The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype (2007)
- Authors:
- USP affiliated authors: SILVA, ALFREDO RIBEIRO DA - FMRP ; ZUCOLOTO, SÉRGIO - FMRP
- Unidade: FMRP
- Assunto: NEOPLASIAS MAMÁRIAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Laboratory Investigation
- ISSN: 1530-0307
- Volume/Número/Paginação/Ano: v. 87, p. 47A, res. 193, 2007
- Conference titles: United States and Canadian Academy of Pathology (USCAP)
-
ABNT
RIBEIRO-SILVA, Alfredo e ZUCOLOTO, Sérgio. The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 11 set. 2024. , 2007 -
APA
Ribeiro-Silva, A., & Zucoloto, S. (2007). The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. Baltimore: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Ribeiro-Silva A, Zucoloto S. The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. 2007 ; 87 47A.[citado 2024 set. 11 ] -
Vancouver
Ribeiro-Silva A, Zucoloto S. The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype. Laboratory Investigation. 2007 ; 87 47A.[citado 2024 set. 11 ] - BRCA1 expression in sporadic breast carcinomas: a clinicopathologic and immuno-histochemical study
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