Endostatin gene therapy enhances the efficacy of IL-2 in suppressing metastatic renal cell carcinoma in mice (2010)
- Authors:
- Autor USP: CHAMMAS, ROGER - FM
- Unidade: FM
- DOI: 10.1007/s00262-010-0865-6
- Subjects: CAMUNDONGOS; MODELOS ANIMAIS; GENES (TERAPIA); METÁSTASE NEOPLÁSICA (PREVENÇÃO E CONTROLE)
- Language: Inglês
- Imprenta:
- Source:
- Título: Cancer Immunology Immunotherapy
- ISSN: 0340-7004
- Volume/Número/Paginação/Ano: v. 59, n. 9, p. 1357-1365, 2010
- Este artigo possui versão em acesso aberto
- URL de acesso aberto
- Versão do Documento: Versão submetida (Pré-print)
-
Status: Artigo possui versão em acesso aberto em repositório (Green Open Access) -
ABNT
ROCHA, Flávia Gomes de Góes et al. Endostatin gene therapy enhances the efficacy of IL-2 in suppressing metastatic renal cell carcinoma in mice. Cancer Immunology Immunotherapy, v. 59, n. 9, p. 1357-1365, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00262-010-0865-6. Acesso em: 14 mar. 2026. -
APA
Rocha, F. G. de G., Chaves, K. C. B., Chammas, R., Peron, J. P. S., Rizzo, L. V., Schor, N. S., & Bellini, · M. H. (2010). Endostatin gene therapy enhances the efficacy of IL-2 in suppressing metastatic renal cell carcinoma in mice. Cancer Immunology Immunotherapy, 59( 9), 1357-1365. doi:10.1007/s00262-010-0865-6 -
NLM
Rocha FG de G, Chaves KCB, Chammas R, Peron JPS, Rizzo LV, Schor NS, Bellini · MH. Endostatin gene therapy enhances the efficacy of IL-2 in suppressing metastatic renal cell carcinoma in mice [Internet]. Cancer Immunology Immunotherapy. 2010 ; 59( 9): 1357-1365.[citado 2026 mar. 14 ] Available from: https://doi.org/10.1007/s00262-010-0865-6 -
Vancouver
Rocha FG de G, Chaves KCB, Chammas R, Peron JPS, Rizzo LV, Schor NS, Bellini · MH. Endostatin gene therapy enhances the efficacy of IL-2 in suppressing metastatic renal cell carcinoma in mice [Internet]. Cancer Immunology Immunotherapy. 2010 ; 59( 9): 1357-1365.[citado 2026 mar. 14 ] Available from: https://doi.org/10.1007/s00262-010-0865-6 - Bradykinin-dependent pathways are activated within tumor microenvironments new targets for therapy
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