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Trabalho de evento-resumo periodico
Quantitative assessment of PTEN loss (qPTEN) is strongly associated with biochemical recurrence and may improve treatment decisions after surgery (2020)
Jamaspishvili, Tamara; Patel, Palak; Niu, Yi; Vidotti, Thiago; Ouellet, Veronique; Mes-Masson, Anne-Marie; Lotan, Tamara; Squire, Jeremy Andrew ; Peng, Yingwei; Siemens, D.; Berman, David
Fonte: Laboratory Investigation. Nome do evento: USCAP Annual Meeting. Unidade: FMRP
Assuntos: BIOQUÍMICA, CIRURGIA, PROSTATECTOMIA, PRÓSTATA, NEOPLASIAS
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ABNT
JAMASPISHVILI, Tamara et al. Quantitative assessment of PTEN loss (qPTEN) is strongly associated with biochemical recurrence and may improve treatment decisions after surgery. Laboratory Investigation. London: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 03 nov. 2025. , 2020
APA
Jamaspishvili, T., Patel, P., Niu, Y., Vidotti, T., Ouellet, V., Mes-Masson, A. -M., et al. (2020). Quantitative assessment of PTEN loss (qPTEN) is strongly associated with biochemical recurrence and may improve treatment decisions after surgery. Laboratory Investigation. London: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Jamaspishvili T, Patel P, Niu Y, Vidotti T, Ouellet V, Mes-Masson A-M, Lotan T, Squire JA, Peng Y, Siemens D, Berman D. Quantitative assessment of PTEN loss (qPTEN) is strongly associated with biochemical recurrence and may improve treatment decisions after surgery. Laboratory Investigation. 2020 ; 100 905.[citado 2025 nov. 03 ]
Vancouver
Jamaspishvili T, Patel P, Niu Y, Vidotti T, Ouellet V, Mes-Masson A-M, Lotan T, Squire JA, Peng Y, Siemens D, Berman D. Quantitative assessment of PTEN loss (qPTEN) is strongly associated with biochemical recurrence and may improve treatment decisions after surgery. Laboratory Investigation. 2020 ; 100 905.[citado 2025 nov. 03 ]
Artigo de periodico
Use of multicolor fluorescence in situ hybridization to detect deletions in clinical tissue sections (2018)
Yoshimoto, Maisa; Ludkovski, Olga; Good, Jennifer; Pereira, Ciro; Gooding, Robert J.; McGowan-Jordan, Jean; Boag, Alexander; Evans, Andrew; Tsao, Ming-Sound; Nuin, Paulo; Squire, Jeremy Andrew
Fonte: Laboratory Investigation. Unidade: FMRP
Assuntos: DELEÇÃO DE GENES, HIBRIDIZAÇÃO, GENES SUPRESSORES DE TUMOR, NEOPLASIAS, GENÉTICA
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ABNT
YOSHIMOTO, Maisa et al. Use of multicolor fluorescence in situ hybridization to detect deletions in clinical tissue sections. Laboratory Investigation, v. 98, n. 4, p. 403-413, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41374-017-0007-2. Acesso em: 03 nov. 2025.
APA
Yoshimoto, M., Ludkovski, O., Good, J., Pereira, C., Gooding, R. J., McGowan-Jordan, J., et al. (2018). Use of multicolor fluorescence in situ hybridization to detect deletions in clinical tissue sections. Laboratory Investigation, 98( 4), 403-413. doi:10.1038/s41374-017-0007-2
NLM
Yoshimoto M, Ludkovski O, Good J, Pereira C, Gooding RJ, McGowan-Jordan J, Boag A, Evans A, Tsao M-S, Nuin P, Squire JA. Use of multicolor fluorescence in situ hybridization to detect deletions in clinical tissue sections [Internet]. Laboratory Investigation. 2018 ; 98( 4): 403-413.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/s41374-017-0007-2
Vancouver
Yoshimoto M, Ludkovski O, Good J, Pereira C, Gooding RJ, McGowan-Jordan J, Boag A, Evans A, Tsao M-S, Nuin P, Squire JA. Use of multicolor fluorescence in situ hybridization to detect deletions in clinical tissue sections [Internet]. Laboratory Investigation. 2018 ; 98( 4): 403-413.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/s41374-017-0007-2
Trabalho de evento-resumo periodico
Compensated versus decompensated advanced HCV-related chronic liver disease: a morphologic and morphometric autopsy study (2015)
Dettoni, Juliano Bertollo; Cogliati, Bruno ; Castro, Isaac de; Alves, Venâncio Avancini Ferreira
Fonte: Laboratory Investigation. Nome do evento: Annual Meeting Abstracts. Unidades: FMVZ, FM
Assuntos: CIRROSE HEPÁTICA, NECRÓPSIA ANIMAL
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ABNT
DETTONI, Juliano Bertollo et al. Compensated versus decompensated advanced HCV-related chronic liver disease: a morphologic and morphometric autopsy study. Laboratory Investigation. New York: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo. Disponível em: http://www.nature.com/labinvest/journal/v95/n1s/pdf/labinvest201519a.pdf?WT.ec_id=LABINVEST-201502. Acesso em: 03 nov. 2025. , 2015
APA
Dettoni, J. B., Cogliati, B., Castro, I. de, & Alves, V. A. F. (2015). Compensated versus decompensated advanced HCV-related chronic liver disease: a morphologic and morphometric autopsy study. Laboratory Investigation. New York: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo. Recuperado de http://www.nature.com/labinvest/journal/v95/n1s/pdf/labinvest201519a.pdf?WT.ec_id=LABINVEST-201502
NLM
Dettoni JB, Cogliati B, Castro I de, Alves VAF. Compensated versus decompensated advanced HCV-related chronic liver disease: a morphologic and morphometric autopsy study [Internet]. Laboratory Investigation. 2015 ; 95( S1): 418A.[citado 2025 nov. 03 ] Available from: http://www.nature.com/labinvest/journal/v95/n1s/pdf/labinvest201519a.pdf?WT.ec_id=LABINVEST-201502
Vancouver
Dettoni JB, Cogliati B, Castro I de, Alves VAF. Compensated versus decompensated advanced HCV-related chronic liver disease: a morphologic and morphometric autopsy study [Internet]. Laboratory Investigation. 2015 ; 95( S1): 418A.[citado 2025 nov. 03 ] Available from: http://www.nature.com/labinvest/journal/v95/n1s/pdf/labinvest201519a.pdf?WT.ec_id=LABINVEST-201502
Trabalho de evento-resumo periodico
A broad immunohistochemistry panel in Hepatocellular Carcinoma (HCC) seems to segregate molecular subtypes by grade: a cross-sectional study in 80 brazilian autopsies (2014)
Felipe-Silva, Aloisio; Wakamatsu, A; Cirqueira, C S; Cassol, C; Alves, V
Fonte: Laboratory Investigation. Nome do evento: Annual Meeting of the United States and Canadian Academy of Pathology (USCAP). Unidade: HU
Assuntos: CARCINOMA HEPATOCELULAR, IMUNOHISTOQUÍMICA, BIOMARCADORES
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ABNT
FELIPE-SILVA, Aloisio et al. A broad immunohistochemistry panel in Hepatocellular Carcinoma (HCC) seems to segregate molecular subtypes by grade: a cross-sectional study in 80 brazilian autopsies. Laboratory Investigation. Baltimore: Hospital Universitário, Universidade de São Paulo. . Acesso em: 03 nov. 2025. , 2014
APA
Felipe-Silva, A., Wakamatsu, A., Cirqueira, C. S., Cassol, C., & Alves, V. (2014). A broad immunohistochemistry panel in Hepatocellular Carcinoma (HCC) seems to segregate molecular subtypes by grade: a cross-sectional study in 80 brazilian autopsies. Laboratory Investigation. Baltimore: Hospital Universitário, Universidade de São Paulo.
NLM
Felipe-Silva A, Wakamatsu A, Cirqueira CS, Cassol C, Alves V. A broad immunohistochemistry panel in Hepatocellular Carcinoma (HCC) seems to segregate molecular subtypes by grade: a cross-sectional study in 80 brazilian autopsies. Laboratory Investigation. 2014 ; 94( S1): 419A res. 1718.[citado 2025 nov. 03 ]
Vancouver
Felipe-Silva A, Wakamatsu A, Cirqueira CS, Cassol C, Alves V. A broad immunohistochemistry panel in Hepatocellular Carcinoma (HCC) seems to segregate molecular subtypes by grade: a cross-sectional study in 80 brazilian autopsies. Laboratory Investigation. 2014 ; 94( S1): 419A res. 1718.[citado 2025 nov. 03 ]
Artigo de periodico
Bradykinin inhibits hepatic gluconeogenesis in obese mice (2012)
Barros, Carlos Castilho; Haro, Anderson; Russo, Fernanda Jacqueline; Schadock, Ines; Almeida, Sandro Soares; Reis, Felipe Castellani; Moraes, Milton Rocha; Haidar, Andre; Hirata, Aparecida Emiko; Mori, Marcelo; Bacurau, Reury Frank Pereira; Würtele, Martin; Bader, Michael; Pesquero, João Bosco; Araujo, Ronaldo Carvalho
Fonte: Laboratory Investigation. Unidade: EACH
Assuntos: BRADICININA, RECEPTORES, CININAS, LEPTINA, OBESIDADE
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ABNT
BARROS, Carlos Castilho et al. Bradykinin inhibits hepatic gluconeogenesis in obese mice. Laboratory Investigation, n. 92, p. 1419-1427, 2012Tradução . . Disponível em: https://doi.org/10.1038/labinvest.2012.105. Acesso em: 03 nov. 2025.
APA
Barros, C. C., Haro, A., Russo, F. J., Schadock, I., Almeida, S. S., Reis, F. C., et al. (2012). Bradykinin inhibits hepatic gluconeogenesis in obese mice. Laboratory Investigation, ( 92), 1419-1427. doi:10.1038/labinvest.2012.105
NLM
Barros CC, Haro A, Russo FJ, Schadock I, Almeida SS, Reis FC, Moraes MR, Haidar A, Hirata AE, Mori M, Bacurau RFP, Würtele M, Bader M, Pesquero JB, Araujo RC. Bradykinin inhibits hepatic gluconeogenesis in obese mice [Internet]. Laboratory Investigation. 2012 ;( 92): 1419-1427.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/labinvest.2012.105
Vancouver
Barros CC, Haro A, Russo FJ, Schadock I, Almeida SS, Reis FC, Moraes MR, Haidar A, Hirata AE, Mori M, Bacurau RFP, Würtele M, Bader M, Pesquero JB, Araujo RC. Bradykinin inhibits hepatic gluconeogenesis in obese mice [Internet]. Laboratory Investigation. 2012 ;( 92): 1419-1427.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/labinvest.2012.105
Artigo de periodico
Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis (2011)
Zecchin, Karina G.; Rossato, Franco A.; Raposo, Helena F.; Melo, Daniela R.; Alberici, Luciane Carla; Oliveira, Helena C. F.; Castilho, Roger F.; Coletta, Ricardo D.; Vercesi, Aníbal E.; Graner, Edgard
Fonte: Laboratory Investigation. Unidade: FCFRP
Assuntos: APOPTOSE, MELANOMA
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ABNT
ZECCHIN, Karina G. et al. Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis. Laboratory Investigation, v. 91, n. 2, p. 232-240, 2011Tradução . . Disponível em: https://doi.org/10.1038/labinvest.2010.157. Acesso em: 03 nov. 2025.
APA
Zecchin, K. G., Rossato, F. A., Raposo, H. F., Melo, D. R., Alberici, L. C., Oliveira, H. C. F., et al. (2011). Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis. Laboratory Investigation, 91( 2), 232-240. doi:10.1038/labinvest.2010.157
NLM
Zecchin KG, Rossato FA, Raposo HF, Melo DR, Alberici LC, Oliveira HCF, Castilho RF, Coletta RD, Vercesi AE, Graner E. Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis [Internet]. Laboratory Investigation. 2011 ; 91( 2): 232-240.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/labinvest.2010.157
Vancouver
Zecchin KG, Rossato FA, Raposo HF, Melo DR, Alberici LC, Oliveira HCF, Castilho RF, Coletta RD, Vercesi AE, Graner E. Inhibition of fatty acid synthase in melanoma cells activates the intrinsic pathway of apoptosis [Internet]. Laboratory Investigation. 2011 ; 91( 2): 232-240.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/labinvest.2010.157
Artigo de periodico
Bone marrow mononuclear cells attenuate fibrosis development after severe acute kidney injury (2010)
Semedo, Patrícia; Donizetti-Oliveira, Cassiano; Burgos-Silva, Marina; Cenedeze, Marco Antonio; Malheiros, Denise Maria Avancini Costa; Pacheco-Silva, Alvaro; Câmara, Niels Olsen Saraiva
Fonte: Laboratory Investigation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
SEMEDO, Patrícia et al. Bone marrow mononuclear cells attenuate fibrosis development after severe acute kidney injury. Laboratory Investigation, v. 90, n. 5, p. 685-695, 2010Tradução . . Disponível em: https://doi.org/10.1038/labinvest.2010.45. Acesso em: 03 nov. 2025.
APA
Semedo, P., Donizetti-Oliveira, C., Burgos-Silva, M., Cenedeze, M. A., Malheiros, D. M. A. C., Pacheco-Silva, A., & Câmara, N. O. S. (2010). Bone marrow mononuclear cells attenuate fibrosis development after severe acute kidney injury. Laboratory Investigation, 90( 5), 685-695. doi:10.1038/labinvest.2010.45
NLM
Semedo P, Donizetti-Oliveira C, Burgos-Silva M, Cenedeze MA, Malheiros DMAC, Pacheco-Silva A, Câmara NOS. Bone marrow mononuclear cells attenuate fibrosis development after severe acute kidney injury [Internet]. Laboratory Investigation. 2010 ; 90( 5): 685-695.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/labinvest.2010.45
Vancouver
Semedo P, Donizetti-Oliveira C, Burgos-Silva M, Cenedeze MA, Malheiros DMAC, Pacheco-Silva A, Câmara NOS. Bone marrow mononuclear cells attenuate fibrosis development after severe acute kidney injury [Internet]. Laboratory Investigation. 2010 ; 90( 5): 685-695.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1038/labinvest.2010.45
Artigo de periodico
Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis (2010)
Celes, Mara Rúbia Nunes; Malvestio, Lygia M.; Torres-Dueñas, Diego; Blefari, Valdecir; Campos, Érica Carolina; Ramos, Simone Gusmão; Prado, Cibele Maria; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Fonte: Laboratory Investigation. Unidade: FMRP
Assuntos: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
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ABNT
CELES, Mara Rúbia Nunes et al. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, v. 90, n. 4, p. 531-542, 2010Tradução . . Disponível em: https://doi.org/10.1186/cc9157. Acesso em: 03 nov. 2025.
APA
Celes, M. R. N., Malvestio, L. M., Torres-Dueñas, D., Blefari, V., Campos, É. C., Ramos, S. G., et al. (2010). Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, 90( 4), 531-542. doi:10.1186/cc9157
NLM
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1186/cc9157
Vancouver
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1186/cc9157
Trabalho de evento-resumo periodico
Significance and relationship between cytokeratins, p63, 'beta'-catenin and matrix metalloproteinases expression in DMBA-induced mammary tumors in mice fed with fat diet (2007)
Ribeiro-Silva, Alfredo; Ramalho, Leandra Naíra Zambelli; Ramalho, Fernando S.; Garcia, Sérgio Brito; Zucoloto, Sérgio
Fonte: Laboratory Investigation. Nome do evento: United States and Canadian Academy of Pathology (USCAP). Unidade: FMRP
Assunto: NEOPLASIAS MAMÁRIAS
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ABNT
RIBEIRO-SILVA, Alfredo et al. Significance and relationship between cytokeratins, p63, 'beta'-catenin and matrix metalloproteinases expression in DMBA-induced mammary tumors in mice fed with fat diet. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 03 nov. 2025. , 2007
APA
Ribeiro-Silva, A., Ramalho, L. N. Z., Ramalho, F. S., Garcia, S. B., & Zucoloto, S. (2007). Significance and relationship between cytokeratins, p63, 'beta'-catenin and matrix metalloproteinases expression in DMBA-induced mammary tumors in mice fed with fat diet. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Ribeiro-Silva A, Ramalho LNZ, Ramalho FS, Garcia SB, Zucoloto S. Significance and relationship between cytokeratins, p63, 'beta'-catenin and matrix metalloproteinases expression in DMBA-induced mammary tumors in mice fed with fat diet. Laboratory Investigation. 2007 ; 87 47A.[citado 2025 nov. 03 ]
Vancouver
Ribeiro-Silva A, Ramalho LNZ, Ramalho FS, Garcia SB, Zucoloto S. Significance and relationship between cytokeratins, p63, 'beta'-catenin and matrix metalloproteinases expression in DMBA-induced mammary tumors in mice fed with fat diet. Laboratory Investigation. 2007 ; 87 47A.[citado 2025 nov. 03 ]
Trabalho de evento-resumo periodico
Apoptosis-related proteins in cytokeratin 5-positive in breast carcinomas (2007)
Ribeiro-Silva, Alfredo
Fonte: Laboratory Investigation. Nome do evento: United States and Canadian Academy of Pathology (USCAP). Unidade: FMRP
Assuntos: CARCINOMA, GENES, PROLIFERAÇÃO CELULAR
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ABNT
RIBEIRO-SILVA, Alfredo. Apoptosis-related proteins in cytokeratin 5-positive in breast carcinomas. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 03 nov. 2025. , 2007
APA
Ribeiro-Silva, A. (2007). Apoptosis-related proteins in cytokeratin 5-positive in breast carcinomas. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Ribeiro-Silva A. Apoptosis-related proteins in cytokeratin 5-positive in breast carcinomas. Laboratory Investigation. 2007 ; 87 S244.[citado 2025 nov. 03 ]
Vancouver
Ribeiro-Silva A. Apoptosis-related proteins in cytokeratin 5-positive in breast carcinomas. Laboratory Investigation. 2007 ; 87 S244.[citado 2025 nov. 03 ]
Trabalho de evento-resumo periodico
The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype (2007)
Ribeiro-Silva, Alfredo; Zucoloto, Sérgio
Fonte: Laboratory Investigation. Nome do evento: United States and Canadian Academy of Pathology (USCAP). Unidade: FMRP
Assunto: NEOPLASIAS MAMÁRIAS
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ABNT
RIBEIRO-SILVA, Alfredo e ZUCOLOTO, Sérgio. The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 03 nov. 2025. , 2007
APA
Ribeiro-Silva, A., & Zucoloto, S. (2007). The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Ribeiro-Silva A, Zucoloto S. The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. 2007 ; 87 47A.[citado 2025 nov. 03 ]
Vancouver
Ribeiro-Silva A, Zucoloto S. The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. 2007 ; 87 47A.[citado 2025 nov. 03 ]
Trabalho de evento-resumo periodico
Sequential analyses of the growth of activated stromal foci with increased expression of CD34, CD68, 'alfa'-smooth muscle actin, and COX-2 in the rat colon carcinogenesis: putative markers of field cancerization and relationship with increased crypt fission rates (2007)
Garcia, Sérgio Brito; Turatti, A.; Ribeiro-Silva, Alfredo; Zucoloto, Sérgio
Fonte: Laboratory Investigation. Nome do evento: United States and Canadian Academy of Pathology (USCAP). Unidade: FMRP
Assunto: CARCINOGÊNESE ANIMAL
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ABNT
GARCIA, Sérgio Brito et al. Sequential analyses of the growth of activated stromal foci with increased expression of CD34, CD68, 'alfa'-smooth muscle actin, and COX-2 in the rat colon carcinogenesis: putative markers of field cancerization and relationship with increased crypt fission rates. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 03 nov. 2025. , 2007
APA
Garcia, S. B., Turatti, A., Ribeiro-Silva, A., & Zucoloto, S. (2007). Sequential analyses of the growth of activated stromal foci with increased expression of CD34, CD68, 'alfa'-smooth muscle actin, and COX-2 in the rat colon carcinogenesis: putative markers of field cancerization and relationship with increased crypt fission rates. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Garcia SB, Turatti A, Ribeiro-Silva A, Zucoloto S. Sequential analyses of the growth of activated stromal foci with increased expression of CD34, CD68, 'alfa'-smooth muscle actin, and COX-2 in the rat colon carcinogenesis: putative markers of field cancerization and relationship with increased crypt fission rates. Laboratory Investigation. 2007 ; 87 114A.[citado 2025 nov. 03 ]
Vancouver
Garcia SB, Turatti A, Ribeiro-Silva A, Zucoloto S. Sequential analyses of the growth of activated stromal foci with increased expression of CD34, CD68, 'alfa'-smooth muscle actin, and COX-2 in the rat colon carcinogenesis: putative markers of field cancerization and relationship with increased crypt fission rates. Laboratory Investigation. 2007 ; 87 114A.[citado 2025 nov. 03 ]
Trabalho de evento-resumo periodico
Over-Expression of the N-myc Downstrean-Regulated Gene 1 (NDRG1) Is Associated with Poorer Prognosis in Breast Cancer (2006)
Nagai, M. A.; Nishimoto, I. N.; Mourão Neto, M.; Santos, O. P.; Soares, F. A.
Fonte: Laboratory Investigation. Nome do evento: Annual Meeting of the United-States-and-Canadian-Academy-of-Pathology. Unidade: FM
Assuntos: NEOPLASIAS MAMÁRIAS, PROGNÓSTICO, EXPRESSÃO GÊNICA, PROLIFERAÇÃO CELULAR, CARCINOMA, APOPTOSE, RESUMOS (EVENTOS)
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ABNT
NAGAI, M. A. et al. Over-Expression of the N-myc Downstrean-Regulated Gene 1 (NDRG1) Is Associated with Poorer Prognosis in Breast Cancer. Laboratory Investigation. Baltimore: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://www.nature.com/labinvest/journal/v86/n1s/pdf/3700606a.pdf. Acesso em: 03 nov. 2025. , 2006
APA
Nagai, M. A., Nishimoto, I. N., Mourão Neto, M., Santos, O. P., & Soares, F. A. (2006). Over-Expression of the N-myc Downstrean-Regulated Gene 1 (NDRG1) Is Associated with Poorer Prognosis in Breast Cancer. Laboratory Investigation. Baltimore: Faculdade de Medicina, Universidade de São Paulo. Recuperado de http://www.nature.com/labinvest/journal/v86/n1s/pdf/3700606a.pdf
NLM
Nagai MA, Nishimoto IN, Mourão Neto M, Santos OP, Soares FA. Over-Expression of the N-myc Downstrean-Regulated Gene 1 (NDRG1) Is Associated with Poorer Prognosis in Breast Cancer [Internet]. Laboratory Investigation. 2006 ; 86( suppl. 1): 35A-36A.[citado 2025 nov. 03 ] Available from: http://www.nature.com/labinvest/journal/v86/n1s/pdf/3700606a.pdf
Vancouver
Nagai MA, Nishimoto IN, Mourão Neto M, Santos OP, Soares FA. Over-Expression of the N-myc Downstrean-Regulated Gene 1 (NDRG1) Is Associated with Poorer Prognosis in Breast Cancer [Internet]. Laboratory Investigation. 2006 ; 86( suppl. 1): 35A-36A.[citado 2025 nov. 03 ] Available from: http://www.nature.com/labinvest/journal/v86/n1s/pdf/3700606a.pdf
Artigo de periodico
Renal injuri in apolipoprotein E-deficient mice (2002)
Wen, Min; Segerer, Stephan; Dantas, Márcio; Brown, Paul A.; Hudkins, Kelly L.; Goodpaster, Tracy; Kirk, Elizabeth; LeBoeuf, Renée (Department of OF Medicine, University of Washington, Seattle, Washington, USA); Alpers, Charles E.
Fonte: Laboratory Investigation. Unidade: FMRP
Assuntos: RIM (PATOLOGIA), NEFROPATIAS
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ABNT
WEN, Min et al. Renal injuri in apolipoprotein E-deficient mice. Laboratory Investigation, v. 82, p. 8, 2002Tradução . . Acesso em: 03 nov. 2025.
APA
Wen, M., Segerer, S., Dantas, M., Brown, P. A., Hudkins, K. L., Goodpaster, T., et al. (2002). Renal injuri in apolipoprotein E-deficient mice. Laboratory Investigation, 82, 8.
NLM
Wen M, Segerer S, Dantas M, Brown PA, Hudkins KL, Goodpaster T, Kirk E, Alpers CE. Renal injuri in apolipoprotein E-deficient mice. Laboratory Investigation. 2002 ; 82 8.[citado 2025 nov. 03 ]
Vancouver
Wen M, Segerer S, Dantas M, Brown PA, Hudkins KL, Goodpaster T, Kirk E, Alpers CE. Renal injuri in apolipoprotein E-deficient mice. Laboratory Investigation. 2002 ; 82 8.[citado 2025 nov. 03 ]
Artigo de periodico
Molecular and standard approaches to the diagnosis of mycrobacterial granulomatous lymphadenitis in paraffin-embedded tissue (2002)
Patzina, Rosely Antunes; Andrade Junior, Heitor Franco de ; Brito, Thales de ; Caiaffa Filho, H H ; Kauffman, Mônica Rebeca; Pagliari, Carla ; Lucena, Andréa; Matta, Vania Lucia Ribeiro da ; Duarte, Maria Irma Seixas
Fonte: Laboratory Investigation. Unidade: FM
Assunto: PATOLOGIA
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ABNT
PATZINA, Rosely Antunes et al. Molecular and standard approaches to the diagnosis of mycrobacterial granulomatous lymphadenitis in paraffin-embedded tissue. Laboratory Investigation, v. 82, n. 8, p. 1095-1097, 2002Tradução . . Acesso em: 03 nov. 2025.
APA
Patzina, R. A., Andrade Junior, H. F. de, Brito, T. de, Caiaffa Filho, H. H., Kauffman, M. R., Pagliari, C., et al. (2002). Molecular and standard approaches to the diagnosis of mycrobacterial granulomatous lymphadenitis in paraffin-embedded tissue. Laboratory Investigation, 82( 8), 1095-1097.
NLM
Patzina RA, Andrade Junior HF de, Brito T de, Caiaffa Filho HH, Kauffman MR, Pagliari C, Lucena A, Matta VLR da, Duarte MIS. Molecular and standard approaches to the diagnosis of mycrobacterial granulomatous lymphadenitis in paraffin-embedded tissue. Laboratory Investigation. 2002 ; 82( 8): 1095-1097.[citado 2025 nov. 03 ]
Vancouver
Patzina RA, Andrade Junior HF de, Brito T de, Caiaffa Filho HH, Kauffman MR, Pagliari C, Lucena A, Matta VLR da, Duarte MIS. Molecular and standard approaches to the diagnosis of mycrobacterial granulomatous lymphadenitis in paraffin-embedded tissue. Laboratory Investigation. 2002 ; 82( 8): 1095-1097.[citado 2025 nov. 03 ]
Artigo de periodico
Heterogeneity of the angiogenic response induced in different normal adult tissues by vascular permeability factor: vascular endothelial growth factor (2000)
Pettersson, Anna; Nagy, J. A.; Brown, Lawrence F.; Sundberg, Christian; Morgan, Ellen; Jungles, Steven; Carter, Robert; Krieger, José Eduardo; Manseau, Eleanor J.; Harvey, V. Susan; Eckelhoefer, Isabelle A.; Feng, Dian; Dvorak, Ann M.; Mulligan, Richard C.
Fonte: Laboratory Investigation. Unidade: FM
Assunto: MEDICINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PETTERSSON, Anna et al. Heterogeneity of the angiogenic response induced in different normal adult tissues by vascular permeability factor: vascular endothelial growth factor. Laboratory Investigation, v. 80, n. 1 , p. 99-115, 2000Tradução . . Acesso em: 03 nov. 2025.
APA
Pettersson, A., Nagy, J. A., Brown, L. F., Sundberg, C., Morgan, E., Jungles, S., et al. (2000). Heterogeneity of the angiogenic response induced in different normal adult tissues by vascular permeability factor: vascular endothelial growth factor. Laboratory Investigation, 80( 1 ), 99-115.
NLM
Pettersson A, Nagy JA, Brown LF, Sundberg C, Morgan E, Jungles S, Carter R, Krieger JE, Manseau EJ, Harvey VS, Eckelhoefer IA, Feng D, Dvorak AM, Mulligan RC. Heterogeneity of the angiogenic response induced in different normal adult tissues by vascular permeability factor: vascular endothelial growth factor. Laboratory Investigation. 2000 ; 80( 1 ): 99-115.[citado 2025 nov. 03 ]
Vancouver
Pettersson A, Nagy JA, Brown LF, Sundberg C, Morgan E, Jungles S, Carter R, Krieger JE, Manseau EJ, Harvey VS, Eckelhoefer IA, Feng D, Dvorak AM, Mulligan RC. Heterogeneity of the angiogenic response induced in different normal adult tissues by vascular permeability factor: vascular endothelial growth factor. Laboratory Investigation. 2000 ; 80( 1 ): 99-115.[citado 2025 nov. 03 ]
Artigo de periodico
Quantification and morphological demonstration of reactive oxygen species produced by walker 256 tumor cells in vitro (1994)
Soares, Fernando Augusto; Shanghnesy, S G; Maclarkey, W R; Orr, F W
Fonte: Laboratory Investigation. Unidade: FMRP
Assunto: PATOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOARES, Fernando Augusto et al. Quantification and morphological demonstration of reactive oxygen species produced by walker 256 tumor cells in vitro. Laboratory Investigation, v. 71, n. 4 , p. 480-9, 1994Tradução . . Acesso em: 03 nov. 2025.
APA
Soares, F. A., Shanghnesy, S. G., Maclarkey, W. R., & Orr, F. W. (1994). Quantification and morphological demonstration of reactive oxygen species produced by walker 256 tumor cells in vitro. Laboratory Investigation, 71( 4 ), 480-9.
NLM
Soares FA, Shanghnesy SG, Maclarkey WR, Orr FW. Quantification and morphological demonstration of reactive oxygen species produced by walker 256 tumor cells in vitro. Laboratory Investigation. 1994 ;71( 4 ): 480-9.[citado 2025 nov. 03 ]
Vancouver
Soares FA, Shanghnesy SG, Maclarkey WR, Orr FW. Quantification and morphological demonstration of reactive oxygen species produced by walker 256 tumor cells in vitro. Laboratory Investigation. 1994 ;71( 4 ): 480-9.[citado 2025 nov. 03 ]
Trabalho de evento-resumo periodico
Dna content and proliferanting cell nuclear antigen (pcna) in persistent neonatal hyperinsulinemic hypoglicemia (pnhh) (1994)
Zerbini, Maria Cláudia Nogueira; Wakamatsu, A; Almeida, W L; Lopes, L H A C; Maksoud, João Gilberto; Marujo, W C
Fonte: Laboratory Investigation. Nome do evento: Annual Meeting of the United States and Canadian Academy of Pathology. Unidade: FM
Assunto: PEDIATRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZERBINI, Maria Cláudia Nogueira et al. Dna content and proliferanting cell nuclear antigen (pcna) in persistent neonatal hyperinsulinemic hypoglicemia (pnhh). Laboratory Investigation. Baltimore: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 03 nov. 2025. , 1994
APA
Zerbini, M. C. N., Wakamatsu, A., Almeida, W. L., Lopes, L. H. A. C., Maksoud, J. G., & Marujo, W. C. (1994). Dna content and proliferanting cell nuclear antigen (pcna) in persistent neonatal hyperinsulinemic hypoglicemia (pnhh). Laboratory Investigation. Baltimore: Faculdade de Medicina, Universidade de São Paulo.
NLM
Zerbini MCN, Wakamatsu A, Almeida WL, Lopes LHAC, Maksoud JG, Marujo WC. Dna content and proliferanting cell nuclear antigen (pcna) in persistent neonatal hyperinsulinemic hypoglicemia (pnhh). Laboratory Investigation. 1994 ;70( ja 1994): 149a.[citado 2025 nov. 03 ]
Vancouver
Zerbini MCN, Wakamatsu A, Almeida WL, Lopes LHAC, Maksoud JG, Marujo WC. Dna content and proliferanting cell nuclear antigen (pcna) in persistent neonatal hyperinsulinemic hypoglicemia (pnhh). Laboratory Investigation. 1994 ;70( ja 1994): 149a.[citado 2025 nov. 03 ]
Artigo de periodico
Effect of urethane on isoproterenol-stimulated parotid glands of mice (1973)
Guimarães, Sérgio Augusto Catanzaro; Baserga, Renato
Fonte: Laboratory Investigation. Unidade: FOB
Assuntos: GLÂNDULA PARÓTIDA, DNA, RNA RIBOSSÔMICO, PROLIFERAÇÃO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUIMARÃES, Sérgio Augusto Catanzaro e BASERGA, Renato. Effect of urethane on isoproterenol-stimulated parotid glands of mice. Laboratory Investigation, v. 29, n. 4, p. 367-373, 1973Tradução . . Acesso em: 03 nov. 2025.
APA
Guimarães, S. A. C., & Baserga, R. (1973). Effect of urethane on isoproterenol-stimulated parotid glands of mice. Laboratory Investigation, 29( 4), 367-373.
NLM
Guimarães SAC, Baserga R. Effect of urethane on isoproterenol-stimulated parotid glands of mice. Laboratory Investigation. 1973 ; 29( 4): 367-373.[citado 2025 nov. 03 ]
Vancouver
Guimarães SAC, Baserga R. Effect of urethane on isoproterenol-stimulated parotid glands of mice. Laboratory Investigation. 1973 ; 29( 4): 367-373.[citado 2025 nov. 03 ]

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