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Artigo de periodico
12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers (2024)
Sousa, Carlos Alberto Rodrigues de ; Nogueira, Luciana Fidalgo Ramos ; Moreno, Claudia Roberta de Castro ; Marqueze, Elaine Cristina ; Cipolla Neto, José
Source: Frontiers in Nutrition. Unidades: FSP, ICB
Subjects: FISIOLOGIA, RITMOS BIOLÓGICOS, HOMEOSTASE, GORDURAS, COMORBIDADE, DIABETES MELLITUS, OBESIDADE, MELATONINA, TRABALHO NOTURNO, MULHERES, PESOS E MEDIDAS CORPORAIS, ESTUDOS RANDOMIZADOS
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SOUSA, Carlos Alberto Rodrigues de et al. 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers. Frontiers in Nutrition, v. 11, p. 11 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fnut.2024.1285398. Acesso em: 14 jul. 2024.
APA
Sousa, C. A. R. de, Nogueira, L. F. R., Moreno, C. R. de C., Marqueze, E. C., & Cipolla Neto, J. (2024). 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers. Frontiers in Nutrition, 11, 11 . doi:10.3389/fnut.2024.1285398
NLM
Sousa CAR de, Nogueira LFR, Moreno CR de C, Marqueze EC, Cipolla Neto J. 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers [Internet]. Frontiers in Nutrition. 2024 ; 11 11 .[citado 2024 jul. 14 ] Available from: https://doi.org/10.3389/fnut.2024.1285398
Vancouver
Sousa CAR de, Nogueira LFR, Moreno CR de C, Marqueze EC, Cipolla Neto J. 12-week melatonin administration had no effect on diabetes risk markers and fat intake in overweight women night workers [Internet]. Frontiers in Nutrition. 2024 ; 11 11 .[citado 2024 jul. 14 ] Available from: https://doi.org/10.3389/fnut.2024.1285398
Artigo de periodico
Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model (2024)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Experimental Neurology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS MORTAS, ASTRÓCITOS, NEURÓGLIA, CAMUNDONGOS, RATOS, TÉCNICAS ESTEREOTÁXICAS, IMUNOHISTOQUÍMICA, REAÇÃO EM CADEIA POR POLIMERASE
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FERREIRA, Ana Flávia Fernandes et al. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, v. 377, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2024.114780. Acesso em: 14 jul. 2024.
APA
Ferreira, A. F. F., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2024). Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, 377, 15 . doi:10.1016/j.expneurol.2024.114780
NLM
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Vancouver
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Artigo de periodico
Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats (2024)
Dellacqua, Lais Oliveira; Gomes, Paula Magalhães ; Batista, Julia Santos; Michelini, Lisete Compagno ; Antunes, Vagner Roberto
Source: Journal of Applied Physiology. Unidade: ICB
Subjects: FISIOLOGIA, BARORREFLEXO, FISIOLOGIA CARDIOVASCULAR, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, HIPERTENSÃO, ENVELHECIMENTO, TREINAMENTO AERÓBIO, PLASTICIDADE NEURONAL, RATOS WISTAR
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DELLACQUA, Lais Oliveira et al. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats. Journal of Applied Physiology, v. 136, n. 1, p. 189-198, 2024Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.00433.2023. Acesso em: 14 jul. 2024.
APA
Dellacqua, L. O., Gomes, P. M., Batista, J. S., Michelini, L. C., & Antunes, V. R. (2024). Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats. Journal of Applied Physiology, 136( 1), 189-198. doi:10.1152/japplphysiol.00433.2023
NLM
Dellacqua LO, Gomes PM, Batista JS, Michelini LC, Antunes VR. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats [Internet]. Journal of Applied Physiology. 2024 ; 136( 1): 189-198.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1152/japplphysiol.00433.2023
Vancouver
Dellacqua LO, Gomes PM, Batista JS, Michelini LC, Antunes VR. Exercise-induced neuroplasticity in autonomic nuclei restores the cardiac vagal tone and baroreflex dysfunction in aged hypertensive rats [Internet]. Journal of Applied Physiology. 2024 ; 136( 1): 189-198.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1152/japplphysiol.00433.2023
Artigo de periodico
Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats (2024)
Florido Neto, Armando Ribeiro ; Agostini, Lucas ; Silva Junior, Luciano Pedro da ; Nunes, Maria Tereza
Source: Endocrinology. Unidades: ICB, FSP
Subjects: FISIOLOGIA, RATOS WISTAR, MODELOS ANIMAIS DE DOENÇAS, GLICEMIA, CITOCINAS, DIABETES MELLITUS, HIPOTIREOIDISMO ANIMAL, RESISTÊNCIA À INSULINA, FÁRMACOS (METABOLISMO E NUTRIÇÃO), TRIIODOTIRONINA, HORMÔNIOS TIREOIDIANOS
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FLORIDO NETO, Armando Ribeiro et al. Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats. Endocrinology, v. 165, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqae066. Acesso em: 14 jul. 2024.
APA
Florido Neto, A. R., Agostini, L., Silva Junior, L. P. da, & Nunes, M. T. (2024). Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats. Endocrinology, 165, 14 . doi:https://doi.org/10.1210/endocr/bqae066
NLM
Florido Neto AR, Agostini L, Silva Junior LP da, Nunes MT. Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats [Internet]. Endocrinology. 2024 ; 165 14 .[citado 2024 jul. 14 ] Available from: https://doi.org/10.1210/endocr/bqae066
Vancouver
Florido Neto AR, Agostini L, Silva Junior LP da, Nunes MT. Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats [Internet]. Endocrinology. 2024 ; 165 14 .[citado 2024 jul. 14 ] Available from: https://doi.org/10.1210/endocr/bqae066
Artigo de periodico
Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure (2023)
Fontes, Milene T.; Arruda Junior, Daniel F.; Santos, Danúbia Silva dos ; Silva, João Carlos Ribeiro ; Antônio, Edinei L.; Tucci, Paulo F. J. ; Rossoni, Luciana Venturini ; Girardi, Adriana C. C.
Source: Life Sciences. Unidades: ICB, FM
Assunto: FISIOLOGIA
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FONTES, Milene T. et al. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, v. 323, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121648. Acesso em: 14 jul. 2024.
APA
Fontes, M. T., Arruda Junior, D. F., Santos, D. S. dos, Silva, J. C. R., Antônio, E. L., Tucci, P. F. J., et al. (2023). Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure. Life Sciences, 323, 1-10. doi:10.1016/j.lfs.2023.121648
NLM
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Vancouver
Fontes MT, Arruda Junior DF, Santos DS dos, Silva JCR, Antônio EL, Tucci PFJ, Rossoni LV, Girardi ACC. Dipeptidyl peptidase 4 inhibition rescues PKA-eNOS signaling and suppresses aortic hypercontractility in male rats with heart failure [Internet]. Life Sciences. 2023 ; 323 1-10.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121648
Artigo de periodico
miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction (2023)
Silva, Camila Stephanie Balbino ; Couto, Gisele Kruger ; Lino, Caroline Antunes ; Silva, Tábatha de Oliveira ; Lunardon, Guilherme ; Huang, Zhan-Peng ; Wang, Da Zhi ; Festuccia, William Tadeu Lara ; Barreto-Chaves, Maria Luiza Morais ; Rossoni, Luciana Venturini ; Diniz, Gabriela Placoná
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, FISIOLOGIA, AORTA DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, VASOS SANGUÍNEOS, TECIDO ADIPOSO, MICRORNAS, OBESIDADE, REAÇÃO EM CADEIA POR POLIMERASE, WESTERN BLOTTING
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SILVA, Camila Stephanie Balbino et al. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, v. 316, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121416. Acesso em: 14 jul. 2024.
APA
Silva, C. S. B., Couto, G. K., Lino, C. A., Silva, T. de O., Lunardon, G., Huang, Z. -P., et al. (2023). miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, 316, 1-10. doi:10.1016/j.lfs.2023.121416
NLM
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Vancouver
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Artigo de periodico
Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats (2023)
Gomes, Paula Magalhães ; Batista, Julia Santos; Sá, Renato Willian Martins ; Antunes, Vagner Roberto
Source: Experimental Physiology. Unidade: ICB
Subjects: FISIOLOGIA, LÍQUIDO CEFALORRAQUIDIANO, PRESSÃO SANGUÍNEA, HIPERTENSÃO, DOENÇAS CARDIOVASCULARES EM ANIMAL, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, NEUROGENÉTICA, FLUXO SANGUÍNEO, RESISTÊNCIA VASCULAR
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GOMES, Paula Magalhães et al. Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats. Experimental Physiology, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1113/EP090912op. Acesso em: 14 jul. 2024.
APA
Gomes, P. M., Batista, J. S., Sá, R. W. M., & Antunes, V. R. (2023). Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats. Experimental Physiology, 1-10. doi:10.1113/EP090912op
NLM
Gomes PM, Batista JS, Sá RWM, Antunes VR. Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats [Internet]. Experimental Physiology. 2023 ; 1-10.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1113/EP090912op
Vancouver
Gomes PM, Batista JS, Sá RWM, Antunes VR. Short exposure to high salt in drinking solution leads to a cardiovascular phenotype of hypertension without changes in the blood volume of rats [Internet]. Experimental Physiology. 2023 ; 1-10.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1113/EP090912op
Artigo de periodico
Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy (2023)
Chaves Filho, Adriano de Britto ; Peixoto, Albert Souza ; Castro, Érique de ; Silva, Tiago Eugênio Oliveira da; Perandini, Luiz Augusto Buoro ; Moreira, Rafael Junges ; Silva, Railmara Pereira da ; Silva, Beatriz Pereira da; Moretti, Eduardo Hermógenes ; Steiner, Alexandre Alarcon ; Miyamoto, Sayuri ; Yoshinaga, Marcos Yukio ; Festuccia, William Tadeu Lara
Source: Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. Unidades: ICB, IQ
Subjects: FISIOLOGIA, IMUNOLOGIA, PEROXISSOMOS, OXIDAÇÃO, TECIDO ADIPOSO, ADIPOSIDADE, FÍGADO GORDUROSO, CAMUNDONGOS, ÁCIDOS GRAXOS
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CHAVES FILHO, Adriano de Britto et al. Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids, v. 1868, n. 3, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.bbalip.2022.159264. Acesso em: 14 jul. 2024.
APA
Chaves Filho, A. de B., Peixoto, A. S., Castro, É. de, Silva, T. E. O. da, Perandini, L. A. B., Moreira, R. J., et al. (2023). Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids, 1868( 3), 1-14. doi:10.1016/j.bbalip.2022.159264
NLM
Chaves Filho A de B, Peixoto AS, Castro É de, Silva TEO da, Perandini LAB, Moreira RJ, Silva RP da, Silva BP da, Moretti EH, Steiner AA, Miyamoto S, Yoshinaga MY, Festuccia WTL. Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy [Internet]. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. 2023 ; 1868( 3): 1-14.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.bbalip.2022.159264
Vancouver
Chaves Filho A de B, Peixoto AS, Castro É de, Silva TEO da, Perandini LAB, Moreira RJ, Silva RP da, Silva BP da, Moretti EH, Steiner AA, Miyamoto S, Yoshinaga MY, Festuccia WTL. Futile cycle of β-oxidation and de novo lipogenesis are associated with essential fatty acids depletion in lipoatrophy [Internet]. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. 2023 ; 1868( 3): 1-14.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.bbalip.2022.159264
Artigo de periodico
Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats (2022)
Sá, Renato Willian Martins ; Theparambil, Shefeeq M. ; Santos, Karoline Martins dos ; Christie, Isabel N. ; Marina, Nephtali ; Cardoso, Barbara V.; Hosford, Patrick S. ; Antunes, Vagner Roberto
Source: Molecular and Cellular Neuroscience. Unidade: ICB
Subjects: FISIOLOGIA, HIPOTÁLAMO, RATOS, PRESSÃO SANGUÍNEA, ADENOSINA, ASTRÓCITOS, IMUNOHISTOQUÍMICA, NUCLEOTÍDEOS
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SÁ, Renato Willian Martins et al. Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats. Molecular and Cellular Neuroscience, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mcn.2022.103806. Acesso em: 14 jul. 2024.
APA
Sá, R. W. M., Theparambil, S. M., Santos, K. M. dos, Christie, I. N., Marina, N., Cardoso, B. V., et al. (2022). Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats. Molecular and Cellular Neuroscience. doi:10.1016/j.mcn.2022.103806
NLM
Sá RWM, Theparambil SM, Santos KM dos, Christie IN, Marina N, Cardoso BV, Hosford PS, Antunes VR. Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats [Internet]. Molecular and Cellular Neuroscience. 2022 ;[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.mcn.2022.103806
Vancouver
Sá RWM, Theparambil SM, Santos KM dos, Christie IN, Marina N, Cardoso BV, Hosford PS, Antunes VR. Salt-loading promotes extracellular ATP release mediated by glial cells in the hypothalamic paraventricular nucleus of rats [Internet]. Molecular and Cellular Neuroscience. 2022 ;[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.mcn.2022.103806
Artigo de periodico
Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model (2022)
Ferreira, Ana Flávia Fernandes ; Singulani, Monique Patricio ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: IQ, ICB, FM
Subjects: FISIOLOGIA, NEUROBIOLOGIA, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, MESENCÉFALO, GÂNGLIOS, RECEPTORES DE DOPAMINA, CAMUNDONGOS, IMUNOHISTOQUÍMICA, IMMUNOBLOTTING
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FERREIRA, Ana Flávia Fernandes et al. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, v. 59, p. 1543–1559, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02723-8. Acesso em: 14 jul. 2024.
APA
Ferreira, A. F. F., Singulani, M. P., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2022). Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, 59, 1543–1559. doi:10.1007/s12035-022-02723-8
NLM
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Vancouver
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Artigo de periodico
Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats (2022)
Moraes, Ruan Carlos Macêdo de ; Lima, Gabriely Cristina Alves; Cardinali, Camila Aparecida Errerias Fernandes ; Gonçalves, Alisson Carvalho de ; Portari, Guilherme Vannucchi ; Guerra-Shinohara, Elvira Maria ; Leboucher, Antoine ; Donato Junior, José ; Kleinridders, André ; Torrão, Andréa da Silva
Source: Life Sciences. Unidades: FCF, ICB, FM
Subjects: DOENÇA DE ALZHEIMER, HIPOTÁLAMO, FISIOLOGIA, VITAMINAS, DEMÊNCIA, INSULINA, DOENÇAS METABÓLICAS, METABOLISMO ANIMAL, RATOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇAS NEURODEGENERATIVAS
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MORAES, Ruan Carlos Macêdo de et al. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, v. 306, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120841. Acesso em: 14 jul. 2024.
APA
Moraes, R. C. M. de, Lima, G. C. A., Cardinali, C. A. E. F., Gonçalves, A. C. de, Portari, G. V., Guerra-Shinohara, E. M., et al. (2022). Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, 306, 1-13. doi:10.1016/j.lfs.2022.120841
NLM
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Vancouver
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Artigo de periodico
Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction (2021)
Fragas, Matheus Garcia ; Cândido, Vanessa Brito ; Davanzo, Gustavo Gastão ; Santos, Carla Rocha; Ceroni, Alexandre; Michelini, Lisete Compagno
Source: AJP: Regulatory, Integrative and Comparative Physiology. Unidade: ICB
Subjects: FISIOLOGIA, BARREIRA HEMATO-ENCEFÁLICA, EXERCÍCIO FÍSICO, HIPERTENSÃO, TREINAMENTO AERÓBIO, RATOS, MODELOS ANIMAIS DE DOENÇAS, HIPOTÁLAMO, PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA
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ABNT
FRAGAS, Matheus Garcia et al. Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction. AJP: Regulatory, Integrative and Comparative Physiology, v. 321, n. 5, p. R732–R741, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00154.2020. Acesso em: 14 jul. 2024.
APA
Fragas, M. G., Cândido, V. B., Davanzo, G. G., Santos, C. R., Ceroni, A., & Michelini, L. C. (2021). Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction. AJP: Regulatory, Integrative and Comparative Physiology, 321( 5), R732–R741. doi:10.1152/ajpregu.00154.2020
NLM
Fragas MG, Cândido VB, Davanzo GG, Santos CR, Ceroni A, Michelini LC. Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction [Internet]. AJP: Regulatory, Integrative and Comparative Physiology. 2021 ; 321( 5): R732–R741.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1152/ajpregu.00154.2020
Vancouver
Fragas MG, Cândido VB, Davanzo GG, Santos CR, Ceroni A, Michelini LC. Transcytosis within PVN capillaries: a mechanism determining both hypertension-induced blood-brain barrier dysfunction and exercise-induced correction [Internet]. AJP: Regulatory, Integrative and Comparative Physiology. 2021 ; 321( 5): R732–R741.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1152/ajpregu.00154.2020
Artigo de periodico
Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice (2021)
Castro, Érique de ; Vieira, Thayna dos Santos ; Oliveira, Tiago E.; Silva, Milene Ortiz ; Andrade, Maynara Lucca; Tomazelli, Caroline Aparecida ; Peixoto, Albert Souza ; Sobrinho, Cleyton Roberto ; Moreno, Mayara Franzoi ; Gilio, Gustavo Renan ; Moreira, Rafael Junges ; Guimarães, Raphael Campos ; Perandini, Luiz Augusto Buoro ; Chimin, Patricia ; Reckziegel, Patricia ; Moretti, Eduardo Hermógenes ; Steiner, Alexandre Alarcon ; Laplante, Mathieu ; Festuccia, William Tadeu Lara
Source: AJP: Endocrinology and Metabolism. Unidade: ICB
Subjects: IMUNOLOGIA, FISIOLOGIA, FÁRMACOS IMUNOSSUPRESSORES, TECIDO ADIPOSO, METABOLISMO ENERGÉTICO, EXPRESSÃO GÊNICA, GLICOSE, CAMUNDONGOS, TERMOGÊNESE, GENES, GORDURAS
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ABNT
CASTRO, Érique de et al. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice. AJP: Endocrinology and Metabolism, v. 321, p. E592–E605, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00587.2020. Acesso em: 14 jul. 2024.
APA
Castro, É. de, Vieira, T. dos S., Oliveira, T. E., Silva, M. O., Andrade, M. L., Tomazelli, C. A., et al. (2021). Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice. AJP: Endocrinology and Metabolism, 321, E592–E605. doi:10.1152/ajpendo.00587.2020
NLM
Castro É de, Vieira T dos S, Oliveira TE, Silva MO, Andrade ML, Tomazelli CA, Peixoto AS, Sobrinho CR, Moreno MF, Gilio GR, Moreira RJ, Guimarães RC, Perandini LAB, Chimin P, Reckziegel P, Moretti EH, Steiner AA, Laplante M, Festuccia WTL. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice [Internet]. AJP: Endocrinology and Metabolism. 2021 ; 321 E592–E605.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1152/ajpendo.00587.2020
Vancouver
Castro É de, Vieira T dos S, Oliveira TE, Silva MO, Andrade ML, Tomazelli CA, Peixoto AS, Sobrinho CR, Moreno MF, Gilio GR, Moreira RJ, Guimarães RC, Perandini LAB, Chimin P, Reckziegel P, Moretti EH, Steiner AA, Laplante M, Festuccia WTL. Adipocyte-specific mTORC2 deficiency impairs BAT and iWAT thermogenic capacity without affecting glucose uptake and energy expenditure in coldacclimated mice [Internet]. AJP: Endocrinology and Metabolism. 2021 ; 321 E592–E605.[citado 2024 jul. 14 ] Available from: https://doi.org/10.1152/ajpendo.00587.2020

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