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Artigo de periodico
Ethanol: striking the cardiovascular system by harming the gut microbiota (2021)
Silva, Carla Brigagão Pacheco da ; Oliveira, Jefferson Elias ; McCarthy, Cameron; Wenceslau, Camilla F.; Carlos, Daniela; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology : Heart and Circulatory Physiology. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR, ÁLCOOL, DOENÇAS CARDIOVASCULARES, PREBIÓTICOS, PROBIÓTICOS
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ABNT
SILVA, Carla Brigagão Pacheco da et al. Ethanol: striking the cardiovascular system by harming the gut microbiota. American Journal of Physiology : Heart and Circulatory Physiology, v. 321, n. 2, p. H275-H291, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00225.2021. Acesso em: 14 nov. 2024.
APA
Silva, C. B. P. da, Oliveira, J. E., McCarthy, C., Wenceslau, C. F., Carlos, D., & Tostes, R. de C. A. (2021). Ethanol: striking the cardiovascular system by harming the gut microbiota. American Journal of Physiology : Heart and Circulatory Physiology, 321( 2), H275-H291. doi:10.1152/ajpheart.00225.2021
NLM
Silva CBP da, Oliveira JE, McCarthy C, Wenceslau CF, Carlos D, Tostes R de CA. Ethanol: striking the cardiovascular system by harming the gut microbiota [Internet]. American Journal of Physiology : Heart and Circulatory Physiology. 2021 ; 321( 2): H275-H291.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/ajpheart.00225.2021
Vancouver
Silva CBP da, Oliveira JE, McCarthy C, Wenceslau CF, Carlos D, Tostes R de CA. Ethanol: striking the cardiovascular system by harming the gut microbiota [Internet]. American Journal of Physiology : Heart and Circulatory Physiology. 2021 ; 321( 2): H275-H291.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/ajpheart.00225.2021
Artigo de periodico
Angiotensin-II activates vascular inflammasome and induces vascular damage (2021)
Cáu, Stêfany Bruno de Assis; Bruder-Nascimento, Ariane; Silva, Marcondes Alves Barbosa da; Ramalho, Fernanda Naira Zambelli; Mestriner, Fabiola Leslie Antunes Cardoso; Lopes, Rheure Alves Moreira; Ferreira, Nathanne dos Santos; Tostes, Rita de Cássia Aleixo ; Nascimento, Thiago Bruder do
Source: Vascular Pharmacology. Unidade: FMRP
Subjects: ANGIOTENSINA II, CÉLULAS CULTIVADAS, DOENÇAS CARDIOVASCULARES
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ABNT
CÁU, Stêfany Bruno de Assis et al. Angiotensin-II activates vascular inflammasome and induces vascular damage. Vascular Pharmacology, v. 139, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106881. Acesso em: 14 nov. 2024.
APA
Cáu, S. B. de A., Bruder-Nascimento, A., Silva, M. A. B. da, Ramalho, F. N. Z., Mestriner, F. L. A. C., Lopes, R. A. M., et al. (2021). Angiotensin-II activates vascular inflammasome and induces vascular damage. Vascular Pharmacology, 139, 1-10. doi:10.1016/j.vph.2021.106881
NLM
Cáu SB de A, Bruder-Nascimento A, Silva MAB da, Ramalho FNZ, Mestriner FLAC, Lopes RAM, Ferreira N dos S, Tostes R de CA, Nascimento TB do. Angiotensin-II activates vascular inflammasome and induces vascular damage [Internet]. Vascular Pharmacology. 2021 ; 139 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.vph.2021.106881
Vancouver
Cáu SB de A, Bruder-Nascimento A, Silva MAB da, Ramalho FNZ, Mestriner FLAC, Lopes RAM, Ferreira N dos S, Tostes R de CA, Nascimento TB do. Angiotensin-II activates vascular inflammasome and induces vascular damage [Internet]. Vascular Pharmacology. 2021 ; 139 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.vph.2021.106881
Artigo de periodico
Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations (2021)
Prado, Alejandro F.; Batista, Rose Inês Matos ; Tanus-Santos, José Eduardo ; Gerlach, Raquel Fernanda
Source: Biomolecules. Unidades: FMRP, FORP
Subjects: METALOPROTEINASES, HIPERTENSÃO, ESTRESSE OXIDATIVO, ÓXIDO NÍTRICO, FISIOPATOLOGIA, DOENÇAS CARDIOVASCULARES
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ABNT
PRADO, Alejandro F. et al. Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations. Biomolecules, v. 11, n. 4, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3390/biom11040585. Acesso em: 14 nov. 2024.
APA
Prado, A. F., Batista, R. I. M., Tanus-Santos, J. E., & Gerlach, R. F. (2021). Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations. Biomolecules, 11( 4), 1-17. doi:10.3390/biom11040585
NLM
Prado AF, Batista RIM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations [Internet]. Biomolecules. 2021 ; 11( 4): 1-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/biom11040585
Vancouver
Prado AF, Batista RIM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinases and arterial hypertension: Role of oxidative stress and nitric oxide in vascular functional and structural alterations [Internet]. Biomolecules. 2021 ; 11( 4): 1-17.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/biom11040585
Artigo de periodico
Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases (2019)
Costa, Rafael Menezes da; Rodrigues, Daniel; Pereira, Camila André; Silva, Josiane F. da; Alves, Juliano Vilela; Lobato, Nubia Souza; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: ESTRESSE OXIDATIVO, DOENÇAS CARDIOVASCULARES, DOENÇAS METABÓLICAS
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ABNT
COSTA, Rafael Menezes da et al. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases. Frontiers in Pharmacology, v. 10, 2019Tradução . . Disponível em: https://doi.org/10.3389/fphar.2019.00382. Acesso em: 14 nov. 2024.
APA
Costa, R. M. da, Rodrigues, D., Pereira, C. A., Silva, J. F. da, Alves, J. V., Lobato, N. S., & Tostes, R. de C. A. (2019). Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases. Frontiers in Pharmacology, 10. doi:10.3389/fphar.2019.00382
NLM
Costa RM da, Rodrigues D, Pereira CA, Silva JF da, Alves JV, Lobato NS, Tostes R de CA. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphar.2019.00382
Vancouver
Costa RM da, Rodrigues D, Pereira CA, Silva JF da, Alves JV, Lobato NS, Tostes R de CA. Nrf2 as a potential mediator of cardiovascular risk in metabolic diseases [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphar.2019.00382
Artigo de periodico
Ameliorative effect of low-dose spironolactone on obesity and insulin resistance is through replenishment of estrogen in ovariectomized rats (2019)
Olatunji, Lawrence A.; Adeyanju, Oluwaseun A.; Michael, Olugbenga S.; Usman, Taofeek O.; Tostes, Rita de Cássia Aleixo; Soladoye, Ayodele O.
Source: Canadian Journal of Physiology and Pharmacology. Unidade: FMRP
Subjects: TERAPIA DE REPOSIÇÃO DE ESTRÓGENOS, ALDOSTERONA, DOENÇAS CARDIOVASCULARES, OBESIDADE
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ABNT
OLATUNJI, Lawrence A. et al. Ameliorative effect of low-dose spironolactone on obesity and insulin resistance is through replenishment of estrogen in ovariectomized rats. Canadian Journal of Physiology and Pharmacology, v. 97, n. 1, p. 65-74, 2019Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2018-0416. Acesso em: 14 nov. 2024.
APA
Olatunji, L. A., Adeyanju, O. A., Michael, O. S., Usman, T. O., Tostes, R. de C. A., & Soladoye, A. O. (2019). Ameliorative effect of low-dose spironolactone on obesity and insulin resistance is through replenishment of estrogen in ovariectomized rats. Canadian Journal of Physiology and Pharmacology, 97( 1), 65-74. doi:10.1139/cjpp-2018-0416
NLM
Olatunji LA, Adeyanju OA, Michael OS, Usman TO, Tostes R de CA, Soladoye AO. Ameliorative effect of low-dose spironolactone on obesity and insulin resistance is through replenishment of estrogen in ovariectomized rats [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 1): 65-74.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2018-0416
Vancouver
Olatunji LA, Adeyanju OA, Michael OS, Usman TO, Tostes R de CA, Soladoye AO. Ameliorative effect of low-dose spironolactone on obesity and insulin resistance is through replenishment of estrogen in ovariectomized rats [Internet]. Canadian Journal of Physiology and Pharmacology. 2019 ; 97( 1): 65-74.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2018-0416
Artigo de periodico
Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries (2017)
Simplicio, Janaina A.; Vale, Gabriel T. do; Gonzaga, Natália A.; Leite, Letícia N.; Hipólito, Ulisses V.; Pereira, Camila A.; Tostes, Rita de Cássia Aleixo; Tirapelli, Carlos Renato
Source: Journal of Physiology and Biochemistry. Unidades: FMRP, EERP
Subjects: ETANOL, HIPERTENSÃO, ESTRESSE OXIDATIVO, DOENÇAS CARDIOVASCULARES, ENDOTÉLIO VASCULAR, GLICOPROTEÍNAS DA MEMBRANA
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ABNT
SIMPLICIO, Janaina A. et al. Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries. Journal of Physiology and Biochemistry, v. 73, n. 1, p. 5-16, 2017Tradução . . Disponível em: https://doi.org/10.1007/s13105-016-0519-z. Acesso em: 14 nov. 2024.
APA
Simplicio, J. A., Vale, G. T. do, Gonzaga, N. A., Leite, L. N., Hipólito, U. V., Pereira, C. A., et al. (2017). Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries. Journal of Physiology and Biochemistry, 73( 1), 5-16. doi:10.1007/s13105-016-0519-z
NLM
Simplicio JA, Vale GT do, Gonzaga NA, Leite LN, Hipólito UV, Pereira CA, Tostes R de CA, Tirapelli CR. Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries [Internet]. Journal of Physiology and Biochemistry. 2017 ; 73( 1): 5-16.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s13105-016-0519-z
Vancouver
Simplicio JA, Vale GT do, Gonzaga NA, Leite LN, Hipólito UV, Pereira CA, Tostes R de CA, Tirapelli CR. Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries [Internet]. Journal of Physiology and Biochemistry. 2017 ; 73( 1): 5-16.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s13105-016-0519-z
Artigo de periodico-carta/editorial
Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta] (2016)
Amaral, Jefferson H.; Tanus-Santos, José Eduardo
Source: Clinica Chimica Acta. Unidade: FMRP
Subjects: DOENÇAS CARDIOVASCULARES, VEGETARIANISMO
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ABNT
AMARAL, Jefferson H. e TANUS-SANTOS, José Eduardo. Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta]. Clinica Chimica Acta. Amsterdam: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.cca.2016.08.020. Acesso em: 14 nov. 2024. , 2016
APA
Amaral, J. H., & Tanus-Santos, J. E. (2016). Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta]. Clinica Chimica Acta. Amsterdam: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1016/j.cca.2016.08.020
NLM
Amaral JH, Tanus-Santos JE. Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta] [Internet]. Clinica Chimica Acta. 2016 ; 462 40.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cca.2016.08.020
Vancouver
Amaral JH, Tanus-Santos JE. Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta] [Internet]. Clinica Chimica Acta. 2016 ; 462 40.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cca.2016.08.020
Trabalho de evento-resumo
NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice (2016)
Pereira, Camila A.; Ferreira, Nathanne S.; Zanotto, Camila Z.; Carlos, Daniela; Tostes, Rita de Cássia Aleixo
Source: Final Program. Conference titles: Council on Hypertension 2016. Unidade: FMRP
Subjects: INFLAMAÇÃO, DIABETES MELLITUS, DOENÇAS CARDIOVASCULARES
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ABNT
PEREIRA, Camila A. et al. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. 2016, Anais.. Orlando: AHA, 2016. . Acesso em: 14 nov. 2024.
APA
Pereira, C. A., Ferreira, N. S., Zanotto, C. Z., Carlos, D., & Tostes, R. de C. A. (2016). NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. In Final Program. Orlando: AHA.
NLM
Pereira CA, Ferreira NS, Zanotto CZ, Carlos D, Tostes R de CA. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. Final Program. 2016 ;[citado 2024 nov. 14 ]
Vancouver
Pereira CA, Ferreira NS, Zanotto CZ, Carlos D, Tostes R de CA. NLRP3 inflammasome activation by mitochondrial DNA contributes to oxidative stress and inflammation in the vasculature of type diabetic mice. Final Program. 2016 ;[citado 2024 nov. 14 ]
Artigo de periodico
Plasma matrix metalloproteinase-9 levels, MMP-9 gene haplotypes, and cardiovascular risk in obese subjects (2016)
Luizon, Marcelo Rizzatti; Belo, Vanessa A.; Fernandes, Karla S.; Andrade, Vanessa L.; Tanus-Santos, José Eduardo; Sandrim, Valéria Cristina
Source: Molecular Biology Reports. Unidade: FMRP
Subjects: BIOMARCADORES, DOENÇAS CARDIOVASCULARES, POLIMORFISMO, OBESIDADE, FATORES DE RISCO
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ABNT
LUIZON, Marcelo Rizzatti et al. Plasma matrix metalloproteinase-9 levels, MMP-9 gene haplotypes, and cardiovascular risk in obese subjects. Molecular Biology Reports, v. 43, n. 6, p. 463-471, 2016Tradução . . Disponível em: https://doi.org/10.1007/s11033-016-3993-z. Acesso em: 14 nov. 2024.
APA
Luizon, M. R., Belo, V. A., Fernandes, K. S., Andrade, V. L., Tanus-Santos, J. E., & Sandrim, V. C. (2016). Plasma matrix metalloproteinase-9 levels, MMP-9 gene haplotypes, and cardiovascular risk in obese subjects. Molecular Biology Reports, 43( 6), 463-471. doi:10.1007/s11033-016-3993-z
NLM
Luizon MR, Belo VA, Fernandes KS, Andrade VL, Tanus-Santos JE, Sandrim VC. Plasma matrix metalloproteinase-9 levels, MMP-9 gene haplotypes, and cardiovascular risk in obese subjects [Internet]. Molecular Biology Reports. 2016 ; 43( 6): 463-471.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s11033-016-3993-z
Vancouver
Luizon MR, Belo VA, Fernandes KS, Andrade VL, Tanus-Santos JE, Sandrim VC. Plasma matrix metalloproteinase-9 levels, MMP-9 gene haplotypes, and cardiovascular risk in obese subjects [Internet]. Molecular Biology Reports. 2016 ; 43( 6): 463-471.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s11033-016-3993-z
Artigo de periodico
Reduced levels of potential circulating biomarkers of cardiovascular diseaes in apparently healthy vegetarian men (2016)
Navarro, Julio Acosta; Gouveia, Luiza Antoniazzi de; Rocha-Penha, Liliam; Cinegaglia, Naiara; Belo, Vanessa; Castro, Michele Mazzaron de; Sandrim, Valéria Cristina
Source: Clinica Chimica Acta. Unidade: FMRP
Subjects: DOENÇAS CARDIOVASCULARES, BIOMARCADORES, VEGETARIANISMO
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ABNT
NAVARRO, Julio Acosta et al. Reduced levels of potential circulating biomarkers of cardiovascular diseaes in apparently healthy vegetarian men. Clinica Chimica Acta, v. 461, p. 100-113, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.cca.2016.08.002. Acesso em: 14 nov. 2024.
APA
Navarro, J. A., Gouveia, L. A. de, Rocha-Penha, L., Cinegaglia, N., Belo, V., Castro, M. M. de, & Sandrim, V. C. (2016). Reduced levels of potential circulating biomarkers of cardiovascular diseaes in apparently healthy vegetarian men. Clinica Chimica Acta, 461, 100-113. doi:10.1016/j.cca.2016.08.002
NLM
Navarro JA, Gouveia LA de, Rocha-Penha L, Cinegaglia N, Belo V, Castro MM de, Sandrim VC. Reduced levels of potential circulating biomarkers of cardiovascular diseaes in apparently healthy vegetarian men [Internet]. Clinica Chimica Acta. 2016 ; 461 100-113.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cca.2016.08.002
Vancouver
Navarro JA, Gouveia LA de, Rocha-Penha L, Cinegaglia N, Belo V, Castro MM de, Sandrim VC. Reduced levels of potential circulating biomarkers of cardiovascular diseaes in apparently healthy vegetarian men [Internet]. Clinica Chimica Acta. 2016 ; 461 100-113.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.cca.2016.08.002
Artigo de periodico
Genetic effects of eNOS polymorphisms on biomarkers related to cardiovascular status in a population coexposed to methylmercury and lead (2015)
Barcelos, Gustavo Rafael Mazzaron; De Marco, Kátia Cristina; Rezende, Vânia Braghini de; Braga, Gilberto Úbida Leite; Antunes, Lusânia Maria Greggi; Tanus-Santos, José Eduardo; Barbosa Júnior, Fernando
Source: Archives of Environmental Contamination and Toxicology. Unidades: FCFRP, FMRP
Subjects: BIOMARCADORES (METABOLISMO), DOENÇAS CARDIOVASCULARES, POLUIÇÃO AMBIENTAL, CHUMBO (TOXICIDADE), ÓXIDO NÍTRICO
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ABNT
BARCELOS, Gustavo Rafael Mazzaron et al. Genetic effects of eNOS polymorphisms on biomarkers related to cardiovascular status in a population coexposed to methylmercury and lead. Archives of Environmental Contamination and Toxicology, v. 69, n. 2, p. 173-180, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00244-015-0137-8. Acesso em: 14 nov. 2024.
APA
Barcelos, G. R. M., De Marco, K. C., Rezende, V. B. de, Braga, G. Ú. L., Antunes, L. M. G., Tanus-Santos, J. E., & Barbosa Júnior, F. (2015). Genetic effects of eNOS polymorphisms on biomarkers related to cardiovascular status in a population coexposed to methylmercury and lead. Archives of Environmental Contamination and Toxicology, 69( 2), 173-180. doi:10.1007/s00244-015-0137-8
NLM
Barcelos GRM, De Marco KC, Rezende VB de, Braga GÚL, Antunes LMG, Tanus-Santos JE, Barbosa Júnior F. Genetic effects of eNOS polymorphisms on biomarkers related to cardiovascular status in a population coexposed to methylmercury and lead [Internet]. Archives of Environmental Contamination and Toxicology. 2015 ; 69( 2): 173-180.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00244-015-0137-8
Vancouver
Barcelos GRM, De Marco KC, Rezende VB de, Braga GÚL, Antunes LMG, Tanus-Santos JE, Barbosa Júnior F. Genetic effects of eNOS polymorphisms on biomarkers related to cardiovascular status in a population coexposed to methylmercury and lead [Internet]. Archives of Environmental Contamination and Toxicology. 2015 ; 69( 2): 173-180.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00244-015-0137-8
Artigo de periodico
Angiotensin II induces Fat1 expression/activation and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation (2014)
Bruder-Nascimento, T.; Chinnasamy, P.; Riascos-Bernal, D. F.; Cau, S. B.; Callera, G. E.; Touyz, R. M.; Tostes, Rita de Cassia Aleixo; Sibinga, N. E. S.
Source: Journal of Molecular and Cellular Cardiology. Unidade: FMRP
Subjects: ANGIOTENSINAS, CÉLULAS MUSCULARES, DOENÇAS CARDIOVASCULARES
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ABNT
BRUDER-NASCIMENTO, T. et al. Angiotensin II induces Fat1 expression/activation and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation. Journal of Molecular and Cellular Cardiology, v. 66, p. 18-26, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.yjmcc.2013.10.013. Acesso em: 14 nov. 2024.
APA
Bruder-Nascimento, T., Chinnasamy, P., Riascos-Bernal, D. F., Cau, S. B., Callera, G. E., Touyz, R. M., et al. (2014). Angiotensin II induces Fat1 expression/activation and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation. Journal of Molecular and Cellular Cardiology, 66, 18-26. doi:10.1016/j.yjmcc.2013.10.013
NLM
Bruder-Nascimento T, Chinnasamy P, Riascos-Bernal DF, Cau SB, Callera GE, Touyz RM, Tostes R de CA, Sibinga NES. Angiotensin II induces Fat1 expression/activation and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation [Internet]. Journal of Molecular and Cellular Cardiology. 2014 ; 66 18-26.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.yjmcc.2013.10.013
Vancouver
Bruder-Nascimento T, Chinnasamy P, Riascos-Bernal DF, Cau SB, Callera GE, Touyz RM, Tostes R de CA, Sibinga NES. Angiotensin II induces Fat1 expression/activation and vascular smooth muscle cell migration via Nox1-dependent reactive oxygen species generation [Internet]. Journal of Molecular and Cellular Cardiology. 2014 ; 66 18-26.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.yjmcc.2013.10.013
Artigo de periodico
Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats (2013)
Cáu, Stêfany Bruno de Assis; Barato, Renan C.; Celes, Mara Rúbia Nunes; Muniz, Jaqueline J.; Rossi, Marcos Antonio; Tanus-Santos, José Eduardo
Source: Cardiovascular Drugs and Therapy. Unidade: FMRP
Subjects: EMBOLIA PULMONAR, DOENÇAS CARDIOVASCULARES, METALOPROTEINASES, MODELOS ANIMAIS
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ABNT
CÁU, Stêfany Bruno de Assis et al. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats. Cardiovascular Drugs and Therapy, v. 27, n. 4, p. 259-267, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10557-013-6458-9. Acesso em: 14 nov. 2024.
APA
Cáu, S. B. de A., Barato, R. C., Celes, M. R. N., Muniz, J. J., Rossi, M. A., & Tanus-Santos, J. E. (2013). Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats. Cardiovascular Drugs and Therapy, 27( 4), 259-267. doi:10.1007/s10557-013-6458-9
NLM
Cáu SB de A, Barato RC, Celes MRN, Muniz JJ, Rossi MA, Tanus-Santos JE. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 4): 259-267.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s10557-013-6458-9
Vancouver
Cáu SB de A, Barato RC, Celes MRN, Muniz JJ, Rossi MA, Tanus-Santos JE. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 4): 259-267.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s10557-013-6458-9
Artigo de periodico
Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy (2013)
Rizzi, Elen; Ceron, Carla S.; Guimarães, Danielle A.; Prado, Cibele Maria; Rossi, Marcos Antonio; Gerlach, Raquel Fernanda; Santos, José Eduardo Tanus dos
Source: Experimental and Molecular Pathology. Unidades: FMRP, FORP
Subjects: METALOPROTEINASES, HIPERTENSÃO, DOENÇAS CARDIOVASCULARES
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ABNT
RIZZI, Elen et al. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy. Experimental and Molecular Pathology, v. 94, n. 1, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.yexmp.2012.10.010. Acesso em: 14 nov. 2024.
APA
Rizzi, E., Ceron, C. S., Guimarães, D. A., Prado, C. M., Rossi, M. A., Gerlach, R. F., & Santos, J. E. T. dos. (2013). Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy. Experimental and Molecular Pathology, 94( 1), 1-9. doi:10.1016/j.yexmp.2012.10.010
NLM
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Santos JET dos. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy [Internet]. Experimental and Molecular Pathology. 2013 ; 94( 1): 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.yexmp.2012.10.010
Vancouver
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Santos JET dos. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy [Internet]. Experimental and Molecular Pathology. 2013 ; 94( 1): 1-9.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.yexmp.2012.10.010
Artigo de periodico
BK channels in cardiovascular diseases and aging (2013)
Carvalho de Souza, João Luís; Varanda, Wamberto Antonio; Tostes, Rita de Cassia Aleixo; Chignalia, Andreia Z.
Source: Aging and Disease. Unidade: FMRP
Subjects: ENVELHECIMENTO, DOENÇAS CARDIOVASCULARES, MÚSCULO LISO VASCULAR, CANAIS IÔNICOS, POTÁSSIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO DE SOUZA, João Luís et al. BK channels in cardiovascular diseases and aging. Aging and Disease, v. 4, n. 1, p. 38-49, 2013Tradução . . Acesso em: 14 nov. 2024.
APA
Carvalho de Souza, J. L., Varanda, W. A., Tostes, R. de C. A., & Chignalia, A. Z. (2013). BK channels in cardiovascular diseases and aging. Aging and Disease, 4( 1), 38-49.
NLM
Carvalho de Souza JL, Varanda WA, Tostes R de CA, Chignalia AZ. BK channels in cardiovascular diseases and aging. Aging and Disease. 2013 ; 4( 1): 38-49.[citado 2024 nov. 14 ]
Vancouver
Carvalho de Souza JL, Varanda WA, Tostes R de CA, Chignalia AZ. BK channels in cardiovascular diseases and aging. Aging and Disease. 2013 ; 4( 1): 38-49.[citado 2024 nov. 14 ]
Artigo de periodico
Early effects of high-fat diet on neurovascular function and focal ischemic brain injury (2013)
Li, Weiguo; Prakash, Roshini; Chawla, Dhruv; Du, Wenting; Didion, Sean P.; Filosa, Jessica A.; Zhang, Quanguang; Brann, Darrell W.; Lima, Victor V.; Passaglia, Rita de Cassia Aleixo Tostes; Ergul, Adviye
Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: FMRP
Subjects: DIETA, GORDURAS, ISQUEMIA CEREBRAL, HEMORRAGIA, DOENÇAS CARDIOVASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LI, Weiguo et al. Early effects of high-fat diet on neurovascular function and focal ischemic brain injury. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 304, n. 11, p. R1001-R1008, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00523.2012. Acesso em: 14 nov. 2024.
APA
Li, W., Prakash, R., Chawla, D., Du, W., Didion, S. P., Filosa, J. A., et al. (2013). Early effects of high-fat diet on neurovascular function and focal ischemic brain injury. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 304( 11), R1001-R1008. doi:10.1152/ajpregu.00523.2012
NLM
Li W, Prakash R, Chawla D, Du W, Didion SP, Filosa JA, Zhang Q, Brann DW, Lima VV, Passaglia R de CAT, Ergul A. Early effects of high-fat diet on neurovascular function and focal ischemic brain injury [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2013 ; 304( 11): R1001-R1008.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/ajpregu.00523.2012
Vancouver
Li W, Prakash R, Chawla D, Du W, Didion SP, Filosa JA, Zhang Q, Brann DW, Lima VV, Passaglia R de CAT, Ergul A. Early effects of high-fat diet on neurovascular function and focal ischemic brain injury [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2013 ; 304( 11): R1001-R1008.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/ajpregu.00523.2012
Artigo de periodico
Imbalanced matrix metalloproteinases in cardiovascular complications of end-stage kidney disease: a potential pharmacological target (2012)
Marson, Bernardo P.; Figueiredo, Carlos E. Poli de; Santos, José Eduardo Tanus dos
Source: Basic and Clinical Pharmacology and toxicology. Unidade: FMRP
Subjects: ARTERIOSCLEROSE, DOENÇAS CARDIOVASCULARES, INSUFICIÊNCIA RENAL CRÔNICA, METALOPROTEINASES, UREMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARSON, Bernardo P. e FIGUEIREDO, Carlos E. Poli de e SANTOS, José Eduardo Tanus dos. Imbalanced matrix metalloproteinases in cardiovascular complications of end-stage kidney disease: a potential pharmacological target. Basic and Clinical Pharmacology and toxicology, v. 110, n. 5, p. 409-415, 2012Tradução . . Disponível em: https://doi.org/10.1111/j.1742-7843.2012.00863.x. Acesso em: 14 nov. 2024.
APA
Marson, B. P., Figueiredo, C. E. P. de, & Santos, J. E. T. dos. (2012). Imbalanced matrix metalloproteinases in cardiovascular complications of end-stage kidney disease: a potential pharmacological target. Basic and Clinical Pharmacology and toxicology, 110( 5), 409-415. doi:10.1111/j.1742-7843.2012.00863.x
NLM
Marson BP, Figueiredo CEP de, Santos JET dos. Imbalanced matrix metalloproteinases in cardiovascular complications of end-stage kidney disease: a potential pharmacological target [Internet]. Basic and Clinical Pharmacology and toxicology. 2012 ; 110( 5): 409-415.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/j.1742-7843.2012.00863.x
Vancouver
Marson BP, Figueiredo CEP de, Santos JET dos. Imbalanced matrix metalloproteinases in cardiovascular complications of end-stage kidney disease: a potential pharmacological target [Internet]. Basic and Clinical Pharmacology and toxicology. 2012 ; 110( 5): 409-415.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/j.1742-7843.2012.00863.x
Artigo de periodico
Testosterone and vascular function in aging (2012)
Lopes, Rheure Alves Moreira; Neves, Karla Bianca; Carneiro, Fernando S.; Passaglia, Rita de Cassia Aleixo Tostes
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: TESTOSTERONA (METABOLISMO), ENVELHECIMENTO, DOENÇAS CARDIOVASCULARES, ENDOTÉLIO VASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Rheure Alves Moreira et al. Testosterone and vascular function in aging. Frontiers in Physiology, v. 3, 2012Tradução . . Disponível em: https://doi.org/10.3389/fphys.2012.00089. Acesso em: 14 nov. 2024.
APA
Lopes, R. A. M., Neves, K. B., Carneiro, F. S., & Passaglia, R. de C. A. T. (2012). Testosterone and vascular function in aging. Frontiers in Physiology, 3. doi:10.3389/fphys.2012.00089
NLM
Lopes RAM, Neves KB, Carneiro FS, Passaglia R de CAT. Testosterone and vascular function in aging [Internet]. Frontiers in Physiology. 2012 ; 3[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphys.2012.00089
Vancouver
Lopes RAM, Neves KB, Carneiro FS, Passaglia R de CAT. Testosterone and vascular function in aging [Internet]. Frontiers in Physiology. 2012 ; 3[citado 2024 nov. 14 ] Available from: https://doi.org/10.3389/fphys.2012.00089
Artigo de periodico
Genetic variants in matrix metalloproteinase-9 gene modify metalloproteinase-9 levels in black subjects (2012)
Metzger, Ingrid; Luizon, Marcelo R.; Lacchini, Riccardo ; Tanus-Santos, José Eduardo
Source: DNA and Cell Biology. Unidade: FMRP
Subjects: METALOPROTEINASES (EFEITOS), DOENÇAS CARDIOVASCULARES, POLIMORFISMO, NEGROS, FARMACOGENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
METZGER, Ingrid et al. Genetic variants in matrix metalloproteinase-9 gene modify metalloproteinase-9 levels in black subjects. DNA and Cell Biology, v. 31, n. 4, p. 504-510, 2012Tradução . . Disponível em: https://doi.org/10.1089/dna.2011.1388. Acesso em: 14 nov. 2024.
APA
Metzger, I., Luizon, M. R., Lacchini, R., & Tanus-Santos, J. E. (2012). Genetic variants in matrix metalloproteinase-9 gene modify metalloproteinase-9 levels in black subjects. DNA and Cell Biology, 31( 4), 504-510. doi:10.1089/dna.2011.1388
NLM
Metzger I, Luizon MR, Lacchini R, Tanus-Santos JE. Genetic variants in matrix metalloproteinase-9 gene modify metalloproteinase-9 levels in black subjects [Internet]. DNA and Cell Biology. 2012 ; 31( 4): 504-510.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1089/dna.2011.1388
Vancouver
Metzger I, Luizon MR, Lacchini R, Tanus-Santos JE. Genetic variants in matrix metalloproteinase-9 gene modify metalloproteinase-9 levels in black subjects [Internet]. DNA and Cell Biology. 2012 ; 31( 4): 504-510.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1089/dna.2011.1388
Artigo de periodico
Intron 4 polymorphism of the endothelial nitric oxide synthase (eNOS) gene is associated with decreased NO production in a mercury-exposed population (2012)
Marco, Katia Cristina de; Antunes, Lusania Maria Greggi; Tanus-Santos, José Eduardo; Barbosa Júnior, Fernando
Source: Science of the Total Environment. Unidades: FCFRP, FMRP
Subjects: ÓXIDO NÍTRICO, SISTEMA CARDIOVASCULAR (REGULAÇÃO), POLIMORFISMO, MERCÚRIO (ELEMENTO QUÍMICO) (EXPOSIÇÕES), DOENÇAS CARDIOVASCULARES, POPULAÇÃO (AVALIAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARCO, Katia Cristina de et al. Intron 4 polymorphism of the endothelial nitric oxide synthase (eNOS) gene is associated with decreased NO production in a mercury-exposed population. Science of the Total Environment, v. 414, p. 708-712, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.scitotenv.2011.11.010. Acesso em: 14 nov. 2024.
APA
Marco, K. C. de, Antunes, L. M. G., Tanus-Santos, J. E., & Barbosa Júnior, F. (2012). Intron 4 polymorphism of the endothelial nitric oxide synthase (eNOS) gene is associated with decreased NO production in a mercury-exposed population. Science of the Total Environment, 414, 708-712. doi:10.1016/j.scitotenv.2011.11.010
NLM
Marco KC de, Antunes LMG, Tanus-Santos JE, Barbosa Júnior F. Intron 4 polymorphism of the endothelial nitric oxide synthase (eNOS) gene is associated with decreased NO production in a mercury-exposed population [Internet]. Science of the Total Environment. 2012 ; 414 708-712.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.scitotenv.2011.11.010
Vancouver
Marco KC de, Antunes LMG, Tanus-Santos JE, Barbosa Júnior F. Intron 4 polymorphism of the endothelial nitric oxide synthase (eNOS) gene is associated with decreased NO production in a mercury-exposed population [Internet]. Science of the Total Environment. 2012 ; 414 708-712.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.scitotenv.2011.11.010

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