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Artigo de periodico
A Brazilian multicentre study evaluating pregnancies induced by cabergoline in patients harboring prolactinomas (2020)
Anna, B. G. Sant; Musolino, N. R. C; Gadelha, M. R; Marques, C; Castro, Margaret de ; Elias, P. C. L.; Vilar, L; Lyra, R; Martins, M. R. A; Francisco, Rossana Pulcineli Vieira
Fonte: Pituitary. Unidades: FMRP, HU
Assuntos: ABORTO, AGENTES DOPAMINÉRGICOS, COMPLICAÇÕES DO TRABALHO DE PARTO, ESTUDOS RETROSPECTIVOS
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ABNT
ANNA, B. G. Sant et al. A Brazilian multicentre study evaluating pregnancies induced by cabergoline in patients harboring prolactinomas. Pituitary, v. 23, n. 2, p. 120-128, 2020Tradução . . Disponível em: https://doi.org/10.1007/s11102-019-01008-z. Acesso em: 16 out. 2025.
APA
Anna, B. G. S., Musolino, N. R. C., Gadelha, M. R., Marques, C., Castro, M. de, Elias, P. C. L., et al. (2020). A Brazilian multicentre study evaluating pregnancies induced by cabergoline in patients harboring prolactinomas. Pituitary, 23( 2), 120-128. doi:10.1007/s11102-019-01008-z
NLM
Anna BGS, Musolino NRC, Gadelha MR, Marques C, Castro M de, Elias PCL, Vilar L, Lyra R, Martins MRA, Francisco RPV. A Brazilian multicentre study evaluating pregnancies induced by cabergoline in patients harboring prolactinomas [Internet]. Pituitary. 2020 ; 23( 2): 120-128.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-019-01008-z
Vancouver
Anna BGS, Musolino NRC, Gadelha MR, Marques C, Castro M de, Elias PCL, Vilar L, Lyra R, Martins MRA, Francisco RPV. A Brazilian multicentre study evaluating pregnancies induced by cabergoline in patients harboring prolactinomas [Internet]. Pituitary. 2020 ; 23( 2): 120-128.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-019-01008-z
Artigo de periodico
Filamin A and DRD2 expression in corticotrophinomas (2019)
Sickler, Thais; Trarbach, Ericka Barbosa; Frassetto, Fernando Pereira; Dettoni, Juliano Bertollo; Alves, Venâncio Avancini Ferreira; Fragoso, Maria Candida Barisson Villares; Machado, Marcio Carlos; Cardoso, Ellison Fernando; Bronstein, Marcello Delano; Glezer, Andrea
Fonte: Pituitary. Unidade: FM
Assuntos: PROTEÍNAS CONTRÁTEIS, RECEPTORES DE DOPAMINA, ADENOMA, NEOPLASIAS HIPOFISÁRIAS, HORMÔNIO ADRENOCORTICOTRÓFICO
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ABNT
SICKLER, Thais et al. Filamin A and DRD2 expression in corticotrophinomas. Pituitary, v. 22, n. 2, p. 163-169, 2019Tradução . . Disponível em: https://doi.org/10.1007/s11102-019-00947-x. Acesso em: 16 out. 2025.
APA
Sickler, T., Trarbach, E. B., Frassetto, F. P., Dettoni, J. B., Alves, V. A. F., Fragoso, M. C. B. V., et al. (2019). Filamin A and DRD2 expression in corticotrophinomas. Pituitary, 22( 2), 163-169. doi:10.1007/s11102-019-00947-x
NLM
Sickler T, Trarbach EB, Frassetto FP, Dettoni JB, Alves VAF, Fragoso MCBV, Machado MC, Cardoso EF, Bronstein MD, Glezer A. Filamin A and DRD2 expression in corticotrophinomas [Internet]. Pituitary. 2019 ; 22( 2): 163-169.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-019-00947-x
Vancouver
Sickler T, Trarbach EB, Frassetto FP, Dettoni JB, Alves VAF, Fragoso MCBV, Machado MC, Cardoso EF, Bronstein MD, Glezer A. Filamin A and DRD2 expression in corticotrophinomas [Internet]. Pituitary. 2019 ; 22( 2): 163-169.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-019-00947-x
Artigo de periodico
Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats (2017)
Santos-Junior, Nilton Nascimento; Costa, Luís Henrique Angenendt; Catalão, Carlos Henrique Rocha; Kanashiro, Alexandre; Sharshar, Tarek; Rocha, Maria José Alves da
Fonte: Pituitary. Unidade: FORP
Assuntos: CITOCINAS, SEPSE, SISTEMA NERVOSO CENTRAL, VASOPRESSINAS, OCITOCINA
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ABNT
SANTOS-JUNIOR, Nilton Nascimento et al. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, v. 20, n. 5, p. 515-521, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11102-017-0812-z. Acesso em: 16 out. 2025.
APA
Santos-Junior, N. N., Costa, L. H. A., Catalão, C. H. R., Kanashiro, A., Sharshar, T., & Rocha, M. J. A. da. (2017). Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, 20( 5), 515-521. doi:10.1007/s11102-017-0812-z
NLM
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Vancouver
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Artigo de periodico
Influence of growth hormone receptor (GHR) exon 3 and -202A/C IGFBP-3 genetic polymorphisms on clinical and biochemical features and therapeutic outcome of patients with acromegaly (2015)
Jallad, Raquel S.; Trarbach, Ericka B.; Duarte, Felipe H.; Jorge, Alexander A. L.; Bronstein, Marcello D.
Fonte: Pituitary. Unidade: FM
Assuntos: POLIMORFISMO, RECEPTORES DA SOMATOTROFINA, GENÓTIPOS, RESULTADO DE TRATAMENTO, AVALIAÇÃO DE PROCESSOS E RESULTADOS, ACROMEGALIA, GENÓTIPOS
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ABNT
JALLAD, Raquel S. et al. Influence of growth hormone receptor (GHR) exon 3 and -202A/C IGFBP-3 genetic polymorphisms on clinical and biochemical features and therapeutic outcome of patients with acromegaly. Pituitary, v. 18, n. 5, p. 666-673, 2015Tradução . . Disponível em: http://link.springer.com/article/10.1007%2Fs11102-014-0629-y. Acesso em: 16 out. 2025.
APA
Jallad, R. S., Trarbach, E. B., Duarte, F. H., Jorge, A. A. L., & Bronstein, M. D. (2015). Influence of growth hormone receptor (GHR) exon 3 and -202A/C IGFBP-3 genetic polymorphisms on clinical and biochemical features and therapeutic outcome of patients with acromegaly. Pituitary, 18( 5), 666-673. doi:10.1007/s11102-014-0629-y
NLM
Jallad RS, Trarbach EB, Duarte FH, Jorge AAL, Bronstein MD. Influence of growth hormone receptor (GHR) exon 3 and -202A/C IGFBP-3 genetic polymorphisms on clinical and biochemical features and therapeutic outcome of patients with acromegaly [Internet]. Pituitary. 2015 ; 18( 5): 666-673.[citado 2025 out. 16 ] Available from: http://link.springer.com/article/10.1007%2Fs11102-014-0629-y
Vancouver
Jallad RS, Trarbach EB, Duarte FH, Jorge AAL, Bronstein MD. Influence of growth hormone receptor (GHR) exon 3 and -202A/C IGFBP-3 genetic polymorphisms on clinical and biochemical features and therapeutic outcome of patients with acromegaly [Internet]. Pituitary. 2015 ; 18( 5): 666-673.[citado 2025 out. 16 ] Available from: http://link.springer.com/article/10.1007%2Fs11102-014-0629-y
Artigo de periodico
Frequent development of combined pituitary hormone deficiency in patients initially diagnosed as isolated growth hormone deficiency: a long term follow-up of patients from a single center (2015)
Otto, Aline P.; França, Marcela M.; Correa, Fernanda A.; Costalonga, Everlayny F.; Leite, Claudia C.; Mendonca, Berenice B.; Arnhold, Ivo J. P.; Carvalho, Luciani R. S.; Jorge, Alexander A. L.
Fonte: Pituitary. Unidade: FM
Assuntos: HORMÔNIOS HIPOFISÁRIOS (DEFICIÊNCIA), ESTUDOS LONGITUDINAIS, CRIANÇAS, SEGUIMENTOS, ESTUDOS DE COORTES
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ABNT
OTTO, Aline P. et al. Frequent development of combined pituitary hormone deficiency in patients initially diagnosed as isolated growth hormone deficiency: a long term follow-up of patients from a single center. Pituitary, v. 18, n. 4, p. 561-567, 2015Tradução . . Disponível em: http://link.springer.com/article/10.1007%2Fs11102-014-0610-9. Acesso em: 16 out. 2025.
APA
Otto, A. P., França, M. M., Correa, F. A., Costalonga, E. F., Leite, C. C., Mendonca, B. B., et al. (2015). Frequent development of combined pituitary hormone deficiency in patients initially diagnosed as isolated growth hormone deficiency: a long term follow-up of patients from a single center. Pituitary, 18( 4), 561-567. doi:10.1007/s11102-014-0610-9
NLM
Otto AP, França MM, Correa FA, Costalonga EF, Leite CC, Mendonca BB, Arnhold IJP, Carvalho LRS, Jorge AAL. Frequent development of combined pituitary hormone deficiency in patients initially diagnosed as isolated growth hormone deficiency: a long term follow-up of patients from a single center [Internet]. Pituitary. 2015 ; 18( 4): 561-567.[citado 2025 out. 16 ] Available from: http://link.springer.com/article/10.1007%2Fs11102-014-0610-9
Vancouver
Otto AP, França MM, Correa FA, Costalonga EF, Leite CC, Mendonca BB, Arnhold IJP, Carvalho LRS, Jorge AAL. Frequent development of combined pituitary hormone deficiency in patients initially diagnosed as isolated growth hormone deficiency: a long term follow-up of patients from a single center [Internet]. Pituitary. 2015 ; 18( 4): 561-567.[citado 2025 out. 16 ] Available from: http://link.springer.com/article/10.1007%2Fs11102-014-0610-9
Artigo de periodico
Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation (2013)
Syro, Luis V.; Sundsbak, Jamie L.; Scheithauer, Bernd W.; Toledo, Rodrigo A.; Camargo, Mauricio; Heyer, Christina M.; Sekiya, Tomoko; Uribe, Humberto; Escobar, Jorge I.; Vasquez, Martin; Rotondo, Fabio; Toledo, Sergio P. A.; Kovacs, Kalman; Horvath, Eva; Babovic-Vuksanovic, Dusica; Harris, Peter C.
Fonte: Pituitary. Unidade: FM
Assuntos: NEOPLASIAS DAS GLÂNDULAS SUPRARRENAIS, ADENOMA, ACROMEGALIA, CISTOS, RIM (PATOLOGIA), IMUNOHISTOQUÍMICA, POLIMORFISMO
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ABNT
SYRO, Luis V. et al. Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation. Pituitary, v. 15, n. 3, p. 342-349, 2013Tradução . . Disponível em: http://link.springer.com/article/10.1007/s11102-011-0325-0. Acesso em: 16 out. 2025.
APA
Syro, L. V., Sundsbak, J. L., Scheithauer, B. W., Toledo, R. A., Camargo, M., Heyer, C. M., et al. (2013). Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation. Pituitary, 15( 3), 342-349. doi:10.1007/s11102-011-0325-0
NLM
Syro LV, Sundsbak JL, Scheithauer BW, Toledo RA, Camargo M, Heyer CM, Sekiya T, Uribe H, Escobar JI, Vasquez M, Rotondo F, Toledo SPA, Kovacs K, Horvath E, Babovic-Vuksanovic D, Harris PC. Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation [Internet]. Pituitary. 2013 ; 15( 3): 342-349.[citado 2025 out. 16 ] Available from: http://link.springer.com/article/10.1007/s11102-011-0325-0
Vancouver
Syro LV, Sundsbak JL, Scheithauer BW, Toledo RA, Camargo M, Heyer CM, Sekiya T, Uribe H, Escobar JI, Vasquez M, Rotondo F, Toledo SPA, Kovacs K, Horvath E, Babovic-Vuksanovic D, Harris PC. Somatotroph pituitary adenoma with acromegaly and autosomal dominant polycystic kidney disease: SSTR5 polymorphism and PKD1 mutation [Internet]. Pituitary. 2013 ; 15( 3): 342-349.[citado 2025 out. 16 ] Available from: http://link.springer.com/article/10.1007/s11102-011-0325-0
Artigo de periodico
Possible role of a radiation-induced p53 mutation in a Nelson’s syndrome patient with a fatal outcome (2011)
Pinto, Emilia Modolo; Siqueira, Sheila A. C.; Cukier, Priscilla; Fragoso, Maria C. B. V.; Lin, Chin Jia; Mendonca, Berenice Bilharinho de
Fonte: Pituitary. Unidade: FM
Assuntos: NEOPLASIAS DAS GLÂNDULAS SUPRARRENAIS, GLÂNDULA PRITUITÁRIA (FISIOPATOLOGIA;EFEITOS DE RADIAÇÃO;;COMPLICAÇÕES), ADNEOMA (MORTALIDADE)
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ABNT
PINTO, Emilia Modolo et al. Possible role of a radiation-induced p53 mutation in a Nelson’s syndrome patient with a fatal outcome. Pituitary, v. 14, p. 400-404, 2011Tradução . . Disponível em: https://doi.org/10.1007/s11102-009-0194-y. Acesso em: 16 out. 2025.
APA
Pinto, E. M., Siqueira, S. A. C., Cukier, P., Fragoso, M. C. B. V., Lin, C. J., & Mendonca, B. B. de. (2011). Possible role of a radiation-induced p53 mutation in a Nelson’s syndrome patient with a fatal outcome. Pituitary, 14, 400-404. doi:10.1007/s11102-009-0194-y
NLM
Pinto EM, Siqueira SAC, Cukier P, Fragoso MCBV, Lin CJ, Mendonca BB de. Possible role of a radiation-induced p53 mutation in a Nelson’s syndrome patient with a fatal outcome [Internet]. Pituitary. 2011 ; 14 400-404.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-009-0194-y
Vancouver
Pinto EM, Siqueira SAC, Cukier P, Fragoso MCBV, Lin CJ, Mendonca BB de. Possible role of a radiation-induced p53 mutation in a Nelson’s syndrome patient with a fatal outcome [Internet]. Pituitary. 2011 ; 14 400-404.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-009-0194-y
Artigo de periodico
Hepatocyte growth factor-regulated tyrosine kinase substrate (HGS) and guanylate kinase 1 (GUK1) are differentially expressed in GH-secreting adenomas (2006)
Rocha, Anderson Alves da; Giorgi, Ricardo Rodrigues; Sá, Sandra Valeria de; Cavaleiro, Ana Mercedes; Correa-Giannella, Maria Lucia; Fortes, Maria Angela Henriques Zanella; Cavaleiro, Ana Mercedes; Machado, Marcio Carlos; Cescato, Valter Angelo; Bronstein, Marcello Delano; Giannella Neto, Daniel
Fonte: Pituitary. Unidade: FM
Assuntos: ADENOMA (SECREÇÃO), FÍGADO (FISIOPATOLOGIA), HEPATOPATIAS (ENZIMOLOGIA)
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ABNT
ROCHA, Anderson Alves da et al. Hepatocyte growth factor-regulated tyrosine kinase substrate (HGS) and guanylate kinase 1 (GUK1) are differentially expressed in GH-secreting adenomas. Pituitary, v. 9, p. 83-92, 2006Tradução . . Disponível em: https://doi.org/10.1007/s11102-006-9277-1. Acesso em: 16 out. 2025.
APA
Rocha, A. A. da, Giorgi, R. R., Sá, S. V. de, Cavaleiro, A. M., Correa-Giannella, M. L., Fortes, M. A. H. Z., et al. (2006). Hepatocyte growth factor-regulated tyrosine kinase substrate (HGS) and guanylate kinase 1 (GUK1) are differentially expressed in GH-secreting adenomas. Pituitary, 9, 83-92. doi:10.1007/s11102-006-9277-1
NLM
Rocha AA da, Giorgi RR, Sá SV de, Cavaleiro AM, Correa-Giannella ML, Fortes MAHZ, Cavaleiro AM, Machado MC, Cescato VA, Bronstein MD, Giannella Neto D. Hepatocyte growth factor-regulated tyrosine kinase substrate (HGS) and guanylate kinase 1 (GUK1) are differentially expressed in GH-secreting adenomas [Internet]. Pituitary. 2006 ; 9 83-92.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-006-9277-1
Vancouver
Rocha AA da, Giorgi RR, Sá SV de, Cavaleiro AM, Correa-Giannella ML, Fortes MAHZ, Cavaleiro AM, Machado MC, Cescato VA, Bronstein MD, Giannella Neto D. Hepatocyte growth factor-regulated tyrosine kinase substrate (HGS) and guanylate kinase 1 (GUK1) are differentially expressed in GH-secreting adenomas [Internet]. Pituitary. 2006 ; 9 83-92.[citado 2025 out. 16 ] Available from: https://doi.org/10.1007/s11102-006-9277-1
Trabalho de evento-resumo periodico
Evaluation of disease status in treated acromegaly with sensitive growth hormone assay, insulin-like growth factor I and IGF-binding protein-3 (IGFBP-3) (2000)
Costa, A. C. F.; Rossi, Antônio; Martinelli Júnior, Carlos Eduardo; Machado, Hélio Rubens; Moreira, Ayrton Custódio
Fonte: Pituitary. Nome do evento: European Workshop on Pituitary Disease. Unidade: FMRP
Assuntos: PATOLOGIA, SISTEMA NERVOSO CENTRAL
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ABNT
COSTA, A. C. F. et al. Evaluation of disease status in treated acromegaly with sensitive growth hormone assay, insulin-like growth factor I and IGF-binding protein-3 (IGFBP-3). Pituitary. Dordrecht: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 16 out. 2025. , 2000
APA
Costa, A. C. F., Rossi, A., Martinelli Júnior, C. E., Machado, H. R., & Moreira, A. C. (2000). Evaluation of disease status in treated acromegaly with sensitive growth hormone assay, insulin-like growth factor I and IGF-binding protein-3 (IGFBP-3). Pituitary. Dordrecht: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Costa ACF, Rossi A, Martinelli Júnior CE, Machado HR, Moreira AC. Evaluation of disease status in treated acromegaly with sensitive growth hormone assay, insulin-like growth factor I and IGF-binding protein-3 (IGFBP-3). Pituitary. 2000 ; 3( 1): 26 res. 8.[citado 2025 out. 16 ]
Vancouver
Costa ACF, Rossi A, Martinelli Júnior CE, Machado HR, Moreira AC. Evaluation of disease status in treated acromegaly with sensitive growth hormone assay, insulin-like growth factor I and IGF-binding protein-3 (IGFBP-3). Pituitary. 2000 ; 3( 1): 26 res. 8.[citado 2025 out. 16 ]

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