The differencial regulation of human telomerase reverse transcriptase and vascular endothelial growth factor may contribute to the clinically more aggressive behavior of p63-positive breast carcinomas (2005)
- Authors:
- USP affiliated authors: SILVA, ALFREDO RIBEIRO DA - FMRP ; ZUCOLOTO, SÉRGIO - FMRP
- Unidade: FMRP
- Subjects: FATORES DE CRESCIMENTO; ENDOTÉLIO VASCULAR; METALOPROTEINASES; MAMA
- Language: Inglês
- Imprenta:
- Source:
- Título: The International Journal of Biological Morkers
- ISSN: 0393-6155
- Volume/Número/Paginação/Ano: v. 20, n. 4, p. 227-234, 2005
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ABNT
RIBEIRO-SILVA, Alfredo et al. The differencial regulation of human telomerase reverse transcriptase and vascular endothelial growth factor may contribute to the clinically more aggressive behavior of p63-positive breast carcinomas. The International Journal of Biological Morkers, v. 20, n. 4, p. 227-234, 2005Tradução . . Acesso em: 19 fev. 2026. -
APA
Ribeiro-Silva, A., Becker de Moura, H., Ribeiro do Vale, F., & Zucoloto, S. (2005). The differencial regulation of human telomerase reverse transcriptase and vascular endothelial growth factor may contribute to the clinically more aggressive behavior of p63-positive breast carcinomas. The International Journal of Biological Morkers, 20( 4), 227-234. -
NLM
Ribeiro-Silva A, Becker de Moura H, Ribeiro do Vale F, Zucoloto S. The differencial regulation of human telomerase reverse transcriptase and vascular endothelial growth factor may contribute to the clinically more aggressive behavior of p63-positive breast carcinomas. The International Journal of Biological Morkers. 2005 ; 20( 4): 227-234.[citado 2026 fev. 19 ] -
Vancouver
Ribeiro-Silva A, Becker de Moura H, Ribeiro do Vale F, Zucoloto S. The differencial regulation of human telomerase reverse transcriptase and vascular endothelial growth factor may contribute to the clinically more aggressive behavior of p63-positive breast carcinomas. The International Journal of Biological Morkers. 2005 ; 20( 4): 227-234.[citado 2026 fev. 19 ] - Expression of apoptosis-related protein Bcl-2 correlates with breast carcinomas of luminal of basal-like subtypes
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