IL-17 mediates articular hypernociception in antigen-induced arthritis in mice (2010)
- Authors:
- USP affiliated authors: CUNHA, THIAGO MATTAR - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; FERREIRA, SERGIO HENRIQUE - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.pain.2009.11.006
- Subjects: NEUTRÓFILOS; ARTRITE REUMATOIDE; CITOCINAS
- Language: Inglês
- Imprenta:
- Source:
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
PINTO, Larissa G. et al. IL-17 mediates articular hypernociception in antigen-induced arthritis in mice. Pain, v. 148, n. 2, p. 247-256, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2009.11.006. Acesso em: 09 jan. 2026. -
APA
Pinto, L. G., Cunha, T. M., Vieira, S. M., Lemos, H. P., Verri Júnior, W. A., Cunha, F. de Q., & Ferreira, S. H. (2010). IL-17 mediates articular hypernociception in antigen-induced arthritis in mice. Pain, 148( 2), 247-256. doi:10.1016/j.pain.2009.11.006 -
NLM
Pinto LG, Cunha TM, Vieira SM, Lemos HP, Verri Júnior WA, Cunha F de Q, Ferreira SH. IL-17 mediates articular hypernociception in antigen-induced arthritis in mice [Internet]. Pain. 2010 ; 148( 2): 247-256.[citado 2026 jan. 09 ] Available from: https://doi.org/10.1016/j.pain.2009.11.006 -
Vancouver
Pinto LG, Cunha TM, Vieira SM, Lemos HP, Verri Júnior WA, Cunha F de Q, Ferreira SH. IL-17 mediates articular hypernociception in antigen-induced arthritis in mice [Internet]. Pain. 2010 ; 148( 2): 247-256.[citado 2026 jan. 09 ] Available from: https://doi.org/10.1016/j.pain.2009.11.006 - The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia
- Role of CCR2 de novo expression in neutrophils in experimental arthritis
- Involvement of nuclear factor kappa B in the maintenance of persistent inflammatory hypernociception
- Kaurenoic acid from Sphagneticola trilobata inhibits inflammatory pain: effect on cytokine production and activation of the NO–Cyclic GMP–protein kinase G–ATP-sensitive potassium channel signaling pathway
- Fractalkine mediates inflammatory pain through activation of satellite glial cells
- Participation of nonpeptidergic nociceptors in inflammatory pain
- Acetic acid and Phenyl-P-Benzoquinone-induced overt pain-like behavior depends on spinal activation of map kinases and PI3K in mice
- Granulocyte-colony stimulating factor (G-CSF) induces mechanical hyperalgesia via spinal activiation of MAP kinases and 'PL IND.3'K in mice
- Investigation of the mechanisms involved in the analgesic effect of choline
- The activation of peripheral kappa opioid receptors directly blocks inflammatory hyperalgesia through the stimulation of PI3Kgamma/AKT?NO signaling pathway
Informações sobre o DOI: 10.1016/j.pain.2009.11.006 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
