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Artigo de periodico
Association of three diferent mutations in the CLCN1 Gene Modulating the phenotype in a consanguineous family with myotonia congenita (2021)
Souza, Lucas Santos; Calyjur, Priscila; Ribeiro Jr, Antonio Fernando; Gurgel-Giannetti, Juliana; Pavanello, Rita de Cássia Mingroni; Zatz, Mayana ; Vainzof, Mariz
Fonte: Journal of Molecular Neuroscience. Unidade: IB
Assuntos: DOENÇAS NEUROMUSCULARES, MUTAÇÃO GENÉTICA, MIOPATIAS CONGÊNITAS ESTRUTURAIS, DOENÇAS GENÉTICAS, FENÓTIPOS
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SOUZA, Lucas Santos et al. Association of three diferent mutations in the CLCN1 Gene Modulating the phenotype in a consanguineous family with myotonia congenita. Journal of Molecular Neuroscience, n. Ja 2021, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12031-020-01785-4. Acesso em: 20 out. 2025.
APA
Souza, L. S., Calyjur, P., Ribeiro Jr, A. F., Gurgel-Giannetti, J., Pavanello, R. de C. M., Zatz, M., & Vainzof, M. (2021). Association of three diferent mutations in the CLCN1 Gene Modulating the phenotype in a consanguineous family with myotonia congenita. Journal of Molecular Neuroscience, ( Ja 2021). doi:10.1007/s12031-020-01785-4
NLM
Souza LS, Calyjur P, Ribeiro Jr AF, Gurgel-Giannetti J, Pavanello R de CM, Zatz M, Vainzof M. Association of three diferent mutations in the CLCN1 Gene Modulating the phenotype in a consanguineous family with myotonia congenita [Internet]. Journal of Molecular Neuroscience. 2021 ;( Ja 2021):[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-020-01785-4
Vancouver
Souza LS, Calyjur P, Ribeiro Jr AF, Gurgel-Giannetti J, Pavanello R de CM, Zatz M, Vainzof M. Association of three diferent mutations in the CLCN1 Gene Modulating the phenotype in a consanguineous family with myotonia congenita [Internet]. Journal of Molecular Neuroscience. 2021 ;( Ja 2021):[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-020-01785-4
Artigo de periodico
Expression of MicroRNAs miR-145, miR-181c, miR-199a and miR-1183 in the blood and hippocampus of patients with mesial temporal lobe epilepsy (2019)
Lourenço, Luana Grupioni; Freitas-Lima, Priscila; Bisson, Gabriela Silva ; Assirati Júnior, João Alberto; Matias, Caio Marconato; Cirino, Mucio Luiz de Assis; Tirapelli, Luis Fernando; Velasco, Tonicarlo Rodrigues ; Sakamoto, Américo Ceiki; Carlotti Júnior, Carlos Gilberto ; Tirapelli, Daniela Pretti da Cunha
Fonte: Journal of Molecular Neuroscience. Unidades: EERP, FMRP
Assuntos: MICRORNAS, EPILEPSIA DO LOBO TEMPORAL, BIOMARCADORES, HIPOCAMPO
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LOURENÇO, Luana Grupioni et al. Expression of MicroRNAs miR-145, miR-181c, miR-199a and miR-1183 in the blood and hippocampus of patients with mesial temporal lobe epilepsy. Journal of Molecular Neuroscience, v. 69, n. 4, p. 580-587, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12031-019-01386-w. Acesso em: 20 out. 2025.
APA
Lourenço, L. G., Freitas-Lima, P., Bisson, G. S., Assirati Júnior, J. A., Matias, C. M., Cirino, M. L. de A., et al. (2019). Expression of MicroRNAs miR-145, miR-181c, miR-199a and miR-1183 in the blood and hippocampus of patients with mesial temporal lobe epilepsy. Journal of Molecular Neuroscience, 69( 4), 580-587. doi:10.1007/s12031-019-01386-w
NLM
Lourenço LG, Freitas-Lima P, Bisson GS, Assirati Júnior JA, Matias CM, Cirino ML de A, Tirapelli LF, Velasco TR, Sakamoto AC, Carlotti Júnior CG, Tirapelli DP da C. Expression of MicroRNAs miR-145, miR-181c, miR-199a and miR-1183 in the blood and hippocampus of patients with mesial temporal lobe epilepsy [Internet]. Journal of Molecular Neuroscience. 2019 ; 69( 4): 580-587.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-019-01386-w
Vancouver
Lourenço LG, Freitas-Lima P, Bisson GS, Assirati Júnior JA, Matias CM, Cirino ML de A, Tirapelli LF, Velasco TR, Sakamoto AC, Carlotti Júnior CG, Tirapelli DP da C. Expression of MicroRNAs miR-145, miR-181c, miR-199a and miR-1183 in the blood and hippocampus of patients with mesial temporal lobe epilepsy [Internet]. Journal of Molecular Neuroscience. 2019 ; 69( 4): 580-587.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-019-01386-w
Artigo de periodico
The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease (2017)
Garcia, P. C.; Real, C. C.; Britto, Luiz Roberto Giorgetti de
Fonte: Journal of Molecular Neuroscience. Unidade: ICB
Assunto: FISIOLOGIA
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GARCIA, P. C. e REAL, C. C. e BRITTO, Luiz Roberto Giorgetti de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, v. 61, n. 4, p. 542-552, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12031-017-0896-y. Acesso em: 20 out. 2025.
APA
Garcia, P. C., Real, C. C., & Britto, L. R. G. de. (2017). The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease. Journal of Molecular Neuroscience, 61( 4), 542-552. doi:10.1007/s12031-017-0896-y
NLM
Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
Vancouver
Garcia PC, Real CC, Britto LRG de. The impact of short and long-term exercise on the expression of arc and AMPARs during evolution of the 6-Hydroxy-Dopamine animal model of Parkinson's disease [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 4): 542-552.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-017-0896-y
Artigo de periodico
Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. (2017)
Gregório, Caroline Cristiano Real ; Garcia, Priscila Crespo ; Britto, Luiz Roberto Giorgetti de
Fonte: Journal of Molecular Neuroscience. Unidade: ICB
Assuntos: FISIOLOGIA, DOENÇA DE PARKINSON, PREVENÇÃO DE DOENÇAS ANIMAIS, RATOS WISTAR, ASTRÓCITOS, RECEPTORES DE DOPAMINA, NEURÔNIOS, MODELOS ANIMAIS DE DOENÇAS
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GREGÓRIO, Caroline Cristiano Real e GARCIA, Priscila Crespo e BRITTO, Luiz Roberto Giorgetti de. Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. Journal of Molecular Neuroscience, v. 63, p. 36-49, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12031-017-0955-4. Acesso em: 20 out. 2025.
APA
Gregório, C. C. R., Garcia, P. C., & Britto, L. R. G. de. (2017). Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. Journal of Molecular Neuroscience, 63, 36-49. doi:10.1007/s12031-017-0955-4
NLM
Gregório CCR, Garcia PC, Britto LRG de. Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. [Internet]. Journal of Molecular Neuroscience. 2017 ; 63 36-49.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-017-0955-4
Vancouver
Gregório CCR, Garcia PC, Britto LRG de. Treadmill exercise prevents increase of neuroinflammation markers involved in the dopaminergic damage of the 6-OHDA Parkinson's disease model. [Internet]. Journal of Molecular Neuroscience. 2017 ; 63 36-49.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-017-0955-4
Artigo de periodico
Central Nervous System and Vertebrae Development in Horses: a Chronological Study with Differential Temporal Expression of Nestin and GFAP (2017)
Rigoglio, Nathia N; Barreto, Rodrigo S. N; Favaron, Phelipe O; Jacob, Júlio C. F; Smith, Lawrence C; Gastal, Melba O; Gastal, Eduardo L; Miglino, Maria Angélica
Fonte: Journal of Molecular Neuroscience. Unidade: FMVZ
Assuntos: EQUINOS, NEURÓGLIA, ORGANOGÊNESE ANIMAL, PROTEÍNAS, SISTEMA NERVOSO CENTRAL
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RIGOGLIO, Nathia N et al. Central Nervous System and Vertebrae Development in Horses: a Chronological Study with Differential Temporal Expression of Nestin and GFAP. Journal of Molecular Neuroscience, v. 61, n. 1, p. 61-78, 2017Tradução . . Disponível em: https://doi.org/10.1007/s12031-016-0805-9. Acesso em: 20 out. 2025.
APA
Rigoglio, N. N., Barreto, R. S. N., Favaron, P. O., Jacob, J. C. F., Smith, L. C., Gastal, M. O., et al. (2017). Central Nervous System and Vertebrae Development in Horses: a Chronological Study with Differential Temporal Expression of Nestin and GFAP. Journal of Molecular Neuroscience, 61( 1), 61-78. doi:10.1007/s12031-016-0805-9
NLM
Rigoglio NN, Barreto RSN, Favaron PO, Jacob JCF, Smith LC, Gastal MO, Gastal EL, Miglino MA. Central Nervous System and Vertebrae Development in Horses: a Chronological Study with Differential Temporal Expression of Nestin and GFAP [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 1): 61-78.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-016-0805-9
Vancouver
Rigoglio NN, Barreto RSN, Favaron PO, Jacob JCF, Smith LC, Gastal MO, Gastal EL, Miglino MA. Central Nervous System and Vertebrae Development in Horses: a Chronological Study with Differential Temporal Expression of Nestin and GFAP [Internet]. Journal of Molecular Neuroscience. 2017 ; 61( 1): 61-78.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-016-0805-9
Artigo de periodico
Tobacco nitrosamine N-nitrosonornicotine as inhibitor of neuronal nicotinic acetylcholine receptors (2013)
Nunes Alves, Ariane Ferreira; Nery, Arthur Andrade; Ulrich, Henning
Fonte: Journal of Molecular Neuroscience. Unidade: IQ
Assuntos: RECEPTORES, FUMO
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ABNT
NUNES ALVES, Ariane Ferreira e NERY, Arthur Andrade e ULRICH, Henning. Tobacco nitrosamine N-nitrosonornicotine as inhibitor of neuronal nicotinic acetylcholine receptors. Journal of Molecular Neuroscience, v. 49, n. 1, p. 52-61, 2013Tradução . . Disponível em: https://doi.org/10.1007/s12031-012-9859-5. Acesso em: 20 out. 2025.
APA
Nunes Alves, A. F., Nery, A. A., & Ulrich, H. (2013). Tobacco nitrosamine N-nitrosonornicotine as inhibitor of neuronal nicotinic acetylcholine receptors. Journal of Molecular Neuroscience, 49( 1), 52-61. doi:10.1007/s12031-012-9859-5
NLM
Nunes Alves AF, Nery AA, Ulrich H. Tobacco nitrosamine N-nitrosonornicotine as inhibitor of neuronal nicotinic acetylcholine receptors [Internet]. Journal of Molecular Neuroscience. 2013 ; 49( 1): 52-61.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-012-9859-5
Vancouver
Nunes Alves AF, Nery AA, Ulrich H. Tobacco nitrosamine N-nitrosonornicotine as inhibitor of neuronal nicotinic acetylcholine receptors [Internet]. Journal of Molecular Neuroscience. 2013 ; 49( 1): 52-61.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-012-9859-5
Artigo de periodico
Family-Based Association Analysis of Serotonin Genes in Pathological Gambling Disorder: Evidence of Vulnerability Risk in the 5HT-2A Receptor Gene (2013)
Wilson, David; Lobo, Daniela Sabbatini da Silva; Tavares, Hermano; Gentil, Valentim; Vallada, Homero
Fonte: Journal of Molecular Neuroscience. Unidade: FM
Assuntos: RELAÇÕES FAMILIARES, DOENÇAS GENÉTICAS (DIAGNÓSTICO), FATORES DE RISCO, SEROTONINA (ANÁLISE), GENES (ANÁLISE), GENÓTIPOS, COMPORTAMENTO, VULNERABILIDADE, POLIMORFISMO
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ABNT
WILSON, David et al. Family-Based Association Analysis of Serotonin Genes in Pathological Gambling Disorder: Evidence of Vulnerability Risk in the 5HT-2A Receptor Gene. Journal of Molecular Neuroscience, v. 49, n. 3, p. 550-553, 2013Tradução . . Disponível em: http://link.springer.com/content/pdf/10.1007%2Fs12031-012-9846-x.pdf. Acesso em: 20 out. 2025.
APA
Wilson, D., Lobo, D. S. da S., Tavares, H., Gentil, V., & Vallada, H. (2013). Family-Based Association Analysis of Serotonin Genes in Pathological Gambling Disorder: Evidence of Vulnerability Risk in the 5HT-2A Receptor Gene. Journal of Molecular Neuroscience, 49( 3), 550-553. doi:10.1007/s12031-012-9846-x
NLM
Wilson D, Lobo DS da S, Tavares H, Gentil V, Vallada H. Family-Based Association Analysis of Serotonin Genes in Pathological Gambling Disorder: Evidence of Vulnerability Risk in the 5HT-2A Receptor Gene [Internet]. Journal of Molecular Neuroscience. 2013 ; 49( 3): 550-553.[citado 2025 out. 20 ] Available from: http://link.springer.com/content/pdf/10.1007%2Fs12031-012-9846-x.pdf
Vancouver
Wilson D, Lobo DS da S, Tavares H, Gentil V, Vallada H. Family-Based Association Analysis of Serotonin Genes in Pathological Gambling Disorder: Evidence of Vulnerability Risk in the 5HT-2A Receptor Gene [Internet]. Journal of Molecular Neuroscience. 2013 ; 49( 3): 550-553.[citado 2025 out. 20 ] Available from: http://link.springer.com/content/pdf/10.1007%2Fs12031-012-9846-x.pdf
Artigo de periodico
Muscle phenotypic variability in limb girdle muscular dystrophy 2 G (2013)
Paim, Julia F; Cotta, Ana; Vargas, Antonio P; Navarro, Monica M; Valicek, Jaquelin; Carvalho, Elmano; da-Cunha-Junior, Antonio; Plentz, Estevão; Braz, Shelida v; Takata, Reinaldo I; Almeida, Camila F; Vainzof, Mariz
Fonte: Journal of Molecular Neuroscience. Unidade: IB
Assuntos: DISTROFIA MUSCULAR, MUTAÇÃO GENÉTICA, MIOPATIAS CONGÊNITAS ESTRUTURAIS
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ABNT
PAIM, Julia F et al. Muscle phenotypic variability in limb girdle muscular dystrophy 2 G. Journal of Molecular Neuroscience, v. 50, n. 2, p. 339-344, 2013Tradução . . Disponível em: https://doi.org/10.1007/s12031-013-9987-6. Acesso em: 20 out. 2025.
APA
Paim, J. F., Cotta, A., Vargas, A. P., Navarro, M. M., Valicek, J., Carvalho, E., et al. (2013). Muscle phenotypic variability in limb girdle muscular dystrophy 2 G. Journal of Molecular Neuroscience, 50( 2), 339-344. doi:10.1007/s12031-013-9987-6
NLM
Paim JF, Cotta A, Vargas AP, Navarro MM, Valicek J, Carvalho E, da-Cunha-Junior A, Plentz E, Braz S v, Takata RI, Almeida CF, Vainzof M. Muscle phenotypic variability in limb girdle muscular dystrophy 2 G [Internet]. Journal of Molecular Neuroscience. 2013 ; 50( 2): 339-344.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-013-9987-6
Vancouver
Paim JF, Cotta A, Vargas AP, Navarro MM, Valicek J, Carvalho E, da-Cunha-Junior A, Plentz E, Braz S v, Takata RI, Almeida CF, Vainzof M. Muscle phenotypic variability in limb girdle muscular dystrophy 2 G [Internet]. Journal of Molecular Neuroscience. 2013 ; 50( 2): 339-344.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-013-9987-6
Artigo de periodico
Catechol-O-methyltransferase (COMT) val158met Polymorphism as a Risk Factor for PTSD After Urban Violence (2011)
Valente, Nina Leão Marques; Vallada, Homero; Cordeiro, Quirino; Bressan, Rodrigo Affonseca; Andreoli, Sergio Baxter; Mari, Jair Jesus; Mello, Marcelo Feijó de
Fonte: Journal of Molecular Neuroscience. Unidade: FM
Assuntos: TRANSTORNO DO STRESS PÓS-TRAUMÁTICO (ENZIMOLOGIA), VIOLÊNCIA URBANA, POLIMORFISMO, FATORES DE RISCO
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ABNT
VALENTE, Nina Leão Marques et al. Catechol-O-methyltransferase (COMT) val158met Polymorphism as a Risk Factor for PTSD After Urban Violence. Journal of Molecular Neuroscience, v. 43, n. 3, p. 516-523, 2011Tradução . . Disponível em: https://doi.org/10.1007/s12031-010-9474-2. Acesso em: 20 out. 2025.
APA
Valente, N. L. M., Vallada, H., Cordeiro, Q., Bressan, R. A., Andreoli, S. B., Mari, J. J., & Mello, M. F. de. (2011). Catechol-O-methyltransferase (COMT) val158met Polymorphism as a Risk Factor for PTSD After Urban Violence. Journal of Molecular Neuroscience, 43( 3), 516-523. doi:10.1007/s12031-010-9474-2
NLM
Valente NLM, Vallada H, Cordeiro Q, Bressan RA, Andreoli SB, Mari JJ, Mello MF de. Catechol-O-methyltransferase (COMT) val158met Polymorphism as a Risk Factor for PTSD After Urban Violence [Internet]. Journal of Molecular Neuroscience. 2011 ; 43( 3): 516-523.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9474-2
Vancouver
Valente NLM, Vallada H, Cordeiro Q, Bressan RA, Andreoli SB, Mari JJ, Mello MF de. Catechol-O-methyltransferase (COMT) val158met Polymorphism as a Risk Factor for PTSD After Urban Violence [Internet]. Journal of Molecular Neuroscience. 2011 ; 43( 3): 516-523.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9474-2
Artigo de periodico
Directed differentiation of neural progenitors into neurons is accompanied by altered expression of P2X purinergic receptors (2011)
Schwindt, Telma Tiemi; Trujillo, Cleber Augusto; Negraes, Priscilla D; Lameu, Claudiana ; Ulrich, Henning
Fonte: Journal of Molecular Neuroscience. Unidade: IQ
Assuntos: RECEPTORES, NEUROGENÉTICA
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ABNT
SCHWINDT, Telma Tiemi et al. Directed differentiation of neural progenitors into neurons is accompanied by altered expression of P2X purinergic receptors. Journal of Molecular Neuroscience, v. 44, n. 5, p. 141-146, 2011Tradução . . Disponível em: https://doi.org/10.1007/s12031-010-9417-y. Acesso em: 20 out. 2025.
APA
Schwindt, T. T., Trujillo, C. A., Negraes, P. D., Lameu, C., & Ulrich, H. (2011). Directed differentiation of neural progenitors into neurons is accompanied by altered expression of P2X purinergic receptors. Journal of Molecular Neuroscience, 44( 5), 141-146. doi:10.1007/s12031-010-9417-y
NLM
Schwindt TT, Trujillo CA, Negraes PD, Lameu C, Ulrich H. Directed differentiation of neural progenitors into neurons is accompanied by altered expression of P2X purinergic receptors [Internet]. Journal of Molecular Neuroscience. 2011 ; 44( 5): 141-146.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9417-y
Vancouver
Schwindt TT, Trujillo CA, Negraes PD, Lameu C, Ulrich H. Directed differentiation of neural progenitors into neurons is accompanied by altered expression of P2X purinergic receptors [Internet]. Journal of Molecular Neuroscience. 2011 ; 44( 5): 141-146.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9417-y
Artigo de periodico
Candidate-Gene Approach in Posttraumatic Stress Disorder After Urban Violence: Association Analysis of the Genes Encoding Serotonin Transporter, Dopamine Transporter, and BDNF (2011)
Valente, Nina Leão Marques; Vallada, Homero; Cordeiro, Quirino; Miguita, Karen; Bressan, Rodrigo Affonseca; Andreoli, Sergio Baxter; Mari, Jair Jesus; Mello, Marcelo Feijó de
Fonte: Journal of Molecular Neuroscience. Unidade: FM
Assuntos: TRANSTORNO DO STRESS PÓS-TRAUMÁTICO (ENZIMOLOGIA), VIOLÊNCIA URBANA, POLIMORFISMO, GENES
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ABNT
VALENTE, Nina Leão Marques et al. Candidate-Gene Approach in Posttraumatic Stress Disorder After Urban Violence: Association Analysis of the Genes Encoding Serotonin Transporter, Dopamine Transporter, and BDNF. Journal of Molecular Neuroscience, v. 44, n. 1, p. 59-67, 2011Tradução . . Disponível em: https://doi.org/10.1007/s12031-011-9513-7. Acesso em: 20 out. 2025.
APA
Valente, N. L. M., Vallada, H., Cordeiro, Q., Miguita, K., Bressan, R. A., Andreoli, S. B., et al. (2011). Candidate-Gene Approach in Posttraumatic Stress Disorder After Urban Violence: Association Analysis of the Genes Encoding Serotonin Transporter, Dopamine Transporter, and BDNF. Journal of Molecular Neuroscience, 44( 1), 59-67. doi:10.1007/s12031-011-9513-7
NLM
Valente NLM, Vallada H, Cordeiro Q, Miguita K, Bressan RA, Andreoli SB, Mari JJ, Mello MF de. Candidate-Gene Approach in Posttraumatic Stress Disorder After Urban Violence: Association Analysis of the Genes Encoding Serotonin Transporter, Dopamine Transporter, and BDNF [Internet]. Journal of Molecular Neuroscience. 2011 ; 44( 1): 59-67.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-011-9513-7
Vancouver
Valente NLM, Vallada H, Cordeiro Q, Miguita K, Bressan RA, Andreoli SB, Mari JJ, Mello MF de. Candidate-Gene Approach in Posttraumatic Stress Disorder After Urban Violence: Association Analysis of the Genes Encoding Serotonin Transporter, Dopamine Transporter, and BDNF [Internet]. Journal of Molecular Neuroscience. 2011 ; 44( 1): 59-67.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-011-9513-7
Artigo de periodico
Population and computational analysis of the MGEA6 P521A variation as a risk factor for familial Idiopathic Basal Ganglia Calcification (Fahr’s Disease) (2011)
Lemos, Roberta R.; Oliveira, Danyllo F; Zatz, Mayana ; Oliveira, João R. M
Fonte: Journal of Molecular Neuroscience. Unidade: IB
Assuntos: DOENÇAS GENÉTICAS, POLIMORFISMO
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ABNT
LEMOS, Roberta R. et al. Population and computational analysis of the MGEA6 P521A variation as a risk factor for familial Idiopathic Basal Ganglia Calcification (Fahr’s Disease). Journal of Molecular Neuroscience, v. 43, n. 3, p. 333-336, 2011Tradução . . Disponível em: https://doi.org/10.1007/s12031-010-9445-7. Acesso em: 20 out. 2025.
APA
Lemos, R. R., Oliveira, D. F., Zatz, M., & Oliveira, J. R. M. (2011). Population and computational analysis of the MGEA6 P521A variation as a risk factor for familial Idiopathic Basal Ganglia Calcification (Fahr’s Disease). Journal of Molecular Neuroscience, 43( 3), 333-336. doi:10.1007/s12031-010-9445-7
NLM
Lemos RR, Oliveira DF, Zatz M, Oliveira JRM. Population and computational analysis of the MGEA6 P521A variation as a risk factor for familial Idiopathic Basal Ganglia Calcification (Fahr’s Disease) [Internet]. Journal of Molecular Neuroscience. 2011 ; 43( 3): 333-336.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9445-7
Vancouver
Lemos RR, Oliveira DF, Zatz M, Oliveira JRM. Population and computational analysis of the MGEA6 P521A variation as a risk factor for familial Idiopathic Basal Ganglia Calcification (Fahr’s Disease) [Internet]. Journal of Molecular Neuroscience. 2011 ; 43( 3): 333-336.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9445-7
Trabalho de evento-anais periodico
Nicotinic acetylcholine receptors modulation by a snake toxin (2010)
Nery, Arthur Andrade; Trujillo, Cleber Augusto; Lameu, Claudiana ; Konno, Katsuhiro; Camargo, Antonio Carlos Martins de; Ulrich, Henning ; Hayashi, Mirian Akemi Furuie
Fonte: Journal of Molecular Neuroscience. Nome do evento: International Symposium on Cholinergic Mechanisms. Unidade: IQ
Assuntos: RECEPTORES, BIOQUÍMICA
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ABNT
NERY, Arthur Andrade et al. Nicotinic acetylcholine receptors modulation by a snake toxin. Journal of Molecular Neuroscience. Totowa: Instituto de Química, Universidade de São Paulo. . Acesso em: 20 out. 2025. , 2010
APA
Nery, A. A., Trujillo, C. A., Lameu, C., Konno, K., Camargo, A. C. M. de, Ulrich, H., & Hayashi, M. A. F. (2010). Nicotinic acetylcholine receptors modulation by a snake toxin. Journal of Molecular Neuroscience. Totowa: Instituto de Química, Universidade de São Paulo.
NLM
Nery AA, Trujillo CA, Lameu C, Konno K, Camargo ACM de, Ulrich H, Hayashi MAF. Nicotinic acetylcholine receptors modulation by a snake toxin. Journal of Molecular Neuroscience. 2010 ; 40( 1-2): 252-254.[citado 2025 out. 20 ]
Vancouver
Nery AA, Trujillo CA, Lameu C, Konno K, Camargo ACM de, Ulrich H, Hayashi MAF. Nicotinic acetylcholine receptors modulation by a snake toxin. Journal of Molecular Neuroscience. 2010 ; 40( 1-2): 252-254.[citado 2025 out. 20 ]
Artigo de periodico
Alpha7 nicotinic acetylcholine receptor expression and activity during neuronal differentiation of PC12 pheochromocytoma cells (2010)
Nery, Arthur Andrade; Resende, Rodrigo Ribeiro; Martins, Antonio Henrique; Trujillo, Cleber Augusto; Eterovic, Vesna A; Ulrich, Henning
Fonte: Journal of Molecular Neuroscience. Unidade: IQ
Assuntos: RECEPTORES, BIOQUÍMICA
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ABNT
NERY, Arthur Andrade et al. Alpha7 nicotinic acetylcholine receptor expression and activity during neuronal differentiation of PC12 pheochromocytoma cells. Journal of Molecular Neuroscience, v. 41, n. 3, p. 329-339, 2010Tradução . . Disponível em: https://doi.org/10.1007/s12031-010-9369-2. Acesso em: 20 out. 2025.
APA
Nery, A. A., Resende, R. R., Martins, A. H., Trujillo, C. A., Eterovic, V. A., & Ulrich, H. (2010). Alpha7 nicotinic acetylcholine receptor expression and activity during neuronal differentiation of PC12 pheochromocytoma cells. Journal of Molecular Neuroscience, 41( 3), 329-339. doi:10.1007/s12031-010-9369-2
NLM
Nery AA, Resende RR, Martins AH, Trujillo CA, Eterovic VA, Ulrich H. Alpha7 nicotinic acetylcholine receptor expression and activity during neuronal differentiation of PC12 pheochromocytoma cells [Internet]. Journal of Molecular Neuroscience. 2010 ; 41( 3): 329-339.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9369-2
Vancouver
Nery AA, Resende RR, Martins AH, Trujillo CA, Eterovic VA, Ulrich H. Alpha7 nicotinic acetylcholine receptor expression and activity during neuronal differentiation of PC12 pheochromocytoma cells [Internet]. Journal of Molecular Neuroscience. 2010 ; 41( 3): 329-339.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9369-2
Artigo de periodico
Plasticity of opioid receptors in the female periaqueductal Gray: multiparity-induced increase in the activity of genes encoding for mu and kappa receptors and a post-translational decrease in delta receptor expression (2010)
Teodorov, Elizabeth; Bernardi, Maria Martha; Ferrari, Merari de Fátima Ramires ; Fior-Chadi, Débora Rejane ; Felicio, Luciano Freitas
Fonte: Journal of Molecular Neuroscience. Unidades: IB, FMVZ
Assuntos: WESTERN BLOTTING, FARMACODINÂMICA, TÉCNICAS E PROCEDIMENTOS DE LABORATÓRIO, EXPRESSÃO GÊNICA
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ABNT
TEODOROV, Elizabeth et al. Plasticity of opioid receptors in the female periaqueductal Gray: multiparity-induced increase in the activity of genes encoding for mu and kappa receptors and a post-translational decrease in delta receptor expression. Journal of Molecular Neuroscience, v. 43, n. 2, p. 175-181, 2010Tradução . . Disponível em: https://doi.org/10.1007/s12031-010-9407-0. Acesso em: 20 out. 2025.
APA
Teodorov, E., Bernardi, M. M., Ferrari, M. de F. R., Fior-Chadi, D. R., & Felicio, L. F. (2010). Plasticity of opioid receptors in the female periaqueductal Gray: multiparity-induced increase in the activity of genes encoding for mu and kappa receptors and a post-translational decrease in delta receptor expression. Journal of Molecular Neuroscience, 43( 2), 175-181. doi:10.1007/s12031-010-9407-0
NLM
Teodorov E, Bernardi MM, Ferrari M de FR, Fior-Chadi DR, Felicio LF. Plasticity of opioid receptors in the female periaqueductal Gray: multiparity-induced increase in the activity of genes encoding for mu and kappa receptors and a post-translational decrease in delta receptor expression [Internet]. Journal of Molecular Neuroscience. 2010 ; 43( 2): 175-181.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9407-0
Vancouver
Teodorov E, Bernardi MM, Ferrari M de FR, Fior-Chadi DR, Felicio LF. Plasticity of opioid receptors in the female periaqueductal Gray: multiparity-induced increase in the activity of genes encoding for mu and kappa receptors and a post-translational decrease in delta receptor expression [Internet]. Journal of Molecular Neuroscience. 2010 ; 43( 2): 175-181.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-010-9407-0
Artigo de periodico
Relationship between expression of voltage-dependent anion channel (VDAC) isoforms and type of hexokinase binding sites on brain mitochondria (2010)
Poleti, Mirele Daiana; Tesch, Andréa Cristina; Crepaldi, Carla Rossini; Souza, Gustavo Henrique Martins Ferreira; Eberlin, Marcos Nogueira; César, Marcelo Cerqueira
Fonte: Journal of Molecular Neuroscience. Unidade: FZEA
Assuntos: NEUROCIÊNCIAS, APOPTOSE, CÉREBRO, MITOCÔNDRIAS
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ABNT
POLETI, Mirele Daiana et al. Relationship between expression of voltage-dependent anion channel (VDAC) isoforms and type of hexokinase binding sites on brain mitochondria. Journal of Molecular Neuroscience, v. 41, n. 1, p. 48-54, 2010Tradução . . Acesso em: 20 out. 2025.
APA
Poleti, M. D., Tesch, A. C., Crepaldi, C. R., Souza, G. H. M. F., Eberlin, M. N., & César, M. C. (2010). Relationship between expression of voltage-dependent anion channel (VDAC) isoforms and type of hexokinase binding sites on brain mitochondria. Journal of Molecular Neuroscience, 41( 1), 48-54.
NLM
Poleti MD, Tesch AC, Crepaldi CR, Souza GHMF, Eberlin MN, César MC. Relationship between expression of voltage-dependent anion channel (VDAC) isoforms and type of hexokinase binding sites on brain mitochondria. Journal of Molecular Neuroscience. 2010 ; 41( 1): 48-54.[citado 2025 out. 20 ]
Vancouver
Poleti MD, Tesch AC, Crepaldi CR, Souza GHMF, Eberlin MN, César MC. Relationship between expression of voltage-dependent anion channel (VDAC) isoforms and type of hexokinase binding sites on brain mitochondria. Journal of Molecular Neuroscience. 2010 ; 41( 1): 48-54.[citado 2025 out. 20 ]
Trabalho de evento-anais periodico
Nicotine effects on LPS-induced NF-'capa'B activity and IL-1'beta' expression in the cerebellum (2010)
Cafe-Mendes, Cecília Cerqueira; Markus, Regina P ; Scavone, Cristoforo ; Malta, Marília Brinati; Garay-Malpartida, Humberto Miguel; Marcourakis, Tania
Fonte: Journal of Molecular Neuroscience. Nome do evento: International Symposium on Cholinergic Mechanisms. Unidades: IB, ICB, EACH, FCF
Assuntos: FUMO (EFEITOS), CEREBELO
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ABNT
CAFE-MENDES, Cecília Cerqueira et al. Nicotine effects on LPS-induced NF-'capa'B activity and IL-1'beta' expression in the cerebellum. Journal of Molecular Neuroscience. Totowa: Instituto de Biociências, Universidade de São Paulo. . Acesso em: 20 out. 2025. , 2010
APA
Cafe-Mendes, C. C., Markus, R. P., Scavone, C., Malta, M. B., Garay-Malpartida, H. M., & Marcourakis, T. (2010). Nicotine effects on LPS-induced NF-'capa'B activity and IL-1'beta' expression in the cerebellum. Journal of Molecular Neuroscience. Totowa: Instituto de Biociências, Universidade de São Paulo.
NLM
Cafe-Mendes CC, Markus RP, Scavone C, Malta MB, Garay-Malpartida HM, Marcourakis T. Nicotine effects on LPS-induced NF-'capa'B activity and IL-1'beta' expression in the cerebellum. Journal of Molecular Neuroscience. 2010 ; 40( 1-2): 254-261.[citado 2025 out. 20 ]
Vancouver
Cafe-Mendes CC, Markus RP, Scavone C, Malta MB, Garay-Malpartida HM, Marcourakis T. Nicotine effects on LPS-induced NF-'capa'B activity and IL-1'beta' expression in the cerebellum. Journal of Molecular Neuroscience. 2010 ; 40( 1-2): 254-261.[citado 2025 out. 20 ]
Artigo de periodico
Expression profile and distribution of Efhcl gene transcript during rodent brain development (2009)
Conte, Fábio F.; Ribeiro, A. O.; Marchesini, Rafael B.; Pascoal, Vinicíus D. B.; Lopes-Cendes, Iscia; Gilioli, R.; Sbragia, L.; Silva, J. M.; Oliveira, A. R.; Bittencourt, Jackson Cioni
Fonte: Journal of Molecular Neuroscience. Unidade: ICB
Assunto: ANATOMIA
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ABNT
CONTE, Fábio F. et al. Expression profile and distribution of Efhcl gene transcript during rodent brain development. Journal of Molecular Neuroscience, v. 39, n. 1-2, p. 69-77, 2009Tradução . . Acesso em: 20 out. 2025.
APA
Conte, F. F., Ribeiro, A. O., Marchesini, R. B., Pascoal, V. D. B., Lopes-Cendes, I., Gilioli, R., et al. (2009). Expression profile and distribution of Efhcl gene transcript during rodent brain development. Journal of Molecular Neuroscience, 39( 1-2), 69-77.
NLM
Conte FF, Ribeiro AO, Marchesini RB, Pascoal VDB, Lopes-Cendes I, Gilioli R, Sbragia L, Silva JM, Oliveira AR, Bittencourt JC. Expression profile and distribution of Efhcl gene transcript during rodent brain development. Journal of Molecular Neuroscience. 2009 ; 39( 1-2): 69-77.[citado 2025 out. 20 ]
Vancouver
Conte FF, Ribeiro AO, Marchesini RB, Pascoal VDB, Lopes-Cendes I, Gilioli R, Sbragia L, Silva JM, Oliveira AR, Bittencourt JC. Expression profile and distribution of Efhcl gene transcript during rodent brain development. Journal of Molecular Neuroscience. 2009 ; 39( 1-2): 69-77.[citado 2025 out. 20 ]
Artigo de periodico
The gentics of Alzheimer´s Disease in Brazil: 10 years of analysis in a unique population (2009)
Oliveira, João Ricardo Mendes de; Nishimura, Agnes Lumi; Lemos, R. R; Zatz, Mayana
Fonte: Journal of Molecular Neuroscience. Unidade: IB
Assuntos: DOENÇA DE ALZHEIMER, GENÉTICA
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ABNT
OLIVEIRA, João Ricardo Mendes de et al. The gentics of Alzheimer´s Disease in Brazil: 10 years of analysis in a unique population. Journal of Molecular Neuroscience, v. 37, n. 1, p. 74-79, 2009Tradução . . Disponível em: https://doi.org/10.1007/s12031-008-9124-0. Acesso em: 20 out. 2025.
APA
Oliveira, J. R. M. de, Nishimura, A. L., Lemos, R. R., & Zatz, M. (2009). The gentics of Alzheimer´s Disease in Brazil: 10 years of analysis in a unique population. Journal of Molecular Neuroscience, 37( 1), 74-79. doi:10.1007/s12031-008-9124-0
NLM
Oliveira JRM de, Nishimura AL, Lemos RR, Zatz M. The gentics of Alzheimer´s Disease in Brazil: 10 years of analysis in a unique population [Internet]. Journal of Molecular Neuroscience. 2009 ; 37( 1): 74-79.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-008-9124-0
Vancouver
Oliveira JRM de, Nishimura AL, Lemos RR, Zatz M. The gentics of Alzheimer´s Disease in Brazil: 10 years of analysis in a unique population [Internet]. Journal of Molecular Neuroscience. 2009 ; 37( 1): 74-79.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-008-9124-0
Artigo de periodico
Animal models for genetic neuromuscular diseases (2008)
Vainzof, Mariz ; Ayub-Guerrieri, Danielle; Onofre, Paula C. G; Martins, Poliana C. M.; Lopes, Vanessa F; Zilberztajn, Dinorah; Maia, Lucas S.; Sell, Karen; Yamamoto, Lydia U
Fonte: Journal of Molecular Neuroscience. Unidade: IB
Assuntos: MODELOS ANIMAIS, DOENÇAS NEUROMUSCULARES, PROTEÍNAS MUSCULARES, DISTROFIA MUSCULAR
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ABNT
VAINZOF, Mariz et al. Animal models for genetic neuromuscular diseases. Journal of Molecular Neuroscience, v. 34, n. 3, p. 241-248, 2008Tradução . . Disponível em: https://doi.org/10.1007/s12031-007-9023-9. Acesso em: 20 out. 2025.
APA
Vainzof, M., Ayub-Guerrieri, D., Onofre, P. C. G., Martins, P. C. M., Lopes, V. F., Zilberztajn, D., et al. (2008). Animal models for genetic neuromuscular diseases. Journal of Molecular Neuroscience, 34( 3), 241-248. doi:10.1007/s12031-007-9023-9
NLM
Vainzof M, Ayub-Guerrieri D, Onofre PCG, Martins PCM, Lopes VF, Zilberztajn D, Maia LS, Sell K, Yamamoto LU. Animal models for genetic neuromuscular diseases [Internet]. Journal of Molecular Neuroscience. 2008 ; 34( 3): 241-248.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-007-9023-9
Vancouver
Vainzof M, Ayub-Guerrieri D, Onofre PCG, Martins PCM, Lopes VF, Zilberztajn D, Maia LS, Sell K, Yamamoto LU. Animal models for genetic neuromuscular diseases [Internet]. Journal of Molecular Neuroscience. 2008 ; 34( 3): 241-248.[citado 2025 out. 20 ] Available from: https://doi.org/10.1007/s12031-007-9023-9

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