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  • Source: Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. Unidade: ICB

    Subjects: PARASITOLOGIA, GENÉTICA BACTERIANA, CROMATOGRAFIA LÍQUIDA, ÁCIDOS, CLOSTRIDIUM, ESTEROIDES, ESPECTROMETRIA

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      HARRIS, Spencer C. et al. Identification of a gene encoding a flavoprotein involved in bile acid metabolism by the human gut bacterium Clostridium scindens ATCC 35704. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids, v. 1863, n. 3, p. 276-283, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbalip.2017.12.001. Acesso em: 27 ago. 2024.
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      Harris, S. C., Devendran, S., Alves, J. M. P., Mythen, S. M., Hylemon, P. B., & Ridlon, J. M. (2018). Identification of a gene encoding a flavoprotein involved in bile acid metabolism by the human gut bacterium Clostridium scindens ATCC 35704. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids, 1863( 3), 276-283. doi:10.1016/j.bbalip.2017.12.001
    • NLM

      Harris SC, Devendran S, Alves JMP, Mythen SM, Hylemon PB, Ridlon JM. Identification of a gene encoding a flavoprotein involved in bile acid metabolism by the human gut bacterium Clostridium scindens ATCC 35704 [Internet]. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. 2018 ; 1863( 3): 276-283.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.bbalip.2017.12.001
    • Vancouver

      Harris SC, Devendran S, Alves JMP, Mythen SM, Hylemon PB, Ridlon JM. Identification of a gene encoding a flavoprotein involved in bile acid metabolism by the human gut bacterium Clostridium scindens ATCC 35704 [Internet]. Biochimica and Biophysica Acta. Molecular and Cell Biology of Lipids. 2018 ; 1863( 3): 276-283.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.bbalip.2017.12.001
  • Source: Journal of Infectious Diseases. Unidade: ICB

    Subjects: PARASITOLOGIA, PLASMODIUM, PROTEÍNAS, MALÁRIA, VACINAS

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      NTUMNGIA, Francis B. et al. Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a. Journal of Infectious Diseases, v. 218, n. 7, p. 1110-1118, 2018Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiy273. Acesso em: 27 ago. 2024.
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      Ntumngia, F. B., Thomson-Luque, R., Galusic, S., Frato, G., Frischmann, S., Peabody, D. S., et al. (2018). Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a. Journal of Infectious Diseases, 218( 7), 1110-1118. doi:10.1093/infdis/jiy273
    • NLM

      Ntumngia FB, Thomson-Luque R, Galusic S, Frato G, Frischmann S, Peabody DS, Chackerian B, Ferreira MU, King CL, Adams JH. Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a [Internet]. Journal of Infectious Diseases. 2018 ; 218( 7): 1110-1118.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1093/infdis/jiy273
    • Vancouver

      Ntumngia FB, Thomson-Luque R, Galusic S, Frato G, Frischmann S, Peabody DS, Chackerian B, Ferreira MU, King CL, Adams JH. Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a [Internet]. Journal of Infectious Diseases. 2018 ; 218( 7): 1110-1118.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1093/infdis/jiy273
  • Source: mBio. Unidade: ICB

    Subjects: PARASITOLOGIA, MEDULA ÓSSEA, PLASMODIUM, EXPRESSÃO GÊNICA, MALÁRIA

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      OBALDIA, Nicanor et al. Bone marrow is a major parasite reservoir in Plasmodium vivax infection. mBio, v. 9, n. 3, 2018Tradução . . Disponível em: https://doi.org/10.1128/mBio.00625-18. Acesso em: 27 ago. 2024.
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      Obaldia, N., Meibalan, E., Sa, J. M., Ma, S., Clark, M. A., Mejia, P., et al. (2018). Bone marrow is a major parasite reservoir in Plasmodium vivax infection. mBio, 9( 3). doi:10.1128/mBio.00625-18
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      Obaldia N, Meibalan E, Sa JM, Ma S, Clark MA, Mejia P, Barros RRM, Otero W, Ferreira MU, Mitchell JR, Milner DA, Huttenhower C, Duraisingh MT, Wellems TE, Marti M. Bone marrow is a major parasite reservoir in Plasmodium vivax infection [Internet]. mBio. 2018 ; 9( 3):[citado 2024 ago. 27 ] Available from: https://doi.org/10.1128/mBio.00625-18
    • Vancouver

      Obaldia N, Meibalan E, Sa JM, Ma S, Clark MA, Mejia P, Barros RRM, Otero W, Ferreira MU, Mitchell JR, Milner DA, Huttenhower C, Duraisingh MT, Wellems TE, Marti M. Bone marrow is a major parasite reservoir in Plasmodium vivax infection [Internet]. mBio. 2018 ; 9( 3):[citado 2024 ago. 27 ] Available from: https://doi.org/10.1128/mBio.00625-18
  • Source: Protein Expression and Purification. Unidades: IQSC, ICB

    Subjects: NEOPLASIAS, PARASITOLOGIA, ESCHERICHIA COLI, BIOMARCADORES

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      MIRANDA, Beatriz Nogueira Messias de et al. Heterologous expression of Homo sapiens alpha-folate receptors in E. colli by fusion with a trigger factor for enhanced solubilization. Protein Expression and Purification, v. 142, p. 75-80, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.pep.2017.10.006. Acesso em: 27 ago. 2024.
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      Miranda, B. N. M. de, Fotoran, W. L., Canduri, F., Souza, D. H. F. de, Wunderlich, G., & Carrilho, E. (2018). Heterologous expression of Homo sapiens alpha-folate receptors in E. colli by fusion with a trigger factor for enhanced solubilization. Protein Expression and Purification, 142, 75-80. doi:10.1016/j.pep.2017.10.006
    • NLM

      Miranda BNM de, Fotoran WL, Canduri F, Souza DHF de, Wunderlich G, Carrilho E. Heterologous expression of Homo sapiens alpha-folate receptors in E. colli by fusion with a trigger factor for enhanced solubilization [Internet]. Protein Expression and Purification. 2018 ; 142 75-80.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.pep.2017.10.006
    • Vancouver

      Miranda BNM de, Fotoran WL, Canduri F, Souza DHF de, Wunderlich G, Carrilho E. Heterologous expression of Homo sapiens alpha-folate receptors in E. colli by fusion with a trigger factor for enhanced solubilization [Internet]. Protein Expression and Purification. 2018 ; 142 75-80.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.pep.2017.10.006
  • Source: Antimicrobial Agents and Chemotherapy. Unidade: ICB

    Subjects: PARASITOLOGIA, ÁCIDOS, METABOLISMO, CLOSTRIDIUM

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      DODEN, Heidi et al. Metabolism of oxo-bile acids and characterization of recombinant 12α-hydroxysteroid dehydrogenases from bile acid 7α-dehydroxylating human gut bacteria. Antimicrobial Agents and Chemotherapy, v. 84, n. 10, p. e00235-18, 2018Tradução . . Disponível em: https://doi.org/10.1128/AEM.00235-18. Acesso em: 27 ago. 2024.
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      Doden, H., Sallam, L. A., Devendran, S., Ly, L., Doden, G., Daniel, S. L., et al. (2018). Metabolism of oxo-bile acids and characterization of recombinant 12α-hydroxysteroid dehydrogenases from bile acid 7α-dehydroxylating human gut bacteria. Antimicrobial Agents and Chemotherapy, 84( 10), e00235-18. doi:10.1128/AEM.00235-18
    • NLM

      Doden H, Sallam LA, Devendran S, Ly L, Doden G, Daniel SL, Alves JMP, Ridlon JM. Metabolism of oxo-bile acids and characterization of recombinant 12α-hydroxysteroid dehydrogenases from bile acid 7α-dehydroxylating human gut bacteria [Internet]. Antimicrobial Agents and Chemotherapy. 2018 ; 84( 10): e00235-18.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1128/AEM.00235-18
    • Vancouver

      Doden H, Sallam LA, Devendran S, Ly L, Doden G, Daniel SL, Alves JMP, Ridlon JM. Metabolism of oxo-bile acids and characterization of recombinant 12α-hydroxysteroid dehydrogenases from bile acid 7α-dehydroxylating human gut bacteria [Internet]. Antimicrobial Agents and Chemotherapy. 2018 ; 84( 10): e00235-18.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1128/AEM.00235-18
  • Source: Developmental Cell. Unidade: ICB

    Subjects: FLUXO SANGUÍNEO, CÉLULAS CULTIVADAS DE TUMOR, METÁSTASE NEOPLÁSICA, NEOPLASIAS

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      FREITAS, Vanessa Morais e HILFENHAUS, Georg e ARISPE, Maria Luisa Iruela. Metastasis of circulating tumor cells: speed matters. Developmental Cell, v. 45, n. 1, p. 3-5, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.devcel.2018.03.005. Acesso em: 27 ago. 2024.
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      Freitas, V. M., Hilfenhaus, G., & Arispe, M. L. I. (2018). Metastasis of circulating tumor cells: speed matters. Developmental Cell, 45( 1), 3-5. doi:10.1016/j.devcel.2018.03.005
    • NLM

      Freitas VM, Hilfenhaus G, Arispe MLI. Metastasis of circulating tumor cells: speed matters [Internet]. Developmental Cell. 2018 ; 45( 1): 3-5.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.devcel.2018.03.005
    • Vancouver

      Freitas VM, Hilfenhaus G, Arispe MLI. Metastasis of circulating tumor cells: speed matters [Internet]. Developmental Cell. 2018 ; 45( 1): 3-5.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.devcel.2018.03.005
  • Source: Journal of Proteomics. Unidades: IQ, ICB

    Subjects: TRYPANOSOMA CRUZI, GLICOPROTEÍNAS, ESPECTROMETRIA DE MASSAS

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      ALVES, Maria Júlia Manso et al. Comprehensive glycoprofiling of the epimastigote and trypomastigote stages of Trypanosoma cruzi. Journal of Proteomics, v. 151, p. 182-192, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jprot.2016.05.034. Acesso em: 27 ago. 2024.
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      Alves, M. J. M., Kawahara, R., Viner, R., Colli, W., Mattos, E. C., Thaysen Andersen, M., et al. (2017). Comprehensive glycoprofiling of the epimastigote and trypomastigote stages of Trypanosoma cruzi. Journal of Proteomics, 151, 182-192. doi:10.1016/j.jprot.2016.05.034
    • NLM

      Alves MJM, Kawahara R, Viner R, Colli W, Mattos EC, Thaysen Andersen M, Larsen MR, Palmisano G. Comprehensive glycoprofiling of the epimastigote and trypomastigote stages of Trypanosoma cruzi [Internet]. Journal of Proteomics. 2017 ; 151 182-192.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.jprot.2016.05.034
    • Vancouver

      Alves MJM, Kawahara R, Viner R, Colli W, Mattos EC, Thaysen Andersen M, Larsen MR, Palmisano G. Comprehensive glycoprofiling of the epimastigote and trypomastigote stages of Trypanosoma cruzi [Internet]. Journal of Proteomics. 2017 ; 151 182-192.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.jprot.2016.05.034
  • Source: Fish Physiology and Biochemistry. Unidade: ICB

    Assunto: HISTOLOGIA

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      NÓBREGA, Rafael Henrique et al. Characterization of gonadotropic cells during continuous and seasonal spermatogenesis of two freshwater fish species: a histochemical and immunohistochemical study. Fish Physiology and Biochemistry, v. 43, n. 1, p. 51-63, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10695-016-0267-6. Acesso em: 27 ago. 2024.
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      Nóbrega, R. H., Jesus, L. W. O. de, Honji, R. M., & Borella, M. I. (2017). Characterization of gonadotropic cells during continuous and seasonal spermatogenesis of two freshwater fish species: a histochemical and immunohistochemical study. Fish Physiology and Biochemistry, 43( 1), 51-63. doi:10.1007/s10695-016-0267-6
    • NLM

      Nóbrega RH, Jesus LWO de, Honji RM, Borella MI. Characterization of gonadotropic cells during continuous and seasonal spermatogenesis of two freshwater fish species: a histochemical and immunohistochemical study [Internet]. Fish Physiology and Biochemistry. 2017 ; 43( 1): 51-63.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1007/s10695-016-0267-6
    • Vancouver

      Nóbrega RH, Jesus LWO de, Honji RM, Borella MI. Characterization of gonadotropic cells during continuous and seasonal spermatogenesis of two freshwater fish species: a histochemical and immunohistochemical study [Internet]. Fish Physiology and Biochemistry. 2017 ; 43( 1): 51-63.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1007/s10695-016-0267-6
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SOUZA, Arnaldo H. de et al. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, v. 439, p. 354-362, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.09.022. Acesso em: 27 ago. 2024.
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      Souza, A. H. de, Santos, L. R. B., Roma, L. P., Bensellam, M., Carpinelli, A. R., & Jonas, J. -C. (2017). NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, 439, 354-362. doi:10.1016/j.mce.2016.09.022
    • NLM

      Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
    • Vancouver

      Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
  • Source: Nature Communications. Unidade: ICB

    Subjects: MICROBIOLOGIA, LINFÓCITOS T, VACINAS, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS

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      NIZARD, Mevyn et al. Induction of resident memory T cells enhances the efficacy of cancer. Nature Communications, v. 8, n. 15221, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1038/ncomms15221. Acesso em: 27 ago. 2024.
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      Nizard, M., Roussel, H., Diniz, M. de O., Karaki, S., Tran, T., Voron, T., et al. (2017). Induction of resident memory T cells enhances the efficacy of cancer. Nature Communications, 8( 15221), 1-11. doi:10.1038/ncomms15221
    • NLM

      Nizard M, Roussel H, Diniz M de O, Karaki S, Tran T, Voron T, Dransart E, Sandoval F, Riquet M, Rance B, Marcheteau E, Fabre E, Mandavit M, Terme M, Blanc C, Escudie J-B, Gibault L, Barthes FLP, Granier C, Ferreira LC de S, Badoual C, Johannes L, Tartour E. Induction of resident memory T cells enhances the efficacy of cancer [Internet]. Nature Communications. 2017 ; 8( 15221): 1-11.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1038/ncomms15221
    • Vancouver

      Nizard M, Roussel H, Diniz M de O, Karaki S, Tran T, Voron T, Dransart E, Sandoval F, Riquet M, Rance B, Marcheteau E, Fabre E, Mandavit M, Terme M, Blanc C, Escudie J-B, Gibault L, Barthes FLP, Granier C, Ferreira LC de S, Badoual C, Johannes L, Tartour E. Induction of resident memory T cells enhances the efficacy of cancer [Internet]. Nature Communications. 2017 ; 8( 15221): 1-11.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1038/ncomms15221
  • Source: Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      FERREIRA-NETO, H. C. et al. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation. Neuroscience, v. 349, p. 253-263, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2017.02.05. Acesso em: 27 ago. 2024.
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      Ferreira-Neto, H. C., Ribeiro, I. M. R., Moreira, T. dos S., Yao, S. T., & Antunes, V. R. (2017). Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation. Neuroscience, 349, 253-263. doi:10.1016/j.neuroscience.2017.02.05
    • NLM

      Ferreira-Neto HC, Ribeiro IMR, Moreira T dos S, Yao ST, Antunes VR. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation [Internet]. Neuroscience. 2017 ; 349 253-263.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.05
    • Vancouver

      Ferreira-Neto HC, Ribeiro IMR, Moreira T dos S, Yao ST, Antunes VR. Purinergic P2 receptors in the paraventricular nucleus of the hypothalamus are involved in hyperosmotic-induced sympathoexcitation [Internet]. Neuroscience. 2017 ; 349 253-263.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.neuroscience.2017.02.05
  • Source: Annals of Human Genetics. Unidade: ICB

    Assunto: HISTOLOGIA

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      COSTA-URRUTIA, Paula et al. Genetic obesity risk and attenuation effect of physical fitness in mexican-mestizo population: a case-control study. Annals of Human Genetics, v. 81, n. 3, p. 106-116, 2017Tradução . . Disponível em: https://doi.org/10.1111/ahg.12190. Acesso em: 27 ago. 2024.
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      Costa-Urrutia, P., Abud, C., Franco-Trecu, V., Colistro, V., Rodríguez‐Arellano, M. E., Vázquez‐Pérez, J., et al. (2017). Genetic obesity risk and attenuation effect of physical fitness in mexican-mestizo population: a case-control study. Annals of Human Genetics, 81( 3), 106-116. doi:10.1111/ahg.12190
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      Costa-Urrutia P, Abud C, Franco-Trecu V, Colistro V, Rodríguez‐Arellano ME, Vázquez‐Pérez J, Granados J, Seelaender M. Genetic obesity risk and attenuation effect of physical fitness in mexican-mestizo population: a case-control study [Internet]. Annals of Human Genetics. 2017 ; 81( 3): 106-116.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1111/ahg.12190
    • Vancouver

      Costa-Urrutia P, Abud C, Franco-Trecu V, Colistro V, Rodríguez‐Arellano ME, Vázquez‐Pérez J, Granados J, Seelaender M. Genetic obesity risk and attenuation effect of physical fitness in mexican-mestizo population: a case-control study [Internet]. Annals of Human Genetics. 2017 ; 81( 3): 106-116.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1111/ahg.12190
  • Source: Acta Physiologica. Unidade: ICB

    Assunto: FISIOLOGIA

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      TEIXEIRA, S. da Silva et al. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice. Acta Physiologica, v. 220, n. 2, p. 238-250, 2017Tradução . . Disponível em: https://doi.org/10.1111/apha.12821. Acesso em: 27 ago. 2024.
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      Teixeira, S. da S., Filgueira, C., Sieglaff, D. H., Benod , C., Villagomez, R., Minze, L. J., et al. (2017). 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice. Acta Physiologica, 220( 2), 238-250. doi:10.1111/apha.12821
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      Teixeira S da S, Filgueira C, Sieglaff DH, Benod C, Villagomez R, Minze LJ, Zhang A, Webb P, Nunes MT. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice [Internet]. Acta Physiologica. 2017 ; 220( 2): 238-250.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1111/apha.12821
    • Vancouver

      Teixeira S da S, Filgueira C, Sieglaff DH, Benod C, Villagomez R, Minze LJ, Zhang A, Webb P, Nunes MT. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice [Internet]. Acta Physiologica. 2017 ; 220( 2): 238-250.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1111/apha.12821
  • Source: Tropical Medicine & International Health. Unidade: ICB

    Assunto: PARASITOLOGIA

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      SOUZA, Higo Fernando Santos et al. Development and in vitro/in vivo evaluation of a novel benznidazole liquid dosage form using a quality-by-design approach. Tropical Medicine & International Health, v. 22, n. 12, p. 1514-1522, 2017Tradução . . Disponível em: https://doi.org/10.1111/tmi.12980. Acesso em: 27 ago. 2024.
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      Souza, H. F. S., Real, D., Leonardi, D., Rocha, S. C., Alonso, V., Serra, E., et al. (2017). Development and in vitro/in vivo evaluation of a novel benznidazole liquid dosage form using a quality-by-design approach. Tropical Medicine & International Health, 22( 12), 1514-1522. doi:10.1111/tmi.12980
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      Souza HFS, Real D, Leonardi D, Rocha SC, Alonso V, Serra E, Silber AM, Salomon CJ. Development and in vitro/in vivo evaluation of a novel benznidazole liquid dosage form using a quality-by-design approach [Internet]. Tropical Medicine & International Health. 2017 ; 22( 12): 1514-1522.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1111/tmi.12980
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      Souza HFS, Real D, Leonardi D, Rocha SC, Alonso V, Serra E, Silber AM, Salomon CJ. Development and in vitro/in vivo evaluation of a novel benznidazole liquid dosage form using a quality-by-design approach [Internet]. Tropical Medicine & International Health. 2017 ; 22( 12): 1514-1522.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1111/tmi.12980
  • Source: Cancer Genomics & Proteomics. Unidade: ICB

    Assunto: HISTOLOGIA

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      PAIVA, Marcelo M. e KIMURA, Edna Teruko e COLTRI, Patricia Pereira. miR18a and miR19a recruit specific proteins for splicing in thyroid cancer cells. Cancer Genomics & Proteomics, v. 14, n. 5, p. 373-381, 2017Tradução . . Disponível em: https://doi.org/10.21873/cgp.20047. Acesso em: 27 ago. 2024.
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      Paiva, M. M., Kimura, E. T., & Coltri, P. P. (2017). miR18a and miR19a recruit specific proteins for splicing in thyroid cancer cells. Cancer Genomics & Proteomics, 14( 5), 373-381. doi:10.21873/cgp.20047
    • NLM

      Paiva MM, Kimura ET, Coltri PP. miR18a and miR19a recruit specific proteins for splicing in thyroid cancer cells [Internet]. Cancer Genomics & Proteomics. 2017 ; 14( 5): 373-381.[citado 2024 ago. 27 ] Available from: https://doi.org/10.21873/cgp.20047
    • Vancouver

      Paiva MM, Kimura ET, Coltri PP. miR18a and miR19a recruit specific proteins for splicing in thyroid cancer cells [Internet]. Cancer Genomics & Proteomics. 2017 ; 14( 5): 373-381.[citado 2024 ago. 27 ] Available from: https://doi.org/10.21873/cgp.20047
  • Source: Neurotoxicity Research. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      RAVELLI, Katherine Garcia et al. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, v. 31, n. 3, p. 327-333, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12640-016-9684-7. Acesso em: 27 ago. 2024.
    • APA

      Ravelli, K. G., Rosário, B. dos A., Camarini, R., Hernandes, M. S., & Britto, L. R. G. de. (2017). Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice. Neurotoxicity Research, 31( 3), 327-333. doi:10.1007/s12640-016-9684-7
    • NLM

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1007/s12640-016-9684-7
    • Vancouver

      Ravelli KG, Rosário B dos A, Camarini R, Hernandes MS, Britto LRG de. Intracerebroventricular streptozotocin as a model of Alzheimer’s disease: neurochemical and behavioral characterization in mice [Internet]. Neurotoxicity Research. 2017 ; 31( 3): 327-333.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1007/s12640-016-9684-7
  • Source: Endocrinology. Unidade: ICB

    Assunto: HISTOLOGIA

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      DAUBRIAC, Julien et al. Hormonal and growth regulation of epithelial and stromal cells from the normal and malignant endometrium by pigment epithelium-derived factor. Endocrinology, v. 158, n. 9, p. 2754-2773, 2017Tradução . . Disponível em: https://doi.org/10.1210/en.2017-00028. Acesso em: 27 ago. 2024.
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      Daubriac, J., Pandya, U. M., Huang, K. -T., Pavlides, S. C., Gama, P., Blank, S. V., et al. (2017). Hormonal and growth regulation of epithelial and stromal cells from the normal and malignant endometrium by pigment epithelium-derived factor. Endocrinology, 158( 9), 2754-2773. doi:10.1210/en.2017-00028
    • NLM

      Daubriac J, Pandya UM, Huang K-T, Pavlides SC, Gama P, Blank SV, Pratibha Shukla, Crawford SE, Gold LI. Hormonal and growth regulation of epithelial and stromal cells from the normal and malignant endometrium by pigment epithelium-derived factor [Internet]. Endocrinology. 2017 ; 158( 9): 2754-2773.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1210/en.2017-00028
    • Vancouver

      Daubriac J, Pandya UM, Huang K-T, Pavlides SC, Gama P, Blank SV, Pratibha Shukla, Crawford SE, Gold LI. Hormonal and growth regulation of epithelial and stromal cells from the normal and malignant endometrium by pigment epithelium-derived factor [Internet]. Endocrinology. 2017 ; 158( 9): 2754-2773.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1210/en.2017-00028
  • Source: Placenta. Unidade: ICB

    Assunto: HISTOLOGIA

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      SANCHES, Juliane C. et al. Distinct effects of short- and long-term type 1 diabetes to the placental extracellular matrix and fetal development in mice. Placenta, v. 53, p. 1-7, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.placenta.2017.03.005. Acesso em: 27 ago. 2024.
    • APA

      Sanches, J. C., Favaro, R. R., Barrence, F. C., Bevilacqua, E. M. A. F., Fortes, Z. B., & Zorn, T. M. T. (2017). Distinct effects of short- and long-term type 1 diabetes to the placental extracellular matrix and fetal development in mice. Placenta, 53, 1-7. doi:10.1016/j.placenta.2017.03.005
    • NLM

      Sanches JC, Favaro RR, Barrence FC, Bevilacqua EMAF, Fortes ZB, Zorn TMT. Distinct effects of short- and long-term type 1 diabetes to the placental extracellular matrix and fetal development in mice [Internet]. Placenta. 2017 ; 53 1-7.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.placenta.2017.03.005
    • Vancouver

      Sanches JC, Favaro RR, Barrence FC, Bevilacqua EMAF, Fortes ZB, Zorn TMT. Distinct effects of short- and long-term type 1 diabetes to the placental extracellular matrix and fetal development in mice [Internet]. Placenta. 2017 ; 53 1-7.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.placenta.2017.03.005
  • Source: Journal of Diabetes and its Complications. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      FILGUEIRAS, Luciano Ribeiro et al. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, v. 31, n. 2, p. 334-339, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2016.08.001. Acesso em: 27 ago. 2024.
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      Filgueiras, L. R., Brandt, S. L., Ramalho, T. R. de O., Jancar, S., & Serezani, C. H. (2017). Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, 31( 2), 334-339. doi:10.1016/j.jdiacomp.2016.08.001
    • NLM

      Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001
    • Vancouver

      Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.08.001
  • Source: Molecular and Cellular Endocrinology. Unidades: FMRP, ICB

    Subjects: ANATOMIA, FISIOLOGIA

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    • ABNT

      SILVEIRA, Marina Augusto et al. STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, v. 448, p. 55-65, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2017.03.024. Acesso em: 27 ago. 2024.
    • APA

      Silveira, M. A., Furigo, I. C., Zampieri, T. T., Bohlen, T. M., Paula, D. G. de, Franci, C. R., et al. (2017). STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, 448, 55-65. doi:10.1016/j.mce.2017.03.024
    • NLM

      Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024
    • Vancouver

      Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 ago. 27 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024

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