Filtros : "Araújo, Eliseu Frank de" Removido: "PARACOCCIDIOIDOMICOSE" Limpar

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  • Source: PLoS Pathogens. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      ARAÚJO, Eliseu Frank de et al. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, v. 12, n. 12, p. 1-29, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1006115. Acesso em: 02 out. 2024.
    • APA

      Araújo, E. F. de, Medeiros, D. H., Galdino, N. A. de L., Condino-Neto, A., Calich, V. L. G., & Loures, F. V. (2016). Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, 12( 12), 1-29. doi:10.1371/journal.ppat.1006115
    • NLM

      Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 out. 02 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
    • Vancouver

      Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 out. 02 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
  • Source: Frontiers in Microbiology. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE (RESISTÊNCIA), INFLAMAÇÃO

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      FERIOTTI, Claudia et al. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, v. 6, n. 913, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00913. Acesso em: 02 out. 2024.
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      Feriotti, C., Bazan, S. B., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2015). Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, 6( 913), 1-13. doi:10.3389/fmicb.2015.00913
    • NLM

      Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 out. 02 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
    • Vancouver

      Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 out. 02 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
  • Source: Frontiers in Microbiology. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDES, PARACOCCIDIOIDES BRASILIENSIS, CÉLULAS DENDRITICAS

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      LOURES, Flávio Vieira et al. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, v. 6, n. 261, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00261. Acesso em: 02 out. 2024.
    • APA

      Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., & Calich, V. L. G. (2015). TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, 6( 261), 1-11. doi:10.3389/fmicb.2015.00261
    • NLM

      Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 out. 02 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
    • Vancouver

      Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 out. 02 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
  • Source: Mediators of Inflammation. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDES BRASILIENSIS, MACRÓFAGOS, INFECÇÕES RESPIRATÓRIAS

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      RIBEIRO, Laura Rossetti Rios et al. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, v. 2015, p. 1-17, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/852574. Acesso em: 02 out. 2024.
    • APA

      Ribeiro, L. R. R., Loures, F. V., Araújo, E. F. de, Feriotti, C., Costa, T. A. da, Serezani, C. H., et al. (2015). Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, 2015, 1-17. doi:10.1155/2015/852574
    • NLM

      Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 out. 02 ] Available from: https://doi.org/10.1155/2015/852574
    • Vancouver

      Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 out. 02 ] Available from: https://doi.org/10.1155/2015/852574
  • Source: PLOS ONE. Unidade: ICB

    Assunto: IMUNOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      FERIOTTI, Claudia et al. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, v. 8, n. 1, p. 1-17, 2013Tradução . . Disponível em: https://doi.org/10.1590/S0004-27302013000100003. Acesso em: 02 out. 2024.
    • APA

      Feriotti, C., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2013). Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, 8( 1), 1-17. doi:10.1590/S0004-27302013000100003
    • NLM

      Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 out. 02 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
    • Vancouver

      Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 out. 02 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      LOURES, Flávio Vieira et al. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, v. 79, n. 6, p. 2470-2480, 2011Tradução . . Disponível em: https://doi.org/10.1128/IAI.00375-10. Acesso em: 02 out. 2024.
    • APA

      Loures, F. V., Pina, A., Felonato, M., Feriotti, C., Araújo, E. F. de, & Calich, V. L. G. (2011). MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, 79( 6), 2470-2480. doi:10.1128/IAI.00375-10
    • NLM

      Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 out. 02 ] Available from: https://doi.org/10.1128/IAI.00375-10
    • Vancouver

      Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 out. 02 ] Available from: https://doi.org/10.1128/IAI.00375-10
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FELONATO, Maíra et al. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, v. 78, n. 11, p. 4922-4935, 2010Tradução . . Disponível em: https://doi.org/10.1128/IAI.00297-10. Acesso em: 02 out. 2024.
    • APA

      Felonato, M., Pina, A., Bernardino, S., Loures, F. V., Araújo, E. F. de, & Calich, V. L. G. (2010). CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, 78( 11), 4922-4935. doi:10.1128/IAI.00297-10
    • NLM

      Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 out. 02 ] Available from: https://doi.org/10.1128/IAI.00297-10
    • Vancouver

      Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 out. 02 ] Available from: https://doi.org/10.1128/IAI.00297-10
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flávio Vieira et al. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, v. 78, n. 3, p. 1078-1088, 2010Tradução . . Acesso em: 02 out. 2024.
    • APA

      Loures, F. V., Pina, A., Felonato, M., Araújo, E. F. de, Leite, K. R. M., & Calich, V. L. G. (2010). Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, 78( 3), 1078-1088.
    • NLM

      Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 out. 02 ]
    • Vancouver

      Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 out. 02 ]

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