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Artigo de periodico
Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides (2016)
Reglodi, Dora; Renaud, Justine; Tamas, Andrea; Tizabi, Yousef; Socías, Sergio B; Del Bel, Elaine Aparecida; Raisman-Vozari , Rita
Source: Progress in Neurobiology. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, ANTIBIÓTICOS, ESTRESSE OXIDATIVO, ANTIOXIDANTES, TERAPÊUTICA, COMPOSTOS FENÓLICOS
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ABNT
REGLODI, Dora et al. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides. Progress in Neurobiology, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.pneurobio.2015.10.004. Acesso em: 15 nov. 2024.
APA
Reglodi, D., Renaud, J., Tamas, A., Tizabi, Y., Socías, S. B., Del Bel, E. A., & Raisman-Vozari , R. (2016). Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides. Progress in Neurobiology. doi:10.1016/j.pneurobio.2015.10.004
NLM
Reglodi D, Renaud J, Tamas A, Tizabi Y, Socías SB, Del Bel EA, Raisman-Vozari R. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides [Internet]. Progress in Neurobiology. 2016 ;[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.pneurobio.2015.10.004
Vancouver
Reglodi D, Renaud J, Tamas A, Tizabi Y, Socías SB, Del Bel EA, Raisman-Vozari R. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides [Internet]. Progress in Neurobiology. 2016 ;[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.pneurobio.2015.10.004
Artigo de periodico
Sk3 channel overexpression in mice causes hippocampal shrinkage associated with cognitive impairments (2016)
Martin, Sabine; Lazzarini, Marcio; Dullin, Christian; Balakrishnan, Saju; Gomes, Felipe V.; Ninkovic, Milena; El Hady, Ahmed; Pardo, Luis A.; Del Bel, Elaine Aparecida
Source: Molecular Neurobiology. Unidade: FORP
Subjects: DOENÇA DE ALZHEIMER, MEMÓRIA, APRENDIZAGEM, TRANSPORTE DE POTÁSSIO, ESQUIZOFRENIA, DOENÇAS NEURODEGENERATIVAS
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ABNT
MARTIN, Sabine et al. Sk3 channel overexpression in mice causes hippocampal shrinkage associated with cognitive impairments. Molecular Neurobiology, v. jan., 2016Tradução . . Disponível em: https://doi.org/10.1007/s12035-015-9680-6. Acesso em: 15 nov. 2024.
APA
Martin, S., Lazzarini, M., Dullin, C., Balakrishnan, S., Gomes, F. V., Ninkovic, M., et al. (2016). Sk3 channel overexpression in mice causes hippocampal shrinkage associated with cognitive impairments. Molecular Neurobiology, jan. doi:10.1007/s12035-015-9680-6
NLM
Martin S, Lazzarini M, Dullin C, Balakrishnan S, Gomes FV, Ninkovic M, El Hady A, Pardo LA, Del Bel EA. Sk3 channel overexpression in mice causes hippocampal shrinkage associated with cognitive impairments [Internet]. Molecular Neurobiology. 2016 ; jan.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12035-015-9680-6
Vancouver
Martin S, Lazzarini M, Dullin C, Balakrishnan S, Gomes FV, Ninkovic M, El Hady A, Pardo LA, Del Bel EA. Sk3 channel overexpression in mice causes hippocampal shrinkage associated with cognitive impairments [Internet]. Molecular Neurobiology. 2016 ; jan.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12035-015-9680-6
Artigo de periodico
Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects (2016)
Breuer, Aviva; Haj, Christeene G.; Fogaça, Manoela V.; Gomes, Felipe V.; Silva, Nicole Rodrigues; Pedrazzi, João Francisco; Del Bel, Elaine Aparecida; Hallak, Jaime Eduardo Cecílio; Crippa, José Alexandre de Souza; Zuardi, Antônio Waldo; Mechoulam, Raphael; Guimarães, Francisco Silveira
Source: PLOS ONE. Unidades: FORP, FMRP
Subjects: CANABINOIDES, FLUORAÇÃO, NEUROPSIQUIATRIA, FARMACOLOGIA
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ABNT
BREUER, Aviva et al. Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects. PLOS ONE, v. 11, n. 8, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0162087. Acesso em: 15 nov. 2024.
APA
Breuer, A., Haj, C. G., Fogaça, M. V., Gomes, F. V., Silva, N. R., Pedrazzi, J. F., et al. (2016). Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects. PLOS ONE, 11( 8). doi:10.1371/journal.pone.0162087
NLM
Breuer A, Haj CG, Fogaça MV, Gomes FV, Silva NR, Pedrazzi JF, Del Bel EA, Hallak JEC, Crippa JA de S, Zuardi AW, Mechoulam R, Guimarães FS. Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects [Internet]. PLOS ONE. 2016 ; 11( 8):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0162087
Vancouver
Breuer A, Haj CG, Fogaça MV, Gomes FV, Silva NR, Pedrazzi JF, Del Bel EA, Hallak JEC, Crippa JA de S, Zuardi AW, Mechoulam R, Guimarães FS. Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects [Internet]. PLOS ONE. 2016 ; 11( 8):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0162087
Artigo de periodico
Cannabidiol attenuates haloperidol-induced catalepsy and c-Fos protein expression in the dorsolateral striatum via 5-HT1A receptors in mice (2016)
Sonego, Andreza B.; Gomes, Felipe V.; Del Bel, Elaine Aparecida; Guimarães, Francisco Silveira
Source: Behavioural Brain Research. Unidades: FORP, FMRP
Subjects: RECEPTORES, CANABINOIDES, CATALEPSIA, ANTIPSICÓTICOS, CANNABIS
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ABNT
SONEGO, Andreza B. et al. Cannabidiol attenuates haloperidol-induced catalepsy and c-Fos protein expression in the dorsolateral striatum via 5-HT1A receptors in mice. Behavioural Brain Research, v. 309, p. 22-28, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2016.04.042. Acesso em: 15 nov. 2024.
APA
Sonego, A. B., Gomes, F. V., Del Bel, E. A., & Guimarães, F. S. (2016). Cannabidiol attenuates haloperidol-induced catalepsy and c-Fos protein expression in the dorsolateral striatum via 5-HT1A receptors in mice. Behavioural Brain Research, 309, 22-28. doi:10.1016/j.bbr.2016.04.042
NLM
Sonego AB, Gomes FV, Del Bel EA, Guimarães FS. Cannabidiol attenuates haloperidol-induced catalepsy and c-Fos protein expression in the dorsolateral striatum via 5-HT1A receptors in mice [Internet]. Behavioural Brain Research. 2016 ; 309 22-28.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.bbr.2016.04.042
Vancouver
Sonego AB, Gomes FV, Del Bel EA, Guimarães FS. Cannabidiol attenuates haloperidol-induced catalepsy and c-Fos protein expression in the dorsolateral striatum via 5-HT1A receptors in mice [Internet]. Behavioural Brain Research. 2016 ; 309 22-28.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.bbr.2016.04.042
Artigo de periodico
Evaluation of TgH(CX3CR1-EGFP) mice implanted with mCherry-GL261 cells as an in vivo model for morphometrical analysis of glioma-microglia interaction (2016)
Resende, Fernando F. B.; Bai, Xianshu; Del Bel, Elaine Aparecida; Kirchhoff, Frank; Scheller, Anja; Titze-de-Almeida, Ricardo
Source: BMC Cancer. Unidade: FORP
Subjects: GLIOMA, NEOPLASIAS CEREBRAIS, MORFOLOGIA
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ABNT
RESENDE, Fernando F. B. et al. Evaluation of TgH(CX3CR1-EGFP) mice implanted with mCherry-GL261 cells as an in vivo model for morphometrical analysis of glioma-microglia interaction. BMC Cancer, v. 16, n. 1, 2016Tradução . . Disponível em: https://doi.org/10.1186/s12885-016-2118-3. Acesso em: 15 nov. 2024.
APA
Resende, F. F. B., Bai, X., Del Bel, E. A., Kirchhoff, F., Scheller, A., & Titze-de-Almeida, R. (2016). Evaluation of TgH(CX3CR1-EGFP) mice implanted with mCherry-GL261 cells as an in vivo model for morphometrical analysis of glioma-microglia interaction. BMC Cancer, 16( 1). doi:10.1186/s12885-016-2118-3
NLM
Resende FFB, Bai X, Del Bel EA, Kirchhoff F, Scheller A, Titze-de-Almeida R. Evaluation of TgH(CX3CR1-EGFP) mice implanted with mCherry-GL261 cells as an in vivo model for morphometrical analysis of glioma-microglia interaction [Internet]. BMC Cancer. 2016 ; 16( 1):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1186/s12885-016-2118-3
Vancouver
Resende FFB, Bai X, Del Bel EA, Kirchhoff F, Scheller A, Titze-de-Almeida R. Evaluation of TgH(CX3CR1-EGFP) mice implanted with mCherry-GL261 cells as an in vivo model for morphometrical analysis of glioma-microglia interaction [Internet]. BMC Cancer. 2016 ; 16( 1):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1186/s12885-016-2118-3
Artigo de periodico
Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons (2016)
Salum, Cristiane; Schmidt, Fanny; Michel, Patrick P.; Del Bel, Elaine Aparecida; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidade: FORP
Subjects: ÓXIDO NÍTRICO, NEUROENDOCRINOLOGIA
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ABNT
SALUM, Cristiane et al. Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons. Neurotoxicity Research, v. 29, n. 1, p. 92–104, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9562-8. Acesso em: 15 nov. 2024.
APA
Salum, C., Schmidt, F., Michel, P. P., Del Bel, E. A., & Raisman-Vozari, R. (2016). Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons. Neurotoxicity Research, 29( 1), 92–104. doi:10.1007/s12640-015-9562-8
NLM
Salum C, Schmidt F, Michel PP, Del Bel EA, Raisman-Vozari R. Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons [Internet]. Neurotoxicity Research. 2016 ; 29( 1): 92–104.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-015-9562-8
Vancouver
Salum C, Schmidt F, Michel PP, Del Bel EA, Raisman-Vozari R. Signaling mechanisms in the nitric oxide donor and amphetamine-induced dopamine release in mesencephalic primary cultured neurons [Internet]. Neurotoxicity Research. 2016 ; 29( 1): 92–104.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-015-9562-8
Artigo de periodico
CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration (2016)
Tristão, Fabrine Sales Massafera; Lazzarini, Márcio; Martin, Sabine; Amar, Majid; Stühmer, Walter; Kirchhoff, Frank; Gomes, Lucas Araújo Caldi; Lanfumey, Laurance; Prediger, Rui D.; Sepulveda, Julia E.; Del Bel, Elaine Aparecida; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, INFLAMAÇÃO, DEGENERAÇÃO NEURAL
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ABNT
TRISTÃO, Fabrine Sales Massafera et al. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, v. 29, n. 3, p. 364-380, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9557-5. Acesso em: 15 nov. 2024.
APA
Tristão, F. S. M., Lazzarini, M., Martin, S., Amar, M., Stühmer, W., Kirchhoff, F., et al. (2016). CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, 29( 3), 364-380. doi:10.1007/s12640-015-9557-5
NLM
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Vancouver
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Artigo de periodico
Cytoarchitecture of nitrergic neurons in the human striatum and subthalamic nucleus (2016)
Santos-Lobato, Bruno Lopes dos; Del Bel, Elaine Aparecida; Pittella, José Eymard Homem; Tumas, Vitor
Source: Brain Research Bulletin. Unidades: FORP, FMRP
Subjects: NEUROCIÊNCIAS, GÂNGLIOS, CÉREBRO, ÓXIDO NÍTRICO, NEURÔNIOS
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ABNT
SANTOS-LOBATO, Bruno Lopes dos et al. Cytoarchitecture of nitrergic neurons in the human striatum and subthalamic nucleus. Brain Research Bulletin, v. 124, p. 129-135, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2016.04.003. Acesso em: 15 nov. 2024.
APA
Santos-Lobato, B. L. dos, Del Bel, E. A., Pittella, J. E. H., & Tumas, V. (2016). Cytoarchitecture of nitrergic neurons in the human striatum and subthalamic nucleus. Brain Research Bulletin, 124, 129-135. doi:10.1016/j.brainresbull.2016.04.003
NLM
Santos-Lobato BL dos, Del Bel EA, Pittella JEH, Tumas V. Cytoarchitecture of nitrergic neurons in the human striatum and subthalamic nucleus [Internet]. Brain Research Bulletin. 2016 ; 124 129-135.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.brainresbull.2016.04.003
Vancouver
Santos-Lobato BL dos, Del Bel EA, Pittella JEH, Tumas V. Cytoarchitecture of nitrergic neurons in the human striatum and subthalamic nucleus [Internet]. Brain Research Bulletin. 2016 ; 124 129-135.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.brainresbull.2016.04.003
Artigo de periodico
Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways (2016)
Santa-Cecília, Flávia V.; Socias, Benjamin; Ouidja, Mohand O.; Sepulveda-Diaz, Julia E.; Acuña, Leonardo; Silva, Rangel L.; Michel, Patrick P.; Del Bel, Elaine Aparecida; Cunha, Thiago Mattar; Raisman-Vozari, Rita
Source: Neurotoxicity Research. Unidades: FORP, FMRP
Subjects: DOENÇAS NEURODEGENERATIVAS, CITOCINAS, DOENÇA DE PARKINSON, FÁRMACOS
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ABNT
SANTA-CECÍLIA, Flávia V. et al. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, v. 29, n. 4, p. 447-459, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9592-2. Acesso em: 15 nov. 2024.
APA
Santa-Cecília, F. V., Socias, B., Ouidja, M. O., Sepulveda-Diaz, J. E., Acuña, L., Silva, R. L., et al. (2016). Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, 29( 4), 447-459. doi:10.1007/s12640-015-9592-2
NLM
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-015-9592-2
Vancouver
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-015-9592-2
Artigo de periodico
Antidyskinetic effect of 7-nitroindazole and sodium nitroprusside associated with amantadine in a rat model of parkinson’s disease (2016)
Bortolanza, Mariza; Santos, Keila D. Bariotto dos; Pereira, Maurício dos Santos; Silva, Célia Aparecida da; Del Bel, Elaine Aparecida
Source: Neurotoxicity Research. Unidade: FORP
Subjects: GLUTAMATOS, ÓXIDO NÍTRICO, AGENTES DOPAMINÉRGICOS, DOENÇA DE PARKINSON
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ABNT
BORTOLANZA, Mariza et al. Antidyskinetic effect of 7-nitroindazole and sodium nitroprusside associated with amantadine in a rat model of parkinson’s disease. Neurotoxicity Research, v. 30, n. 1, p. 88-100, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-016-9618-4. Acesso em: 15 nov. 2024.
APA
Bortolanza, M., Santos, K. D. B. dos, Pereira, M. dos S., Silva, C. A. da, & Del Bel, E. A. (2016). Antidyskinetic effect of 7-nitroindazole and sodium nitroprusside associated with amantadine in a rat model of parkinson’s disease. Neurotoxicity Research, 30( 1), 88-100. doi:10.1007/s12640-016-9618-4
NLM
Bortolanza M, Santos KDB dos, Pereira M dos S, Silva CA da, Del Bel EA. Antidyskinetic effect of 7-nitroindazole and sodium nitroprusside associated with amantadine in a rat model of parkinson’s disease [Internet]. Neurotoxicity Research. 2016 ; 30( 1): 88-100.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-016-9618-4
Vancouver
Bortolanza M, Santos KDB dos, Pereira M dos S, Silva CA da, Del Bel EA. Antidyskinetic effect of 7-nitroindazole and sodium nitroprusside associated with amantadine in a rat model of parkinson’s disease [Internet]. Neurotoxicity Research. 2016 ; 30( 1): 88-100.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s12640-016-9618-4
Artigo de periodico
Fluorinated Cannabidiol derivatives: enhancement of Activity in Mice Models Predictive of Anxiolytic, Antidepressant and Antipsychotic Effects (2016)
Breuer, Aviva; Haj, Christeene G.; Fogaça, Manoela V.; Gomes, Felipe V.; Silva, Nicole Rodrigues; Pedrazzi, João Francisco; Del Bel, Elaine Aparecida; Hallak, Jaime Eduardo Cecílio; Crippa, José Alexandre de Souza; Zuardi, Antônio Waldo; Mechoulam, Raphael; Guimarães, Francisco Silveira
Source: PLoS ONE. Unidades: FORP, FMRP
Subjects: CANABINOIDES, FLUORAÇÃO, NEUROPSIQUIATRIA, FARMACOLOGIA
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ABNT
BREUER, Aviva et al. Fluorinated Cannabidiol derivatives: enhancement of Activity in Mice Models Predictive of Anxiolytic, Antidepressant and Antipsychotic Effects. PLoS ONE, v. 11, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0158779. Acesso em: 15 nov. 2024.
APA
Breuer, A., Haj, C. G., Fogaça, M. V., Gomes, F. V., Silva, N. R., Pedrazzi, J. F., et al. (2016). Fluorinated Cannabidiol derivatives: enhancement of Activity in Mice Models Predictive of Anxiolytic, Antidepressant and Antipsychotic Effects. PLoS ONE, 11. doi:10.1371/journal.pone.0158779
NLM
Breuer A, Haj CG, Fogaça MV, Gomes FV, Silva NR, Pedrazzi JF, Del Bel EA, Hallak JEC, Crippa JA de S, Zuardi AW, Mechoulam R, Guimarães FS. Fluorinated Cannabidiol derivatives: enhancement of Activity in Mice Models Predictive of Anxiolytic, Antidepressant and Antipsychotic Effects [Internet]. PLoS ONE. 2016 ; 11[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0158779
Vancouver
Breuer A, Haj CG, Fogaça MV, Gomes FV, Silva NR, Pedrazzi JF, Del Bel EA, Hallak JEC, Crippa JA de S, Zuardi AW, Mechoulam R, Guimarães FS. Fluorinated Cannabidiol derivatives: enhancement of Activity in Mice Models Predictive of Anxiolytic, Antidepressant and Antipsychotic Effects [Internet]. PLoS ONE. 2016 ; 11[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0158779
Artigo de periodico
Co-administration of cannabidiol and capsazepine reduces L-DOPA-induced dyskinesia in mice: possible mechanism of action (2016)
Pereira, Maurício dos Santos; Silva, Célia Aparecida da; Guimarães, Francisco Silveira; Del Bel, Elaine Aparecida
Source: Neurobiology of Disease. Unidades: FORP, FMRP
Subjects: CANNABIS, CANABINOIDES, FARMACOLOGIA, DOENÇA DE PARKINSON
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ABNT
PEREIRA, Maurício dos Santos et al. Co-administration of cannabidiol and capsazepine reduces L-DOPA-induced dyskinesia in mice: possible mechanism of action. Neurobiology of Disease, v. 94, p. 179-195, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.nbd.2016.06.013. Acesso em: 15 nov. 2024.
APA
Pereira, M. dos S., Silva, C. A. da, Guimarães, F. S., & Del Bel, E. A. (2016). Co-administration of cannabidiol and capsazepine reduces L-DOPA-induced dyskinesia in mice: possible mechanism of action. Neurobiology of Disease, 94, 179-195. doi:10.1016/j.nbd.2016.06.013
NLM
Pereira M dos S, Silva CA da, Guimarães FS, Del Bel EA. Co-administration of cannabidiol and capsazepine reduces L-DOPA-induced dyskinesia in mice: possible mechanism of action [Internet]. Neurobiology of Disease. 2016 ; 94 179-195.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.nbd.2016.06.013
Vancouver
Pereira M dos S, Silva CA da, Guimarães FS, Del Bel EA. Co-administration of cannabidiol and capsazepine reduces L-DOPA-induced dyskinesia in mice: possible mechanism of action [Internet]. Neurobiology of Disease. 2016 ; 94 179-195.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.nbd.2016.06.013

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