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Artigo de periodico
Extracellular vesicles from the dermatophyte trichophyton interdigitale modulate macrophage and keratinocyte functions (2018)
Bitencourt, Tamires Aparecida; Rezende, Caroline Patini; Quaresemin, Natália Renault; Moreno, Pedro; Hatanaka, Otavio; Rossi, Antônio; Martinez-Rossi, Nilce Maria; Almeida, Fausto
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: ARTHRODERMATACEAE, INFECÇÕES BACTERIANAS E MICOSES, IMUNOLOGIA, MACRÓFAGOS, NANOPARTÍCULAS
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ABNT
BITENCOURT, Tamires Aparecida et al. Extracellular vesicles from the dermatophyte trichophyton interdigitale modulate macrophage and keratinocyte functions. Frontiers in Immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.02343. Acesso em: 17 ago. 2024.
APA
Bitencourt, T. A., Rezende, C. P., Quaresemin, N. R., Moreno, P., Hatanaka, O., Rossi, A., et al. (2018). Extracellular vesicles from the dermatophyte trichophyton interdigitale modulate macrophage and keratinocyte functions. Frontiers in Immunology, 9. doi:10.3389/fimmu.2018.02343
NLM
Bitencourt TA, Rezende CP, Quaresemin NR, Moreno P, Hatanaka O, Rossi A, Martinez-Rossi NM, Almeida F. Extracellular vesicles from the dermatophyte trichophyton interdigitale modulate macrophage and keratinocyte functions [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fimmu.2018.02343
Vancouver
Bitencourt TA, Rezende CP, Quaresemin NR, Moreno P, Hatanaka O, Rossi A, Martinez-Rossi NM, Almeida F. Extracellular vesicles from the dermatophyte trichophyton interdigitale modulate macrophage and keratinocyte functions [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fimmu.2018.02343
Artigo de periodico
The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β (2018)
Ruiz-Miyazawa, Kenji W.; Borghi, Sergio M.; Ribeiro, Felipe A. Pinho; Ferrari, Larissa Staurengo; Fattori, Victor; Fernandes, Glaura S. A.; Casella, Antonio M.; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Casagrande, Rubia; Verri, Waldiceu A.
Source: Journal of Functional Foods. Unidade: FMRP
Subjects: ARTRITE, DOR, INFLAMAÇÃO, RATOS, MACRÓFAGOS
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ABNT
RUIZ-MIYAZAWA, Kenji W. et al. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, v. 48, p. 106-116, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jff.2018.06.025. Acesso em: 17 ago. 2024.
APA
Ruiz-Miyazawa, K. W., Borghi, S. M., Ribeiro, F. A. P., Ferrari, L. S., Fattori, V., Fernandes, G. S. A., et al. (2018). The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, 48, 106-116. doi:10.1016/j.jff.2018.06.025
NLM
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Vancouver
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Artigo de periodico
The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression (2018)
Amano, Mariane Tami ; Castoldi, Angela ; Andrade-Oliveira, Vinicius ; Latância, Marcela Teatin ; Terra, Fernanda Fernandes ; Correa-Costa, Matheus ; Breda, Cristiane Naffah Souza ; Felizardo, Raphael José Ferreira ; Pereira, Welbert Oliveira de ; Silva, Marina Burgos da ; Miyagi, Mariana Yasue Saito ; Aguiar, Cristhiane Favero ; Hiyane, Meire Ioshie ; Silva, João Santana da ; Moura, Ivan Cruz ; Câmara, Niels Olsen Saraiva
Source: International Immunopharmacology. Unidades: ICB, FMRP, FCF, BIOTECNOLOGIA
Subjects: INSUFICIÊNCIA RENAL AGUDA, NEFROPATIAS, MACRÓFAGOS, IMUNOLOGIA
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ABNT
AMANO, Mariane Tami et al. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, v. 64, p. 151-161, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.08.020. Acesso em: 17 ago. 2024.
APA
Amano, M. T., Castoldi, A., Andrade-Oliveira, V., Latância, M. T., Terra, F. F., Correa-Costa, M., et al. (2018). The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, 64, 151-161. doi:10.1016/j.intimp.2018.08.020
NLM
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Vancouver
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Artigo de periodico
Ureaplasma diversum and its membrane-associated lipoproteins activate inflammatory genes through the NF-κB pathway via toll-like receptor 4 (2018)
Santos Junior, Manoel Neres ; Rezende, Izadora de Souza ; Souza, Clarissa Leal Silva e; Barbosa, Maysa Santos; Campos, Guilherme Barreto; Brito, Laís Ferraz ; Queiroz, Éllunny Chaves; Barbosa, Elaine Novaes; Teixeira, Mariana M.; Silva, Letícia O. da; Silva, Lucas Santana Coelho; Nascimento, Flávia de Souza; Silva, Tassyo L. da; Martens, Adam Arai; Siqueira, Adriano Felipe Perez ; Assumpção, Mayra Elena Ortiz D'Ávila ; Santelli, Glaucia Maria Machado ; Bastos, Bruno Lopes; Guimarães, Ana Marcia de Sá ; Timenetsky, Jorge ; Marques, Lucas Miranda
Source: Frontiers in Microbiology. Unidades: FMVZ, ICB
Subjects: BOVINOS, VULVOVAGINITE ANIMAL, PATOLOGIAS INERENTES À FÊMEA ANIMAL, LIPOPROTEÍNAS, INFLAMAÇÃO, MACRÓFAGOS
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ABNT
SANTOS JUNIOR, Manoel Neres et al. Ureaplasma diversum and its membrane-associated lipoproteins activate inflammatory genes through the NF-κB pathway via toll-like receptor 4. Frontiers in Microbiology, v. 9, p. 14 , 2018Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2018.01538. Acesso em: 17 ago. 2024.
APA
Santos Junior, M. N., Rezende, I. de S., Souza, C. L. S. e, Barbosa, M. S., Campos, G. B., Brito, L. F., et al. (2018). Ureaplasma diversum and its membrane-associated lipoproteins activate inflammatory genes through the NF-κB pathway via toll-like receptor 4. Frontiers in Microbiology, 9, 14 . doi:10.3389/fmicb.2018.01538
NLM
Santos Junior MN, Rezende I de S, Souza CLS e, Barbosa MS, Campos GB, Brito LF, Queiroz ÉC, Barbosa EN, Teixeira MM, Silva LO da, Silva LSC, Nascimento F de S, Silva TL da, Martens AA, Siqueira AFP, Assumpção MEOD'Á, Santelli GMM, Bastos BL, Guimarães AM de S, Timenetsky J, Marques LM. Ureaplasma diversum and its membrane-associated lipoproteins activate inflammatory genes through the NF-κB pathway via toll-like receptor 4 [Internet]. Frontiers in Microbiology. 2018 ; 9 14 .[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fmicb.2018.01538
Vancouver
Santos Junior MN, Rezende I de S, Souza CLS e, Barbosa MS, Campos GB, Brito LF, Queiroz ÉC, Barbosa EN, Teixeira MM, Silva LO da, Silva LSC, Nascimento F de S, Silva TL da, Martens AA, Siqueira AFP, Assumpção MEOD'Á, Santelli GMM, Bastos BL, Guimarães AM de S, Timenetsky J, Marques LM. Ureaplasma diversum and its membrane-associated lipoproteins activate inflammatory genes through the NF-κB pathway via toll-like receptor 4 [Internet]. Frontiers in Microbiology. 2018 ; 9 14 .[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fmicb.2018.01538
Artigo de periodico
Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis (2018)
Staurengo-Ferrari, Larissa; Trevelin, Silvia C.; Fattori, Victor; Nascimento, Daniele C.; Lima, Kalil A. de; Pelayo, Jacinta S.; Figueiredo, Florêncio; Casagrande, Rubia; Fukada, Sandra Yasuyo; Teixeira, Mauro M.; Cunha, Thiago Mattar; Liew, Foo Y.; Oliveira, Rene D.; Louzada Júnior, Paulo; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias; Verri, Waldiceu A.
Source: Frontiers in Immunology. Unidades: FCFRP, FMRP
Subjects: MACRÓFAGOS, STAPHYLOCOCCUS, INTERLEUCINAS, ÓXIDO NÍTRICO, ARTRITE
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ABNT
STAURENGO-FERRARI, Larissa et al. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis. Frontiers in Immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00962. Acesso em: 17 ago. 2024.
APA
Staurengo-Ferrari, L., Trevelin, S. C., Fattori, V., Nascimento, D. C., Lima, K. A. de, Pelayo, J. S., et al. (2018). Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis. Frontiers in Immunology, 9. doi:10.3389/fimmu.2018.00962
NLM
Staurengo-Ferrari L, Trevelin SC, Fattori V, Nascimento DC, Lima KA de, Pelayo JS, Figueiredo F, Casagrande R, Fukada SY, Teixeira MM, Cunha TM, Liew FY, Oliveira RD, Louzada Júnior P, Cunha F de Q, Alves Filho JCF, Verri WA. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fimmu.2018.00962
Vancouver
Staurengo-Ferrari L, Trevelin SC, Fattori V, Nascimento DC, Lima KA de, Pelayo JS, Figueiredo F, Casagrande R, Fukada SY, Teixeira MM, Cunha TM, Liew FY, Oliveira RD, Louzada Júnior P, Cunha F de Q, Alves Filho JCF, Verri WA. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fimmu.2018.00962
Artigo de periodico
Purinergic cooperation between P2Y2 and P2X7 receptors promote cutaneous leishmaniasis control: involvement of pannexin-1 and leukotrienes (2018)
Thorstenberg, Maria Luiza; Ferreira, Marcos Vinícius Rangel; Amorim, Natália; Canetti, Claudio; Morrone, Fernanda B.; Alves Filho, José Carlos Farias; Silva, Robson Coutinho
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: LEISHMANIA, ADENOSINA TRIFOSFATO, RECEPTORES, MACRÓFAGOS
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ABNT
THORSTENBERG, Maria Luiza et al. Purinergic cooperation between P2Y2 and P2X7 receptors promote cutaneous leishmaniasis control: involvement of pannexin-1 and leukotrienes. Frontiers in Immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.01531. Acesso em: 17 ago. 2024.
APA
Thorstenberg, M. L., Ferreira, M. V. R., Amorim, N., Canetti, C., Morrone, F. B., Alves Filho, J. C. F., & Silva, R. C. (2018). Purinergic cooperation between P2Y2 and P2X7 receptors promote cutaneous leishmaniasis control: involvement of pannexin-1 and leukotrienes. Frontiers in Immunology, 9. doi:10.3389/fimmu.2018.01531
NLM
Thorstenberg ML, Ferreira MVR, Amorim N, Canetti C, Morrone FB, Alves Filho JCF, Silva RC. Purinergic cooperation between P2Y2 and P2X7 receptors promote cutaneous leishmaniasis control: involvement of pannexin-1 and leukotrienes [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fimmu.2018.01531
Vancouver
Thorstenberg ML, Ferreira MVR, Amorim N, Canetti C, Morrone FB, Alves Filho JCF, Silva RC. Purinergic cooperation between P2Y2 and P2X7 receptors promote cutaneous leishmaniasis control: involvement of pannexin-1 and leukotrienes [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 ago. 17 ] Available from: https://doi.org/10.3389/fimmu.2018.01531
Artigo de periodico
Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner (2018)
Wanderley, Carlos W.; Colon, David F.; Luiz, Joao Paulo Mesquita; Oliveira, Francisco F.; Viacava, Paula R.; Leite, Caio A.; Pereira, Janaina A.; Silva, Camila M.; Silva, Cassia R.; Silva, Rangel L.; Hernandez, Cesar A. Speck; Mota, Jose M.; Alves Filho, José Carlos Farias; Lima-Júnior, Roberto C.; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz
Source: Cancer Research. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS, IMUNOLOGIA CELULAR, NEOPLASIAS, MACRÓFAGOS
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ABNT
WANDERLEY, Carlos W. et al. Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner. Cancer Research, v. 78, n. 20, p. 5891-5900, 2018Tradução . . Disponível em: https://doi.org/10.1158/0008-5472.can-17-3480. Acesso em: 17 ago. 2024.
APA
Wanderley, C. W., Colon, D. F., Luiz, J. P. M., Oliveira, F. F., Viacava, P. R., Leite, C. A., et al. (2018). Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner. Cancer Research, 78( 20), 5891-5900. doi:10.1158/0008-5472.can-17-3480
NLM
Wanderley CW, Colon DF, Luiz JPM, Oliveira FF, Viacava PR, Leite CA, Pereira JA, Silva CM, Silva CR, Silva RL, Hernandez CAS, Mota JM, Alves Filho JCF, Lima-Júnior RC, Cunha TM, Cunha F de Q. Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner [Internet]. Cancer Research. 2018 ; 78( 20): 5891-5900.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1158/0008-5472.can-17-3480
Vancouver
Wanderley CW, Colon DF, Luiz JPM, Oliveira FF, Viacava PR, Leite CA, Pereira JA, Silva CM, Silva CR, Silva RL, Hernandez CAS, Mota JM, Alves Filho JCF, Lima-Júnior RC, Cunha TM, Cunha F de Q. Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner [Internet]. Cancer Research. 2018 ; 78( 20): 5891-5900.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1158/0008-5472.can-17-3480
Artigo de periodico
Macrophage-derived GPNMB accelerates skin healing (2018)
Silva, Walison N; Prazeres, Pedro H. D. M; Paiva, Ana E; Lousado, Luiza; Turquetti, Anaelise O. M; Barreto, Rodrigo S. N; Alvarenga, Erika Costa de; Miglino, Maria Angélica; Gonçalves, Ricardo; Mintz, Akiva; Birbrair, Alexander
Source: Experimental Dermatology. Unidade: FMVZ
Subjects: PELE, CICATRIZAÇÃO, MACRÓFAGOS
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ABNT
SILVA, Walison N et al. Macrophage-derived GPNMB accelerates skin healing. Experimental Dermatology, v. 27, n. 6, p. 630-635, 2018Tradução . . Disponível em: https://doi.org/10.1111/exd.13524. Acesso em: 17 ago. 2024.
APA
Silva, W. N., Prazeres, P. H. D. M., Paiva, A. E., Lousado, L., Turquetti, A. O. M., Barreto, R. S. N., et al. (2018). Macrophage-derived GPNMB accelerates skin healing. Experimental Dermatology, 27( 6), 630-635. doi:10.1111/exd.13524
NLM
Silva WN, Prazeres PHDM, Paiva AE, Lousado L, Turquetti AOM, Barreto RSN, Alvarenga EC de, Miglino MA, Gonçalves R, Mintz A, Birbrair A. Macrophage-derived GPNMB accelerates skin healing [Internet]. Experimental Dermatology. 2018 ; 27( 6): 630-635.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1111/exd.13524
Vancouver
Silva WN, Prazeres PHDM, Paiva AE, Lousado L, Turquetti AOM, Barreto RSN, Alvarenga EC de, Miglino MA, Gonçalves R, Mintz A, Birbrair A. Macrophage-derived GPNMB accelerates skin healing [Internet]. Experimental Dermatology. 2018 ; 27( 6): 630-635.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1111/exd.13524
Artigo de periodico
Leishmanicidal effects of piperlongumine (piplartine) and its putative metabolites (2018)
Moreira, Fernanda de Lima ; Riul, Thalita Bachelli ; Moreira, Marcela de Lima; Pilon, Alan Cesar; Dias-Baruffi, Marcelo ; Araújo, Márcio S. S.; Lopes, Norberto Peporine ; Oliveira, Anderson Rodrigo Moraes de
Source: Planta Medica. Unidades: FCFRP, FFCLRP
Subjects: ANTI-HELMÍNTICOS, FARMACOLOGIA, METABÓLITOS, MACRÓFAGOS
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ABNT
MOREIRA, Fernanda de Lima et al. Leishmanicidal effects of piperlongumine (piplartine) and its putative metabolites. Planta Medica, v. 84, n. 15, p. 1141-1148, 2018Tradução . . Disponível em: https://doi.org/10.1055/a-0614-2680. Acesso em: 17 ago. 2024.
APA
Moreira, F. de L., Riul, T. B., Moreira, M. de L., Pilon, A. C., Dias-Baruffi, M., Araújo, M. S. S., et al. (2018). Leishmanicidal effects of piperlongumine (piplartine) and its putative metabolites. Planta Medica, 84( 15), 1141-1148. doi:10.1055/a-0614-2680
NLM
Moreira F de L, Riul TB, Moreira M de L, Pilon AC, Dias-Baruffi M, Araújo MSS, Lopes NP, Oliveira ARM de. Leishmanicidal effects of piperlongumine (piplartine) and its putative metabolites [Internet]. Planta Medica. 2018 ; 84( 15): 1141-1148.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1055/a-0614-2680
Vancouver
Moreira F de L, Riul TB, Moreira M de L, Pilon AC, Dias-Baruffi M, Araújo MSS, Lopes NP, Oliveira ARM de. Leishmanicidal effects of piperlongumine (piplartine) and its putative metabolites [Internet]. Planta Medica. 2018 ; 84( 15): 1141-1148.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1055/a-0614-2680
Artigo de periodico
Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages (2017)
Flatow, Elizabeth A.; Komegae, Evilin N.; Fonseca, Monique T.; Brito, Camila F.; Musteata, Florin M.; Antunes-Rodrigues, José; Steiner, Alexandre Alarcon
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. Unidade: FMRP
Subjects: CITOCINAS, FEBRE, PRIVAÇÃO DE ALIMENTOS, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, MACRÓFAGOS
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ABNT
FLATOW, Elizabeth A. et al. Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, v. 313, p. R572-582, 2017Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00171.2017. Acesso em: 17 ago. 2024.
APA
Flatow, E. A., Komegae, E. N., Fonseca, M. T., Brito, C. F., Musteata, F. M., Antunes-Rodrigues, J., & Steiner, A. A. (2017). Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 313, R572-582. doi:10.1152/ajpregu.00171.2017
NLM
Flatow EA, Komegae EN, Fonseca MT, Brito CF, Musteata FM, Antunes-Rodrigues J, Steiner AA. Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2017 ; 313 R572-582.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1152/ajpregu.00171.2017
Vancouver
Flatow EA, Komegae EN, Fonseca MT, Brito CF, Musteata FM, Antunes-Rodrigues J, Steiner AA. Elucidating the role of leptin in systemic inflammation: a study targeting physiological leptin levels in rats and their macrophages [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2017 ; 313 R572-582.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1152/ajpregu.00171.2017
Artigo de periodico
Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis (2017)
Dias-Teixeira, Karina Luiza; Calegari-Silva, Teresa C.; Medina, Jorge M.; Vivarini, Áislan C.; Cavalcanti, Átila; Teteo, Nataly; Santana, Alynne Karen M.; Real, Fernando; Gomes, Ciro M.; Pereira, Renata Meirelles Santos; Fasel, Nicolas; Silva, João Santana da; Aktas, Bertal H.; Lopes, Ulisses G.
Source: Scientific Reports. Unidade: FMRP
Subjects: LEISHMANIA, MACRÓFAGOS, RETÍCULO ENDOPLASMÁTICO
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ABNT
DIAS-TEIXEIRA, Karina Luiza et al. Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis. Scientific Reports, v. 7, n. 1, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-17252-x. Acesso em: 17 ago. 2024.
APA
Dias-Teixeira, K. L., Calegari-Silva, T. C., Medina, J. M., Vivarini, Á. C., Cavalcanti, Á., Teteo, N., et al. (2017). Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis. Scientific Reports, 7( 1). doi:10.1038/s41598-017-17252-x
NLM
Dias-Teixeira KL, Calegari-Silva TC, Medina JM, Vivarini ÁC, Cavalcanti Á, Teteo N, Santana AKM, Real F, Gomes CM, Pereira RMS, Fasel N, Silva JS da, Aktas BH, Lopes UG. Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 ago. 17 ] Available from: https://doi.org/10.1038/s41598-017-17252-x
Vancouver
Dias-Teixeira KL, Calegari-Silva TC, Medina JM, Vivarini ÁC, Cavalcanti Á, Teteo N, Santana AKM, Real F, Gomes CM, Pereira RMS, Fasel N, Silva JS da, Aktas BH, Lopes UG. Emerging role for the PERK/eIF2α/ATF4 in human cutaneous leishmaniasis [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 ago. 17 ] Available from: https://doi.org/10.1038/s41598-017-17252-x
Artigo de periodico
Murine alveolar macrophages are highly susceptible to replication of Coxiella burnetii phase II in vitro (2016)
Fernandes, Talita D.; Cunha, Larissa D.; Ribeiro, Juliana M.; Massis, Liliana M.; Lima-Junior, Djalma S.; Newton, Hayley J.; Zamboni, Dario Simões
Source: Infection and Immunity. Unidade: FMRP
Subjects: FEBRE Q, MACRÓFAGOS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS, MICROBIOLOGIA, SISTEMA IMUNE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERNANDES, Talita D. et al. Murine alveolar macrophages are highly susceptible to replication of Coxiella burnetii phase II in vitro. Infection and Immunity, v. 84, n. 9, p. 2439-2448, 2016Tradução . . Disponível em: https://doi.org/10.1128/iai.00411-16. Acesso em: 17 ago. 2024.
APA
Fernandes, T. D., Cunha, L. D., Ribeiro, J. M., Massis, L. M., Lima-Junior, D. S., Newton, H. J., & Zamboni, D. S. (2016). Murine alveolar macrophages are highly susceptible to replication of Coxiella burnetii phase II in vitro. Infection and Immunity, 84( 9), 2439-2448. doi:10.1128/iai.00411-16
NLM
Fernandes TD, Cunha LD, Ribeiro JM, Massis LM, Lima-Junior DS, Newton HJ, Zamboni DS. Murine alveolar macrophages are highly susceptible to replication of Coxiella burnetii phase II in vitro [Internet]. Infection and Immunity. 2016 ; 84( 9): 2439-2448.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1128/iai.00411-16
Vancouver
Fernandes TD, Cunha LD, Ribeiro JM, Massis LM, Lima-Junior DS, Newton HJ, Zamboni DS. Murine alveolar macrophages are highly susceptible to replication of Coxiella burnetii phase II in vitro [Internet]. Infection and Immunity. 2016 ; 84( 9): 2439-2448.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1128/iai.00411-16
Artigo de periodico
Neuro-immune interactions drive tissue programming in intestinal macrophages (2016)
Gabanyi, Ilana; Muller, Paul A; Feighery, Linda; Oliveira, Thiago Y; Costa-Pinto, Frederico Azevedo da ; Mucida, Daniel
Source: Cell. Unidade: FMVZ
Subjects: MACRÓFAGOS, INTESTINOS, NEUROIMUNOMODULAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GABANYI, Ilana et al. Neuro-immune interactions drive tissue programming in intestinal macrophages. Cell, v. 164, n. 3, p. 378-391, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.cell.2015.12.023. Acesso em: 17 ago. 2024.
APA
Gabanyi, I., Muller, P. A., Feighery, L., Oliveira, T. Y., Costa-Pinto, F. A. da, & Mucida, D. (2016). Neuro-immune interactions drive tissue programming in intestinal macrophages. Cell, 164( 3), 378-391. doi:10.1016/j.cell.2015.12.023
NLM
Gabanyi I, Muller PA, Feighery L, Oliveira TY, Costa-Pinto FA da, Mucida D. Neuro-immune interactions drive tissue programming in intestinal macrophages [Internet]. Cell. 2016 ; 164( 3): 378-391.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.cell.2015.12.023
Vancouver
Gabanyi I, Muller PA, Feighery L, Oliveira TY, Costa-Pinto FA da, Mucida D. Neuro-immune interactions drive tissue programming in intestinal macrophages [Internet]. Cell. 2016 ; 164( 3): 378-391.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.cell.2015.12.023
Artigo de periodico
Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages (2016)
Bradley, William P.; Boyer, Mark A.; Nguyen, Hieu T.; Birdwell, L. Dillon; Yu, Janet; Ribeiro, Juliana M.; Weiss, Susan R.; Zamboni, Dario Simões; Roy, Craig R.; Shin, Sunny
Source: Infection and Immunity. Unidade: FMRP
Subjects: FEBRE Q, MACRÓFAGOS, SISTEMA IMUNE, MICROBIOLOGIA, CÉLULAS CULTIVADAS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRADLEY, William P. et al. Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages. Infection and Immunity, v. 84, n. 4, p. 998-1015, 2016Tradução . . Disponível em: https://doi.org/10.1128/iai.01536-15. Acesso em: 17 ago. 2024.
APA
Bradley, W. P., Boyer, M. A., Nguyen, H. T., Birdwell, L. D., Yu, J., Ribeiro, J. M., et al. (2016). Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages. Infection and Immunity, 84( 4), 998-1015. doi:10.1128/iai.01536-15
NLM
Bradley WP, Boyer MA, Nguyen HT, Birdwell LD, Yu J, Ribeiro JM, Weiss SR, Zamboni DS, Roy CR, Shin S. Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages [Internet]. Infection and Immunity. 2016 ; 84( 4): 998-1015.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1128/iai.01536-15
Vancouver
Bradley WP, Boyer MA, Nguyen HT, Birdwell LD, Yu J, Ribeiro JM, Weiss SR, Zamboni DS, Roy CR, Shin S. Primary role for toll-like receptor-driven tumor necrosis factor rather than cytosolic immune detection in restricting Coxiella burnetii phase II replication within mouse macrophages [Internet]. Infection and Immunity. 2016 ; 84( 4): 998-1015.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1128/iai.01536-15
Artigo de periodico
Electrophysiological characterization of the first Tityus serrulatus alpha-like toxin, Ts5: evidence of a pro-inflammatory toxin on macrophages (2015)
Pucca, Manuela B; Peigneur, Steve; Cologna, Camila T; Cerni, Felipe A; Zoccal, Karina F; Bordon, Karla de Castro Figueiredo; Faccioli, Lucia Helena; Tytgat, Jan; Arantes, Eliane Candiani
Source: Biochimie. Unidade: FCFRP
Subjects: TOXINAS, VENENOS DE ORIGEM ANIMAL, ESCORPIÕES, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PUCCA, Manuela B et al. Electrophysiological characterization of the first Tityus serrulatus alpha-like toxin, Ts5: evidence of a pro-inflammatory toxin on macrophages. Biochimie, v. 115, p. 8-16, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.biochi.2015.04.010. Acesso em: 17 ago. 2024.
APA
Pucca, M. B., Peigneur, S., Cologna, C. T., Cerni, F. A., Zoccal, K. F., Bordon, K. de C. F., et al. (2015). Electrophysiological characterization of the first Tityus serrulatus alpha-like toxin, Ts5: evidence of a pro-inflammatory toxin on macrophages. Biochimie, 115, 8-16. doi:10.1016/j.biochi.2015.04.010
NLM
Pucca MB, Peigneur S, Cologna CT, Cerni FA, Zoccal KF, Bordon K de CF, Faccioli LH, Tytgat J, Arantes EC. Electrophysiological characterization of the first Tityus serrulatus alpha-like toxin, Ts5: evidence of a pro-inflammatory toxin on macrophages [Internet]. Biochimie. 2015 ; 115 8-16.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.biochi.2015.04.010
Vancouver
Pucca MB, Peigneur S, Cologna CT, Cerni FA, Zoccal KF, Bordon K de CF, Faccioli LH, Tytgat J, Arantes EC. Electrophysiological characterization of the first Tityus serrulatus alpha-like toxin, Ts5: evidence of a pro-inflammatory toxin on macrophages [Internet]. Biochimie. 2015 ; 115 8-16.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.biochi.2015.04.010
Artigo de periodico
A dual role for P2X7 receptor during Porphyromonas gingivalis infection (2015)
Ramos-Junior, E. S; Morandini, A. C; Almeida-da-Silva, C. L. C; Franco, E. J; Potempa, J; Nguyen, K. A; Oliveira, A. C; Zamboni, Dario Simões; Ojcius, D. M; Scharfstein, J; Coutinho-Silva, R
Source: Journal of Dental Research. Unidade: FMRP
Subjects: CITOCINAS, MACRÓFAGOS, PERIODONTITE, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RAMOS-JUNIOR, E. S et al. A dual role for P2X7 receptor during Porphyromonas gingivalis infection. Journal of Dental Research, v. 94, n. 9, p. 1233-1242, 2015Tradução . . Disponível em: https://doi.org/10.1177/0022034515593465. Acesso em: 17 ago. 2024.
APA
Ramos-Junior, E. S., Morandini, A. C., Almeida-da-Silva, C. L. C., Franco, E. J., Potempa, J., Nguyen, K. A., et al. (2015). A dual role for P2X7 receptor during Porphyromonas gingivalis infection. Journal of Dental Research, 94( 9), 1233-1242. doi:10.1177/0022034515593465
NLM
Ramos-Junior ES, Morandini AC, Almeida-da-Silva CLC, Franco EJ, Potempa J, Nguyen KA, Oliveira AC, Zamboni DS, Ojcius DM, Scharfstein J, Coutinho-Silva R. A dual role for P2X7 receptor during Porphyromonas gingivalis infection [Internet]. Journal of Dental Research. 2015 ; 94( 9): 1233-1242.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1177/0022034515593465
Vancouver
Ramos-Junior ES, Morandini AC, Almeida-da-Silva CLC, Franco EJ, Potempa J, Nguyen KA, Oliveira AC, Zamboni DS, Ojcius DM, Scharfstein J, Coutinho-Silva R. A dual role for P2X7 receptor during Porphyromonas gingivalis infection [Internet]. Journal of Dental Research. 2015 ; 94( 9): 1233-1242.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1177/0022034515593465
Artigo de periodico
Mycobacterium tuberculosis expressing phospholipase C subverts PGE2 synthesis and induces necrosis in alveolar macrophages (2014)
Assis, Patrícia A; Espíndola, Milena S; Paula-Silva, Francisco Wanderley Garcia de; Rios, Wendy Martin; Pereira, Priscilla A. T; Leão, Sylvia C; Silva, Célio Lopes; Faccioli, Lucia Helena
Source: BMC Microbiology. Unidades: FORP, FMRP, FCFRP
Subjects: MYCOBACTERIUM TUBERCULOSIS, FOSFOLIPASES, PROSTAGLANDINAS, NECROSE, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ASSIS, Patrícia A et al. Mycobacterium tuberculosis expressing phospholipase C subverts PGE2 synthesis and induces necrosis in alveolar macrophages. BMC Microbiology, v. 14, 2014Tradução . . Disponível em: http://www.biomedcentral.com/1471-2180/14/128. Acesso em: 17 ago. 2024.
APA
Assis, P. A., Espíndola, M. S., Paula-Silva, F. W. G. de, Rios, W. M., Pereira, P. A. T., Leão, S. C., et al. (2014). Mycobacterium tuberculosis expressing phospholipase C subverts PGE2 synthesis and induces necrosis in alveolar macrophages. BMC Microbiology, 14. doi:10.1186/1471-2180-14-128
NLM
Assis PA, Espíndola MS, Paula-Silva FWG de, Rios WM, Pereira PAT, Leão SC, Silva CL, Faccioli LH. Mycobacterium tuberculosis expressing phospholipase C subverts PGE2 synthesis and induces necrosis in alveolar macrophages [Internet]. BMC Microbiology. 2014 ; 14[citado 2024 ago. 17 ] Available from: http://www.biomedcentral.com/1471-2180/14/128
Vancouver
Assis PA, Espíndola MS, Paula-Silva FWG de, Rios WM, Pereira PAT, Leão SC, Silva CL, Faccioli LH. Mycobacterium tuberculosis expressing phospholipase C subverts PGE2 synthesis and induces necrosis in alveolar macrophages [Internet]. BMC Microbiology. 2014 ; 14[citado 2024 ago. 17 ] Available from: http://www.biomedcentral.com/1471-2180/14/128
Artigo de periodico
Celecoxib improves host defense through prostaglandin inhibition during Histoplasma capsulatum infection (2013)
Pereira, Priscilla Aparecida Tartari; Trindade, Bruno Caetano; Secatto, Adriana; Nicolete, Roberto; Peres-Buzalaf, Camila; Ramos, Simone Gusmão; Sadikot, Ruxana; Bitencourt, Cláudia da Silva; Faccioli, Lucia Helena
Source: Mediators of Inflammation. Unidades: FMRP, FCFRP
Subjects: INIBIDORES DE ENZIMAS, HISTOPLASMA, INTERFERONS, LEUCOTRIENOS B, MACRÓFAGOS, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Priscilla Aparecida Tartari et al. Celecoxib improves host defense through prostaglandin inhibition during Histoplasma capsulatum infection. Mediators of Inflammation, v. 2013, 2013Tradução . . Disponível em: https://doi.org/10.1155/2013/950981. Acesso em: 17 ago. 2024.
APA
Pereira, P. A. T., Trindade, B. C., Secatto, A., Nicolete, R., Peres-Buzalaf, C., Ramos, S. G., et al. (2013). Celecoxib improves host defense through prostaglandin inhibition during Histoplasma capsulatum infection. Mediators of Inflammation, 2013. doi:10.1155/2013/950981
NLM
Pereira PAT, Trindade BC, Secatto A, Nicolete R, Peres-Buzalaf C, Ramos SG, Sadikot R, Bitencourt C da S, Faccioli LH. Celecoxib improves host defense through prostaglandin inhibition during Histoplasma capsulatum infection [Internet]. Mediators of Inflammation. 2013 ; 2013[citado 2024 ago. 17 ] Available from: https://doi.org/10.1155/2013/950981
Vancouver
Pereira PAT, Trindade BC, Secatto A, Nicolete R, Peres-Buzalaf C, Ramos SG, Sadikot R, Bitencourt C da S, Faccioli LH. Celecoxib improves host defense through prostaglandin inhibition during Histoplasma capsulatum infection [Internet]. Mediators of Inflammation. 2013 ; 2013[citado 2024 ago. 17 ] Available from: https://doi.org/10.1155/2013/950981
Artigo de periodico
Prior reproductive experience alters prolactin-induced macrophage responses in pregnant rats (2013)
Carvalho-Freitas, Maria Isabel Roth de; Anselmo-Franci, Janete Aparecida; Palermo-Neto, João; Felício, Luciano Freitas
Source: Journal of Reproductive Immunology. Unidades: FORP, FMVZ
Subjects: MACRÓFAGOS, PRENHEZ, PROLACTINA, RATOS, REPRODUÇÃO ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO-FREITAS, Maria Isabel Roth de et al. Prior reproductive experience alters prolactin-induced macrophage responses in pregnant rats. Journal of Reproductive Immunology, v. 99, n. 1-2, p. 54-61, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.jri.2013.03.005. Acesso em: 17 ago. 2024.
APA
Carvalho-Freitas, M. I. R. de, Anselmo-Franci, J. A., Palermo-Neto, J., & Felício, L. F. (2013). Prior reproductive experience alters prolactin-induced macrophage responses in pregnant rats. Journal of Reproductive Immunology, 99( 1-2), 54-61. doi:10.1016/j.jri.2013.03.005
NLM
Carvalho-Freitas MIR de, Anselmo-Franci JA, Palermo-Neto J, Felício LF. Prior reproductive experience alters prolactin-induced macrophage responses in pregnant rats [Internet]. Journal of Reproductive Immunology. 2013 ; 99( 1-2): 54-61.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jri.2013.03.005
Vancouver
Carvalho-Freitas MIR de, Anselmo-Franci JA, Palermo-Neto J, Felício LF. Prior reproductive experience alters prolactin-induced macrophage responses in pregnant rats [Internet]. Journal of Reproductive Immunology. 2013 ; 99( 1-2): 54-61.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jri.2013.03.005
Artigo de periodico
IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages (2012)
Jiang, Hui-Rong; Milovanovic, Marija; Allan, Debbie; Niedbala, Wanda; Besnard, Anne-Galle; Fukada, Sandra Y.; Alves Filho, José Carlos Farias; Togbe, Dieudonnée; Goodyear, Carl S.; Linington, Christopher; Xu, Damo; Lukic, Miodrag L.; Liew, Foo Y.
Source: European Journal of Immunology. Unidade: FMRP
Subjects: MACRÓFAGOS, DOENÇAS AUTOIMUNES (EXPERIMENTAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JIANG, Hui-Rong et al. IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages. European Journal of Immunology, v. 42, n. 7, p. 1804-1814, 2012Tradução . . Disponível em: https://doi.org/10.1002/eji.201141947. Acesso em: 17 ago. 2024.
APA
Jiang, H. -R., Milovanovic, M., Allan, D., Niedbala, W., Besnard, A. -G., Fukada, S. Y., et al. (2012). IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages. European Journal of Immunology, 42( 7), 1804-1814. doi:10.1002/eji.201141947
NLM
Jiang H-R, Milovanovic M, Allan D, Niedbala W, Besnard A-G, Fukada SY, Alves Filho JCF, Togbe D, Goodyear CS, Linington C, Xu D, Lukic ML, Liew FY. IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages [Internet]. European Journal of Immunology. 2012 ; 42( 7): 1804-1814.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1002/eji.201141947
Vancouver
Jiang H-R, Milovanovic M, Allan D, Niedbala W, Besnard A-G, Fukada SY, Alves Filho JCF, Togbe D, Goodyear CS, Linington C, Xu D, Lukic ML, Liew FY. IL-33 attenuates EAE by suppressing IL-17 and IFN-y production and inducing alternatively activated macrophages [Internet]. European Journal of Immunology. 2012 ; 42( 7): 1804-1814.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1002/eji.201141947

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