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Artigo de periodico
Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells (2021)
Saito, Renata F.; Rangel, Maria Cristina; Halman, Justin R.; Chandler, Morgan; Andrade, Luciana Nogueira de Sousa; Odete-bustos, Silvina; Furuya, Tatiane Katsue ; Carrasco, Alexis German Murillo; Chaves-filho, Adriano B.; Yoshinaga, Marcos Y.; Chammas, Roger
Source: Nanomedicine-nanotechnology biology and medicine. Unidade: FM
Subjects: RADIAÇÃO (ENERGIA RADIANTE), RADIOTERAPIA, APOPTOSE
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ABNT
SAITO, Renata F. et al. Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells. Nanomedicine-nanotechnology biology and medicine, v. 36, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.nano.2021.102418. Acesso em: 09 nov. 2024.
APA
Saito, R. F., Rangel, M. C., Halman, J. R., Chandler, M., Andrade, L. N. de S., Odete-bustos, S., et al. (2021). Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells. Nanomedicine-nanotechnology biology and medicine, 36. doi:10.1016/j.nano.2021.102418
NLM
Saito RF, Rangel MC, Halman JR, Chandler M, Andrade LN de S, Odete-bustos S, Furuya TK, Carrasco AGM, Chaves-filho AB, Yoshinaga MY, Chammas R. Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells [Internet]. Nanomedicine-nanotechnology biology and medicine. 2021 ; 36[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.nano.2021.102418
Vancouver
Saito RF, Rangel MC, Halman JR, Chandler M, Andrade LN de S, Odete-bustos S, Furuya TK, Carrasco AGM, Chaves-filho AB, Yoshinaga MY, Chammas R. Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells [Internet]. Nanomedicine-nanotechnology biology and medicine. 2021 ; 36[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.nano.2021.102418
Artigo de periodico
Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features (2020)
Bellana, D. L; Biscaiaa, S. M. P; Rossia, G. R; Cristala, A. M; Goncalvesa, J. P; Oliveiraa, C. C; Simasa, F. F; Sabryb, D. A; Rochab, H. A. O; Chammas, Roger
Source: Carbohydrate polymers. Unidade: FM
Subjects: ANIMAIS, APOPTOSE, QUÍMICA VERDE, CAMUNDONGOS, PROLIFERAÇÃO CELULAR
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ABNT
BELLANA, D. L et al. Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features. Carbohydrate polymers, v. 250, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.carbpol.2020.116869. Acesso em: 09 nov. 2024.
APA
Bellana, D. L., Biscaiaa, S. M. P., Rossia, G. R., Cristala, A. M., Goncalvesa, J. P., Oliveiraa, C. C., et al. (2020). Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features. Carbohydrate polymers, 250. doi:10.1016/j.carbpol.2020.116869
NLM
Bellana DL, Biscaiaa SMP, Rossia GR, Cristala AM, Goncalvesa JP, Oliveiraa CC, Simasa FF, Sabryb DA, Rochab HAO, Chammas R. Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features [Internet]. Carbohydrate polymers. 2020 ; 250[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.carbpol.2020.116869
Vancouver
Bellana DL, Biscaiaa SMP, Rossia GR, Cristala AM, Goncalvesa JP, Oliveiraa CC, Simasa FF, Sabryb DA, Rochab HAO, Chammas R. Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features [Internet]. Carbohydrate polymers. 2020 ; 250[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.carbpol.2020.116869
Artigo de periodico
Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death (2017)
Tortelli Junior, Tharcisio Citrangulo; Godoy, Lyris Martins Franco de; Souza, Gustavo Antonio de; Bonatto, Diego; Otake, Andreia Hanada; Saito, Renata de Freitas; Rosa, Jose Cesar; Greene, Lewis Joel; Chammas, Roger
Source: Oncotarget. Unidades: FMRP, FM
Subjects: APOPTOSE, NEOPLASIAS CUTÂNEAS, MANIFESTAÇÕES CUTÂNEAS, MELANOMA
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TORTELLI JUNIOR, Tharcisio Citrangulo et al. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death. Oncotarget, v. 8, n. 26, p. 43114-43129, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.17810. Acesso em: 09 nov. 2024.
APA
Tortelli Junior, T. C., Godoy, L. M. F. de, Souza, G. A. de, Bonatto, D., Otake, A. H., Saito, R. de F., et al. (2017). Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death. Oncotarget, 8( 26), 43114-43129. doi:10.18632/oncotarget.17810
NLM
Tortelli Junior TC, Godoy LMF de, Souza GA de, Bonatto D, Otake AH, Saito R de F, Rosa JC, Greene LJ, Chammas R. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death [Internet]. Oncotarget. 2017 ; 8( 26): 43114-43129.[citado 2024 nov. 09 ] Available from: https://doi.org/10.18632/oncotarget.17810
Vancouver
Tortelli Junior TC, Godoy LMF de, Souza GA de, Bonatto D, Otake AH, Saito R de F, Rosa JC, Greene LJ, Chammas R. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death [Internet]. Oncotarget. 2017 ; 8( 26): 43114-43129.[citado 2024 nov. 09 ] Available from: https://doi.org/10.18632/oncotarget.17810
Artigo de periodico-carta/editorial
Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy (2017)
Chammas, Roger; Andrade, Luciana Nogueira de Sousa; Jancar, Sonia
Source: Nature reviews cancer. Unidades: FM, ICB
Subjects: APOPTOSE, NEOPLASIAS, ONCOGENES, CÉLULAS CULTIVADAS DE TUMOR
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ABNT
CHAMMAS, Roger e ANDRADE, Luciana Nogueira de Sousa e JANCAR, Sonia. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/nrc.2017.15. Acesso em: 09 nov. 2024. , 2017
APA
Chammas, R., Andrade, L. N. de S., & Jancar, S. (2017). Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. doi:10.1038/nrc.2017.15
NLM
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/nrc.2017.15
Vancouver
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/nrc.2017.15
Artigo de periodico
A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production (2013)
Maria, Durvanei Augusto; Souza, Jean Gabriel de; Morais, Katia L. P.; Berra, Carolina Maria; Zampolli, Hamilton de Campos; Demasi, Marilene; Simons, Simone Michaela; Saito, Renata de Freitas; Chammas, Roger; Chudzinski-Tavassi, Ana Marisa
Source: Investigational New Drugs. Unidade: FM
Subjects: ESTRESSE, NEOPLASIAS (TERAPIA), MECANISMOS DE CONTROLE CELULAR, ADENOCARCINOMA, APOPTOSE
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ABNT
MARIA, Durvanei Augusto et al. A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production. Investigational New Drugs, v. 31, n. 3, p. 493-505, 2013Tradução . . Disponível em: http://link.springer.com/article/10.1007/s10637-012-9871-1. Acesso em: 09 nov. 2024.
APA
Maria, D. A., Souza, J. G. de, Morais, K. L. P., Berra, C. M., Zampolli, H. de C., Demasi, M., et al. (2013). A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production. Investigational New Drugs, 31( 3), 493-505. doi:10.1007/s10637-012-9871-1
NLM
Maria DA, Souza JG de, Morais KLP, Berra CM, Zampolli H de C, Demasi M, Simons SM, Saito R de F, Chammas R, Chudzinski-Tavassi AM. A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production [Internet]. Investigational New Drugs. 2013 ; 31( 3): 493-505.[citado 2024 nov. 09 ] Available from: http://link.springer.com/article/10.1007/s10637-012-9871-1
Vancouver
Maria DA, Souza JG de, Morais KLP, Berra CM, Zampolli H de C, Demasi M, Simons SM, Saito R de F, Chammas R, Chudzinski-Tavassi AM. A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production [Internet]. Investigational New Drugs. 2013 ; 31( 3): 493-505.[citado 2024 nov. 09 ] Available from: http://link.springer.com/article/10.1007/s10637-012-9871-1
Artigo de periodico
De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus (2013)
Borges, Beatriz E.; Teixeira, Verônica R.; Appel, Marcia H.; Steclan, Chelin A.; Rigo, Fernanda; Filipak Neto, Francisco; Ferreira, Ana M. da Costa; Chammas, Roger; Zanata, Silvio M.; Nakao, Lia S.
Source: Chemico-Biological Interactions. Unidades: IQ, FM
Subjects: MELANOMA, ESTRESSE OXIDATIVO, PROLIFERAÇÃO CELULAR, APOPTOSE, NEUROBLASTOMA, CITOFISIOLOGIA, METÁSTASE NEOPLÁSICA
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ABNT
BORGES, Beatriz E. et al. De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus. Chemico-Biological Interactions, v. 206, n. 1, p. 37-46, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2013.08.005. Acesso em: 09 nov. 2024.
APA
Borges, B. E., Teixeira, V. R., Appel, M. H., Steclan, C. A., Rigo, F., Filipak Neto, F., et al. (2013). De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus. Chemico-Biological Interactions, 206( 1), 37-46. doi:10.1016/j.cbi.2013.08.005
NLM
Borges BE, Teixeira VR, Appel MH, Steclan CA, Rigo F, Filipak Neto F, Ferreira AM da C, Chammas R, Zanata SM, Nakao LS. De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus [Internet]. Chemico-Biological Interactions. 2013 ; 206( 1): 37-46.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.cbi.2013.08.005
Vancouver
Borges BE, Teixeira VR, Appel MH, Steclan CA, Rigo F, Filipak Neto F, Ferreira AM da C, Chammas R, Zanata SM, Nakao LS. De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus [Internet]. Chemico-Biological Interactions. 2013 ; 206( 1): 37-46.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.cbi.2013.08.005
Artigo de periodico
Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA (2012)
Gimenez, Marcela; Marie, Suely Kazue Nagahashi; Oba-Shinjo, Sueli Mieko; Uno, Miyuki; Silva, Roseli da; Laure, Helen Julie; Izumi, Clarice; Otake, Andreia; Chammas, Roger; Rosa, José César
Source: Proteomics. Unidades: FM, FMRP
Subjects: FOSFOPROTEÍNAS, APOPTOSE, NEOPLASIAS CEREBRAIS, BIOQUÍMICA CELULAR
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ABNT
GIMENEZ, Marcela et al. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA. Proteomics, v. 12, n. 17, p. 2632-2640, 2012Tradução . . Disponível em: https://doi.org/10.1002/pmic.201200034. Acesso em: 09 nov. 2024.
APA
Gimenez, M., Marie, S. K. N., Oba-Shinjo, S. M., Uno, M., Silva, R. da, Laure, H. J., et al. (2012). Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA. Proteomics, 12( 17), 2632-2640. doi:10.1002/pmic.201200034
NLM
Gimenez M, Marie SKN, Oba-Shinjo SM, Uno M, Silva R da, Laure HJ, Izumi C, Otake A, Chammas R, Rosa JC. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA [Internet]. Proteomics. 2012 ; 12( 17): 2632-2640.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/pmic.201200034
Vancouver
Gimenez M, Marie SKN, Oba-Shinjo SM, Uno M, Silva R da, Laure HJ, Izumi C, Otake A, Chammas R, Rosa JC. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA [Internet]. Proteomics. 2012 ; 12( 17): 2632-2640.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/pmic.201200034
Artigo de periodico
Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection (2011)
Oliveira, Felipe L.; Brand, Camila; Paula, Adelzon A.; Arcanjo, Kátia D.; Hsu, Daniel K.; Liu, Fu-Tong; Takiya, Christina M.; Borojevic, Radovan; Chammas, Roger; El-Cheikh, Márcia C.
Source: PLOS ONE. Unidade: FM
Subjects: ESQUISTOSSOMOSE MANSONI (PARASITOLOGIA), APOPTOSE, BIOMARCADORES, CAMUNDONGOS, CÉLULAS DENTRÍTICAS, INFLAMAÇÃO, CITOMETRIA DE FLUXO, FAGÓCITOS, IMUNOHISTOQUÍMICA, LINFÓCITOS
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ABNT
OLIVEIRA, Felipe L. et al. Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection. PLOS ONE, v. 6, n. 5, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0019216. Acesso em: 09 nov. 2024.
APA
Oliveira, F. L., Brand, C., Paula, A. A., Arcanjo, K. D., Hsu, D. K., Liu, F. -T., et al. (2011). Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection. PLOS ONE, 6( 5). doi:10.1371/journal.pone.0019216
NLM
Oliveira FL, Brand C, Paula AA, Arcanjo KD, Hsu DK, Liu F-T, Takiya CM, Borojevic R, Chammas R, El-Cheikh MC. Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection [Internet]. PLOS ONE. 2011 ; 6( 5):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0019216
Vancouver
Oliveira FL, Brand C, Paula AA, Arcanjo KD, Hsu DK, Liu F-T, Takiya CM, Borojevic R, Chammas R, El-Cheikh MC. Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection [Internet]. PLOS ONE. 2011 ; 6( 5):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0019216
Artigo de periodico
Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells (2010)
Carneiro, Marcella Lemos Brettas; Porfírio, Elaine Paulucio; Otake, Andréia Hanada; Chammas, Roger; Báo, Sônia Nair; Guillo, Lidia Andreu
Source: Brazilian Archives of Biology and Technology. Unidade: FM
Subjects: CICLO CELULAR, MELANOMA (MORFOLOGIA), APOPTOSE
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ABNT
CARNEIRO, Marcella Lemos Brettas et al. Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells. Brazilian Archives of Biology and Technology, v. 53, n. 2, p. 343-352, 2010Tradução . . Disponível em: https://doi.org/10.1590/S1516-89132010000600004. Acesso em: 09 nov. 2024.
APA
Carneiro, M. L. B., Porfírio, E. P., Otake, A. H., Chammas, R., Báo, S. N., & Guillo, L. A. (2010). Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells. Brazilian Archives of Biology and Technology, 53( 2), 343-352. doi:10.1590/S1516-89132010000600004
NLM
Carneiro MLB, Porfírio EP, Otake AH, Chammas R, Báo SN, Guillo LA. Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells [Internet]. Brazilian Archives of Biology and Technology. 2010 ; 53( 2): 343-352.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/S1516-89132010000600004
Vancouver
Carneiro MLB, Porfírio EP, Otake AH, Chammas R, Báo SN, Guillo LA. Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells [Internet]. Brazilian Archives of Biology and Technology. 2010 ; 53( 2): 343-352.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/S1516-89132010000600004
Artigo de periodico
Sialylation of beta1 integrins blocks cell adhesion to galectin-3 and protects cells against galectin-3-induced apoptosis (2008)
Zhuo, Ya; Chammas, Roger; Bellis, Susan L
Source: The Journal of biological chemistry. Unidade: FM
Subjects: APOPTOSE, INTEGRINAS, NEOPLASIAS DO COLON (FISIOPATOLOGIA), CARCINOMA
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ABNT
ZHUO, Ya e CHAMMAS, Roger e BELLIS, Susan L. Sialylation of beta1 integrins blocks cell adhesion to galectin-3 and protects cells against galectin-3-induced apoptosis. The Journal of biological chemistry, v. 283, n. 32, p. 22177-22185, 2008Tradução . . Disponível em: https://doi.org/10.1074/jbc.M8000015200. Acesso em: 09 nov. 2024.
APA
Zhuo, Y., Chammas, R., & Bellis, S. L. (2008). Sialylation of beta1 integrins blocks cell adhesion to galectin-3 and protects cells against galectin-3-induced apoptosis. The Journal of biological chemistry, 283( 32), 22177-22185. doi:10.1074/jbc.M8000015200
NLM
Zhuo Y, Chammas R, Bellis SL. Sialylation of beta1 integrins blocks cell adhesion to galectin-3 and protects cells against galectin-3-induced apoptosis [Internet]. The Journal of biological chemistry. 2008 ; 283( 32): 22177-22185.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1074/jbc.M8000015200
Vancouver
Zhuo Y, Chammas R, Bellis SL. Sialylation of beta1 integrins blocks cell adhesion to galectin-3 and protects cells against galectin-3-induced apoptosis [Internet]. The Journal of biological chemistry. 2008 ; 283( 32): 22177-22185.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1074/jbc.M8000015200
Artigo de periodico
ADAM23 silencing in breast cancer is associated with capase-1 and DAPK-1 downregulation and resistance to anoikis (2007)
Silva Pinto, C.; Verbisck, N. V.; Chammas, Roger; Costa, E. T.; Costa, F. F.; Camargo, A. A.
Source: Applied Cancer Research. Conference titles: São Paulo Research Conference-Cancer. Unidade: FM
Subjects: NEOPLASIAS MAMÁRIAS, APOPTOSE, BIOMARCADORES
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ABNT
SILVA PINTO, C. et al. ADAM23 silencing in breast cancer is associated with capase-1 and DAPK-1 downregulation and resistance to anoikis. Applied Cancer Research, v. 27, n. 2, p. 71, 2007Tradução . . Acesso em: 09 nov. 2024.
APA
Silva Pinto, C., Verbisck, N. V., Chammas, R., Costa, E. T., Costa, F. F., & Camargo, A. A. (2007). ADAM23 silencing in breast cancer is associated with capase-1 and DAPK-1 downregulation and resistance to anoikis. Applied Cancer Research, 27( 2), 71.
NLM
Silva Pinto C, Verbisck NV, Chammas R, Costa ET, Costa FF, Camargo AA. ADAM23 silencing in breast cancer is associated with capase-1 and DAPK-1 downregulation and resistance to anoikis. Applied Cancer Research. 2007 ; 27( 2): 71.[citado 2024 nov. 09 ]
Vancouver
Silva Pinto C, Verbisck NV, Chammas R, Costa ET, Costa FF, Camargo AA. ADAM23 silencing in breast cancer is associated with capase-1 and DAPK-1 downregulation and resistance to anoikis. Applied Cancer Research. 2007 ; 27( 2): 71.[citado 2024 nov. 09 ]
Trabalho de evento-resumo
Evidence for a dual role of galectin-3 within tumor microenvironment (2006)
Andrade, Luciana Nogueira de Sousa; Pinheiro, Mônica de Campos; Nonogaki, Sueli; Chammas, Roger
Source: Resumos. Conference titles: Congresso da Sociedade Brasileira de Biologia Celular. Unidade: FM
Subjects: APOPTOSE, INFLAMAÇÃO, MELANOMA, NEOPLASIAS
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ABNT
ANDRADE, Luciana Nogueira de Sousa et al. Evidence for a dual role of galectin-3 within tumor microenvironment. 2006, Anais.. Búzios: SBBC/SIMEC, 2006. . Acesso em: 09 nov. 2024.
APA
Andrade, L. N. de S., Pinheiro, M. de C., Nonogaki, S., & Chammas, R. (2006). Evidence for a dual role of galectin-3 within tumor microenvironment. In Resumos. Búzios: SBBC/SIMEC.
NLM
Andrade LN de S, Pinheiro M de C, Nonogaki S, Chammas R. Evidence for a dual role of galectin-3 within tumor microenvironment. Resumos. 2006 ;[citado 2024 nov. 09 ]
Vancouver
Andrade LN de S, Pinheiro M de C, Nonogaki S, Chammas R. Evidence for a dual role of galectin-3 within tumor microenvironment. Resumos. 2006 ;[citado 2024 nov. 09 ]
Trabalho de evento-resumo
Cytotoxicity and induction of apoptosis by curcumin (curcuma longa linn.) in human melanoma cells (2006)
Carneiro, Marcella Lemos Brettas; Matuda, Larissa; Otake, Andréia H; Chammas, Roger; Báo, Sônia Nair; Guillo, Lidia Andreu
Conference titles: Congresso da Sociedade Brasileira de Biologia Celular. Unidade: FM
Subjects: APOPTOSE, CÉLULAS MORTAS, CITOTOXICIDADE IMUNOLÓGICA, MELANOMA
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ABNT
CARNEIRO, Marcella Lemos Brettas et al. Cytotoxicity and induction of apoptosis by curcumin (curcuma longa linn.) in human melanoma cells. 2006, Anais.. Búzios: SBBC/SIMEC, 2006. . Acesso em: 09 nov. 2024.
APA
Carneiro, M. L. B., Matuda, L., Otake, A. H., Chammas, R., Báo, S. N., & Guillo, L. A. (2006). Cytotoxicity and induction of apoptosis by curcumin (curcuma longa linn.) in human melanoma cells. In . Búzios: SBBC/SIMEC.
NLM
Carneiro MLB, Matuda L, Otake AH, Chammas R, Báo SN, Guillo LA. Cytotoxicity and induction of apoptosis by curcumin (curcuma longa linn.) in human melanoma cells. 2006 ;[citado 2024 nov. 09 ]
Vancouver
Carneiro MLB, Matuda L, Otake AH, Chammas R, Báo SN, Guillo LA. Cytotoxicity and induction of apoptosis by curcumin (curcuma longa linn.) in human melanoma cells. 2006 ;[citado 2024 nov. 09 ]
Trabalho de evento-resumo
Evidence for a dual role of galectin-3 within tumor microenvironment (2006)
Andrade, L. N. S; Pinheiro, M. C; Nonogaki, S; Chammas, R
Source: Programa. Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental. Unidade: FM
Subjects: MELANOMA, INFLAMAÇÃO, NEOPLASIAS, APOPTOSE, RESUMOS
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ABNT
ANDRADE, L. N. S et al. Evidence for a dual role of galectin-3 within tumor microenvironment. 2006, Anais.. São Paulo: FeSBE, 2006. . Acesso em: 09 nov. 2024.
APA
Andrade, L. N. S., Pinheiro, M. C., Nonogaki, S., & Chammas, R. (2006). Evidence for a dual role of galectin-3 within tumor microenvironment. In Programa. São Paulo: FeSBE.
NLM
Andrade LNS, Pinheiro MC, Nonogaki S, Chammas R. Evidence for a dual role of galectin-3 within tumor microenvironment. Programa. 2006 ;[citado 2024 nov. 09 ]
Vancouver
Andrade LNS, Pinheiro MC, Nonogaki S, Chammas R. Evidence for a dual role of galectin-3 within tumor microenvironment. Programa. 2006 ;[citado 2024 nov. 09 ]
Trabalho de evento-resumo periodico
Transient inflammatory response induced by apoptotic cells is an important mediator of melanoma cell engraftment and growth (2005)
Correa, Mariangela; Machado Jr., Joel; Carneiro, Célio R. W; Pesquero, João Bosco; Bader, Michael; Travassos, Luiz R; Chammas, Roger; Jasiulionis, Miriam G
Source: International Journal of Cancer. Unidade: FM
Subjects: MELANOMA, APOPTOSE, INFLAMAÇÃO, NEUTRÓFILOS, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CORREA, Mariangela et al. Transient inflammatory response induced by apoptotic cells is an important mediator of melanoma cell engraftment and growth. International Journal of Cancer. New York: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://onlinelibrary.wiley.com/doi/10.1002/ijc.20673/full. Acesso em: 09 nov. 2024. , 2005
APA
Correa, M., Machado Jr., J., Carneiro, C. R. W., Pesquero, J. B., Bader, M., Travassos, L. R., et al. (2005). Transient inflammatory response induced by apoptotic cells is an important mediator of melanoma cell engraftment and growth. International Journal of Cancer. New York: Faculdade de Medicina, Universidade de São Paulo. doi:10.1002/ijc.20673
NLM
Correa M, Machado Jr. J, Carneiro CRW, Pesquero JB, Bader M, Travassos LR, Chammas R, Jasiulionis MG. Transient inflammatory response induced by apoptotic cells is an important mediator of melanoma cell engraftment and growth [Internet]. International Journal of Cancer. 2005 ; 114( 3): 356-363.[citado 2024 nov. 09 ] Available from: http://onlinelibrary.wiley.com/doi/10.1002/ijc.20673/full
Vancouver
Correa M, Machado Jr. J, Carneiro CRW, Pesquero JB, Bader M, Travassos LR, Chammas R, Jasiulionis MG. Transient inflammatory response induced by apoptotic cells is an important mediator of melanoma cell engraftment and growth [Internet]. International Journal of Cancer. 2005 ; 114( 3): 356-363.[citado 2024 nov. 09 ] Available from: http://onlinelibrary.wiley.com/doi/10.1002/ijc.20673/full
Trabalho de evento-resumo
Ganglioside GD3 modulates chemotherapy-induced cell death in vitro (2004)
Otake, A. H.; Chamma,Roger
Conference titles: Brazilian Symposium on Extracellular Matrix. Unidade: FM
Subjects: APOPTOSE, GÂNGLIOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OTAKE, A. H. e CHAMMA, Roger. Ganglioside GD3 modulates chemotherapy-induced cell death in vitro. 2004, Anais.. Angra dos Reis, Rio de Janeiro, Brazil: Faculdade de Medicina, Universidade de São Paulo, 2004. . Acesso em: 09 nov. 2024.
APA
Otake, A. H., & Chamma, R. (2004). Ganglioside GD3 modulates chemotherapy-induced cell death in vitro. In . Angra dos Reis, Rio de Janeiro, Brazil: Faculdade de Medicina, Universidade de São Paulo.
NLM
Otake AH, Chamma R. Ganglioside GD3 modulates chemotherapy-induced cell death in vitro. 2004 ;[citado 2024 nov. 09 ]
Vancouver
Otake AH, Chamma R. Ganglioside GD3 modulates chemotherapy-induced cell death in vitro. 2004 ;[citado 2024 nov. 09 ]
Trabalho de evento-anais periodico
Efeito anti-proliferatico causado por emulsão lípidica contendo 7-cetocolesterol (7KC) (2004)
Favero, G. M.; Otake, A. H.; Caldini, Elia Tamaso Espin Garcia; Maranhão, Raul Cavalcante ; Chammas, Roger; Bydlowski, Sérgio Paulo
Conference titles: Fesbe - Reunião Anual da federação de Sociedade de Biologia. Unidades: FM, FCF
Subjects: PROLIFERAÇÃO CELULAR, APOPTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAVERO, G. M. et al. Efeito anti-proliferatico causado por emulsão lípidica contendo 7-cetocolesterol (7KC). . Águas de Lindóia, SP, Brasil: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 09 nov. 2024. , 2004
APA
Favero, G. M., Otake, A. H., Caldini, E. T. E. G., Maranhão, R. C., Chammas, R., & Bydlowski, S. P. (2004). Efeito anti-proliferatico causado por emulsão lípidica contendo 7-cetocolesterol (7KC). Águas de Lindóia, SP, Brasil: Faculdade de Medicina, Universidade de São Paulo.
NLM
Favero GM, Otake AH, Caldini ETEG, Maranhão RC, Chammas R, Bydlowski SP. Efeito anti-proliferatico causado por emulsão lípidica contendo 7-cetocolesterol (7KC). 2004 ;[citado 2024 nov. 09 ]
Vancouver
Favero GM, Otake AH, Caldini ETEG, Maranhão RC, Chammas R, Bydlowski SP. Efeito anti-proliferatico causado por emulsão lípidica contendo 7-cetocolesterol (7KC). 2004 ;[citado 2024 nov. 09 ]
Trabalho de evento
Neutrophil recruitment by apoptotic cells within the tunor microenvironment (2002)
Jasiulionis, M. G.; Correa, M.; Machado Junior, J.; Pesquero, J. B.; Travassos, L. L.; Chammas, R.
Conference titles: Brazilian Symposium on Extracellular Matrix. Unidade: FM
Subjects: APOPTOSE, INFLAMAÇÃO, ONCOLOGIA, NEOPLASIAS (GENÉTICA), MUTAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JASIULIONIS, M. G. et al. Neutrophil recruitment by apoptotic cells within the tunor microenvironment. 2002, Anais.. Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo, 2002. . Acesso em: 09 nov. 2024.
APA
Jasiulionis, M. G., Correa, M., Machado Junior, J., Pesquero, J. B., Travassos, L. L., & Chammas, R. (2002). Neutrophil recruitment by apoptotic cells within the tunor microenvironment. In . Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo.
NLM
Jasiulionis MG, Correa M, Machado Junior J, Pesquero JB, Travassos LL, Chammas R. Neutrophil recruitment by apoptotic cells within the tunor microenvironment. 2002 ;[citado 2024 nov. 09 ]
Vancouver
Jasiulionis MG, Correa M, Machado Junior J, Pesquero JB, Travassos LL, Chammas R. Neutrophil recruitment by apoptotic cells within the tunor microenvironment. 2002 ;[citado 2024 nov. 09 ]
Trabalho de evento
Differential distribution of Galectin-1 and Galectin-3 in the thymus following acute infection by trypanosoma cruzi may be related to intrathymic disturbs (2002)
Silva-Monteiro, E.; Lorenzato, L. R.; Junqueira, M. S.; Chammas, R.; Rabinovich, G. A.; Liu, F. T.; Savino, W.; Villa-Verde, D. M. S.
Conference titles: Brazilian Symposium on Extracellular Matrix. Unidade: FM
Subjects: TIMO (SISTEMAS SANGUÍNEO E IMUNE), MATRIZ EXTRACELULAR, APOPTOSE, IMUNOHISTOQUÍMICA, TRYPANOSOMA CRUZI
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA-MONTEIRO, E. et al. Differential distribution of Galectin-1 and Galectin-3 in the thymus following acute infection by trypanosoma cruzi may be related to intrathymic disturbs. 2002, Anais.. Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo, 2002. . Acesso em: 09 nov. 2024.
APA
Silva-Monteiro, E., Lorenzato, L. R., Junqueira, M. S., Chammas, R., Rabinovich, G. A., Liu, F. T., et al. (2002). Differential distribution of Galectin-1 and Galectin-3 in the thymus following acute infection by trypanosoma cruzi may be related to intrathymic disturbs. In . Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo.
NLM
Silva-Monteiro E, Lorenzato LR, Junqueira MS, Chammas R, Rabinovich GA, Liu FT, Savino W, Villa-Verde DMS. Differential distribution of Galectin-1 and Galectin-3 in the thymus following acute infection by trypanosoma cruzi may be related to intrathymic disturbs. 2002 ;[citado 2024 nov. 09 ]
Vancouver
Silva-Monteiro E, Lorenzato LR, Junqueira MS, Chammas R, Rabinovich GA, Liu FT, Savino W, Villa-Verde DMS. Differential distribution of Galectin-1 and Galectin-3 in the thymus following acute infection by trypanosoma cruzi may be related to intrathymic disturbs. 2002 ;[citado 2024 nov. 09 ]
Trabalho de evento
FGF-2 Motogenic, but not mitogenic, activity depends on glycosphingolipid composition of melannocyte cell membranes (2002)
Otake, A. H.; Duarte, A. P. M.; Chammas, R.
Conference titles: Brazilian Symposium on Extracellular Matrix. Unidade: FM
Subjects: NEOPLASIAS CEREBRAIS (PATOLOGIA), APOPTOSE, PROLIFERAÇÃO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OTAKE, A. H. e DUARTE, A. P. M. e CHAMMAS, R. FGF-2 Motogenic, but not mitogenic, activity depends on glycosphingolipid composition of melannocyte cell membranes. 2002, Anais.. Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo, 2002. . Acesso em: 09 nov. 2024.
APA
Otake, A. H., Duarte, A. P. M., & Chammas, R. (2002). FGF-2 Motogenic, but not mitogenic, activity depends on glycosphingolipid composition of melannocyte cell membranes. In . Rio de Janeiro: Faculdade de Medicina, Universidade de São Paulo.
NLM
Otake AH, Duarte APM, Chammas R. FGF-2 Motogenic, but not mitogenic, activity depends on glycosphingolipid composition of melannocyte cell membranes. 2002 ;[citado 2024 nov. 09 ]
Vancouver
Otake AH, Duarte APM, Chammas R. FGF-2 Motogenic, but not mitogenic, activity depends on glycosphingolipid composition of melannocyte cell membranes. 2002 ;[citado 2024 nov. 09 ]

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