Filtros : "Nutrition metabolism and cardiovascular diseases" Removidos: "AVALIAÇÃO DE RISCO" "Fernandes, Ariana Ester" Limpar

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  • Source: Nutrition metabolism and cardiovascular diseases. Unidade: FM

    Subjects: ADIPOSIDADE, FÍGADO GORDUROSO, FATORES DE RISCO, OBESIDADE

    Acesso à fonteDOIHow to cite
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    • ABNT

      NETTO, Alvaro M et al. Adiposity, hepatic steatosis, and metabolic health transitions in people with obesity: Influences of age and sex. Nutrition metabolism and cardiovascular diseases, v. 33, n. 6, p. 1149-1157, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/56012. Acesso em: 08 out. 2024.
    • APA

      Netto, A. M., Kashiwagi, N. M., Minanni, C. A., Santos Filho, R. D. dos, & Cesena, F. Y. (2023). Adiposity, hepatic steatosis, and metabolic health transitions in people with obesity: Influences of age and sex. Nutrition metabolism and cardiovascular diseases, 33( 6), 1149-1157. doi:10.1016/j.numecd.2023.03.020
    • NLM

      Netto AM, Kashiwagi NM, Minanni CA, Santos Filho RD dos, Cesena FY. Adiposity, hepatic steatosis, and metabolic health transitions in people with obesity: Influences of age and sex [Internet]. Nutrition metabolism and cardiovascular diseases. 2023 ; 33( 6): 1149-1157.[citado 2024 out. 08 ] Available from: https://observatorio.fm.usp.br/handle/OPI/56012
    • Vancouver

      Netto AM, Kashiwagi NM, Minanni CA, Santos Filho RD dos, Cesena FY. Adiposity, hepatic steatosis, and metabolic health transitions in people with obesity: Influences of age and sex [Internet]. Nutrition metabolism and cardiovascular diseases. 2023 ; 33( 6): 1149-1157.[citado 2024 out. 08 ] Available from: https://observatorio.fm.usp.br/handle/OPI/56012
  • Source: Nutrition metabolism and cardiovascular diseases. Unidades: HU, FSP, FM

    Subjects: COMPORTAMENTO ALIMENTAR, ARTERIOSCLEROSE, ARTERIOSCLEROSE CORONÁRIA, ESTUDOS LONGITUDINAIS

    Acesso à fonteDOIHow to cite
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    • ABNT

      ALVES, Mariane de Almeida et al. Dietary patterns and subclinical atherosclerosis incidence and progression: Results from ELSA-Brasil. Nutrition metabolism and cardiovascular diseases, v. 33, n. 1, p. 47-55, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/55959. Acesso em: 08 out. 2024.
    • APA

      Alves, M. de A., Miranda, A. A. M., Cacau, L. T., Levy, J., Generoso, G., Bittencourt, M. S., et al. (2023). Dietary patterns and subclinical atherosclerosis incidence and progression: Results from ELSA-Brasil. Nutrition metabolism and cardiovascular diseases, 33( 1), 47-55. doi:10.1016/j.numecd.2022.09.021
    • NLM

      Alves M de A, Miranda AAM, Cacau LT, Levy J, Generoso G, Bittencourt MS, Lotufo PA, Benseñor IJM, Marchioni DML. Dietary patterns and subclinical atherosclerosis incidence and progression: Results from ELSA-Brasil [Internet]. Nutrition metabolism and cardiovascular diseases. 2023 ; 33( 1): 47-55.[citado 2024 out. 08 ] Available from: https://observatorio.fm.usp.br/handle/OPI/55959
    • Vancouver

      Alves M de A, Miranda AAM, Cacau LT, Levy J, Generoso G, Bittencourt MS, Lotufo PA, Benseñor IJM, Marchioni DML. Dietary patterns and subclinical atherosclerosis incidence and progression: Results from ELSA-Brasil [Internet]. Nutrition metabolism and cardiovascular diseases. 2023 ; 33( 1): 47-55.[citado 2024 out. 08 ] Available from: https://observatorio.fm.usp.br/handle/OPI/55959
  • Source: Nutrition metabolism and cardiovascular diseases. Unidade: FM

    Subjects: CARDIOMEGALIA, SAL (CONDIMENTO), RECEPTORES DE ANGIOTENSINA

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    • ABNT

      DOPONA, E. P. B et al. Myocardial hypertrophy induced by high salt consumption is prevented by angiotensin II AT2 receptor agonist. Nutrition metabolism and cardiovascular diseases, v. 29, n. 3, p. 301-305, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.numecd.2018.11.001. Acesso em: 08 out. 2024.
    • APA

      Dopona, E. P. B., Rocha, V. F., Furukawa, L. N. S., Oliveira, I. B., & Heimann, J. C. (2019). Myocardial hypertrophy induced by high salt consumption is prevented by angiotensin II AT2 receptor agonist. Nutrition metabolism and cardiovascular diseases, 29( 3), 301-305. doi:10.1016/j.numecd.2018.11.001
    • NLM

      Dopona EPB, Rocha VF, Furukawa LNS, Oliveira IB, Heimann JC. Myocardial hypertrophy induced by high salt consumption is prevented by angiotensin II AT2 receptor agonist [Internet]. Nutrition metabolism and cardiovascular diseases. 2019 ; 29( 3): 301-305.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.numecd.2018.11.001
    • Vancouver

      Dopona EPB, Rocha VF, Furukawa LNS, Oliveira IB, Heimann JC. Myocardial hypertrophy induced by high salt consumption is prevented by angiotensin II AT2 receptor agonist [Internet]. Nutrition metabolism and cardiovascular diseases. 2019 ; 29( 3): 301-305.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.numecd.2018.11.001
  • Source: Nutrition metabolism and cardiovascular diseases. Unidade: FM

    Subjects: OBESIDADE, MACRONUTRIENTES, SOBREPESO, INTESTINOS

    Acesso à fonteDOIHow to cite
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    • ABNT

      DUARTE, S. M. B et al. Gut microbiome composition in lean patients with NASH is associated with liver damage independent of caloric intake: A prospective pilot study. Nutrition metabolism and cardiovascular diseases, v. 28, n. 4, p. 369-384, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.numecd.2017.10.014. Acesso em: 08 out. 2024.
    • APA

      Duarte, S. M. B., Sabino, E. C., Carrilho, F. J., & Oliveira, C. P. (2018). Gut microbiome composition in lean patients with NASH is associated with liver damage independent of caloric intake: A prospective pilot study. Nutrition metabolism and cardiovascular diseases, 28( 4), 369-384. doi:10.1016/j.numecd.2017.10.014
    • NLM

      Duarte SMB, Sabino EC, Carrilho FJ, Oliveira CP. Gut microbiome composition in lean patients with NASH is associated with liver damage independent of caloric intake: A prospective pilot study [Internet]. Nutrition metabolism and cardiovascular diseases. 2018 ; 28( 4): 369-384.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.numecd.2017.10.014
    • Vancouver

      Duarte SMB, Sabino EC, Carrilho FJ, Oliveira CP. Gut microbiome composition in lean patients with NASH is associated with liver damage independent of caloric intake: A prospective pilot study [Internet]. Nutrition metabolism and cardiovascular diseases. 2018 ; 28( 4): 369-384.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.numecd.2017.10.014
  • Source: Nutrition metabolism and cardiovascular diseases. Unidade: FM

    Subjects: OBESIDADE MÓRBIDA, VENTRÍCULO CARDÍACO, FATORES DE RISCO, DOENÇAS CARDIOVASCULARES

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    • ABNT

      FERNANDES-CARDOSO, A. et al. Epicardial fat thickness correlates with P-wave duration, left atrial size and decreased left ventricular systolic function in morbid obesity. Nutrition metabolism and cardiovascular diseases, v. 27, n. 8, p. 731-738, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.numecd.2017.05.009. Acesso em: 08 out. 2024.
    • APA

      Fernandes-cardoso, A., Furtado, M. S., Grindler, J., Ferreira, L. A., Andrade, J. L., & Santo, M. A. (2017). Epicardial fat thickness correlates with P-wave duration, left atrial size and decreased left ventricular systolic function in morbid obesity. Nutrition metabolism and cardiovascular diseases, 27( 8), 731-738. doi:10.1016/j.numecd.2017.05.009
    • NLM

      Fernandes-cardoso A, Furtado MS, Grindler J, Ferreira LA, Andrade JL, Santo MA. Epicardial fat thickness correlates with P-wave duration, left atrial size and decreased left ventricular systolic function in morbid obesity [Internet]. Nutrition metabolism and cardiovascular diseases. 2017 ; 27( 8): 731-738.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.numecd.2017.05.009
    • Vancouver

      Fernandes-cardoso A, Furtado MS, Grindler J, Ferreira LA, Andrade JL, Santo MA. Epicardial fat thickness correlates with P-wave duration, left atrial size and decreased left ventricular systolic function in morbid obesity [Internet]. Nutrition metabolism and cardiovascular diseases. 2017 ; 27( 8): 731-738.[citado 2024 out. 08 ] Available from: https://doi.org/10.1016/j.numecd.2017.05.009

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