. H., & Vargaftig, B. B. J. (2006). Systemic administration of interleukin-2 inhibits inflammatory neutrophil migration: role of nitric oxide. British Journal of Pharmacology, 148, 1060-1066. doi:10.1038/sj.bjp.0706835
    • NLM

      Moreno SE, Alves-Filho JC, Bertozi G, Alfaya TM, Thèze J, Ferreira SH, Vargaftig BBJ. Systemic administration of interleukin-2 inhibits inflammatory neutrophil migration: role of nitric oxide [Internet]. British Journal of Pharmacology. 2006 ; 148 1060-1066.[citado 2024 out. 15 ] Available from: https://doi.org/10.1038/sj.bjp.0706835
    • Vancouver

      Moreno SE, Alves-Filho JC, Bertozi G, Alfaya TM, Thèze J, Ferreira SH, Vargaftig BBJ. Systemic administration of interleukin-2 inhibits inflammatory neutrophil migration: role of nitric oxide [Internet]. British Journal of Pharmacology. 2006 ; 148 1060-1066.[citado 2024 out. 15 ] Available from: https://doi.org/10.1038/sj.bjp.0706835
  • Source: SHOCK. Unidade: FMRP

    Subjects: FARMACOLOGIA, SEPSE

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    • ABNT

      TORRES-DUEÑAS, D. et al. Failure of neutrophil migration to infectious focus and cardiovascular changes on sepsis in rats: effects of the inhibition of nitric oxide production, removal of infectious focus, and antimicrobial treatment. SHOCK, v. 25, n. 3, p. 267-276, 2006Tradução . . Acesso em: 15 out. 2024.
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      Torres-Dueñas, D., Benjamim, C. F., Ferreira, S. H., & Cunha, F. de Q. (2006). Failure of neutrophil migration to infectious focus and cardiovascular changes on sepsis in rats: effects of the inhibition of nitric oxide production, removal of infectious focus, and antimicrobial treatment. SHOCK, 25( 3), 267-276.
    • NLM

      Torres-Dueñas D, Benjamim CF, Ferreira SH, Cunha F de Q. Failure of neutrophil migration to infectious focus and cardiovascular changes on sepsis in rats: effects of the inhibition of nitric oxide production, removal of infectious focus, and antimicrobial treatment. SHOCK. 2006 ; 25( 3): 267-276.[citado 2024 out. 15 ]
    • Vancouver

      Torres-Dueñas D, Benjamim CF, Ferreira SH, Cunha F de Q. Failure of neutrophil migration to infectious focus and cardiovascular changes on sepsis in rats: effects of the inhibition of nitric oxide production, removal of infectious focus, and antimicrobial treatment. SHOCK. 2006 ; 25( 3): 267-276.[citado 2024 out. 15 ]
  • Unidade: FMRP

    Subjects: DOR, INFLAMAÇÃO, FARMACOLOGIA

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      VERRI JUNIOR, Waldiceu Aparecido. Inter-Relação e Mecanismos Envolvidos na Indução Hipernociceptiva Mecânica e Migração de Neutrófilos pela IL-15. 2006. Tese (Doutorado) – Universidade de São Paulo, Ribeirão Preto, 2006. . Acesso em: 15 out. 2024.
    • APA

      Verri Junior, W. A. (2006). Inter-Relação e Mecanismos Envolvidos na Indução Hipernociceptiva Mecânica e Migração de Neutrófilos pela IL-15 (Tese (Doutorado). Universidade de São Paulo, Ribeirão Preto.
    • NLM

      Verri Junior WA. Inter-Relação e Mecanismos Envolvidos na Indução Hipernociceptiva Mecânica e Migração de Neutrófilos pela IL-15. 2006 ;[citado 2024 out. 15 ]
    • Vancouver

      Verri Junior WA. Inter-Relação e Mecanismos Envolvidos na Indução Hipernociceptiva Mecânica e Migração de Neutrófilos pela IL-15. 2006 ;[citado 2024 out. 15 ]
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP

    Subjects: ENZIMAS, RADICAIS LIVRES, LEISHMANIA

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      ROMÃO, P. R. T. et al. Glutathione and the redox control system trypanothione/trypanothione reductase ar involved in the protection of Leishmania spp against nitrosothiol-induced cytotoxicity. Brazilian Journal of Medical and Biological Research, v. 39, n. 3, p. 355-363, 2006Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x2006000300006. Acesso em: 15 out. 2024.
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      Romão, P. R. T., Tovar, J., Fonseca, S. G., Moraes, R. H., Cruz, Â. K., Hothersall, J. S., et al. (2006). Glutathione and the redox control system trypanothione/trypanothione reductase ar involved in the protection of Leishmania spp against nitrosothiol-induced cytotoxicity. Brazilian Journal of Medical and Biological Research, 39( 3), 355-363. doi:10.1590/s0100-879x2006000300006
    • NLM

      Romão PRT, Tovar J, Fonseca SG, Moraes RH, Cruz ÂK, Hothersall JS, Noronha-Dutra AA, Ferreira SH, Cunha F de Q. Glutathione and the redox control system trypanothione/trypanothione reductase ar involved in the protection of Leishmania spp against nitrosothiol-induced cytotoxicity [Internet]. Brazilian Journal of Medical and Biological Research. 2006 ; 39( 3): 355-363.[citado 2024 out. 15 ] Available from: https://doi.org/10.1590/s0100-879x2006000300006
    • Vancouver

      Romão PRT, Tovar J, Fonseca SG, Moraes RH, Cruz ÂK, Hothersall JS, Noronha-Dutra AA, Ferreira SH, Cunha F de Q. Glutathione and the redox control system trypanothione/trypanothione reductase ar involved in the protection of Leishmania spp against nitrosothiol-induced cytotoxicity [Internet]. Brazilian Journal of Medical and Biological Research. 2006 ; 39( 3): 355-363.[citado 2024 out. 15 ] Available from: https://doi.org/10.1590/s0100-879x2006000300006
  • Source: European Journal of Pharmacology. Unidade: FMRP

    Subjects: FARMACOLOGIA, DOR, ARTRITE, ARTICULAÇÃO DO JOELHO

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      VALE, Mariana L. et al. Anti-nociceptive effect of thalidomide on zymosan-induced experimental articular incapacitation. European Journal of Pharmacology, v. 536, p. 309-317, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2006.02.051. Acesso em: 15 out. 2024.
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      Vale, M. L., Cunha, F. de Q., Brito, G. A. C., Benevides, V. M., Ferreira, S. H., Poole, S., & Ribeiro, R. A. (2006). Anti-nociceptive effect of thalidomide on zymosan-induced experimental articular incapacitation. European Journal of Pharmacology, 536, 309-317. doi:10.1016/j.ejphar.2006.02.051
    • NLM

      Vale ML, Cunha F de Q, Brito GAC, Benevides VM, Ferreira SH, Poole S, Ribeiro RA. Anti-nociceptive effect of thalidomide on zymosan-induced experimental articular incapacitation [Internet]. European Journal of Pharmacology. 2006 ; 536 309-317.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.ejphar.2006.02.051
    • Vancouver

      Vale ML, Cunha F de Q, Brito GAC, Benevides VM, Ferreira SH, Poole S, Ribeiro RA. Anti-nociceptive effect of thalidomide on zymosan-induced experimental articular incapacitation [Internet]. European Journal of Pharmacology. 2006 ; 536 309-317.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.ejphar.2006.02.051
  • Source: Proceedings of the National Academy of Sciences. Unidade: FMRP

    Subjects: DOR (AVALIAÇÃO), INFLAMAÇÃO, ANALGESIA

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      VERRI JÚNIOR, Waldiceu Aparecido et al. IL-15 mediates immune inflammatory hypernociception by triggerin a sequential release of IFN-'gama' endothelin, and prostaglandin. Proceedings of the National Academy of Sciences, v. 103, n. 25, p. 9721-9725, 2006Tradução . . Disponível em: https://doi.org/10.1073/pnas.0603286103. Acesso em: 15 out. 2024.
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      Verri Júnior, W. A., Cunha, T. M., Parada, C. A., Wei, X. -qing, Ferreira, S. H., Liew, F. Y., & Cunha, F. de Q. (2006). IL-15 mediates immune inflammatory hypernociception by triggerin a sequential release of IFN-'gama' endothelin, and prostaglandin. Proceedings of the National Academy of Sciences, 103( 25), 9721-9725. doi:10.1073/pnas.0603286103
    • NLM

      Verri Júnior WA, Cunha TM, Parada CA, Wei X-qing, Ferreira SH, Liew FY, Cunha F de Q. IL-15 mediates immune inflammatory hypernociception by triggerin a sequential release of IFN-'gama' endothelin, and prostaglandin [Internet]. Proceedings of the National Academy of Sciences. 2006 ; 103( 25): 9721-9725.[citado 2024 out. 15 ] Available from: https://doi.org/10.1073/pnas.0603286103
    • Vancouver

      Verri Júnior WA, Cunha TM, Parada CA, Wei X-qing, Ferreira SH, Liew FY, Cunha F de Q. IL-15 mediates immune inflammatory hypernociception by triggerin a sequential release of IFN-'gama' endothelin, and prostaglandin [Internet]. Proceedings of the National Academy of Sciences. 2006 ; 103( 25): 9721-9725.[citado 2024 out. 15 ] Available from: https://doi.org/10.1073/pnas.0603286103
  • Source: Physical Therapy. Unidade: FMRP

    Subjects: POLISSACARÍDEOS, DOR, INFLAMAÇÃO

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      JORGE, Sérgio et al. Interferential therapy produces antinociception during application in various models of inflammatory pain. Physical Therapy, v. 86, n. 6, p. 800-808, 2006Tradução . . Acesso em: 15 out. 2024.
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      Jorge, S., Parada, C. A., Ferreira, S. H., & Tambeli, C. H. (2006). Interferential therapy produces antinociception during application in various models of inflammatory pain. Physical Therapy, 86( 6), 800-808.
    • NLM

      Jorge S, Parada CA, Ferreira SH, Tambeli CH. Interferential therapy produces antinociception during application in various models of inflammatory pain. Physical Therapy. 2006 ; 86( 6): 800-808.[citado 2024 out. 15 ]
    • Vancouver

      Jorge S, Parada CA, Ferreira SH, Tambeli CH. Interferential therapy produces antinociception during application in various models of inflammatory pain. Physical Therapy. 2006 ; 86( 6): 800-808.[citado 2024 out. 15 ]
  • Source: Nitric Oxide. Unidade: FMRP

    Subjects: ADERÊNCIA CELULAR, ÓXIDO NÍTRICO, NEUTRÓFILOS

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      DAL SECCO, Daniela et al. Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase. Nitric Oxide, v. 15, p. 77-86, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2006.02.004. Acesso em: 15 out. 2024.
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      Dal Secco, D., Moreira, A. P., Freitas, A., Silva, J. S. da, Rossi, M. A., Ferreira, S. H., & Cunha, F. de Q. (2006). Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase. Nitric Oxide, 15, 77-86. doi:10.1016/j.niox.2006.02.004
    • NLM

      Dal Secco D, Moreira AP, Freitas A, Silva JS da, Rossi MA, Ferreira SH, Cunha F de Q. Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase [Internet]. Nitric Oxide. 2006 ; 15 77-86.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.niox.2006.02.004
    • Vancouver

      Dal Secco D, Moreira AP, Freitas A, Silva JS da, Rossi MA, Ferreira SH, Cunha F de Q. Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase [Internet]. Nitric Oxide. 2006 ; 15 77-86.[citado 2024 out. 15 ] Available from: https://doi.org/10.1016/j.niox.2006.02.004

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