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Artigo de periodico
Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies (2015)
Lucena, Camila Ferraz; Roma, Leticia Prates; Graciano, Maria Fernanda Rodrigues; Veras, Katherine; Simões, Daniel; Curi, Rui; Carpinelli, Angelo Rafael
Fonte: Pancreas. Unidade: ICB
Assuntos: FISIOLOGIA, ILHOTAS DE LANGERHANS, SUPLEMENTOS ALIMENTARES PARA ANIMAIS, ÓLEOS DE PEIUXE
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ABNT
LUCENA, Camila Ferraz et al. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, v. 44, n. 2, p. 287-295, 2015Tradução . . Disponível em: https://doi.org/10.1097/MPA.0000000000000249. Acesso em: 19 ago. 2024.
APA
Lucena, C. F., Roma, L. P., Graciano, M. F. R., Veras, K., Simões, D., Curi, R., & Carpinelli, A. R. (2015). Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, 44( 2), 287-295. doi:10.1097/MPA.0000000000000249
NLM
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Vancouver
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Artigo de periodico
Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids (2015)
Fiamoncini, Jarlei ; Lima, Thais Martins de; Hirabara, Sandro Massao; Ecker, Josef; Gorjão, Renata; Romanatto, Talita; Elolimy, Ahmed; Worsch, Stefanie; Laumen, Helmut; Bader, Bernhard; Daniel, Hannelore; Curi, Rui
Fonte: Molecular Nutrition & Food Research. Unidade: ICB
Assuntos: FISIOLOGIA, OLEO DE PEIXE, CAMUNDONGOS, PEROXISSOMOS, OXIDAÇÃO, METABOLISMO ANIMAL
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ABNT
FIAMONCINI, Jarlei et al. Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids. Molecular Nutrition & Food Research, v. 59, n. 8, p. 1573-1583, 2015Tradução . . Disponível em: https://doi.org/10.1002/mnfr.201400743. Acesso em: 19 ago. 2024.
APA
Fiamoncini, J., Lima, T. M. de, Hirabara, S. M., Ecker, J., Gorjão, R., Romanatto, T., et al. (2015). Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids. Molecular Nutrition & Food Research, 59( 8), 1573-1583. doi:10.1002/mnfr.201400743
NLM
Fiamoncini J, Lima TM de, Hirabara SM, Ecker J, Gorjão R, Romanatto T, Elolimy A, Worsch S, Laumen H, Bader B, Daniel H, Curi R. Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids [Internet]. Molecular Nutrition & Food Research. 2015 ; 59( 8): 1573-1583.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1002/mnfr.201400743
Vancouver
Fiamoncini J, Lima TM de, Hirabara SM, Ecker J, Gorjão R, Romanatto T, Elolimy A, Worsch S, Laumen H, Bader B, Daniel H, Curi R. Medium-chain dicarboxylic acylcarnitines as markers of n-3 PUFA-induced peroxisomal oxidation of fatty acids [Internet]. Molecular Nutrition & Food Research. 2015 ; 59( 8): 1573-1583.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1002/mnfr.201400743
Artigo de periodico
DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice (2014)
Amaral, Catia Lira do; Crisma, Amanda Rabello; Masi, Laureane Nunes; Martins, Amanda Roque; Hirabara, Sandro Massao; Curi, Rui
Fonte: Journal of Nutrigenetics and Nutrigenomics. Unidade: ICB
Assuntos: CAMUNDONGOS, MÚSCULO ESQUELÉTICO, ÓLEOS DE PEIXE, TRANSCRIÇÃO GÊNICA
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ABNT
AMARAL, Catia Lira do et al. DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice. Journal of Nutrigenetics and Nutrigenomics, v. 7, n. 4-6, p. 314-326, 2014Tradução . . Disponível em: https://doi.org/10.1159/000381777. Acesso em: 19 ago. 2024.
APA
Amaral, C. L. do, Crisma, A. R., Masi, L. N., Martins, A. R., Hirabara, S. M., & Curi, R. (2014). DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice. Journal of Nutrigenetics and Nutrigenomics, 7( 4-6), 314-326. doi:10.1159/000381777
NLM
Amaral CL do, Crisma AR, Masi LN, Martins AR, Hirabara SM, Curi R. DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice [Internet]. Journal of Nutrigenetics and Nutrigenomics. 2014 ; 7( 4-6): 314-326.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1159/000381777
Vancouver
Amaral CL do, Crisma AR, Masi LN, Martins AR, Hirabara SM, Curi R. DNA methylation changes induced by a high-fat diet and fish oil supplementation in the skeletal muscle of mice [Internet]. Journal of Nutrigenetics and Nutrigenomics. 2014 ; 7( 4-6): 314-326.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1159/000381777
Artigo de periodico
The central role of the gut microbiota in chronic inflammatory diseases (2014)
Ferreira, Caroline Marcantonio; Vieira, Angélica Thomaz; Vinolo, Marco Aurelio Ramirez ; Oliveira, Fernando A. ; Martins, Flaviano dos Santos ; Curi, Rui
Fonte: Journal of Immunology Research. Unidade: ICB
Assuntos: FISIOLOGIA, IMUNOLOGIA CELULAR, SISTEMA IMUNE, CAMUNDONGOS, DOENÇAS IMUNOLÓGICAS, INFLAMAÇÃO
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ABNT
FERREIRA, Caroline Marcantonio et al. The central role of the gut microbiota in chronic inflammatory diseases. Journal of Immunology Research, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/689492. Acesso em: 19 ago. 2024.
APA
Ferreira, C. M., Vieira, A. T., Vinolo, M. A. R., Oliveira, F. A., Martins, F. dos S., & Curi, R. (2014). The central role of the gut microbiota in chronic inflammatory diseases. Journal of Immunology Research, 2014. doi:10.1155/2014/689492
NLM
Ferreira CM, Vieira AT, Vinolo MAR, Oliveira FA, Martins F dos S, Curi R. The central role of the gut microbiota in chronic inflammatory diseases [Internet]. Journal of Immunology Research. 2014 ; 2014[citado 2024 ago. 19 ] Available from: https://doi.org/10.1155/2014/689492
Vancouver
Ferreira CM, Vieira AT, Vinolo MAR, Oliveira FA, Martins F dos S, Curi R. The central role of the gut microbiota in chronic inflammatory diseases [Internet]. Journal of Immunology Research. 2014 ; 2014[citado 2024 ago. 19 ] Available from: https://doi.org/10.1155/2014/689492
Artigo de periodico
DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil (2014)
Amaral, Cátia Lira do; Milagro, Fermín I.; Curi, Rui; Martínez, J. Alfredo
Fonte: BioMed Research International. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
AMARAL, Cátia Lira do et al. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil. BioMed Research International, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/675021. Acesso em: 19 ago. 2024.
APA
Amaral, C. L. do, Milagro, F. I., Curi, R., & Martínez, J. A. (2014). DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil. BioMed Research International, 2014, 1-10. doi:10.1155/2014/675021
NLM
Amaral CL do, Milagro FI, Curi R, Martínez JA. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil [Internet]. BioMed Research International. 2014 ; 2014 1-10.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1155/2014/675021
Vancouver
Amaral CL do, Milagro FI, Curi R, Martínez JA. DNA methylation pattern in overweight women under an energy-restricted diet supplemented with fish oil [Internet]. BioMed Research International. 2014 ; 2014 1-10.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1155/2014/675021
Artigo de periodico
Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice (2014)
Romanatto, Talita; Fiamoncini, Jarlei ; Wang, Bin; Curi, Rui; Kang, Jing X.
Fonte: Biochimica et Biophysica Acta: Molecular Basis Diseases. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ROMANATTO, Talita et al. Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice. Biochimica et Biophysica Acta: Molecular Basis Diseases, v. 1842, n. 2, p. 186-191, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2013.10.017. Acesso em: 19 ago. 2024.
APA
Romanatto, T., Fiamoncini, J., Wang, B., Curi, R., & Kang, J. X. (2014). Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice. Biochimica et Biophysica Acta: Molecular Basis Diseases, 1842( 2), 186-191. doi:10.1016/j.bbadis.2013.10.017
NLM
Romanatto T, Fiamoncini J, Wang B, Curi R, Kang JX. Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice [Internet]. Biochimica et Biophysica Acta: Molecular Basis Diseases. 2014 ; 1842( 2): 186-191.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.bbadis.2013.10.017
Vancouver
Romanatto T, Fiamoncini J, Wang B, Curi R, Kang JX. Elevated tissue omega-3 fatty acid status prevents age-related glucose intolerance in fat-1 transgenic mice [Internet]. Biochimica et Biophysica Acta: Molecular Basis Diseases. 2014 ; 1842( 2): 186-191.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.bbadis.2013.10.017
Artigo de periodico
Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion (2013)
Graciano, Maria Fernanda Rodrigues ; Valle, Maíra Mello Rezende ; Curi, Rui ; Carpinelli, Angelo Rafael
Fonte: Islets. Unidade: ICB
Assuntos: FISIOLOGIA, ILHOTAS DE LANGERHANS, INSULINA, SUPERÓXIDO DISMUTASE
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ABNT
GRACIANO, Maria Fernanda Rodrigues et al. Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion. Islets, v. 5 n. 4, p. 139-148, 2013Tradução . . Disponível em: https://doi.org/10.4161/isl.25459. Acesso em: 19 ago. 2024.
APA
Graciano, M. F. R., Valle, M. M. R., Curi, R., & Carpinelli, A. R. (2013). Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion. Islets, 5 n. 4, 139-148. doi:10.4161/isl.25459
NLM
Graciano MFR, Valle MMR, Curi R, Carpinelli AR. Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion [Internet]. Islets. 2013 ; 5 n. 4 139-148.[citado 2024 ago. 19 ] Available from: https://doi.org/10.4161/isl.25459
Vancouver
Graciano MFR, Valle MMR, Curi R, Carpinelli AR. Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion [Internet]. Islets. 2013 ; 5 n. 4 139-148.[citado 2024 ago. 19 ] Available from: https://doi.org/10.4161/isl.25459
Artigo de periodico
Effects of melatonin on DNA damage induced by cyclophosphamide in rats (2013)
Ferreira, S. G.; Peliciari-Garcia, R. A.; Takahashi-Hyodo, S. A.; Rodrigues, Alice Cristina ; Amaral, F. G.; Berra, Carolina Maria; Bordin, Silvana ; Curi, Rui; Cipolla-Neto, Jose
Fonte: Brazilian Journal of Medical and Biological Research. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FERREIRA, S. G. et al. Effects of melatonin on DNA damage induced by cyclophosphamide in rats. Brazilian Journal of Medical and Biological Research, v. 46, n. 3, p. 278-286, 2013Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20122230. Acesso em: 19 ago. 2024.
APA
Ferreira, S. G., Peliciari-Garcia, R. A., Takahashi-Hyodo, S. A., Rodrigues, A. C., Amaral, F. G., Berra, C. M., et al. (2013). Effects of melatonin on DNA damage induced by cyclophosphamide in rats. Brazilian Journal of Medical and Biological Research, 46( 3), 278-286. doi:10.1590/1414-431X20122230
NLM
Ferreira SG, Peliciari-Garcia RA, Takahashi-Hyodo SA, Rodrigues AC, Amaral FG, Berra CM, Bordin S, Curi R, Cipolla-Neto J. Effects of melatonin on DNA damage induced by cyclophosphamide in rats [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 3): 278-286.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1590/1414-431X20122230
Vancouver
Ferreira SG, Peliciari-Garcia RA, Takahashi-Hyodo SA, Rodrigues AC, Amaral FG, Berra CM, Bordin S, Curi R, Cipolla-Neto J. Effects of melatonin on DNA damage induced by cyclophosphamide in rats [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 3): 278-286.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1590/1414-431X20122230
Artigo de periodico
Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells (2013)
Lima, Larissa de Sá; Kawamoto, Elisa Mitiko ; Munhoz, Carolina Demarchi ; Kinoshita, Paula Fernanda; Orellana, Ana Maria Marques; Curi, Rui; Rossoni, Luciana Venturini ; Avellar, M. C. W.; Scavone, Cristoforo
Fonte: Neuropharmacology. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA
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ABNT
LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 19 ago. 2024.
APA
Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
NLM
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Vancouver
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Artigo de periodico
Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration (2013)
Marçal, Anderson Carlos; Leonelli, Mauro; Fiamoncini, Jarlei ; Deschamps, Francisco C.; Rodrigues, Maria A. M.; Curi, Rui; Carpinelli, Angelo Rafael ; Britto, Luiz Roberto Giorgetti de ; Carvalho, Carla Roberta de Oliveira
Fonte: Cell Biochemistry and Function. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MARÇAL, Anderson Carlos et al. Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration. Cell Biochemistry and Function, v. 31, n. 1, p. 65-74, 2013Tradução . . Disponível em: https://doi.org/10.1002/cbf.2861. Acesso em: 19 ago. 2024.
APA
Marçal, A. C., Leonelli, M., Fiamoncini, J., Deschamps, F. C., Rodrigues, M. A. M., Curi, R., et al. (2013). Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration. Cell Biochemistry and Function, 31( 1), 65-74. doi:10.1002/cbf.2861
NLM
Marçal AC, Leonelli M, Fiamoncini J, Deschamps FC, Rodrigues MAM, Curi R, Carpinelli AR, Britto LRG de, Carvalho CR de O. Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration [Internet]. Cell Biochemistry and Function. 2013 ; 31( 1): 65-74.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1002/cbf.2861
Vancouver
Marçal AC, Leonelli M, Fiamoncini J, Deschamps FC, Rodrigues MAM, Curi R, Carpinelli AR, Britto LRG de, Carvalho CR de O. Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration [Internet]. Cell Biochemistry and Function. 2013 ; 31( 1): 65-74.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1002/cbf.2861
Artigo de periodico
Cessation of physical exercise changes metabolism and modifies the adipocyte cellularity of the periepididymal white adipose tissue in rats (2013)
Sertie, Rogerio A. L.; Andreotti, Sandra; Proença, André Ricardo Gomes; Campaña, Amanda Baroni; Lima-Salgado, Thais M.; Batista Jr., Miguél L.; Seelaender, Marilia Cerqueira Leite; Curi, Rui; Oliveira, Ariclecio C.; Lima, Fabio Bessa
Fonte: Journal of Applied Physiology. Unidade: ICB
Assuntos: FISIOLOGIA, HISTOLOGIA
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ABNT
SERTIE, Rogerio A. L. et al. Cessation of physical exercise changes metabolism and modifies the adipocyte cellularity of the periepididymal white adipose tissue in rats. Journal of Applied Physiology, v. 115, p. 394-402, 2013Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.01272.2012. Acesso em: 19 ago. 2024.
APA
Sertie, R. A. L., Andreotti, S., Proença, A. R. G., Campaña, A. B., Lima-Salgado, T. M., Batista Jr., M. L., et al. (2013). Cessation of physical exercise changes metabolism and modifies the adipocyte cellularity of the periepididymal white adipose tissue in rats. Journal of Applied Physiology, 115, 394-402. doi:10.1152/japplphysiol.01272.2012
NLM
Sertie RAL, Andreotti S, Proença ARG, Campaña AB, Lima-Salgado TM, Batista Jr. ML, Seelaender MCL, Curi R, Oliveira AC, Lima FB. Cessation of physical exercise changes metabolism and modifies the adipocyte cellularity of the periepididymal white adipose tissue in rats [Internet]. Journal of Applied Physiology. 2013 ; 115 394-402.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1152/japplphysiol.01272.2012
Vancouver
Sertie RAL, Andreotti S, Proença ARG, Campaña AB, Lima-Salgado TM, Batista Jr. ML, Seelaender MCL, Curi R, Oliveira AC, Lima FB. Cessation of physical exercise changes metabolism and modifies the adipocyte cellularity of the periepididymal white adipose tissue in rats [Internet]. Journal of Applied Physiology. 2013 ; 115 394-402.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1152/japplphysiol.01272.2012
Artigo de periodico
Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation (2013)
Hatanaka, Elaine; Dermargos, Alexandre; Hirata, Aparecida Emiko; Vinolo, Marco Aurélio Ramirez; Carpinelli, Angelo Rafael ; Newsholme, Philip; Armelin, Hugo Aguirre ; Curi, Rui
Fonte: PLOS ONE. Unidades: ICB, IQ
Assunto: FISIOLOGIA
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ABNT
HATANAKA, Elaine et al. Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation. PLOS ONE, v. 8, p. 1-8, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0058626. Acesso em: 19 ago. 2024.
APA
Hatanaka, E., Dermargos, A., Hirata, A. E., Vinolo, M. A. R., Carpinelli, A. R., Newsholme, P., et al. (2013). Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation. PLOS ONE, 8, 1-8. doi:10.1371/journal.pone.0058626
NLM
Hatanaka E, Dermargos A, Hirata AE, Vinolo MAR, Carpinelli AR, Newsholme P, Armelin HA, Curi R. Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation [Internet]. PLOS ONE. 2013 ; 8 1-8.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1371/journal.pone.0058626
Vancouver
Hatanaka E, Dermargos A, Hirata AE, Vinolo MAR, Carpinelli AR, Newsholme P, Armelin HA, Curi R. Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation [Internet]. PLOS ONE. 2013 ; 8 1-8.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1371/journal.pone.0058626
Artigo de periodico
Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells (2013)
Costa, E. S.; Faiad, O. J.; Landgraf, R. G.; Ferreira, A. K.; Brigatte, P.; Curi, Rui; Cury, Y.
Fonte: Toxicon. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
COSTA, E. S. et al. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells. Toxicon, v. 74, p. 167-178, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.toxicon.2013.08.057. Acesso em: 19 ago. 2024.
APA
Costa, E. S., Faiad, O. J., Landgraf, R. G., Ferreira, A. K., Brigatte, P., Curi, R., & Cury, Y. (2013). Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells. Toxicon, 74, 167-178. doi:10.1016/j.toxicon.2013.08.057
NLM
Costa ES, Faiad OJ, Landgraf RG, Ferreira AK, Brigatte P, Curi R, Cury Y. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells [Internet]. Toxicon. 2013 ; 74 167-178.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.toxicon.2013.08.057
Vancouver
Costa ES, Faiad OJ, Landgraf RG, Ferreira AK, Brigatte P, Curi R, Cury Y. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells [Internet]. Toxicon. 2013 ; 74 167-178.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.toxicon.2013.08.057
Artigo de periodico
Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats (2013)
Vitzel, Kaio Fernando; Bikopoulos, George; Hung, Steven; Curi, Rui; Ceddia, Rolando B.
Fonte: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
VITZEL, Kaio Fernando et al. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats. PLOS ONE, v. 8, n. 8, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0071944. Acesso em: 19 ago. 2024.
APA
Vitzel, K. F., Bikopoulos, G., Hung, S., Curi, R., & Ceddia, R. B. (2013). Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats. PLOS ONE, 8( 8), 1-10. doi:10.1371/journal.pone.0071944
NLM
Vitzel KF, Bikopoulos G, Hung S, Curi R, Ceddia RB. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats [Internet]. PLOS ONE. 2013 ; 8( 8): 1-10.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1371/journal.pone.0071944
Vancouver
Vitzel KF, Bikopoulos G, Hung S, Curi R, Ceddia RB. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats [Internet]. PLOS ONE. 2013 ; 8( 8): 1-10.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1371/journal.pone.0071944
Artigo de periodico
Palmitoleic acid (n-7) increases white adipocyte lipolysis and lipase content in a PPARα-dependent manner (2013)
Lopes, Andressa Bolsoni; Festuccia, William Tadeu Lara ; Farias, Talita da Silva Mendes de; Chimin, Patricia; Torres-Leal, Francisco Leonardo; Derogis, Priscilla Bento Matos Cruz; Andrade, Paula B. de; Miyamoto, Sayuri ; Lima, Fábio Bessa; Curi, Rui; Alonso-Vale, Maria Isabel Cardoso
Fonte: American Journal of Physiology Endocrinology and Metabolism. Unidades: ICB, IQ
Assuntos: LIPASE, ÁCIDOS GRAXOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Andressa Bolsoni et al. Palmitoleic acid (n-7) increases white adipocyte lipolysis and lipase content in a PPARα-dependent manner. American Journal of Physiology Endocrinology and Metabolism, v. 305, n. 9, p. E1093-E1102, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00082.2013. Acesso em: 19 ago. 2024.
APA
Lopes, A. B., Festuccia, W. T. L., Farias, T. da S. M. de, Chimin, P., Torres-Leal, F. L., Derogis, P. B. M. C., et al. (2013). Palmitoleic acid (n-7) increases white adipocyte lipolysis and lipase content in a PPARα-dependent manner. American Journal of Physiology Endocrinology and Metabolism, 305( 9), E1093-E1102. doi:10.1152/ajpendo.00082.2013
NLM
Lopes AB, Festuccia WTL, Farias T da SM de, Chimin P, Torres-Leal FL, Derogis PBMC, Andrade PB de, Miyamoto S, Lima FB, Curi R, Alonso-Vale MIC. Palmitoleic acid (n-7) increases white adipocyte lipolysis and lipase content in a PPARα-dependent manner [Internet]. American Journal of Physiology Endocrinology and Metabolism. 2013 ; 305( 9): E1093-E1102.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1152/ajpendo.00082.2013
Vancouver
Lopes AB, Festuccia WTL, Farias T da SM de, Chimin P, Torres-Leal FL, Derogis PBMC, Andrade PB de, Miyamoto S, Lima FB, Curi R, Alonso-Vale MIC. Palmitoleic acid (n-7) increases white adipocyte lipolysis and lipase content in a PPARα-dependent manner [Internet]. American Journal of Physiology Endocrinology and Metabolism. 2013 ; 305( 9): E1093-E1102.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1152/ajpendo.00082.2013
Artigo de periodico
Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets (2012)
Alves, E. S.; Haidar, A. A.; Quadros, C. D.; Carvalho, D. S.; Morgan, D.; Rocha, M. S.; Curi, Rui; Carpinelli, Angelo Rafael ; Hirata, A. E.
Fonte: Regulatory Peptides. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALVES, E. S. et al. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets. Regulatory Peptides, v. 175, n. 1-3, p. 1-6, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.regpep.2012.01.003. Acesso em: 19 ago. 2024.
APA
Alves, E. S., Haidar, A. A., Quadros, C. D., Carvalho, D. S., Morgan, D., Rocha, M. S., et al. (2012). Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets. Regulatory Peptides, 175( 1-3), 1-6. doi:10.1016/j.regpep.2012.01.003
NLM
Alves ES, Haidar AA, Quadros CD, Carvalho DS, Morgan D, Rocha MS, Curi R, Carpinelli AR, Hirata AE. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets [Internet]. Regulatory Peptides. 2012 ; 175( 1-3): 1-6.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.regpep.2012.01.003
Vancouver
Alves ES, Haidar AA, Quadros CD, Carvalho DS, Morgan D, Rocha MS, Curi R, Carpinelli AR, Hirata AE. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets [Internet]. Regulatory Peptides. 2012 ; 175( 1-3): 1-6.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.regpep.2012.01.003
Artigo de periodico
Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis (2012)
Cappi, Sylas Bezerra; Noritomi, Danilo Teixeira; Velasco, Irineu Tadeu ; Curi, Rui ; Loureiro, Tatiana Carolina Alba ; Soriano, Francisco Garcia
Fonte: Intensive Care Medicine. Unidades: ICB, FM
Assuntos: FISIOLOGIA, LIPÍDEOS, GORDURAS, SEPSE, GLICEMIA, ESTUDOS PROSPECTIVOS, ESTUDOS RANDOMIZADOS, LIPOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAPPI, Sylas Bezerra et al. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis. Intensive Care Medicine, v. 38, p. 634-641, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00134-011-2458-z. Acesso em: 19 ago. 2024.
APA
Cappi, S. B., Noritomi, D. T., Velasco, I. T., Curi, R., Loureiro, T. C. A., & Soriano, F. G. (2012). Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis. Intensive Care Medicine, 38, 634-641. doi:10.1007/s00134-011-2458-z
NLM
Cappi SB, Noritomi DT, Velasco IT, Curi R, Loureiro TCA, Soriano FG. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis [Internet]. Intensive Care Medicine. 2012 ; 38 634-641.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1007/s00134-011-2458-z
Vancouver
Cappi SB, Noritomi DT, Velasco IT, Curi R, Loureiro TCA, Soriano FG. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis [Internet]. Intensive Care Medicine. 2012 ; 38 634-641.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1007/s00134-011-2458-z
Artigo de periodico
Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages (2012)
Magdalon, Juliana; Vinolo, Marco Aurélio Ramirez; Rodrigues, Hosana Gomes; Paschoal, Vivian Almeida; Torres, Rosângela Pavan; Mancini-Filho, Jorge ; Calder, Philip C; Hatanaka, Elaine; Curi, Rui
Fonte: Lipids. Unidades: FCF, ICB
Assuntos: ÁCIDO LINOLEICO, ÁCIDOS GRAXOS, EICOSANOIDES, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAGDALON, Juliana et al. Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages. Lipids, v. 47, n. 8, p. 803-812, 2012Tradução . . Disponível em: https://doi.org/10.1007/s11745-012-3687-9. Acesso em: 19 ago. 2024.
APA
Magdalon, J., Vinolo, M. A. R., Rodrigues, H. G., Paschoal, V. A., Torres, R. P., Mancini-Filho, J., et al. (2012). Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages. Lipids, 47( 8), 803-812. doi:10.1007/s11745-012-3687-9
NLM
Magdalon J, Vinolo MAR, Rodrigues HG, Paschoal VA, Torres RP, Mancini-Filho J, Calder PC, Hatanaka E, Curi R. Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages [Internet]. Lipids. 2012 ; 47( 8): 803-812.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1007/s11745-012-3687-9
Vancouver
Magdalon J, Vinolo MAR, Rodrigues HG, Paschoal VA, Torres RP, Mancini-Filho J, Calder PC, Hatanaka E, Curi R. Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages [Internet]. Lipids. 2012 ; 47( 8): 803-812.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1007/s11745-012-3687-9
Artigo de periodico
Persistence of inflammatory response to intense exercise in diabetic rats (2012)
Bortolon, José Ricardo; Silva Junior, Antonio José de Almeida; Murata, Gilson Masahiro; Newsholme, Philip; Curi, Rui; Pithon-Curi, Tania Cristina; Hatanaka, Elaine
Fonte: Experimental Diabetes Research. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORTOLON, José Ricardo et al. Persistence of inflammatory response to intense exercise in diabetic rats. Experimental Diabetes Research, v. 2012, n. 213986, p. 1-8, 2012Tradução . . Disponível em: https://doi.org/10.1155/2012/213986. Acesso em: 19 ago. 2024.
APA
Bortolon, J. R., Silva Junior, A. J. de A., Murata, G. M., Newsholme, P., Curi, R., Pithon-Curi, T. C., & Hatanaka, E. (2012). Persistence of inflammatory response to intense exercise in diabetic rats. Experimental Diabetes Research, 2012( 213986), 1-8. doi:10.1155/2012/213986
NLM
Bortolon JR, Silva Junior AJ de A, Murata GM, Newsholme P, Curi R, Pithon-Curi TC, Hatanaka E. Persistence of inflammatory response to intense exercise in diabetic rats [Internet]. Experimental Diabetes Research. 2012 ; 2012( 213986): 1-8.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1155/2012/213986
Vancouver
Bortolon JR, Silva Junior AJ de A, Murata GM, Newsholme P, Curi R, Pithon-Curi TC, Hatanaka E. Persistence of inflammatory response to intense exercise in diabetic rats [Internet]. Experimental Diabetes Research. 2012 ; 2012( 213986): 1-8.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1155/2012/213986
Artigo de periodico
Synthesis, biological evaluation and molecular docking studies of 3-(triazolyl)-coumarin derivatives: effect on inducible nitric oxide synthase (2012)
Stefani, Hélio Alexandre ; Gueogjan, Karina; Manarin, Flávia; Farsky, Sandra Helena Poliselli ; Zukerman-Schpector, Julio; Caracelli, Ignez; Rodrigues, Sergio R. Pizano; Muscará, Marcelo Nicolás ; Teixeira, Simone Aparecida; Santin, José R; Machado, Isabel D; Bolonheis, Simone M; Curi, Rui; Vinolo, Marco Aurelio Ramirez
Fonte: European Journal of Medicinal Chemistry. Unidades: ICB, FCF
Assuntos: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
STEFANI, Hélio Alexandre et al. Synthesis, biological evaluation and molecular docking studies of 3-(triazolyl)-coumarin derivatives: effect on inducible nitric oxide synthase. European Journal of Medicinal Chemistry, v. 58, p. 117-127, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.ejmech.2012.10.010. Acesso em: 19 ago. 2024.
APA
Stefani, H. A., Gueogjan, K., Manarin, F., Farsky, S. H. P., Zukerman-Schpector, J., Caracelli, I., et al. (2012). Synthesis, biological evaluation and molecular docking studies of 3-(triazolyl)-coumarin derivatives: effect on inducible nitric oxide synthase. European Journal of Medicinal Chemistry, 58, 117-127. doi:10.1016/j.ejmech.2012.10.010
NLM
Stefani HA, Gueogjan K, Manarin F, Farsky SHP, Zukerman-Schpector J, Caracelli I, Rodrigues SRP, Muscará MN, Teixeira SA, Santin JR, Machado ID, Bolonheis SM, Curi R, Vinolo MAR. Synthesis, biological evaluation and molecular docking studies of 3-(triazolyl)-coumarin derivatives: effect on inducible nitric oxide synthase [Internet]. European Journal of Medicinal Chemistry. 2012 ; 58 117-127.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.ejmech.2012.10.010
Vancouver
Stefani HA, Gueogjan K, Manarin F, Farsky SHP, Zukerman-Schpector J, Caracelli I, Rodrigues SRP, Muscará MN, Teixeira SA, Santin JR, Machado ID, Bolonheis SM, Curi R, Vinolo MAR. Synthesis, biological evaluation and molecular docking studies of 3-(triazolyl)-coumarin derivatives: effect on inducible nitric oxide synthase [Internet]. European Journal of Medicinal Chemistry. 2012 ; 58 117-127.[citado 2024 ago. 19 ] Available from: https://doi.org/10.1016/j.ejmech.2012.10.010

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