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Artigo de periodico
Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis (2018)
Gonçalves, Guilherme Lopes; Pessoa, Juliana Martins Costa; Thieme, Karina ; Lins, Bruna Bezerra; Souza, Maria Oliveira de
Source: Scientific reports. Unidade: ICB
Subjects: NEFROPATIAS, ALBUMINAS, RATOS, CÉLULAS EPITELIAIS, APOPTOSE, IMUNOFLUORESCÊNCIA EM ANIMAL, CITOMETRIA DE FLUXO, IMMUNOBLOTTING, PROTEÍNAS QUINASES, ATIVAÇÃO ENZIMÁTICA
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GONÇALVES, Guilherme Lopes et al. Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis. Scientific reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-36933-9. Acesso em: 18 nov. 2024.
APA
Gonçalves, G. L., Pessoa, J. M. C., Thieme, K., Lins, B. B., & Souza, M. O. de. (2018). Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis. Scientific reports, 8. doi:10.1038/s41598-018-36933-9
NLM
Gonçalves GL, Pessoa JMC, Thieme K, Lins BB, Souza MO de. Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis [Internet]. Scientific reports. 2018 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-018-36933-9
Vancouver
Gonçalves GL, Pessoa JMC, Thieme K, Lins BB, Souza MO de. Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis [Internet]. Scientific reports. 2018 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-018-36933-9
Artigo de periodico
Role of HIF-1α and CASPASE-3 in cystogenesis of odontogenic cysts and tumors (2018)
Costa, Natacha Malu Miranda; Siqueira, Adriane Sousa de; Ribeiro, André Luis Ribeiro; Kataoka, Maria Sueli da Silva; Jaeger, Ruy Gastaldoni ; Alves Junior, Sérgio de Melo; Smith, Andrew M.; Pinheiro, João de Jesus Viana
Source: Clinical Oral Investigations. Unidade: ICB
Subjects: TUMORES ODONTOGÊNICOS, CISTO ODONTOGÊNICO, CISTOS, ANÓXIA, AMELOBLASTOMA, APOPTOSE
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COSTA, Natacha Malu Miranda et al. Role of HIF-1α and CASPASE-3 in cystogenesis of odontogenic cysts and tumors. Clinical Oral Investigations, v. 22, n. 1, p. 141-149, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00784-017-2090-6. Acesso em: 18 nov. 2024.
APA
Costa, N. M. M., Siqueira, A. S. de, Ribeiro, A. L. R., Kataoka, M. S. da S., Jaeger, R. G., Alves Junior, S. de M., et al. (2018). Role of HIF-1α and CASPASE-3 in cystogenesis of odontogenic cysts and tumors. Clinical Oral Investigations, 22( 1), 141-149. doi:10.1007/s00784-017-2090-6
NLM
Costa NMM, Siqueira AS de, Ribeiro ALR, Kataoka MS da S, Jaeger RG, Alves Junior S de M, Smith AM, Pinheiro J de JV. Role of HIF-1α and CASPASE-3 in cystogenesis of odontogenic cysts and tumors [Internet]. Clinical Oral Investigations. 2018 ; 22( 1): 141-149.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00784-017-2090-6
Vancouver
Costa NMM, Siqueira AS de, Ribeiro ALR, Kataoka MS da S, Jaeger RG, Alves Junior S de M, Smith AM, Pinheiro J de JV. Role of HIF-1α and CASPASE-3 in cystogenesis of odontogenic cysts and tumors [Internet]. Clinical Oral Investigations. 2018 ; 22( 1): 141-149.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00784-017-2090-6
Artigo de periodico
Carbon monoxide protects the kidney through the centralcircadian clock and CD39 (2018)
Correa-Costa, Matheus; Gallo, David; Csizmadia, Eva; Gomperts, Edward; Lieberum, Judih-Lisa; Hauser, Carl J.; Xingyue, Ji; Wang, Bingue; Robson, Simnon C.; Otterbien, Leo E.; Câmara, Niels Olsen Saraiva
Source: Proceedings National Academy of Sciences. Unidade: RUSP
Subjects: APOPTOSE, IMUNOLOGIA DE TRANSPLANTES, RITMOS CIRCADIANOS ANIMAL, IMUNIDADE, MONÓXIDO DE CARBONO, CAMUNDONGOS, INFLAMAÇÃO, RIM
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CORREA-COSTA, Matheus et al. Carbon monoxide protects the kidney through the centralcircadian clock and CD39. Proceedings National Academy of Sciences, v. 115, n. 10, p. 2302-2310, 2018Tradução . . Disponível em: https://doi.org/10.1073/pnas.1716747115. Acesso em: 18 nov. 2024.
APA
Correa-Costa, M., Gallo, D., Csizmadia, E., Gomperts, E., Lieberum, J. -L., Hauser, C. J., et al. (2018). Carbon monoxide protects the kidney through the centralcircadian clock and CD39. Proceedings National Academy of Sciences, 115( 10), 2302-2310. doi:10.1073/pnas.1716747115
NLM
Correa-Costa M, Gallo D, Csizmadia E, Gomperts E, Lieberum J-L, Hauser CJ, Xingyue J, Wang B, Robson SC, Otterbien LE, Câmara NOS. Carbon monoxide protects the kidney through the centralcircadian clock and CD39 [Internet]. Proceedings National Academy of Sciences. 2018 ; 115( 10): 2302-2310.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1073/pnas.1716747115
Vancouver
Correa-Costa M, Gallo D, Csizmadia E, Gomperts E, Lieberum J-L, Hauser CJ, Xingyue J, Wang B, Robson SC, Otterbien LE, Câmara NOS. Carbon monoxide protects the kidney through the centralcircadian clock and CD39 [Internet]. Proceedings National Academy of Sciences. 2018 ; 115( 10): 2302-2310.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1073/pnas.1716747115
Artigo de periodico
Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms (2018)
Machado Neto, João Agostinho ; Fenerich, Bruna Alves; Ribeiro, Renata Scopim; Eide, Christopher A; Silva, Juan Luiz Coelho da; Dechandt, Carlos Roberto Porto; Fernandes, Jaqueline Cristina; Alves, Ana Paula Nunes Rodrigues; Scheucher, Priscila Santos; Simões, Belinda Pinto; Alberici, Luciane Carla; Pontes, Lorena Lôbo de Figueiredo; Tognon, Cristina E; Druker, Brian J; Rego, Eduardo Magalhães; Traina, Fabíola
Source: Cell Death & Disease. Unidades: ICB, FMRP, FCFRP, FM
Subjects: METFORMINA, PROLIFERAÇÃO CELULAR, APOPTOSE, MUTAÇÃO GENÉTICA, LEUCEMIA
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MACHADO NETO, João Agostinho et al. Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms. Cell Death & Disease, v. 9, n. 3, p. 16 , 2018Tradução . . Disponível em: https://doi.org/10.1038/s41419-017-0256-4. Acesso em: 18 nov. 2024.
APA
Machado Neto, J. A., Fenerich, B. A., Ribeiro, R. S., Eide, C. A., Silva, J. L. C. da, Dechandt, C. R. P., et al. (2018). Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms. Cell Death & Disease, 9( 3), 16 . doi:10.1038/s41419-017-0256-4
NLM
Machado Neto JA, Fenerich BA, Ribeiro RS, Eide CA, Silva JLC da, Dechandt CRP, Fernandes JC, Alves APNR, Scheucher PS, Simões BP, Alberici LC, Pontes LL de F, Tognon CE, Druker BJ, Rego EM, Traina F. Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms [Internet]. Cell Death & Disease. 2018 ; 9( 3): 16 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41419-017-0256-4
Vancouver
Machado Neto JA, Fenerich BA, Ribeiro RS, Eide CA, Silva JLC da, Dechandt CRP, Fernandes JC, Alves APNR, Scheucher PS, Simões BP, Alberici LC, Pontes LL de F, Tognon CE, Druker BJ, Rego EM, Traina F. Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms [Internet]. Cell Death & Disease. 2018 ; 9( 3): 16 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41419-017-0256-4
Artigo de periodico
Effect of variations in dietary Pi intake on intestinal Pi transporters (NaPi-IIb, PiT-1, and PiT-2) and phosphate-regulating factors (PTH, FGF-23, and MEPE) (2018)
Aniteli, Tatiana Martins; Siqueira, Flávia Ramos de; Reis, Luciene Machado dos ; Dominguez, Wagner Vasques; Oliveira, Elizabeth Maria Costa de; Castelucci, Patricia ; Moysés, Rosa Maria Affonso ; Jorgetti, Vanda
Source: Pflügers Archiv - European journal of physiology. Unidade: ICB
Subjects: APOPTOSE, RATOS WISTAR, UREMIA, FOSFATOS, SÓDIO, INTESTINOS, MATRIZ EXTRACELULAR
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ANITELI, Tatiana Martins et al. Effect of variations in dietary Pi intake on intestinal Pi transporters (NaPi-IIb, PiT-1, and PiT-2) and phosphate-regulating factors (PTH, FGF-23, and MEPE). Pflügers Archiv - European journal of physiology, v. 470, n. 4, p. 623-632, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00424-018-2111-6. Acesso em: 18 nov. 2024.
APA
Aniteli, T. M., Siqueira, F. R. de, Reis, L. M. dos, Dominguez, W. V., Oliveira, E. M. C. de, Castelucci, P., et al. (2018). Effect of variations in dietary Pi intake on intestinal Pi transporters (NaPi-IIb, PiT-1, and PiT-2) and phosphate-regulating factors (PTH, FGF-23, and MEPE). Pflügers Archiv - European journal of physiology, 470( 4), 623-632. doi:10.1007/s00424-018-2111-6
NLM
Aniteli TM, Siqueira FR de, Reis LM dos, Dominguez WV, Oliveira EMC de, Castelucci P, Moysés RMA, Jorgetti V. Effect of variations in dietary Pi intake on intestinal Pi transporters (NaPi-IIb, PiT-1, and PiT-2) and phosphate-regulating factors (PTH, FGF-23, and MEPE) [Internet]. Pflügers Archiv - European journal of physiology. 2018 ; 470( 4): 623-632.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00424-018-2111-6
Vancouver
Aniteli TM, Siqueira FR de, Reis LM dos, Dominguez WV, Oliveira EMC de, Castelucci P, Moysés RMA, Jorgetti V. Effect of variations in dietary Pi intake on intestinal Pi transporters (NaPi-IIb, PiT-1, and PiT-2) and phosphate-regulating factors (PTH, FGF-23, and MEPE) [Internet]. Pflügers Archiv - European journal of physiology. 2018 ; 470( 4): 623-632.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00424-018-2111-6
Artigo de periodico
"Role of the macrophage migration inhibitory factor (MIF) in the survival of first trimester human placenta uder induced stress conditions" (2018)
Letta, Francesca; Ferro, Eloisa Amália Vieira; Benincasa, Linda; Maioli, Emanuela; Paulesu, Luana; Bevilacqua, Estela Maris Andrade Forell
Source: Scientific Reports. Unidade: ICB
Subjects: TROFOBLASTO, HOMEOSTASE, MACRÓFAGOS, ESTRESSE, APOPTOSE, ATIVAÇÃO ENZIMÁTICA, PLACENTA
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LETTA, Francesca et al. "Role of the macrophage migration inhibitory factor (MIF) in the survival of first trimester human placenta uder induced stress conditions". Scientific Reports, v. 8, p. 1-10, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-29797-6. Acesso em: 18 nov. 2024.
APA
Letta, F., Ferro, E. A. V., Benincasa, L., Maioli, E., Paulesu, L., & Bevilacqua, E. M. A. F. (2018). "Role of the macrophage migration inhibitory factor (MIF) in the survival of first trimester human placenta uder induced stress conditions". Scientific Reports, 8, 1-10. doi:10.1038/s41598-018-29797-6
NLM
Letta F, Ferro EAV, Benincasa L, Maioli E, Paulesu L, Bevilacqua EMAF. "Role of the macrophage migration inhibitory factor (MIF) in the survival of first trimester human placenta uder induced stress conditions" [Internet]. Scientific Reports. 2018 ; 8 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-018-29797-6
Vancouver
Letta F, Ferro EAV, Benincasa L, Maioli E, Paulesu L, Bevilacqua EMAF. "Role of the macrophage migration inhibitory factor (MIF) in the survival of first trimester human placenta uder induced stress conditions" [Internet]. Scientific Reports. 2018 ; 8 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-018-29797-6
Artigo de periodico
FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes (2018)
Silva, Paula Maciel da; Caldeira, Izabela Daniel Sardinha; Santos, Icaro de Assis; Nishiyama, Ana Claudia Oliveira Carreira; Siqueira, Flávia Ramos de; Antonioli, Eliane; Goldberg, Anna Carla Renata Krepel; Belizário, José Ernesto; Garay-Malpartida, Humberto Miguel
Source: BMC Cancer. Unidades: ICB, EACH
Subjects: APOPTOSE, CITOCINAS, NEOPLASIAS PROSTÁTICAS, CÉLULAS CULTIVADAS DE TUMOR
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ABNT
SILVA, Paula Maciel da et al. FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes. BMC Cancer, v. 18, n. 1, p. 14, 2018Tradução . . Disponível em: https://doi.org/10.1186/s12885-017-3950-9. Acesso em: 18 nov. 2024.
APA
Silva, P. M. da, Caldeira, I. D. S., Santos, I. de A., Nishiyama, A. C. O. C., Siqueira, F. R. de, Antonioli, E., et al. (2018). FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes. BMC Cancer, 18( 1), 14. doi:10.1186/s12885-017-3950-9
NLM
Silva PM da, Caldeira IDS, Santos I de A, Nishiyama ACOC, Siqueira FR de, Antonioli E, Goldberg ACRK, Belizário JE, Garay-Malpartida HM. FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes [Internet]. BMC Cancer. 2018 ; 18( 1): 14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12885-017-3950-9
Vancouver
Silva PM da, Caldeira IDS, Santos I de A, Nishiyama ACOC, Siqueira FR de, Antonioli E, Goldberg ACRK, Belizário JE, Garay-Malpartida HM. FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes [Internet]. BMC Cancer. 2018 ; 18( 1): 14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12885-017-3950-9
Artigo de periodico
Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity (2018)
Cardoso, Vanessa Gerolde; Gonçalves, Guilherme Lopes; Pessoa, Juliana Martins da Costa; Thieme, Karina ; Lins, Bruna Bezerra; Casare, Fernando Augusto Malavazzi; Ponte, Mariana Charleaux de; Câmara, Niels Olsen Saraiva ; Souza, Maria Oliveira de
Source: BMC Nephrology. Unidade: ICB
Subjects: ANGIOTENSINA II, APOPTOSE, CITOMETRIA DE FLUXO, IMUNOFLUORESCÊNCIA EM ANIMAL, IMMUNOBLOTTING, RATOS WISTAR, CAMUNDONGOS
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ABNT
CARDOSO, Vanessa Gerolde et al. Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity. BMC Nephrology, v. 19, n. 1, p. 12 , 2018Tradução . . Disponível em: https://doi.org/10.1186/s12882-018-0968-4. Acesso em: 18 nov. 2024.
APA
Cardoso, V. G., Gonçalves, G. L., Pessoa, J. M. da C., Thieme, K., Lins, B. B., Casare, F. A. M., et al. (2018). Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity. BMC Nephrology, 19( 1), 12 . doi:10.1186/s12882-018-0968-4
NLM
Cardoso VG, Gonçalves GL, Pessoa JM da C, Thieme K, Lins BB, Casare FAM, Ponte MC de, Câmara NOS, Souza MO de. Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity [Internet]. BMC Nephrology. 2018 ; 19( 1): 12 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12882-018-0968-4
Vancouver
Cardoso VG, Gonçalves GL, Pessoa JM da C, Thieme K, Lins BB, Casare FAM, Ponte MC de, Câmara NOS, Souza MO de. Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity [Internet]. BMC Nephrology. 2018 ; 19( 1): 12 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12882-018-0968-4
Artigo de periodico-carta/editorial
Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy (2017)
Chammas, Roger; Andrade, Luciana Nogueira de Sousa; Jancar, Sonia
Source: Nature reviews cancer. Unidades: FM, ICB
Subjects: APOPTOSE, NEOPLASIAS, ONCOGENES, CÉLULAS CULTIVADAS DE TUMOR
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CHAMMAS, Roger e ANDRADE, Luciana Nogueira de Sousa e JANCAR, Sonia. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/nrc.2017.15. Acesso em: 18 nov. 2024. , 2017
APA
Chammas, R., Andrade, L. N. de S., & Jancar, S. (2017). Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. doi:10.1038/nrc.2017.15
NLM
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/nrc.2017.15
Vancouver
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/nrc.2017.15
Artigo de periodico
Modulation of procaspase-7 self-activation by pest amino acid residues of the n-terminal prodomain and intersubunit linker (2017)
Alves, Juliano; Garay-Malpartida, Humberto Miguel ; Occhiucci, João Marcelo; Belizario, Jose Ernesto
Source: Biochemistry and Cell Biology. Unidades: EACH, ICB
Subjects: BIOLOGIA MOLECULAR, BIOLOGIA CELULAR, AMINOÁCIDOS, APOPTOSE
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ABNT
ALVES, Juliano et al. Modulation of procaspase-7 self-activation by pest amino acid residues of the n-terminal prodomain and intersubunit linker. Biochemistry and Cell Biology, v. 95, n. 6, p. 634-643, 2017Tradução . . Disponível em: https://doi.org/10.1139/bcb-2016-0220. Acesso em: 18 nov. 2024.
APA
Alves, J., Garay-Malpartida, H. M., Occhiucci, J. M., & Belizario, J. E. (2017). Modulation of procaspase-7 self-activation by pest amino acid residues of the n-terminal prodomain and intersubunit linker. Biochemistry and Cell Biology, 95( 6), 634-643. doi:10.1139/bcb-2016-0220
NLM
Alves J, Garay-Malpartida HM, Occhiucci JM, Belizario JE. Modulation of procaspase-7 self-activation by pest amino acid residues of the n-terminal prodomain and intersubunit linker [Internet]. Biochemistry and Cell Biology. 2017 ; 95( 6): 634-643.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/bcb-2016-0220
Vancouver
Alves J, Garay-Malpartida HM, Occhiucci JM, Belizario JE. Modulation of procaspase-7 self-activation by pest amino acid residues of the n-terminal prodomain and intersubunit linker [Internet]. Biochemistry and Cell Biology. 2017 ; 95( 6): 634-643.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1139/bcb-2016-0220
Artigo de periodico
Identification of long noncoding RNAs deregulated in papillary thyroid cancer and correlated with BRAFV600E mutation by bioinformatics integrative analysis (2017)
Goedert, Lucas; Plaça, Jessica Rodrigues; Fuziwara, Cesar Seigi; Machado, Maiaro Cabral Rosa; Plaça, Desirée Rodrigues; Almeida, Palloma Porto; Sanches, Talita Perez; Santos, Jair Figueredo dos; Corveloni, Amanda Cristina; Pereira, Illy Enne Gomes; Castro, Marcela Motta de; Kimura, Edna Teruko ; Silva Júnior, Wilson Araújo da; Espreáfico, Enilza Maria
Source: Scientific Reports. Unidades: ICB, FMRP
Subjects: CARCINOMA PAPILAR, NEOPLASIAS DA TIREOIDE, PROLIFERAÇÃO CELULAR, APOPTOSE, TRANSDUÇÃO DE SINAL CELULAR
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GOEDERT, Lucas et al. Identification of long noncoding RNAs deregulated in papillary thyroid cancer and correlated with BRAFV600E mutation by bioinformatics integrative analysis. Scientific Reports, v. 7, n. 1, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-01957-0. Acesso em: 18 nov. 2024.
APA
Goedert, L., Plaça, J. R., Fuziwara, C. S., Machado, M. C. R., Plaça, D. R., Almeida, P. P., et al. (2017). Identification of long noncoding RNAs deregulated in papillary thyroid cancer and correlated with BRAFV600E mutation by bioinformatics integrative analysis. Scientific Reports, 7( 1), 1-11. doi:10.1038/s41598-017-01957-0
NLM
Goedert L, Plaça JR, Fuziwara CS, Machado MCR, Plaça DR, Almeida PP, Sanches TP, Santos JF dos, Corveloni AC, Pereira IEG, Castro MM de, Kimura ET, Silva Júnior WA da, Espreáfico EM. Identification of long noncoding RNAs deregulated in papillary thyroid cancer and correlated with BRAFV600E mutation by bioinformatics integrative analysis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-017-01957-0
Vancouver
Goedert L, Plaça JR, Fuziwara CS, Machado MCR, Plaça DR, Almeida PP, Sanches TP, Santos JF dos, Corveloni AC, Pereira IEG, Castro MM de, Kimura ET, Silva Júnior WA da, Espreáfico EM. Identification of long noncoding RNAs deregulated in papillary thyroid cancer and correlated with BRAFV600E mutation by bioinformatics integrative analysis [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-017-01957-0
Artigo de periodico
Promising pharmacological profile of a Kunitz-type inhibitor in murine renal cell carcinoma model (2016)
Souza, Jean Gabriel de; Morais, Katia L. P.; Anglés-Cano, Eduardo; Boufleur, Pamela; Mello, Evandro Sobroza de; Maria, Durvanei Augusto; Origassa, Clarice Silvia Taemi; Zampolli, Hamilton de Campos; Câmara, Niels Olsen Saraiva ; Berra, Carolina Maria; Bosch, Rosemary Viola Bosch; Chudzinski-Tavassi, Ana Marisa
Source: OncoTarget. Unidade: ICB
Subjects: NEOPLASIAS RENAIS, NEOPLASIAS PULMONARES, ADENOCARCINOMA, AMBLYOMMA CAJENNENSE, CARRAPATOS, GLÂNDULAS SALIVARES, MEDICAMENTO, METÁSTASE NEOPLÁSICA, APOPTOSE, IMUNOLOGIA
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ABNT
SOUZA, Jean Gabriel de et al. Promising pharmacological profile of a Kunitz-type inhibitor in murine renal cell carcinoma model. OncoTarget, v. 7, n. 38, p. 62255-62266, 2016Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.11555. Acesso em: 18 nov. 2024.
APA
Souza, J. G. de, Morais, K. L. P., Anglés-Cano, E., Boufleur, P., Mello, E. S. de, Maria, D. A., et al. (2016). Promising pharmacological profile of a Kunitz-type inhibitor in murine renal cell carcinoma model. OncoTarget, 7( 38), 62255-62266. doi:10.18632/oncotarget.11555
NLM
Souza JG de, Morais KLP, Anglés-Cano E, Boufleur P, Mello ES de, Maria DA, Origassa CST, Zampolli H de C, Câmara NOS, Berra CM, Bosch RVB, Chudzinski-Tavassi AM. Promising pharmacological profile of a Kunitz-type inhibitor in murine renal cell carcinoma model [Internet]. OncoTarget. 2016 ; 7( 38): 62255-62266.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.11555
Vancouver
Souza JG de, Morais KLP, Anglés-Cano E, Boufleur P, Mello ES de, Maria DA, Origassa CST, Zampolli H de C, Câmara NOS, Berra CM, Bosch RVB, Chudzinski-Tavassi AM. Promising pharmacological profile of a Kunitz-type inhibitor in murine renal cell carcinoma model [Internet]. OncoTarget. 2016 ; 7( 38): 62255-62266.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.11555
Artigo de periodico
Treatment with human wharton's Jelly-derived Mesenchymal stem cells attenuates sepsis-induced kidney injury, liver Injury, and endothelial dysfunction (2016)
Cóndor, Jose M.; Rodrigues, Camila E.; Moreira, Roberto de Sousa; Canale, Daniele; Volpini, Rildo A.; Shimizu, Maria H. M.; Câmara, Niels Olsen Saraiva ; Noronha, Irene de L.; Andrade, Lucia
Source: Stem Cells Translational Medicine. Unidade: ICB
Subjects: APOPTOSE, ENDOTÉLIO, INSUFICIÊNCIA RENAL AGUDA, FISIOPATOLOGIA, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CÓNDOR, Jose M. et al. Treatment with human wharton's Jelly-derived Mesenchymal stem cells attenuates sepsis-induced kidney injury, liver Injury, and endothelial dysfunction. Stem Cells Translational Medicine, v. 5, n. 8, p. 1048-1057, 2016Tradução . . Disponível em: https://doi.org/10.5966/sctm.2015-0138. Acesso em: 18 nov. 2024.
APA
Cóndor, J. M., Rodrigues, C. E., Moreira, R. de S., Canale, D., Volpini, R. A., Shimizu, M. H. M., et al. (2016). Treatment with human wharton's Jelly-derived Mesenchymal stem cells attenuates sepsis-induced kidney injury, liver Injury, and endothelial dysfunction. Stem Cells Translational Medicine, 5( 8), 1048-1057. doi:10.5966/sctm.2015-0138
NLM
Cóndor JM, Rodrigues CE, Moreira R de S, Canale D, Volpini RA, Shimizu MHM, Câmara NOS, Noronha I de L, Andrade L. Treatment with human wharton's Jelly-derived Mesenchymal stem cells attenuates sepsis-induced kidney injury, liver Injury, and endothelial dysfunction [Internet]. Stem Cells Translational Medicine. 2016 ; 5( 8): 1048-1057.[citado 2024 nov. 18 ] Available from: https://doi.org/10.5966/sctm.2015-0138
Vancouver
Cóndor JM, Rodrigues CE, Moreira R de S, Canale D, Volpini RA, Shimizu MHM, Câmara NOS, Noronha I de L, Andrade L. Treatment with human wharton's Jelly-derived Mesenchymal stem cells attenuates sepsis-induced kidney injury, liver Injury, and endothelial dysfunction [Internet]. Stem Cells Translational Medicine. 2016 ; 5( 8): 1048-1057.[citado 2024 nov. 18 ] Available from: https://doi.org/10.5966/sctm.2015-0138
Artigo de periodico
BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity (2016)
Ferreira, Adilson Kleber; Pasqualoto, Kerly Fernanda Mesquita; Kruyt, Frank A. E; Berl, Fanny Palace; Azevedo, Ricardo Alexandre de; Turra, Kely Medeiros; Rodrigues, Cecília Pessoa; Ferreira, Ana Carolina Franco; Salomon, Maria Alejandra Clavijo; Sá Júnior, Paulo Luiz de; Farias, Camyla Fernandes; Figueiredo, Carlos Rogério; Tavares, Leoberto Costa ; Barbuto, José Alexandre Marzagão ; Jorge, Salomão Dória
Source: Toxicology and Applied Pharmacology. Unidades: FCF, ICB
Subjects: MELANOMA, METÁSTASE NEOPLÁSICA, APOPTOSE
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ABNT
FERREIRA, Adilson Kleber et al. BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity. Toxicology and Applied Pharmacology, v. 295, p. 56-67, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2016.02.008. Acesso em: 18 nov. 2024.
APA
Ferreira, A. K., Pasqualoto, K. F. M., Kruyt, F. A. E., Berl, F. P., Azevedo, R. A. de, Turra, K. M., et al. (2016). BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity. Toxicology and Applied Pharmacology, 295, 56-67. doi:10.1016/j.taap.2016.02.008
NLM
Ferreira AK, Pasqualoto KFM, Kruyt FAE, Berl FP, Azevedo RA de, Turra KM, Rodrigues CP, Ferreira ACF, Salomon MAC, Sá Júnior PL de, Farias CF, Figueiredo CR, Tavares LC, Barbuto JAM, Jorge SD. BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity [Internet]. Toxicology and Applied Pharmacology. 2016 ; 295 56-67.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.taap.2016.02.008
Vancouver
Ferreira AK, Pasqualoto KFM, Kruyt FAE, Berl FP, Azevedo RA de, Turra KM, Rodrigues CP, Ferreira ACF, Salomon MAC, Sá Júnior PL de, Farias CF, Figueiredo CR, Tavares LC, Barbuto JAM, Jorge SD. BFD-22 a new potential inhibitor of BRAF inhibits the metastasis of B16F10 melanoma cells and simultaneously increased the tumor immunogenicity [Internet]. Toxicology and Applied Pharmacology. 2016 ; 295 56-67.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.taap.2016.02.008
Artigo de periodico
Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy (2016)
Tamura, Rodrigo Esaki; Soares, Rafael Bento da Silva; Costanzi-Strauss, Eugenia ; Strauss, Bryan Eric
Source: Cancer Biology & Therapy. Unidade: ICB
Subjects: ADENOVÍRUS, APOPTOSE, CARCINOMA, PRÓSTATA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TAMURA, Rodrigo Esaki et al. Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy. Cancer Biology & Therapy, v. 17, n. 12, p. 1221-1230, 2016Tradução . . Disponível em: https://doi.org/10.1080/15384047.2016.1235655. Acesso em: 18 nov. 2024.
APA
Tamura, R. E., Soares, R. B. da S., Costanzi-Strauss, E., & Strauss, B. E. (2016). Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy. Cancer Biology & Therapy, 17( 12), 1221-1230. doi:10.1080/15384047.2016.1235655
NLM
Tamura RE, Soares RB da S, Costanzi-Strauss E, Strauss BE. Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy [Internet]. Cancer Biology & Therapy. 2016 ; 17( 12): 1221-1230.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/15384047.2016.1235655
Vancouver
Tamura RE, Soares RB da S, Costanzi-Strauss E, Strauss BE. Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy [Internet]. Cancer Biology & Therapy. 2016 ; 17( 12): 1221-1230.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/15384047.2016.1235655
Artigo de periodico
Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis (2015)
Harbort, C. J.; Soeiro-Pereira, Paulo Vitor; Bernuth, Horst von; Kaindl, Angela M.; Costa-Carvalho, Beatriz Tavares; Condino-Neto, Antonio; Reichenbach, Janine; Roesler, Joachim; Zychlinsky, Arturo; Amulic, Borko
Source: Blood. Unidade: ICB
Subjects: IMUNOLOGIA, APOPTOSE, NEUTRÓFILOS, CITOCINAS
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ABNT
HARBORT, C. J. et al. Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis. Blood, v. 126, n. 26, p. 2842-2851, 2015Tradução . . Disponível em: https://doi.org/10.1182/blood-2015-05-645424. Acesso em: 18 nov. 2024.
APA
Harbort, C. J., Soeiro-Pereira, P. V., Bernuth, H. von, Kaindl, A. M., Costa-Carvalho, B. T., Condino-Neto, A., et al. (2015). Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis. Blood, 126( 26), 2842-2851. doi:10.1182/blood-2015-05-645424
NLM
Harbort CJ, Soeiro-Pereira PV, Bernuth H von, Kaindl AM, Costa-Carvalho BT, Condino-Neto A, Reichenbach J, Roesler J, Zychlinsky A, Amulic B. Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis [Internet]. Blood. 2015 ; 126( 26): 2842-2851.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1182/blood-2015-05-645424
Vancouver
Harbort CJ, Soeiro-Pereira PV, Bernuth H von, Kaindl AM, Costa-Carvalho BT, Condino-Neto A, Reichenbach J, Roesler J, Zychlinsky A, Amulic B. Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis [Internet]. Blood. 2015 ; 126( 26): 2842-2851.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1182/blood-2015-05-645424
Artigo de periodico
Suppression of MAPK attenuates neuronal cell death induced by activated glia-conditioned medium in alpha-synuclein overexpressing SH-SY5Y cells (2015)
Yshii, Lidia Mitiko; Denadai-Souza, Alexandre; Vasconcelos, Andrea Rodrigues; Avellar, Maria Cristina Werneck de; Scavone, Cristoforo
Source: Journal of Neuroinflammation. Unidade: ICB
Subjects: FARMACOLOGIA, DOENÇA DE PARKINSON, APOPTOSE, DOENÇAS DEGENERATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
YSHII, Lidia Mitiko et al. Suppression of MAPK attenuates neuronal cell death induced by activated glia-conditioned medium in alpha-synuclein overexpressing SH-SY5Y cells. Journal of Neuroinflammation, v. 12, n. 193, p. 1-8, 2015Tradução . . Disponível em: https://doi.org/10.1186/s12974-015-0412-7. Acesso em: 18 nov. 2024.
APA
Yshii, L. M., Denadai-Souza, A., Vasconcelos, A. R., Avellar, M. C. W. de, & Scavone, C. (2015). Suppression of MAPK attenuates neuronal cell death induced by activated glia-conditioned medium in alpha-synuclein overexpressing SH-SY5Y cells. Journal of Neuroinflammation, 12( 193), 1-8. doi:10.1186/s12974-015-0412-7
NLM
Yshii LM, Denadai-Souza A, Vasconcelos AR, Avellar MCW de, Scavone C. Suppression of MAPK attenuates neuronal cell death induced by activated glia-conditioned medium in alpha-synuclein overexpressing SH-SY5Y cells [Internet]. Journal of Neuroinflammation. 2015 ; 12( 193): 1-8.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12974-015-0412-7
Vancouver
Yshii LM, Denadai-Souza A, Vasconcelos AR, Avellar MCW de, Scavone C. Suppression of MAPK attenuates neuronal cell death induced by activated glia-conditioned medium in alpha-synuclein overexpressing SH-SY5Y cells [Internet]. Journal of Neuroinflammation. 2015 ; 12( 193): 1-8.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12974-015-0412-7
Artigo de periodico
Therapeutic applications of TRAIL receptor agonists in cancer and beyond (2015)
Amarante-Mendes, João Gustavo Pessini ; Griffith, Thomas S.
Source: Pharmacology & Therapeutics. Unidade: ICB
Subjects: CITOCINAS, NEOPLASIAS, APOPTOSE
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ABNT
AMARANTE-MENDES, João Gustavo Pessini e GRIFFITH, Thomas S. Therapeutic applications of TRAIL receptor agonists in cancer and beyond. Pharmacology & Therapeutics, v. 155, p. 117-131, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.pharmthera.2015.09.001. Acesso em: 18 nov. 2024.
APA
Amarante-Mendes, J. G. P., & Griffith, T. S. (2015). Therapeutic applications of TRAIL receptor agonists in cancer and beyond. Pharmacology & Therapeutics, 155, 117-131. doi:10.1016/j.pharmthera.2015.09.001
NLM
Amarante-Mendes JGP, Griffith TS. Therapeutic applications of TRAIL receptor agonists in cancer and beyond [Internet]. Pharmacology & Therapeutics. 2015 ; 155 117-131.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.pharmthera.2015.09.001
Vancouver
Amarante-Mendes JGP, Griffith TS. Therapeutic applications of TRAIL receptor agonists in cancer and beyond [Internet]. Pharmacology & Therapeutics. 2015 ; 155 117-131.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.pharmthera.2015.09.001
Artigo de periodico
Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy (2013)
Faião-Flores, Fernanda; Coelho, Paulo Rogério Pinto; Arruda Neto, João Dias de Toledo ; Maria-Engler, Silvya Stuchi ; Tiago, Manoela; Capelozzi, Vera Luiza ; Giorgi, Ricardo Rodrigues; Maria, Durvanei Augusto
Source: Plos One. Unidades: IF, FCF, FM, IPEN
Subjects: APOPTOSE, CULTURA DE CÉLULAS, MELANOMA, CITOMETRIA DE FLUXO, REAÇÃO EM CADEIA POR POLIMERASE, ELETROFORESE EM GEL, IMUNOHISTOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAIÃO-FLORES, Fernanda et al. Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy. Plos One, v. 8, n. 3, p. 12 , 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0059639. Acesso em: 18 nov. 2024.
APA
Faião-Flores, F., Coelho, P. R. P., Arruda Neto, J. D. de T., Maria-Engler, S. S., Tiago, M., Capelozzi, V. L., et al. (2013). Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy. Plos One, 8( 3), 12 . doi:10.1371/journal.pone.0059639
NLM
Faião-Flores F, Coelho PRP, Arruda Neto JD de T, Maria-Engler SS, Tiago M, Capelozzi VL, Giorgi RR, Maria DA. Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy [Internet]. Plos One. 2013 ; 8( 3): 12 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0059639
Vancouver
Faião-Flores F, Coelho PRP, Arruda Neto JD de T, Maria-Engler SS, Tiago M, Capelozzi VL, Giorgi RR, Maria DA. Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy [Internet]. Plos One. 2013 ; 8( 3): 12 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pone.0059639
Artigo de periodico
HOX antisense lincRNA HOXA-AS2 is an apoptosis repressor in all trans retinoic acid treated NB4 promyelocytic leukemia cells (2013)
Zhao, Hang; Xueqing Zhang; Frazão, Josias Brito; Condino Neto, Antonio ; Newburger, Peter E.
Source: Journal of Cellular Biochemistry. Unidade: ICB
Subjects: IMUNOLOGIA, APOPTOSE, SOBREVIVÊNCIA CELULAR, NEUTRÓFILOS, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
ZHAO, Hang et al. HOX antisense lincRNA HOXA-AS2 is an apoptosis repressor in all trans retinoic acid treated NB4 promyelocytic leukemia cells. Journal of Cellular Biochemistry, v. 114, n. 10, p. 2375-2383, 2013Tradução . . Disponível em: https://doi.org/10.1002/jcb.24586. Acesso em: 18 nov. 2024.
APA
Zhao, H., Xueqing Zhang,, Frazão, J. B., Condino Neto, A., & Newburger, P. E. (2013). HOX antisense lincRNA HOXA-AS2 is an apoptosis repressor in all trans retinoic acid treated NB4 promyelocytic leukemia cells. Journal of Cellular Biochemistry, 114( 10), 2375-2383. doi:10.1002/jcb.24586
NLM
Zhao H, Xueqing Zhang, Frazão JB, Condino Neto A, Newburger PE. HOX antisense lincRNA HOXA-AS2 is an apoptosis repressor in all trans retinoic acid treated NB4 promyelocytic leukemia cells [Internet]. Journal of Cellular Biochemistry. 2013 ; 114( 10): 2375-2383.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/jcb.24586
Vancouver
Zhao H, Xueqing Zhang, Frazão JB, Condino Neto A, Newburger PE. HOX antisense lincRNA HOXA-AS2 is an apoptosis repressor in all trans retinoic acid treated NB4 promyelocytic leukemia cells [Internet]. Journal of Cellular Biochemistry. 2013 ; 114( 10): 2375-2383.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/jcb.24586

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