Filtros : "Pain" Removidos: "Alemanha" "Martinez, Francisco Eulógio" Limpar

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  • Source: Pain. Unidade: FOB

    Subjects: DISFUNÇÃO TEMPOROMANDIBULAR, TELEMEDICINA, DIAGNÓSTICO, PANDEMIAS, COVID-19

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    • ABNT

      EXPOSTO, Fernando G. et al. Remote physical examination for temporomandibular disorders. Pain, v. 163, n. 5, p. 936-942, 2022Tradução . . Disponível em: https://doi.org/10.1097/j.pain.0000000000002455. Acesso em: 02 nov. 2024.
    • APA

      Exposto, F. G., Castrillon, E. E., Exposto, C. R., Costa, D. M. F., Gøkhan, M. A., Svensson, P., & Costa, Y. M. (2022). Remote physical examination for temporomandibular disorders. Pain, 163( 5), 936-942. doi:10.1097/j.pain.0000000000002455
    • NLM

      Exposto FG, Castrillon EE, Exposto CR, Costa DMF, Gøkhan MA, Svensson P, Costa YM. Remote physical examination for temporomandibular disorders [Internet]. Pain. 2022 ; 163( 5): 936-942.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/j.pain.0000000000002455
    • Vancouver

      Exposto FG, Castrillon EE, Exposto CR, Costa DMF, Gøkhan MA, Svensson P, Costa YM. Remote physical examination for temporomandibular disorders [Internet]. Pain. 2022 ; 163( 5): 936-942.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/j.pain.0000000000002455
  • Source: Pain. Unidade: FM

    Subjects: ACIDENTE VASCULAR CEREBRAL, DOR, IMAGEM POR RESSONÂNCIA MAGNÉTICA, DIAGNÓSTICO POR IMAGEM

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    • ABNT

      LEMOS, Marcelo Delboni et al. Dissecting neuropathic from poststroke pain: the white matter within. Pain, v. 163, n. 4, p. 765-778, 2022Tradução . . Disponível em: https://doi.org/10.1097/j.pain.0000000000002427. Acesso em: 02 nov. 2024.
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      Lemos, M. D., Faillenot, I., Lucato, L. T., Teixeira, M. J., Barbosa, L. M., Alho, E. J. L., et al. (2022). Dissecting neuropathic from poststroke pain: the white matter within. Pain, 163( 4), 765-778. doi:10.1097/j.pain.0000000000002427
    • NLM

      Lemos MD, Faillenot I, Lucato LT, Teixeira MJ, Barbosa LM, Alho EJL, Conforto AB, Rodrigues AL de L, Galhardoni R, Silva VA da. Dissecting neuropathic from poststroke pain: the white matter within [Internet]. Pain. 2022 ; 163( 4): 765-778.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/j.pain.0000000000002427
    • Vancouver

      Lemos MD, Faillenot I, Lucato LT, Teixeira MJ, Barbosa LM, Alho EJL, Conforto AB, Rodrigues AL de L, Galhardoni R, Silva VA da. Dissecting neuropathic from poststroke pain: the white matter within [Internet]. Pain. 2022 ; 163( 4): 765-778.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/j.pain.0000000000002427
  • Source: Pain. Unidade: FMRP

    Subjects: DOR, NERVOS PERIFÉRICOS, NERVO CIÁTICO, LINFÓCITOS T, SISTEMA IMUNE

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    • ABNT

      DAVOLI-FERREIRA, Marcela et al. Regulatory T cells counteract neuropathic pain through inhibition of the Th1 response at the site of peripheral nerve injury. Pain, v. 161, n. 8, p. 1730-1743, 2020Tradução . . Disponível em: https://doi.org/10.1097/j.pain.0000000000001879. Acesso em: 02 nov. 2024.
    • APA

      Davoli-Ferreira, M., Lima, K. W. A. de, Fonseca, M. das D. M., Guimarães, R. M., Gomes, F. I. F., Cavallini, M. C. M., et al. (2020). Regulatory T cells counteract neuropathic pain through inhibition of the Th1 response at the site of peripheral nerve injury. Pain, 161( 8), 1730-1743. doi:10.1097/j.pain.0000000000001879
    • NLM

      Davoli-Ferreira M, Lima KWA de, Fonseca M das DM, Guimarães RM, Gomes FIF, Cavallini MCM, Quadros AU de, Kusuda R, Cunha F de Q, Alves Filho JCF, Cunha TM. Regulatory T cells counteract neuropathic pain through inhibition of the Th1 response at the site of peripheral nerve injury [Internet]. Pain. 2020 ; 161( 8): 1730-1743.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/j.pain.0000000000001879
    • Vancouver

      Davoli-Ferreira M, Lima KWA de, Fonseca M das DM, Guimarães RM, Gomes FIF, Cavallini MCM, Quadros AU de, Kusuda R, Cunha F de Q, Alves Filho JCF, Cunha TM. Regulatory T cells counteract neuropathic pain through inhibition of the Th1 response at the site of peripheral nerve injury [Internet]. Pain. 2020 ; 161( 8): 1730-1743.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/j.pain.0000000000001879
  • Source: Pain. Unidade: FMRP

    Subjects: APOPTOSE, PROTEÍNAS DE LIGAÇÃO DO CÁLCIO, INFLAMAÇÃO, MODELOS ANIMAIS DE DOENÇAS

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    • ABNT

      LOPES, Alexandre H. et al. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain. Pain, v. 156, n. 3, p. 451-459, 2015Tradução . . Disponível em: https://doi.org/10.1097/01.j.pain.0000460322.72396.53. Acesso em: 02 nov. 2024.
    • APA

      Lopes, A. H., Talbot, J., Silva, R. L., Lima, J. B., França, R. O., Verri Júnior, W. A., et al. (2015). Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain. Pain, 156( 3), 451-459. doi:10.1097/01.j.pain.0000460322.72396.53
    • NLM

      Lopes AH, Talbot J, Silva RL, Lima JB, França RO, Verri Júnior WA, Mascarenhas DPA, Ryffel B, Cunha F de Q, Zamboni DS, Cunha TM. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain [Internet]. Pain. 2015 ; 156( 3): 451-459.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/01.j.pain.0000460322.72396.53
    • Vancouver

      Lopes AH, Talbot J, Silva RL, Lima JB, França RO, Verri Júnior WA, Mascarenhas DPA, Ryffel B, Cunha F de Q, Zamboni DS, Cunha TM. Peripheral NLCR4 inflammasome participates in the genesis of acute inflammatory pain [Internet]. Pain. 2015 ; 156( 3): 451-459.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/01.j.pain.0000460322.72396.53
  • Source: Pain. Unidade: FMRP

    Subjects: DOR, DOENÇAS DO SISTEMA NERVOSO, EXERCÍCIO FÍSICO, ASTRÓCITOS

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    • ABNT

      ALMEIDA, Cayo et al. Exercise therapy normalizes BDNF upregulation and glial hyperactivity in a mouse model of neuropathic pain. Pain, v. 156, n. 3, p. 504-513, 2015Tradução . . Disponível em: https://doi.org/10.1097/01.j.pain.0000460339.23976.12. Acesso em: 02 nov. 2024.
    • APA

      Almeida, C., DeMaman, A., Kusuda, R., Cadetti, F., Ravanelli, M. I., Queiroz, A. L., et al. (2015). Exercise therapy normalizes BDNF upregulation and glial hyperactivity in a mouse model of neuropathic pain. Pain, 156( 3), 504-513. doi:10.1097/01.j.pain.0000460339.23976.12
    • NLM

      Almeida C, DeMaman A, Kusuda R, Cadetti F, Ravanelli MI, Queiroz AL, Sousa TA, Zanon SA, Silveira LR, Lucas G de A. Exercise therapy normalizes BDNF upregulation and glial hyperactivity in a mouse model of neuropathic pain [Internet]. Pain. 2015 ; 156( 3): 504-513.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/01.j.pain.0000460339.23976.12
    • Vancouver

      Almeida C, DeMaman A, Kusuda R, Cadetti F, Ravanelli MI, Queiroz AL, Sousa TA, Zanon SA, Silveira LR, Lucas G de A. Exercise therapy normalizes BDNF upregulation and glial hyperactivity in a mouse model of neuropathic pain [Internet]. Pain. 2015 ; 156( 3): 504-513.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/01.j.pain.0000460339.23976.12
  • Source: Pain. Unidade: FM

    Subjects: DOR (FISIOLOGIA), BLOQUEIO NERVOSO, EXTREMIDADES, VÉRTEBRAS TORÁCICAS

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    • ABNT

      ROCHA, Roberto de Oliveira et al. Thoracic sympathetic block for the treatment of complex regional pain syndrome typo 1: A double blind randomized controlled study. Pain, v. 155, p. 2274-2281, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2014.08.015. Acesso em: 02 nov. 2024.
    • APA

      Rocha, R. de O., Teixeira, M. J., Yeng, L. T., Cantara, M. G., Faria, V. G., Liggieri, V., et al. (2014). Thoracic sympathetic block for the treatment of complex regional pain syndrome typo 1: A double blind randomized controlled study. Pain, 155, 2274-2281. doi:10.1016/j.pain.2014.08.015
    • NLM

      Rocha R de O, Teixeira MJ, Yeng LT, Cantara MG, Faria VG, Liggieri V, Loduca A, Muller BM, Souza ACMS, Andrade DC de. Thoracic sympathetic block for the treatment of complex regional pain syndrome typo 1: A double blind randomized controlled study [Internet]. Pain. 2014 ; 155 2274-2281.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2014.08.015
    • Vancouver

      Rocha R de O, Teixeira MJ, Yeng LT, Cantara MG, Faria VG, Liggieri V, Loduca A, Muller BM, Souza ACMS, Andrade DC de. Thoracic sympathetic block for the treatment of complex regional pain syndrome typo 1: A double blind randomized controlled study [Internet]. Pain. 2014 ; 155 2274-2281.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2014.08.015
  • Source: Pain. Unidade: EE

    Subjects: DOR (PREVALÊNCIA), MÚSCULO ESQUELÉTICO, FATORES DE RISCO, SAÚDE OCUPACIONAL

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    • ABNT

      COGGON, David et al. Disabling musculoskeletal pain in working populations: Is it the job, the person, or the culture?. Pain, v. 154, n. 6, p. 856-863, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2013.02.008. Acesso em: 02 nov. 2024.
    • APA

      Coggon, D., Ntani, G., Palmer, K. T., & Felli, V. E. A. (2013). Disabling musculoskeletal pain in working populations: Is it the job, the person, or the culture? Pain, 154( 6), 856-863. doi:10.1016/j.pain.2013.02.008
    • NLM

      Coggon D, Ntani G, Palmer KT, Felli VEA. Disabling musculoskeletal pain in working populations: Is it the job, the person, or the culture? [Internet]. Pain. 2013 ; 154( 6): 856-863.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2013.02.008
    • Vancouver

      Coggon D, Ntani G, Palmer KT, Felli VEA. Disabling musculoskeletal pain in working populations: Is it the job, the person, or the culture? [Internet]. Pain. 2013 ; 154( 6): 856-863.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2013.02.008
  • Source: Pain. Unidades: EACH, FM

    Subjects: DOR (ANÁLISE QUANTITATIVA), DOR (TRATAMENTO), DOENÇA DE PARKINSON, DOENÇA CRÔNICA, ESTIMULAÇÃO CEREBRAL, CIRURGIA

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    • ABNT

      ANDRADE, Daniel Ciampi Araújo de et al. Subthalamic deep brain stimulation modulates small fiber–dependent sensory thresholds in Parkinson’s disease. Pain, v. 153, n. 5, p. 1107-1113, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2012.02.016. Acesso em: 02 nov. 2024.
    • APA

      Andrade, D. C. A. de, Lefaucheur, J. -P., Galhardoni, R., Ferreira, K. S. L., Paiva, A. R. B. de, Bor-Seng-Shu, E., et al. (2012). Subthalamic deep brain stimulation modulates small fiber–dependent sensory thresholds in Parkinson’s disease. Pain, 153( 5), 1107-1113. doi:10.1016/j.pain.2012.02.016
    • NLM

      Andrade DCA de, Lefaucheur J-P, Galhardoni R, Ferreira KSL, Paiva ARB de, Bor-Seng-Shu E, Silva LA, Myczkowski ML, Marcolin MA, Siqueira SRDT de, Fonoff ET, Barbosa ER, Teixeira MJ. Subthalamic deep brain stimulation modulates small fiber–dependent sensory thresholds in Parkinson’s disease [Internet]. Pain. 2012 ; 153( 5): 1107-1113.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2012.02.016
    • Vancouver

      Andrade DCA de, Lefaucheur J-P, Galhardoni R, Ferreira KSL, Paiva ARB de, Bor-Seng-Shu E, Silva LA, Myczkowski ML, Marcolin MA, Siqueira SRDT de, Fonoff ET, Barbosa ER, Teixeira MJ. Subthalamic deep brain stimulation modulates small fiber–dependent sensory thresholds in Parkinson’s disease [Internet]. Pain. 2012 ; 153( 5): 1107-1113.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2012.02.016
  • Source: Pain. Unidade: FMRP

    Subjects: NEUTRÓFILOS, ARTRITE REUMATOIDE, CITOCINAS

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      PINTO, Larissa G. et al. IL-17 mediates articular hypernociception in antigen-induced arthritis in mice. Pain, v. 148, n. 2, p. 247-256, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2009.11.006. Acesso em: 02 nov. 2024.
    • APA

      Pinto, L. G., Cunha, T. M., Vieira, S. M., Lemos, H. P., Verri Júnior, W. A., Cunha, F. de Q., & Ferreira, S. H. (2010). IL-17 mediates articular hypernociception in antigen-induced arthritis in mice. Pain, 148( 2), 247-256. doi:10.1016/j.pain.2009.11.006
    • NLM

      Pinto LG, Cunha TM, Vieira SM, Lemos HP, Verri Júnior WA, Cunha F de Q, Ferreira SH. IL-17 mediates articular hypernociception in antigen-induced arthritis in mice [Internet]. Pain. 2010 ; 148( 2): 247-256.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2009.11.006
    • Vancouver

      Pinto LG, Cunha TM, Vieira SM, Lemos HP, Verri Júnior WA, Cunha F de Q, Ferreira SH. IL-17 mediates articular hypernociception in antigen-induced arthritis in mice [Internet]. Pain. 2010 ; 148( 2): 247-256.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2009.11.006
  • Source: Pain. Unidade: EE

    Subjects: DOR (TRATAMENTO), ANALGÉSICOS, COGNIÇÃO (ALTERAÇÃO)

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      KENDALL, Sally Elizabeth et al. The cognitive effects of opioids in chronic non-cancer pain. Pain, v. 150, n. 2, p. 225-230, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2010.05.012. Acesso em: 02 nov. 2024.
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      Kendall, S. E., Sjøgren, P., Pimenta, C. A. de M., & Højsted, J. (2010). The cognitive effects of opioids in chronic non-cancer pain. Pain, 150( 2), 225-230. doi:10.1016/j.pain.2010.05.012
    • NLM

      Kendall SE, Sjøgren P, Pimenta CA de M, Højsted J. The cognitive effects of opioids in chronic non-cancer pain [Internet]. Pain. 2010 ; 150( 2): 225-230.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2010.05.012
    • Vancouver

      Kendall SE, Sjøgren P, Pimenta CA de M, Højsted J. The cognitive effects of opioids in chronic non-cancer pain [Internet]. Pain. 2010 ; 150( 2): 225-230.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2010.05.012
  • Source: Pain. Unidade: ICB

    Assunto: FARMACOLOGIA

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      KEEBLE, Julie Elizabeth et al. Hydrogen peroxide is a novel mediator of inflammatory hyperalgesia, acting via transient receptor potential vanilloid 1-dependent and independent mechanisms. Pain, v. 141, n. 1/2, p. 135-42, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2008.10.025. Acesso em: 02 nov. 2024.
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      Keeble, J. E., Bodkin, J. V., Liang, L., Wodarski, R., Davies, M., Fernandes, E. S., et al. (2009). Hydrogen peroxide is a novel mediator of inflammatory hyperalgesia, acting via transient receptor potential vanilloid 1-dependent and independent mechanisms. Pain, 141( 1/2), 135-42. doi:10.1016/j.pain.2008.10.025
    • NLM

      Keeble JE, Bodkin JV, Liang L, Wodarski R, Davies M, Fernandes ES, Coelho C de F, Russell F, Graepel R, Muscará MN, Malcangio M, Brain SD. Hydrogen peroxide is a novel mediator of inflammatory hyperalgesia, acting via transient receptor potential vanilloid 1-dependent and independent mechanisms [Internet]. Pain. 2009 ; 141( 1/2): 135-42.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2008.10.025
    • Vancouver

      Keeble JE, Bodkin JV, Liang L, Wodarski R, Davies M, Fernandes ES, Coelho C de F, Russell F, Graepel R, Muscará MN, Malcangio M, Brain SD. Hydrogen peroxide is a novel mediator of inflammatory hyperalgesia, acting via transient receptor potential vanilloid 1-dependent and independent mechanisms [Internet]. Pain. 2009 ; 141( 1/2): 135-42.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2008.10.025
  • Source: Pain. Unidade: FMRP

    Subjects: DOR (CONTROLE), SEROTONINA, NÚCLEO CELULAR

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      VILLARREAL, Cristiane Flora e PRADO, William Alves do. Modulation of persistent nociceptives inputs in the anterior pretectal nucleus of the rat. Pain, v. 132, p. 42-52, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2007.01.021. Acesso em: 02 nov. 2024.
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      Villarreal, C. F., & Prado, W. A. do. (2007). Modulation of persistent nociceptives inputs in the anterior pretectal nucleus of the rat. Pain, 132, 42-52. doi:10.1016/j.pain.2007.01.021
    • NLM

      Villarreal CF, Prado WA do. Modulation of persistent nociceptives inputs in the anterior pretectal nucleus of the rat [Internet]. Pain. 2007 ; 132 42-52.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2007.01.021
    • Vancouver

      Villarreal CF, Prado WA do. Modulation of persistent nociceptives inputs in the anterior pretectal nucleus of the rat [Internet]. Pain. 2007 ; 132 42-52.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2007.01.021
  • Source: Pain. Unidade: FFCLRP

    Assunto: PSICOFARMACOLOGIA

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      DE LUCA-VINHAS, Maria Cecília Zanoto e MACEDO, Carlos Eduardo e BRANDÃO, Marcus Lira. Pharmacological assessment of the freezing, antinociception, and exploratory behavior organized in the ventrolateral periaqueductal gray. Pain, v. 121, p. 94-104, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2005.12.008. Acesso em: 02 nov. 2024.
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      De Luca-Vinhas, M. C. Z., Macedo, C. E., & Brandão, M. L. (2006). Pharmacological assessment of the freezing, antinociception, and exploratory behavior organized in the ventrolateral periaqueductal gray. Pain, 121, 94-104. doi:10.1016/j.pain.2005.12.008
    • NLM

      De Luca-Vinhas MCZ, Macedo CE, Brandão ML. Pharmacological assessment of the freezing, antinociception, and exploratory behavior organized in the ventrolateral periaqueductal gray [Internet]. Pain. 2006 ; 121 94-104.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2005.12.008
    • Vancouver

      De Luca-Vinhas MCZ, Macedo CE, Brandão ML. Pharmacological assessment of the freezing, antinociception, and exploratory behavior organized in the ventrolateral periaqueductal gray [Internet]. Pain. 2006 ; 121 94-104.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2005.12.008
  • Source: Pain. Unidade: FCFRP

    Subjects: FARMACOLOGIA, ENDOTELINAS

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      CHICHORRO, Juliana Geremias et al. Orofacial cold hyperalgesia due to infraorbital nerve constriction injury in rats: Reversal by endothelin receptor antagonists but not non-steroidal anti-inflammatory drugs. Pain, v. 123, p. 64-74, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2006.02.010. Acesso em: 02 nov. 2024.
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      Chichorro, J. G., Zampronio, A. R., Souza, G. E. P. de, & Rae, G. A. (2006). Orofacial cold hyperalgesia due to infraorbital nerve constriction injury in rats: Reversal by endothelin receptor antagonists but not non-steroidal anti-inflammatory drugs. Pain, 123, 64-74. doi:10.1016/j.pain.2006.02.010
    • NLM

      Chichorro JG, Zampronio AR, Souza GEP de, Rae GA. Orofacial cold hyperalgesia due to infraorbital nerve constriction injury in rats: Reversal by endothelin receptor antagonists but not non-steroidal anti-inflammatory drugs [Internet]. Pain. 2006 ; 123 64-74.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2006.02.010
    • Vancouver

      Chichorro JG, Zampronio AR, Souza GEP de, Rae GA. Orofacial cold hyperalgesia due to infraorbital nerve constriction injury in rats: Reversal by endothelin receptor antagonists but not non-steroidal anti-inflammatory drugs [Internet]. Pain. 2006 ; 123 64-74.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2006.02.010
  • Source: Pain. Unidade: FMRP

    Subjects: DOR, FARMACOLOGIA

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      PARADA, Carlos Amílcar e REICHLING, David B. e LEVINE, J. D. Chronic hyperalgesic priming in the rat involves a novel interaction between cAMP and PKCe second messenger pathways. Pain, v. 113, p. 185-190, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2004.10.021. Acesso em: 02 nov. 2024.
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      Parada, C. A., Reichling, D. B., & Levine, J. D. (2005). Chronic hyperalgesic priming in the rat involves a novel interaction between cAMP and PKCe second messenger pathways. Pain, 113, 185-190. doi:10.1016/j.pain.2004.10.021
    • NLM

      Parada CA, Reichling DB, Levine JD. Chronic hyperalgesic priming in the rat involves a novel interaction between cAMP and PKCe second messenger pathways [Internet]. Pain. 2005 ; 113 185-190.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2004.10.021
    • Vancouver

      Parada CA, Reichling DB, Levine JD. Chronic hyperalgesic priming in the rat involves a novel interaction between cAMP and PKCe second messenger pathways [Internet]. Pain. 2005 ; 113 185-190.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2004.10.021
  • Source: Pain. Unidade: ICB

    Assunto: FISIOLOGIA

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      GUTIÉRREZ, José Maria et al. Snake venom components enhance pain upon subcutaneous injection: an initial examination of spinal cord mediators. Pain, v. 111, p. 65-76, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2004.06.001. Acesso em: 02 nov. 2024.
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      Gutiérrez, J. M., Gutiérrez, J. M., Milligan, E. D., Wieseler-Frank, J., Britto, L. R. G. de, Maier, S. F., et al. (2004). Snake venom components enhance pain upon subcutaneous injection: an initial examination of spinal cord mediators. Pain, 111, 65-76. doi:10.1016/j.pain.2004.06.001
    • NLM

      Gutiérrez JM, Gutiérrez JM, Milligan ED, Wieseler-Frank J, Britto LRG de, Maier SF, Watkins LR, Cury Y. Snake venom components enhance pain upon subcutaneous injection: an initial examination of spinal cord mediators [Internet]. Pain. 2004 ; 111 65-76.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2004.06.001
    • Vancouver

      Gutiérrez JM, Gutiérrez JM, Milligan ED, Wieseler-Frank J, Britto LRG de, Maier SF, Watkins LR, Cury Y. Snake venom components enhance pain upon subcutaneous injection: an initial examination of spinal cord mediators [Internet]. Pain. 2004 ; 111 65-76.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2004.06.001
  • Source: Pain. Unidade: FMRP

    Subjects: FARMACOLOGIA, DOR, NEOPLASIAS ABDOMINAIS, NEOPLASIAS PÉLVICAS, ANALGESIA

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    • ABNT

      OLIVEIRA, Raquel de e REIS, Marlene Paulino dos e PRADO, Wiliam Alves do. The effects of early or late neurolytic sympathetic plexus block on the management of abdominal or pelvic cancer pain. Pain, v. 110, p. 400-408, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2004.04.023. Acesso em: 02 nov. 2024.
    • APA

      Oliveira, R. de, Reis, M. P. dos, & Prado, W. A. do. (2004). The effects of early or late neurolytic sympathetic plexus block on the management of abdominal or pelvic cancer pain. Pain, 110, 400-408. doi:10.1016/j.pain.2004.04.023
    • NLM

      Oliveira R de, Reis MP dos, Prado WA do. The effects of early or late neurolytic sympathetic plexus block on the management of abdominal or pelvic cancer pain [Internet]. Pain. 2004 ; 110 400-408.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2004.04.023
    • Vancouver

      Oliveira R de, Reis MP dos, Prado WA do. The effects of early or late neurolytic sympathetic plexus block on the management of abdominal or pelvic cancer pain [Internet]. Pain. 2004 ; 110 400-408.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.pain.2004.04.023
  • Source: Pain. Unidade: FMRP

    Subjects: DOR, FARMACOLOGIA

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    • ABNT

      PRADO, Wiliam Alves do e VILLARREAL, Cristiane Floral e DEL BEL, Elaine Aparecida. Involvement of the anterior pretectal nucleus in the control of persistent pain: a behavioral and c-Fos expression study in thr rat. Pain, v. 103, p. 163-174, 2003Tradução . . Disponível em: https://doi.org/10.1016/s0304-3959(02)00449-9. Acesso em: 02 nov. 2024.
    • APA

      Prado, W. A. do, Villarreal, C. F., & Del Bel, E. A. (2003). Involvement of the anterior pretectal nucleus in the control of persistent pain: a behavioral and c-Fos expression study in thr rat. Pain, 103, 163-174. doi:10.1016/s0304-3959(02)00449-9
    • NLM

      Prado WA do, Villarreal CF, Del Bel EA. Involvement of the anterior pretectal nucleus in the control of persistent pain: a behavioral and c-Fos expression study in thr rat [Internet]. Pain. 2003 ; 103 163-174.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/s0304-3959(02)00449-9
    • Vancouver

      Prado WA do, Villarreal CF, Del Bel EA. Involvement of the anterior pretectal nucleus in the control of persistent pain: a behavioral and c-Fos expression study in thr rat [Internet]. Pain. 2003 ; 103 163-174.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/s0304-3959(02)00449-9
  • Source: Pain. Unidade: FMRP

    Subjects: FARMACOLOGIA, DOR, HIPERSENSIBILIDADE

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    • ABNT

      SACHS, Daniela et al. Tumour necrosis factor-'alfa', interleukin-1'beta' and interleukin-8 induce persistent mechanical nociceptor hypersensitivity. Pain, v. 96, p. 89-97, 2002Tradução . . Disponível em: http://www.sciencedirect.com/science?_ob=JournalURL&_cdi=4865&_auth=y&_acct=C000049650&_version=1&_urlVersion=0&_userid=972067&md5=7c8c95fbc30de605a60e40dc05910fc6. Acesso em: 02 nov. 2024.
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      Sachs, D., Cunha, F. de Q., Poole, S., & Ferreira, S. H. (2002). Tumour necrosis factor-'alfa', interleukin-1'beta' and interleukin-8 induce persistent mechanical nociceptor hypersensitivity. Pain, 96, 89-97. Recuperado de http://www.sciencedirect.com/science?_ob=JournalURL&_cdi=4865&_auth=y&_acct=C000049650&_version=1&_urlVersion=0&_userid=972067&md5=7c8c95fbc30de605a60e40dc05910fc6
    • NLM

      Sachs D, Cunha F de Q, Poole S, Ferreira SH. Tumour necrosis factor-'alfa', interleukin-1'beta' and interleukin-8 induce persistent mechanical nociceptor hypersensitivity [Internet]. Pain. 2002 ; 96 89-97.[citado 2024 nov. 02 ] Available from: http://www.sciencedirect.com/science?_ob=JournalURL&_cdi=4865&_auth=y&_acct=C000049650&_version=1&_urlVersion=0&_userid=972067&md5=7c8c95fbc30de605a60e40dc05910fc6
    • Vancouver

      Sachs D, Cunha F de Q, Poole S, Ferreira SH. Tumour necrosis factor-'alfa', interleukin-1'beta' and interleukin-8 induce persistent mechanical nociceptor hypersensitivity [Internet]. Pain. 2002 ; 96 89-97.[citado 2024 nov. 02 ] Available from: http://www.sciencedirect.com/science?_ob=JournalURL&_cdi=4865&_auth=y&_acct=C000049650&_version=1&_urlVersion=0&_userid=972067&md5=7c8c95fbc30de605a60e40dc05910fc6
  • Source: Pain. Unidade: FMRP

    Subjects: FARMACOLOGIA, FARMACOCINÉTICA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      PRADO, Wiliam Alves do e FAGANELLO, Fernanda A. The anterior pretectal nucleus participates as a relay station in the glutamate-, but not morphine-induced antinociception from the dorsal raphe nucleus in rats. Pain, v. 88, p. 169-176, 2000Tradução . . Disponível em: https://doi.org/10.1016/s0304-3959(00)00326-2. Acesso em: 02 nov. 2024.
    • APA

      Prado, W. A. do, & Faganello, F. A. (2000). The anterior pretectal nucleus participates as a relay station in the glutamate-, but not morphine-induced antinociception from the dorsal raphe nucleus in rats. Pain, 88, 169-176. doi:10.1016/s0304-3959(00)00326-2
    • NLM

      Prado WA do, Faganello FA. The anterior pretectal nucleus participates as a relay station in the glutamate-, but not morphine-induced antinociception from the dorsal raphe nucleus in rats [Internet]. Pain. 2000 ; 88 169-176.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/s0304-3959(00)00326-2
    • Vancouver

      Prado WA do, Faganello FA. The anterior pretectal nucleus participates as a relay station in the glutamate-, but not morphine-induced antinociception from the dorsal raphe nucleus in rats [Internet]. Pain. 2000 ; 88 169-176.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/s0304-3959(00)00326-2

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