Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells (2010)
- Authors:
- Autor USP: CHAMMAS, ROGER - FM
- Unidade: FM
- DOI: 10.1590/S1516-89132010000600004
- Subjects: CICLO CELULAR; MELANOMA (MORFOLOGIA); APOPTOSE
- Language: Inglês
- Imprenta:
- Source:
- Título: Brazilian Archives of Biology and Technology
- ISSN: 1516-8913
- Volume/Número/Paginação/Ano: v. 53, n. 2, p. 343-352, 2010
- Este artigo possui versão em acesso aberto
- URL de acesso aberto
- PDF de acesso aberto
- Versão do Documento: Versão publicada (Published version)
-
Status: Artigo publicado em periódico de acesso aberto (Gold Open Access) -
ABNT
CARNEIRO, Marcella Lemos Brettas et al. Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells. Brazilian Archives of Biology and Technology, v. 53, n. 2, p. 343-352, 2010Tradução . . Disponível em: https://doi.org/10.1590/S1516-89132010000600004. Acesso em: 13 mar. 2026. -
APA
Carneiro, M. L. B., Porfírio, E. P., Otake, A. H., Chammas, R., Báo, S. N., & Guillo, L. A. (2010). Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells. Brazilian Archives of Biology and Technology, 53( 2), 343-352. doi:10.1590/S1516-89132010000600004 -
NLM
Carneiro MLB, Porfírio EP, Otake AH, Chammas R, Báo SN, Guillo LA. Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells [Internet]. Brazilian Archives of Biology and Technology. 2010 ; 53( 2): 343-352.[citado 2026 mar. 13 ] Available from: https://doi.org/10.1590/S1516-89132010000600004 -
Vancouver
Carneiro MLB, Porfírio EP, Otake AH, Chammas R, Báo SN, Guillo LA. Morphological alterations and G0/G1 cell cycle arrest induced by curcumin in human SK-MEL-37 melanoma cells [Internet]. Brazilian Archives of Biology and Technology. 2010 ; 53( 2): 343-352.[citado 2026 mar. 13 ] Available from: https://doi.org/10.1590/S1516-89132010000600004 - Bradykinin-dependent pathways are activated within tumor microenvironments new targets for therapy
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