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Artigo de periodico
Proteomic profiling of HDL in newly diagnosed breast cancer based on tumor molecular classification and clinical stage of disease (2024)
Santana, Monique de Fátima Mello ; Sawada, Maria Isabela Bloise Alves Caldas ; Souza Junior, Douglas Ricardo de ; Giacaglia, Marcia Benacchio; Reis, Mozania; Xavier, Jacira; Giannella, Maria Lucia Correa ; Soriano, Francisco Garcia ; Gebrim, Luiz Henrique ; Ronsein, Graziella Eliza ; Passarelli, Marisa
Source: Cells. Unidades: FM, IQ
Subjects: NEOPLASIAS MAMÁRIAS, PROTEÔMICA
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SANTANA, Monique de Fátima Mello et al. Proteomic profiling of HDL in newly diagnosed breast cancer based on tumor molecular classification and clinical stage of disease. Cells, v. 13, p. 1-19 art. 1327, 2024Tradução . . Disponível em: https://dx.doi.org/10.3390/cells13161327. Acesso em: 18 nov. 2024.
APA
Santana, M. de F. M., Sawada, M. I. B. A. C., Souza Junior, D. R. de, Giacaglia, M. B., Reis, M., Xavier, J., et al. (2024). Proteomic profiling of HDL in newly diagnosed breast cancer based on tumor molecular classification and clinical stage of disease. Cells, 13, 1-19 art. 1327. doi:10.3390/cells13161327
NLM
Santana M de FM, Sawada MIBAC, Souza Junior DR de, Giacaglia MB, Reis M, Xavier J, Giannella MLC, Soriano FG, Gebrim LH, Ronsein GE, Passarelli M. Proteomic profiling of HDL in newly diagnosed breast cancer based on tumor molecular classification and clinical stage of disease [Internet]. Cells. 2024 ; 13 1-19 art. 1327.[citado 2024 nov. 18 ] Available from: https://dx.doi.org/10.3390/cells13161327
Vancouver
Santana M de FM, Sawada MIBAC, Souza Junior DR de, Giacaglia MB, Reis M, Xavier J, Giannella MLC, Soriano FG, Gebrim LH, Ronsein GE, Passarelli M. Proteomic profiling of HDL in newly diagnosed breast cancer based on tumor molecular classification and clinical stage of disease [Internet]. Cells. 2024 ; 13 1-19 art. 1327.[citado 2024 nov. 18 ] Available from: https://dx.doi.org/10.3390/cells13161327
Artigo de periodico
Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time (2023)
Leite, Giuseppe Gianini Figueiredo ; Brunialti, Milena Karina Colo ; Pietrobom, Paula M. Peçanha ; Ferreira, Paulo Roberto Abrão ; Arakaki, Jaquelina Sonoe Ota ; Cunha Neto, Edecio ; Ferreira, Bianca Lima ; Ronsein, Graziella Eliza ; Tashima, Alexandre Keiji ; Salomão, Reinaldo
Source: iScience. Unidades: FM, IQ
Subjects: COVID-19, PROTEÔMICA
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ABNT
LEITE, Giuseppe Gianini Figueiredo et al. Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time. iScience, v. 26, n. 10, p. 1-16, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.107824. Acesso em: 18 nov. 2024.
APA
Leite, G. G. F., Brunialti, M. K. C., Pietrobom, P. M. P., Ferreira, P. R. A., Arakaki, J. S. O., Cunha Neto, E., et al. (2023). Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time. iScience, 26( 10), 1-16. doi:10.1016/j.isci.2023.107824
NLM
Leite GGF, Brunialti MKC, Pietrobom PMP, Ferreira PRA, Arakaki JSO, Cunha Neto E, Ferreira BL, Ronsein GE, Tashima AK, Salomão R. Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time [Internet]. iScience. 2023 ; 26( 10): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.isci.2023.107824
Vancouver
Leite GGF, Brunialti MKC, Pietrobom PMP, Ferreira PRA, Arakaki JSO, Cunha Neto E, Ferreira BL, Ronsein GE, Tashima AK, Salomão R. Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time [Internet]. iScience. 2023 ; 26( 10): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.isci.2023.107824
Artigo de periodico
Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display (2023)
Carnero, Luis Antonio Rodriguez ; Kuramoto, Andréia; Oliveira, Lea Campos de; Monteiro, Jhonatas Sirino ; Setubal, João Carlos ; Cunha Neto, Edecio ; Sabino, Ester Cerdeira ; Giordano, Ricardo José
Source: Plos Neglected Tropical Diseases. Unidades: IQ, FM, CEBIMAR
Subjects: DOENÇA DE CHAGAS, GENOMAS, ANTICORPOS, TRYPANOSOMA CRUZI
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CARNERO, Luis Antonio Rodriguez et al. Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display. Plos Neglected Tropical Diseases, v. 17, n. 1, p. 1-20 art. e0011019, 2023Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0011019. Acesso em: 18 nov. 2024.
APA
Carnero, L. A. R., Kuramoto, A., Oliveira, L. C. de, Monteiro, J. S., Setubal, J. C., Cunha Neto, E., et al. (2023). Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display. Plos Neglected Tropical Diseases, 17( 1), 1-20 art. e0011019. doi:10.1371/journal.pntd.0011019
NLM
Carnero LAR, Kuramoto A, Oliveira LC de, Monteiro JS, Setubal JC, Cunha Neto E, Sabino EC, Giordano RJ. Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display [Internet]. Plos Neglected Tropical Diseases. 2023 ; 17( 1): 1-20 art. e0011019.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pntd.0011019
Vancouver
Carnero LAR, Kuramoto A, Oliveira LC de, Monteiro JS, Setubal JC, Cunha Neto E, Sabino EC, Giordano RJ. Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display [Internet]. Plos Neglected Tropical Diseases. 2023 ; 17( 1): 1-20 art. e0011019.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1371/journal.pntd.0011019
Artigo de periodico
Cathelicidin protects the brain from mitochondrial DNA damage in health, but not following septic shock (2023)
Barbeiro, Denise Frediani; Ariga, Suely Kubo; Barbeiro, Hermes Vieira ; Souza-Pinto, Nadja Cristhina de ; Silva, Fabiano Pinheiro da
Source: Journal of Intensive and Critical Care. Unidades: FM, IQ
Subjects: IMUNIDADE, CÉREBRO, DANO AO DNA, SEPSE, INFLAMAÇÃO
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ABNT
BARBEIRO, Denise Frediani et al. Cathelicidin protects the brain from mitochondrial DNA damage in health, but not following septic shock. Journal of Intensive and Critical Care, v. 9 p. 1-4, 2023Tradução . . Disponível em: https://dx.doi.org/10.21203/rs.3.rs-514325/v1. Acesso em: 18 nov. 2024.
APA
Barbeiro, D. F., Ariga, S. K., Barbeiro, H. V., Souza-Pinto, N. C. de, & Silva, F. P. da. (2023). Cathelicidin protects the brain from mitochondrial DNA damage in health, but not following septic shock. Journal of Intensive and Critical Care, 9 p. 1-4. doi:10.21203/rs.3.rs-514325/v1
NLM
Barbeiro DF, Ariga SK, Barbeiro HV, Souza-Pinto NC de, Silva FP da. Cathelicidin protects the brain from mitochondrial DNA damage in health, but not following septic shock [Internet]. Journal of Intensive and Critical Care. 2023 ; 9 p. 1-4[citado 2024 nov. 18 ] Available from: https://dx.doi.org/10.21203/rs.3.rs-514325/v1
Vancouver
Barbeiro DF, Ariga SK, Barbeiro HV, Souza-Pinto NC de, Silva FP da. Cathelicidin protects the brain from mitochondrial DNA damage in health, but not following septic shock [Internet]. Journal of Intensive and Critical Care. 2023 ; 9 p. 1-4[citado 2024 nov. 18 ] Available from: https://dx.doi.org/10.21203/rs.3.rs-514325/v1
Artigo de periodico
Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study (2023)
Toyoshima, Marcos Tadashi Kakitani ; Santana, Monique de Fatima Mello; Silva, Amanda R. M; Mello, Gabriela B; Bezerra, Daniele P. Santos; Góes, Marisa F. S; Bosco, Adriana A; Caramelli, Bruno ; Ronsein, Graziella Eliza ; Giannella, Maria Lucia Correa ; Passarelli, Marisa
Source: Diabetology and Metabolic Syndrome. Unidades: FM, IQ
Subjects: PROTEÔMICA, DIABETES MELLITUS, ARTERIOSCLEROSE
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ABNT
TOYOSHIMA, Marcos Tadashi Kakitani et al. Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study. Diabetology and Metabolic Syndrome, v. 15, p. 1-14 art. 42, 2023Tradução . . Disponível em: https://doi.org/10.1186/s13098-023-01007-y. Acesso em: 18 nov. 2024.
APA
Toyoshima, M. T. K., Santana, M. de F. M., Silva, A. R. M., Mello, G. B., Bezerra, D. P. S., Góes, M. F. S., et al. (2023). Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study. Diabetology and Metabolic Syndrome, 15, 1-14 art. 42. doi:10.1186/s13098-023-01007-y
NLM
Toyoshima MTK, Santana M de FM, Silva ARM, Mello GB, Bezerra DPS, Góes MFS, Bosco AA, Caramelli B, Ronsein GE, Giannella MLC, Passarelli M. Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study [Internet]. Diabetology and Metabolic Syndrome. 2023 ; 15 1-14 art. 42.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s13098-023-01007-y
Vancouver
Toyoshima MTK, Santana M de FM, Silva ARM, Mello GB, Bezerra DPS, Góes MFS, Bosco AA, Caramelli B, Ronsein GE, Giannella MLC, Passarelli M. Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study [Internet]. Diabetology and Metabolic Syndrome. 2023 ; 15 1-14 art. 42.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s13098-023-01007-y
Artigo de periodico
Identification of two patterns of mitochondrial DNA-copy number variation in postcentral gyrus during aging, influenced by body mass index and type 2 diabetes (2022)
Sekiya, Felipe Seiti; Silva, Clarisse Pereira Nunes da; Shinjo, Sueli Mieko Oba ; Bezerra, Daniele Pereira Santos ; Ravagnani, Felipe Gustavo ; Pasqualucci, Carlos Augusto Goncalves ; Gil, Saulo ; Gualano, Bruno ; Baptista, Maurício da Silva ; Giannella, Maria Lucia Correa ; Marie, Suely Kazue Nagahashi
Source: Experimental Gerontology. Unidades: FM, IQ
Subjects: ESTRESSE OXIDATIVO, MITOCÔNDRIAS, INFLAMAÇÃO, DOENÇAS CEREBRAIS, ENVELHECIMENTO
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ABNT
SEKIYA, Felipe Seiti et al. Identification of two patterns of mitochondrial DNA-copy number variation in postcentral gyrus during aging, influenced by body mass index and type 2 diabetes. Experimental Gerontology, v. 168, p. 1-8 art. 111932, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2022.111932. Acesso em: 18 nov. 2024.
APA
Sekiya, F. S., Silva, C. P. N. da, Shinjo, S. M. O., Bezerra, D. P. S., Ravagnani, F. G., Pasqualucci, C. A. G., et al. (2022). Identification of two patterns of mitochondrial DNA-copy number variation in postcentral gyrus during aging, influenced by body mass index and type 2 diabetes. Experimental Gerontology, 168, 1-8 art. 111932. doi:10.1016/j.exger.2022.111932
NLM
Sekiya FS, Silva CPN da, Shinjo SMO, Bezerra DPS, Ravagnani FG, Pasqualucci CAG, Gil S, Gualano B, Baptista M da S, Giannella MLC, Marie SKN. Identification of two patterns of mitochondrial DNA-copy number variation in postcentral gyrus during aging, influenced by body mass index and type 2 diabetes [Internet]. Experimental Gerontology. 2022 ; 168 1-8 art. 111932.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.exger.2022.111932
Vancouver
Sekiya FS, Silva CPN da, Shinjo SMO, Bezerra DPS, Ravagnani FG, Pasqualucci CAG, Gil S, Gualano B, Baptista M da S, Giannella MLC, Marie SKN. Identification of two patterns of mitochondrial DNA-copy number variation in postcentral gyrus during aging, influenced by body mass index and type 2 diabetes [Internet]. Experimental Gerontology. 2022 ; 168 1-8 art. 111932.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.exger.2022.111932
Artigo de jornal
Estudo ajuda a entender como portadores de doença renal hereditária desenvolvem disfunção cardíaca (2022)
Amaral, Andressa Godoy ; Onuchic, Luiz Fernando ; Kowaltowski, Alicia Juliana ; Miyamoto, Sayuri
Source: Agência FAPESP. Unidades: FM, IQ
Subjects: NEFROPATIAS, DOENÇAS CARDIOVASCULARES
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ABNT
AMARAL, Andressa Godoy et al. Estudo ajuda a entender como portadores de doença renal hereditária desenvolvem disfunção cardíaca. Tradução . Agência FAPESP, São Paulo, 2022. , n. p.Disponível em: https://agencia.fapesp.br/estudo-ajuda-a-entender-como-portadores-de-doenca-renal-hereditaria-desenvolvem-disfuncao-cardiaca/38215/#:~:text=Ag*C3*AAncia%FAPESP*%*E2*80*93%Estudo%conduzido%no,desenvolvimento%de%disfun*C3*A7*C3*A3o%card*C3*ADaca*2C%hoje. Acesso em: 18 nov. 2024.
APA
Amaral, A. G., Onuchic, L. F., Kowaltowski, A. J., & Miyamoto, S. (2022). Estudo ajuda a entender como portadores de doença renal hereditária desenvolvem disfunção cardíaca. Agência FAPESP. São Paulo: Faculdade de Medicina, Universidade de São Paulo. Recuperado de https://agencia.fapesp.br/estudo-ajuda-a-entender-como-portadores-de-doenca-renal-hereditaria-desenvolvem-disfuncao-cardiaca/38215/#:~:text=Ag*C3*AAncia%FAPESP*%*E2*80*93%Estudo%conduzido%no,desenvolvimento%de%disfun*C3*A7*C3*A3o%card*C3*ADaca*2C%hoje
NLM
Amaral AG, Onuchic LF, Kowaltowski AJ, Miyamoto S. Estudo ajuda a entender como portadores de doença renal hereditária desenvolvem disfunção cardíaca [Internet]. Agência FAPESP. 2022 ;( p.):[citado 2024 nov. 18 ] Available from: https://agencia.fapesp.br/estudo-ajuda-a-entender-como-portadores-de-doenca-renal-hereditaria-desenvolvem-disfuncao-cardiaca/38215/#:~:text=Ag*C3*AAncia%FAPESP*%*E2*80*93%Estudo%conduzido%no,desenvolvimento%de%disfun*C3*A7*C3*A3o%card*C3*ADaca*2C%hoje
Vancouver
Amaral AG, Onuchic LF, Kowaltowski AJ, Miyamoto S. Estudo ajuda a entender como portadores de doença renal hereditária desenvolvem disfunção cardíaca [Internet]. Agência FAPESP. 2022 ;( p.):[citado 2024 nov. 18 ] Available from: https://agencia.fapesp.br/estudo-ajuda-a-entender-como-portadores-de-doenca-renal-hereditaria-desenvolvem-disfuncao-cardiaca/38215/#:~:text=Ag*C3*AAncia%FAPESP*%*E2*80*93%Estudo%conduzido%no,desenvolvimento%de%disfun*C3*A7*C3*A3o%card*C3*ADaca*2C%hoje
Artigo de periodico
Drug repurposing in chagas disease: chloroquine potentiates benznidazole activity against Trypanosoma cruzi In vitro and in vivo (2022)
Pandey, Ramendra Pati; Nascimento, Marilda Savoia ; Franco, Caio Haddad ; Bortoluci, Karina ; Silva, Marcelo Nunes da ; Zingales, Bianca ; Gibald, Daniel; Barrios, Leda Castaño; Vieira, Joseli Lannes ; Cariste, Leonardo Moro; Vasconcelos, Jose Ronnie ; Moraes, Carolina Borsoi ; Freitas Junior, Lucio H; Kalil Filho, Jorge Elias ; Alcântara, Laura; Cunha Neto, Edecio
Source: Antimicrobial Agents and Chemotherapy. Unidades: FM, IQ
Subjects: DOENÇA DE CHAGAS, MEDICAMENTO
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ABNT
PANDEY, Ramendra Pati et al. Drug repurposing in chagas disease: chloroquine potentiates benznidazole activity against Trypanosoma cruzi In vitro and in vivo. Antimicrobial Agents and Chemotherapy, v. 66, n. 11, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1128/aac.00284-22. Acesso em: 18 nov. 2024.
APA
Pandey, R. P., Nascimento, M. S., Franco, C. H., Bortoluci, K., Silva, M. N. da, Zingales, B., et al. (2022). Drug repurposing in chagas disease: chloroquine potentiates benznidazole activity against Trypanosoma cruzi In vitro and in vivo. Antimicrobial Agents and Chemotherapy, 66( 11), 1-17. doi:10.1128/aac.00284-22
NLM
Pandey RP, Nascimento MS, Franco CH, Bortoluci K, Silva MN da, Zingales B, Gibald D, Barrios LC, Vieira JL, Cariste LM, Vasconcelos JR, Moraes CB, Freitas Junior LH, Kalil Filho JE, Alcântara L, Cunha Neto E. Drug repurposing in chagas disease: chloroquine potentiates benznidazole activity against Trypanosoma cruzi In vitro and in vivo [Internet]. Antimicrobial Agents and Chemotherapy. 2022 ; 66( 11): 1-17.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1128/aac.00284-22
Vancouver
Pandey RP, Nascimento MS, Franco CH, Bortoluci K, Silva MN da, Zingales B, Gibald D, Barrios LC, Vieira JL, Cariste LM, Vasconcelos JR, Moraes CB, Freitas Junior LH, Kalil Filho JE, Alcântara L, Cunha Neto E. Drug repurposing in chagas disease: chloroquine potentiates benznidazole activity against Trypanosoma cruzi In vitro and in vivo [Internet]. Antimicrobial Agents and Chemotherapy. 2022 ; 66( 11): 1-17.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1128/aac.00284-22
Artigo de periodico
Disruption of polycystin-1 cleavage leads to cardiac metabolic rewiring in mice (2022)
Amaral, Andressa Godoy ; Silva, Camille Cristine Caldeira da ; Serna, Julian David Cualcialpud ; Sampaio, Kinulpe Honorato ; Freitas, Jessica Quereza de ; Duarte Neto, Amaro Nunes ; Bloise Júnior, Antonio Carlos ; Cassina, Laura ; Yoshinaga, Marcos Yukio ; Chaves Filho, Adriano de Britto ; Qian, Feng ; Miyamoto, Sayuri ; Boletta, Alessandra ; Bordin, Silvana ; Kowaltowski, Alicia Juliana ; Onuchic, Luiz Fernando
Source: Biochimica et Biophysica Acta. Molecular Basis of Disease. Unidades: ICB, FM, IF, IQ
Subjects: NEFROPATIAS, MITOCÔNDRIAS, FISIOLOGIA, CANAIS IÔNICOS, CAMUNDONGOS, CANAIS IÔNICOS, CAMUNDONGOS, MUTAÇÃO, DOENÇAS CARDIOVASCULARES EM ANIMAL
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ABNT
AMARAL, Andressa Godoy et al. Disruption of polycystin-1 cleavage leads to cardiac metabolic rewiring in mice. Biochimica et Biophysica Acta. Molecular Basis of Disease, v. 1868, p. 1-17 art. 166371, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2022.166371. Acesso em: 18 nov. 2024.
APA
Amaral, A. G., Silva, C. C. C. da, Serna, J. D. C., Sampaio, K. H., Freitas, J. Q. de, Duarte Neto, A. N., et al. (2022). Disruption of polycystin-1 cleavage leads to cardiac metabolic rewiring in mice. Biochimica et Biophysica Acta. Molecular Basis of Disease, 1868, 1-17 art. 166371. doi:10.1016/j.bbadis.2022.166371
NLM
Amaral AG, Silva CCC da, Serna JDC, Sampaio KH, Freitas JQ de, Duarte Neto AN, Bloise Júnior AC, Cassina L, Yoshinaga MY, Chaves Filho A de B, Qian F, Miyamoto S, Boletta A, Bordin S, Kowaltowski AJ, Onuchic LF. Disruption of polycystin-1 cleavage leads to cardiac metabolic rewiring in mice [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2022 ; 1868 1-17 art. 166371.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbadis.2022.166371
Vancouver
Amaral AG, Silva CCC da, Serna JDC, Sampaio KH, Freitas JQ de, Duarte Neto AN, Bloise Júnior AC, Cassina L, Yoshinaga MY, Chaves Filho A de B, Qian F, Miyamoto S, Boletta A, Bordin S, Kowaltowski AJ, Onuchic LF. Disruption of polycystin-1 cleavage leads to cardiac metabolic rewiring in mice [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2022 ; 1868 1-17 art. 166371.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbadis.2022.166371
Artigo de periodico
A refined genome phage display methodology delineates the human antibody response in Chagas disease patients (2021)
Teixeira, André Azevedo Reis ; Carnero, Luis Rodriguez ; Kuramoto, Andréia; Tang, Fenny Hui Fen ; Gomes, Carlos Hernique; Pereira, Natalia Bueno; Oliveira, Lea Campos de; Garrini, Regina; Monteiro, Jhonatas Sirino ; Setubal, João Carlos ; Sabino, Ester Cerdeira ; Pasqualini, Renata ; Colli, Walter ; Arap,Wadih ; Alves, Maria Júlia Manso ; Cunha Neto, Edecio ; Giordano, Ricardo José
Source: iScience. Unidades: IQ, FM, BIOINFORMÁTICA
Subjects: GENOMAS, DOENÇA DE CHAGAS, ANTÍGENOS, EPITOPOS, TRYPANOSOMA CRUZI
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ABNT
TEIXEIRA, André Azevedo Reis et al. A refined genome phage display methodology delineates the human antibody response in Chagas disease patients. iScience, v. 24, n. 6, p. 1-27 art. 102540, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2021.102540. Acesso em: 18 nov. 2024.
APA
Teixeira, A. A. R., Carnero, L. R., Kuramoto, A., Tang, F. H. F., Gomes, C. H., Pereira, N. B., et al. (2021). A refined genome phage display methodology delineates the human antibody response in Chagas disease patients. iScience, 24( 6), 1-27 art. 102540. doi:10.1016/j.isci.2021.102540
NLM
Teixeira AAR, Carnero LR, Kuramoto A, Tang FHF, Gomes CH, Pereira NB, Oliveira LC de, Garrini R, Monteiro JS, Setubal JC, Sabino EC, Pasqualini R, Colli W, Arap W, Alves MJM, Cunha Neto E, Giordano RJ. A refined genome phage display methodology delineates the human antibody response in Chagas disease patients [Internet]. iScience. 2021 ; 24( 6): 1-27 art. 102540.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.isci.2021.102540
Vancouver
Teixeira AAR, Carnero LR, Kuramoto A, Tang FHF, Gomes CH, Pereira NB, Oliveira LC de, Garrini R, Monteiro JS, Setubal JC, Sabino EC, Pasqualini R, Colli W, Arap W, Alves MJM, Cunha Neto E, Giordano RJ. A refined genome phage display methodology delineates the human antibody response in Chagas disease patients [Internet]. iScience. 2021 ; 24( 6): 1-27 art. 102540.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.isci.2021.102540
Artigo de periodico
Protocol for design, construction, and selection of genome phage (gPhage) display libraries (2021)
Carnero, Luis Antonio Rodriguez; Teixeira, André Azevedo Reis ; Tang, Fenny Hui Fen; Kuramoto, Andréia; Alves, Maria Júlia Manso ; Colli, Walter ; Setubal, João Carlos ; Cunha Neto, Edecio ; Pasqualini, Renata ; Arap,Wadih ; Giordano, Ricardo José
Source: STAR Protocols. Unidades: IQ, FM
Subjects: ANTÍGENOS, MAPEAMENTO GENÉTICO, GENOMAS
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ABNT
CARNERO, Luis Antonio Rodriguez et al. Protocol for design, construction, and selection of genome phage (gPhage) display libraries. STAR Protocols, v. 2, n. 4, p. 1-24 art. 100936, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.xpro.2021.100936. Acesso em: 18 nov. 2024.
APA
Carnero, L. A. R., Teixeira, A. A. R., Tang, F. H. F., Kuramoto, A., Alves, M. J. M., Colli, W., et al. (2021). Protocol for design, construction, and selection of genome phage (gPhage) display libraries. STAR Protocols, 2( 4), 1-24 art. 100936. doi:10.1016/j.xpro.2021.100936
NLM
Carnero LAR, Teixeira AAR, Tang FHF, Kuramoto A, Alves MJM, Colli W, Setubal JC, Cunha Neto E, Pasqualini R, Arap W, Giordano RJ. Protocol for design, construction, and selection of genome phage (gPhage) display libraries [Internet]. STAR Protocols. 2021 ; 2( 4): 1-24 art. 100936.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.xpro.2021.100936
Vancouver
Carnero LAR, Teixeira AAR, Tang FHF, Kuramoto A, Alves MJM, Colli W, Setubal JC, Cunha Neto E, Pasqualini R, Arap W, Giordano RJ. Protocol for design, construction, and selection of genome phage (gPhage) display libraries [Internet]. STAR Protocols. 2021 ; 2( 4): 1-24 art. 100936.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.xpro.2021.100936
Artigo de periodico
A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome (2021)
Casaro, Mateus Barbosa ; Thomas, Andrew Maltez ; Mendes, Eduardo ; Fukumori, Claudio ; Ribeiro, Willian R.; Oliveira, Fernando A.; Crisma, Amanda Rabello ; Murata, Gilson Masahiro ; Bizzarro, Bruna ; Nunes, Anderson de Sá ; Setubal, João Carlos ; Mayer, Marcia Pinto Alves ; Martins, Flaviano S. ; Vieira, Angélica Thomaz ; Antiorio, Ana Tada Fonseca Brasil; Lima, Wothan Tavares de ; Câmara, Niels Olsen Saraiva ; Curi, Rui ; Dias-Neto, Emmanuel ; Ferreira, Caroline Marcantonio
Source: Microbiome. Unidades: FM, IQ, ICB, FMVZ
Subjects: PROBIÓTICOS, ALERGIA E IMUNOLOGIA, INFLAMAÇÃO, ASMA, IMUNOLOGIA, MICROBIOLOGIA, FARMACOLOGIA, DERMATOPATIAS, CAMUNDONGOS, FENÓTIPOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CASARO, Mateus Barbosa et al. A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome. Microbiome, v. 9, p. 1-16 art. 134, 2021Tradução . . Disponível em: https://doi.org/10.1186/s40168-021-01081-2. Acesso em: 18 nov. 2024.
APA
Casaro, M. B., Thomas, A. M., Mendes, E., Fukumori, C., Ribeiro, W. R., Oliveira, F. A., et al. (2021). A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome. Microbiome, 9, 1-16 art. 134. doi:10.1186/s40168-021-01081-2
NLM
Casaro MB, Thomas AM, Mendes E, Fukumori C, Ribeiro WR, Oliveira FA, Crisma AR, Murata GM, Bizzarro B, Nunes A de S, Setubal JC, Mayer MPA, Martins FS, Vieira AT, Antiorio ATFB, Lima WT de, Câmara NOS, Curi R, Dias-Neto E, Ferreira CM. A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome [Internet]. Microbiome. 2021 ; 9 1-16 art. 134.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s40168-021-01081-2
Vancouver
Casaro MB, Thomas AM, Mendes E, Fukumori C, Ribeiro WR, Oliveira FA, Crisma AR, Murata GM, Bizzarro B, Nunes A de S, Setubal JC, Mayer MPA, Martins FS, Vieira AT, Antiorio ATFB, Lima WT de, Câmara NOS, Curi R, Dias-Neto E, Ferreira CM. A probiotic has differential effects on allergic airway inflammation in A/J and C57BL/6 mice and is correlated with the gut microbiome [Internet]. Microbiome. 2021 ; 9 1-16 art. 134.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s40168-021-01081-2
Artigo de periodico
The role of chronic muscle (in)activity on carnosine homeostasis: a study with spinal-cord injured athletes (2021)
Nemezio, Kleiner ; Yamaguchi, Guilherme de Carvalho; Ramkrapes, Ana Paula Boito ; Schulz, Mariane Leichsenring; Baptista, Igor Luchini ; Riani, Luiz Augusto; Goncalves, Livia de Souza; Sale, Craig ; Medeiros, Marisa Helena Gennari de ; Gualano, Bruno ; Artioli, Guilherme Giannini
Source: American Journal of Physiology. Unidades: IQ, FM, EEFE
Subjects: HOMEOSTASE, LESÕES NO ESPORTE, MEDULA ESPINHAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NEMEZIO, Kleiner et al. The role of chronic muscle (in)activity on carnosine homeostasis: a study with spinal-cord injured athletes. American Journal of Physiology, v. 320, n. 6, p. R824-R832, 2021Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00360.2020. Acesso em: 18 nov. 2024.
APA
Nemezio, K., Yamaguchi, G. de C., Ramkrapes, A. P. B., Schulz, M. L., Baptista, I. L., Riani, L. A., et al. (2021). The role of chronic muscle (in)activity on carnosine homeostasis: a study with spinal-cord injured athletes. American Journal of Physiology, 320( 6), R824-R832. doi:10.1152/ajpregu.00360.2020
NLM
Nemezio K, Yamaguchi G de C, Ramkrapes APB, Schulz ML, Baptista IL, Riani LA, Goncalves L de S, Sale C, Medeiros MHG de, Gualano B, Artioli GG. The role of chronic muscle (in)activity on carnosine homeostasis: a study with spinal-cord injured athletes [Internet]. American Journal of Physiology. 2021 ; 320( 6): R824-R832.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpregu.00360.2020
Vancouver
Nemezio K, Yamaguchi G de C, Ramkrapes APB, Schulz ML, Baptista IL, Riani LA, Goncalves L de S, Sale C, Medeiros MHG de, Gualano B, Artioli GG. The role of chronic muscle (in)activity on carnosine homeostasis: a study with spinal-cord injured athletes [Internet]. American Journal of Physiology. 2021 ; 320( 6): R824-R832.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpregu.00360.2020
Artigo de periodico
Dietary sodium restriction alters muscle lipidomics that relates to insulin resistance in mice (2021)
Pinto, Paula Ramos ; Yoshinaga, Marcos Yukio ; Bianco, Vanessa Del; Bochi, Ana Paula Garcia; Ferreira, Guilherme S; Pinto, Isabella Fernanda Dantas ; Rodrigues, Letícia Gomes ; Nakandakare, Edna Regina ; Okamoto, Maristela Mitiko ; Machado, Ubiratan Fabres ; Miyamoto, Sayuri ; Catanozi, Sergio ; Passarelli, Marisa
Source: Journal of Biological Chemistry. Unidades: FM, ICB, IQ
Subjects: MÚSCULO ESQUELÉTICO, SÓDIO, RESISTÊNCIA À INSULINA, LIPÍDEOS, CAMUNDONGOS, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINTO, Paula Ramos et al. Dietary sodium restriction alters muscle lipidomics that relates to insulin resistance in mice. Journal of Biological Chemistry, v. 296, p. 1-12 art. 100344, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.jbc.2021.100344. Acesso em: 18 nov. 2024.
APA
Pinto, P. R., Yoshinaga, M. Y., Bianco, V. D., Bochi, A. P. G., Ferreira, G. S., Pinto, I. F. D., et al. (2021). Dietary sodium restriction alters muscle lipidomics that relates to insulin resistance in mice. Journal of Biological Chemistry, 296, 1-12 art. 100344. doi:10.1016/j.jbc.2021.100344
NLM
Pinto PR, Yoshinaga MY, Bianco VD, Bochi APG, Ferreira GS, Pinto IFD, Rodrigues LG, Nakandakare ER, Okamoto MM, Machado UF, Miyamoto S, Catanozi S, Passarelli M. Dietary sodium restriction alters muscle lipidomics that relates to insulin resistance in mice [Internet]. Journal of Biological Chemistry. 2021 ; 296 1-12 art. 100344.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jbc.2021.100344
Vancouver
Pinto PR, Yoshinaga MY, Bianco VD, Bochi APG, Ferreira GS, Pinto IFD, Rodrigues LG, Nakandakare ER, Okamoto MM, Machado UF, Miyamoto S, Catanozi S, Passarelli M. Dietary sodium restriction alters muscle lipidomics that relates to insulin resistance in mice [Internet]. Journal of Biological Chemistry. 2021 ; 296 1-12 art. 100344.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jbc.2021.100344
Trabalho de evento-resumo
Recognition of Trypanosoma cruzi epitopes by IgG of benznidazole treated Chagas disease patients (2021)
Carnero, Luis Antonio Rodriguez ; Monteiro, Jhonatas Sirino ; Setubal, João Carlos ; Sabino, Ester Cerdeira ; Cunha Neto, Edécio ; Giordano, Ricardo José
Source: Program and Abstract Book. Conference titles: Congress of the International Union for Pure Applied Biophysics/IUPAB. Unidades: IQ, FM, BIOINFORMÁTICA
Subjects: TRYPANOSOMA CRUZI, DOENÇA DE CHAGAS, EPITOPOS
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ABNT
CARNERO, Luis Antonio Rodriguez et al. Recognition of Trypanosoma cruzi epitopes by IgG of benznidazole treated Chagas disease patients. 2021, Anais.. São Paulo: IUPAB-SBBf-SBBq, 2021. Disponível em: http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf. Acesso em: 18 nov. 2024.
APA
Carnero, L. A. R., Monteiro, J. S., Setubal, J. C., Sabino, E. C., Cunha Neto, E., & Giordano, R. J. (2021). Recognition of Trypanosoma cruzi epitopes by IgG of benznidazole treated Chagas disease patients. In Program and Abstract Book. São Paulo: IUPAB-SBBf-SBBq. Recuperado de http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf
NLM
Carnero LAR, Monteiro JS, Setubal JC, Sabino EC, Cunha Neto E, Giordano RJ. Recognition of Trypanosoma cruzi epitopes by IgG of benznidazole treated Chagas disease patients [Internet]. Program and Abstract Book. 2021 ;[citado 2024 nov. 18 ] Available from: http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf
Vancouver
Carnero LAR, Monteiro JS, Setubal JC, Sabino EC, Cunha Neto E, Giordano RJ. Recognition of Trypanosoma cruzi epitopes by IgG of benznidazole treated Chagas disease patients [Internet]. Program and Abstract Book. 2021 ;[citado 2024 nov. 18 ] Available from: http://easyapp.ekmf.com.br/sg/uploads/eventos/evento8/documentos/abstract_book.pdf
Artigo de periodico
Comparing data-Independent acquisition and parallel reaction monitoring in their abilities to differentiate high-density lipoprotein subclasses (2020)
Silva, Amanda R. M; Toyoshima, Marcos Tadashi Kakitani; Passarelli, Marisa ; Di Mascio, Paolo ; Ronsein, Graziella Eliza
Source: Journal of Proteome Research. Unidades: FM, IQ
Subjects: PROTEÔMICA, LIPOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Amanda R. M et al. Comparing data-Independent acquisition and parallel reaction monitoring in their abilities to differentiate high-density lipoprotein subclasses. Journal of Proteome Research, v. 19, n. 1, p. 248-259, 2020Tradução . . Disponível em: https://doi.org/10.1021/acs.jproteome.9b00511. Acesso em: 18 nov. 2024.
APA
Silva, A. R. M., Toyoshima, M. T. K., Passarelli, M., Di Mascio, P., & Ronsein, G. E. (2020). Comparing data-Independent acquisition and parallel reaction monitoring in their abilities to differentiate high-density lipoprotein subclasses. Journal of Proteome Research, 19( 1), 248-259. doi:10.1021/acs.jproteome.9b00511
NLM
Silva ARM, Toyoshima MTK, Passarelli M, Di Mascio P, Ronsein GE. Comparing data-Independent acquisition and parallel reaction monitoring in their abilities to differentiate high-density lipoprotein subclasses [Internet]. Journal of Proteome Research. 2020 ; 19( 1): 248-259.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1021/acs.jproteome.9b00511
Vancouver
Silva ARM, Toyoshima MTK, Passarelli M, Di Mascio P, Ronsein GE. Comparing data-Independent acquisition and parallel reaction monitoring in their abilities to differentiate high-density lipoprotein subclasses [Internet]. Journal of Proteome Research. 2020 ; 19( 1): 248-259.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1021/acs.jproteome.9b00511
Artigo de periodico
Tributyrin attenuates metabolic and inflammatory changes associated with obesity through a GPR109A-dependent mechanism (2020)
Sato, Fabio Takeo ; Yap, Yu Anne ; Crisma, Amanda Rabello ; Portovedo, Mariana ; Murata, Gilson Masahiro ; Hirabara, Sandro Massao ; Ribeiro, Willian Rodrigues; Ferreira, Caroline Marcantonio ; Cruz, Maysa Mariana ; Pereira, Joice Naiara Bertaglia ; Payolla, Tanyara Baliani ; Guima, Suzana Eiko Sato ; Thomas, Andrew Maltez ; Setubal, João Carlos ; Vale, Maria Isabel Cardoso Alonso ; Santos, Marinilce Fagundes dos ; Curi, Rui ; Marino, Eliana ; Vinolo, Marco A. R.
Source: Cells. Unidades: FM, IQ, ICB, FSP
Subjects: OBESIDADE, RESISTÊNCIA À INSULINA, BIOLOGIA CELULAR, FISIOLOGIA, INFLAMAÇÃO, ÁCIDOS GRAXOS, CAMUNDONGOS, DIETA ANIMAL, ANTI-INFLAMATORIOS, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SATO, Fabio Takeo et al. Tributyrin attenuates metabolic and inflammatory changes associated with obesity through a GPR109A-dependent mechanism. Cells, v. 9, p. 1-18 art. 2007, 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9092007. Acesso em: 18 nov. 2024.
APA
Sato, F. T., Yap, Y. A., Crisma, A. R., Portovedo, M., Murata, G. M., Hirabara, S. M., et al. (2020). Tributyrin attenuates metabolic and inflammatory changes associated with obesity through a GPR109A-dependent mechanism. Cells, 9, 1-18 art. 2007. doi:10.3390/cells9092007
NLM
Sato FT, Yap YA, Crisma AR, Portovedo M, Murata GM, Hirabara SM, Ribeiro WR, Ferreira CM, Cruz MM, Pereira JNB, Payolla TB, Guima SES, Thomas AM, Setubal JC, Vale MICA, Santos MF dos, Curi R, Marino E, Vinolo MAR. Tributyrin attenuates metabolic and inflammatory changes associated with obesity through a GPR109A-dependent mechanism [Internet]. Cells. 2020 ; 9 1-18 art. 2007.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells9092007
Vancouver
Sato FT, Yap YA, Crisma AR, Portovedo M, Murata GM, Hirabara SM, Ribeiro WR, Ferreira CM, Cruz MM, Pereira JNB, Payolla TB, Guima SES, Thomas AM, Setubal JC, Vale MICA, Santos MF dos, Curi R, Marino E, Vinolo MAR. Tributyrin attenuates metabolic and inflammatory changes associated with obesity through a GPR109A-dependent mechanism [Internet]. Cells. 2020 ; 9 1-18 art. 2007.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells9092007
Artigo de periodico
Enrichment of apolipoprotein A-IV and apolipoprotein D in the HDL proteome is associated with HDL functions in diabetic kidney disease without dialysis (2020)
Santana, Monique Fátima Mello; Lira, Aécio Lopes de Araújo; Pinto, Raphael de Souza; Minanni, Carlos André; Silva, Amanda Ribeiro Martins da; Sawada, Maria I. B. A. C; Nakandakare, Edna Regina; Giannella, Maria L. C. Correa; Queiroz, Marcia Silva ; Ronsein, Graziella Eliza ; Passarelli, Marisa
Source: Lipids in Health and Disease. Unidades: FM, IQ
Subjects: ARTERIOSCLEROSE, NEFROPATIAS, APOLIPOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTANA, Monique Fátima Mello et al. Enrichment of apolipoprotein A-IV and apolipoprotein D in the HDL proteome is associated with HDL functions in diabetic kidney disease without dialysis. Lipids in Health and Disease, v. 19, p. 1-14 art. 205, 2020Tradução . . Disponível em: https://doi.org/10.1186/s12944-020-01381-w. Acesso em: 18 nov. 2024.
APA
Santana, M. F. M., Lira, A. L. de A., Pinto, R. de S., Minanni, C. A., Silva, A. R. M. da, Sawada, M. I. B. A. C., et al. (2020). Enrichment of apolipoprotein A-IV and apolipoprotein D in the HDL proteome is associated with HDL functions in diabetic kidney disease without dialysis. Lipids in Health and Disease, 19, 1-14 art. 205. doi:10.1186/s12944-020-01381-w
NLM
Santana MFM, Lira AL de A, Pinto R de S, Minanni CA, Silva ARM da, Sawada MIBAC, Nakandakare ER, Giannella MLCC, Queiroz MS, Ronsein GE, Passarelli M. Enrichment of apolipoprotein A-IV and apolipoprotein D in the HDL proteome is associated with HDL functions in diabetic kidney disease without dialysis [Internet]. Lipids in Health and Disease. 2020 ; 19 1-14 art. 205.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12944-020-01381-w
Vancouver
Santana MFM, Lira AL de A, Pinto R de S, Minanni CA, Silva ARM da, Sawada MIBAC, Nakandakare ER, Giannella MLCC, Queiroz MS, Ronsein GE, Passarelli M. Enrichment of apolipoprotein A-IV and apolipoprotein D in the HDL proteome is associated with HDL functions in diabetic kidney disease without dialysis [Internet]. Lipids in Health and Disease. 2020 ; 19 1-14 art. 205.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12944-020-01381-w
Artigo de periodico
HDL-proteome enrichment with apolipoprotein A-IV compensates for HDL loss of function in diabetes mellitus kidney disease (2020)
Santana, Monique Fátima Mello; Lira, Aécio Lopes de Araújo; Pinto, Raphael de Souza; Minanni, Carlos André; Silva, Amanda Ribeiro Martins da; Sawada, Maria I. B. A. C; Nakandakare, Edna Regina; Giannella, Maria Lucia Cardillo Correa ; Ronsein, Graziella Eliza ; Queiroz, Márcia S; Passarelli, Marisa
Source: Research Square. Unidades: FM, IQ
Subjects: NEFROPATIAS, DIABETES MELLITUS, APOLIPOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTANA, Monique Fátima Mello et al. HDL-proteome enrichment with apolipoprotein A-IV compensates for HDL loss of function in diabetes mellitus kidney disease. Research Square, 2020Tradução . . Disponível em: https://doi.org/10.21203/rs.3.rs-30049/v1. Acesso em: 18 nov. 2024.
APA
Santana, M. F. M., Lira, A. L. de A., Pinto, R. de S., Minanni, C. A., Silva, A. R. M. da, Sawada, M. I. B. A. C., et al. (2020). HDL-proteome enrichment with apolipoprotein A-IV compensates for HDL loss of function in diabetes mellitus kidney disease. Research Square. doi:10.21203/rs.3.rs-30049/v1
NLM
Santana MFM, Lira AL de A, Pinto R de S, Minanni CA, Silva ARM da, Sawada MIBAC, Nakandakare ER, Giannella MLCC, Ronsein GE, Queiroz MS, Passarelli M. HDL-proteome enrichment with apolipoprotein A-IV compensates for HDL loss of function in diabetes mellitus kidney disease [Internet]. Research Square. 2020 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.21203/rs.3.rs-30049/v1
Vancouver
Santana MFM, Lira AL de A, Pinto R de S, Minanni CA, Silva ARM da, Sawada MIBAC, Nakandakare ER, Giannella MLCC, Ronsein GE, Queiroz MS, Passarelli M. HDL-proteome enrichment with apolipoprotein A-IV compensates for HDL loss of function in diabetes mellitus kidney disease [Internet]. Research Square. 2020 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.21203/rs.3.rs-30049/v1
Artigo de periodico
Insulin does not stimulate Beta-alanine transport into human skeletal muscle (2020)
Gonçalves, Lívia de Souza; Kratz, Caroline; Santos, Lívia; Carvalho, Victor Henrique; Sales, Lucas Peixoto; Nemezio, Kleiner; Longobardi, Igor; Riani, Luiz; Lima, Marcelo Miranda de Oliveira; Saito, Tiemi; Fernandes, Alan Lins; Rodrigues, Joice; James, Ruth Margaret; Sale, Craig; Gualano, Bruno ; Geloneze, Bruno; Medeiros, Marisa Helena Gennari de ; Artioli, Guilherme Giannini
Source: American Journal of Physiology. Cell Physiology. Unidades: FM, IQ, EEFE
Subjects: MÚSCULO ESQUELÉTICO, INSULINA, SUPLEMENTOS DIETÉTICOS, PROTEÍNAS DE TRANSPORTE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONÇALVES, Lívia de Souza et al. Insulin does not stimulate Beta-alanine transport into human skeletal muscle. American Journal of Physiology. Cell Physiology, v. 318, p. C777–C786, 2020Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00550.2019. Acesso em: 18 nov. 2024.
APA
Gonçalves, L. de S., Kratz, C., Santos, L., Carvalho, V. H., Sales, L. P., Nemezio, K., et al. (2020). Insulin does not stimulate Beta-alanine transport into human skeletal muscle. American Journal of Physiology. Cell Physiology, 318, C777–C786. doi:10.1152/ajpcell.00550.2019
NLM
Gonçalves L de S, Kratz C, Santos L, Carvalho VH, Sales LP, Nemezio K, Longobardi I, Riani L, Lima MM de O, Saito T, Fernandes AL, Rodrigues J, James RM, Sale C, Gualano B, Geloneze B, Medeiros MHG de, Artioli GG. Insulin does not stimulate Beta-alanine transport into human skeletal muscle [Internet]. American Journal of Physiology. Cell Physiology. 2020 ; 318 C777–C786.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpcell.00550.2019
Vancouver
Gonçalves L de S, Kratz C, Santos L, Carvalho VH, Sales LP, Nemezio K, Longobardi I, Riani L, Lima MM de O, Saito T, Fernandes AL, Rodrigues J, James RM, Sale C, Gualano B, Geloneze B, Medeiros MHG de, Artioli GG. Insulin does not stimulate Beta-alanine transport into human skeletal muscle [Internet]. American Journal of Physiology. Cell Physiology. 2020 ; 318 C777–C786.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1152/ajpcell.00550.2019

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