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Artigo de periodico
Antiviral response induced by toll-like receptor (TLR) 7/TLR8 activation inhibits human immunodeficiency virus Type 1 infection in cord blood macrophages (2022)
Pietrobon, Anna Julia ; Yoshikawa, Fábio Setti Yamada ; Oliveira, Luana Maia; Pereira, Natalli Zanete ; Souza, Taís Aparecida Matozo de ; Bargieri, Bruna Cunha de Alencar ; Duarte, Alberto José da Silva; Sato, Maria Notomi
Fonte: Journal of Infectious Diseases. Unidades: ICB, FM
Assuntos: IMUNOLOGIA, ADJUVANTES IMUNOLÓGICOS, HIV, INFECÇÕES POR HIV, RECÉM-NASCIDO, MACRÓFAGOS, IMUNODEFICIÊNCIA DE VARIÁVEL COMUM, INTERFERON TIPO I, MONÓCITOS
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PIETROBON, Anna Julia et al. Antiviral response induced by toll-like receptor (TLR) 7/TLR8 activation inhibits human immunodeficiency virus Type 1 infection in cord blood macrophages. Journal of Infectious Diseases, v. 225, n. 3, p. 510–519, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab389. Acesso em: 29 jun. 2025.
APA
Pietrobon, A. J., Yoshikawa, F. S. Y., Oliveira, L. M., Pereira, N. Z., Souza, T. A. M. de, Bargieri, B. C. de A., et al. (2022). Antiviral response induced by toll-like receptor (TLR) 7/TLR8 activation inhibits human immunodeficiency virus Type 1 infection in cord blood macrophages. Journal of Infectious Diseases, 225( 3), 510–519. doi:10.1093/infdis/jiab389
NLM
Pietrobon AJ, Yoshikawa FSY, Oliveira LM, Pereira NZ, Souza TAM de, Bargieri BC de A, Duarte AJ da S, Sato MN. Antiviral response induced by toll-like receptor (TLR) 7/TLR8 activation inhibits human immunodeficiency virus Type 1 infection in cord blood macrophages [Internet]. Journal of Infectious Diseases. 2022 ; 225( 3): 510–519.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiab389
Vancouver
Pietrobon AJ, Yoshikawa FSY, Oliveira LM, Pereira NZ, Souza TAM de, Bargieri BC de A, Duarte AJ da S, Sato MN. Antiviral response induced by toll-like receptor (TLR) 7/TLR8 activation inhibits human immunodeficiency virus Type 1 infection in cord blood macrophages [Internet]. Journal of Infectious Diseases. 2022 ; 225( 3): 510–519.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiab389
Artigo de periodico
Plasmodium simium: population genomics reveals the origin of a reverse zoonosis (2021)
Oliveira, Thaís C. de; Rodrigues, Priscila Thihara ; Early, Angela M; Duarte, Ana Maria Ribeiro de Castro ; Buery, Julyana C; Bueno, Marina Galvão ; Catão-Dias, José Luiz ; Cerutti Junior, Crispim; Rona, Luísa D. P; Neafsey, Daniel E. ; Ferreira, Marcelo Urbano
Fonte: Journal of Infectious Diseases. Unidades: FMVZ, ICB
Assuntos: PLASMODIUM, MALÁRIA, ZOONOSES
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OLIVEIRA, Thaís C. de et al. Plasmodium simium: population genomics reveals the origin of a reverse zoonosis. Journal of Infectious Diseases, v. 224, n. 11, p. 1950-1961, 2021Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab214. Acesso em: 29 jun. 2025.
APA
Oliveira, T. C. de, Rodrigues, P. T., Early, A. M., Duarte, A. M. R. de C., Buery, J. C., Bueno, M. G., et al. (2021). Plasmodium simium: population genomics reveals the origin of a reverse zoonosis. Journal of Infectious Diseases, 224( 11), 1950-1961. doi:10.1093/infdis/jiab214
NLM
Oliveira TC de, Rodrigues PT, Early AM, Duarte AMR de C, Buery JC, Bueno MG, Catão-Dias JL, Cerutti Junior C, Rona LDP, Neafsey DE, Ferreira MU. Plasmodium simium: population genomics reveals the origin of a reverse zoonosis [Internet]. Journal of Infectious Diseases. 2021 ; 224( 11): 1950-1961.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiab214
Vancouver
Oliveira TC de, Rodrigues PT, Early AM, Duarte AMR de C, Buery JC, Bueno MG, Catão-Dias JL, Cerutti Junior C, Rona LDP, Neafsey DE, Ferreira MU. Plasmodium simium: population genomics reveals the origin of a reverse zoonosis [Internet]. Journal of Infectious Diseases. 2021 ; 224( 11): 1950-1961.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiab214
Artigo de periodico
Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria (2021)
Bomfim, Caio César Barbosa ; Amaral, Eduardo Pinheiro ; Carvalho, Igor Santiago ; Santos, Gislane Almeida ; Salles, Érika Machado de ; Hastreiter, Araceli Aparecida ; Nascimento, Rogério Silva do; Almeida, Fabricio M.; Simão, Thatiana Lopes Biá Ventura ; Rezende, Andreza Linhares; Hirata, Mario Hiroyuki ; Fock, Ricardo Ambrósio ; Alvarez, José Maria ; Lasunskaia, Elena B ; Lima, Maria Regina D'Império
Fonte: Journal of Infectious Diseases. Unidades: ICB, FCF
Assuntos: TUBERCULOSE PULMONAR, CÉLULAS SANGUINEAS, GRANULOCITOS CITOPLASMATICOS, MEDULA ÓSSEA DE ANIMAL, VIRULÊNCIA, NEUTRÓFILOS, PROLIFERAÇÃO CELULAR, MYCOBACTERIUM BOVIS
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BOMFIM, Caio César Barbosa et al. Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria. Journal of Infectious Diseases, v. 223, n. 3, p. 494–507, 2021Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiaa708. Acesso em: 29 jun. 2025.
APA
Bomfim, C. C. B., Amaral, E. P., Carvalho, I. S., Santos, G. A., Salles, É. M. de, Hastreiter, A. A., et al. (2021). Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria. Journal of Infectious Diseases, 223( 3), 494–507. doi:10.1093/infdis/jiaa708
NLM
Bomfim CCB, Amaral EP, Carvalho IS, Santos GA, Salles ÉM de, Hastreiter AA, Nascimento RS do, Almeida FM, Simão TLBV, Rezende AL, Hirata MH, Fock RA, Alvarez JM, Lasunskaia EB, Lima MRD'I. Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria [Internet]. Journal of Infectious Diseases. 2021 ; 223( 3): 494–507.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiaa708
Vancouver
Bomfim CCB, Amaral EP, Carvalho IS, Santos GA, Salles ÉM de, Hastreiter AA, Nascimento RS do, Almeida FM, Simão TLBV, Rezende AL, Hirata MH, Fock RA, Alvarez JM, Lasunskaia EB, Lima MRD'I. Harmful effects of granulocytic myeloid-derived suppressor cells on tuberculosis caused by hypervirulent mycobacteria [Internet]. Journal of Infectious Diseases. 2021 ; 223( 3): 494–507.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiaa708
Artigo de periodico
ACE2 expression is increased in the lungs of patients with comorbidities associated with severe COVID-19 (2020)
Pinto, Bruna G. G; Oliveira, Antonio Edson Rocha; Singh, Youvika; Gonçalves, André Nicolau Aquime ; Ogava, Rodrigo L. T; Creighton, Rachel; Peron, Jean Pierre Schatzmann ; Nakaya, Helder Takashi Imoto
Fonte: Journal of Infectious Diseases. Unidades: ICB, FCF
Assuntos: ANGIOTENSINAS, COVID-19, COMORBIDADE, GENES, IMUNOLOGIA, INFECÇÕES RESPIRATÓRIAS, PNEUMOPATIAS, DOENÇAS CARDIOVASCULARES
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PINTO, Bruna G. G et al. ACE2 expression is increased in the lungs of patients with comorbidities associated with severe COVID-19. Journal of Infectious Diseases, v. 222, p. 556-563, 2020Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiaa332. Acesso em: 29 jun. 2025.
APA
Pinto, B. G. G., Oliveira, A. E. R., Singh, Y., Gonçalves, A. N. A., Ogava, R. L. T., Creighton, R., et al. (2020). ACE2 expression is increased in the lungs of patients with comorbidities associated with severe COVID-19. Journal of Infectious Diseases, 222, 556-563. doi:10.1093/infdis/jiaa332
NLM
Pinto BGG, Oliveira AER, Singh Y, Gonçalves ANA, Ogava RLT, Creighton R, Peron JPS, Nakaya HTI. ACE2 expression is increased in the lungs of patients with comorbidities associated with severe COVID-19 [Internet]. Journal of Infectious Diseases. 2020 ; 222 556-563.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiaa332
Vancouver
Pinto BGG, Oliveira AER, Singh Y, Gonçalves ANA, Ogava RLT, Creighton R, Peron JPS, Nakaya HTI. ACE2 expression is increased in the lungs of patients with comorbidities associated with severe COVID-19 [Internet]. Journal of Infectious Diseases. 2020 ; 222 556-563.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiaa332
Artigo de periodico
Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a (2018)
Ntumngia, Francis B.; Thomson-Luque, Richard ; Galusic, Sandra ; Frato, Gabriel; Frischmann, Sarah; Peabody, David S.; Chackerian, Bryce ; Ferreira, Marcelo Urbano ; King, Christopher L.; Adams, John H.
Fonte: Journal of Infectious Diseases. Unidade: ICB
Assuntos: PARASITOLOGIA, PLASMODIUM, PROTEÍNAS, MALÁRIA, VACINAS
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NTUMNGIA, Francis B. et al. Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a. Journal of Infectious Diseases, v. 218, n. 7, p. 1110-1118, 2018Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiy273. Acesso em: 29 jun. 2025.
APA
Ntumngia, F. B., Thomson-Luque, R., Galusic, S., Frato, G., Frischmann, S., Peabody, D. S., et al. (2018). Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a. Journal of Infectious Diseases, 218( 7), 1110-1118. doi:10.1093/infdis/jiy273
NLM
Ntumngia FB, Thomson-Luque R, Galusic S, Frato G, Frischmann S, Peabody DS, Chackerian B, Ferreira MU, King CL, Adams JH. Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a [Internet]. Journal of Infectious Diseases. 2018 ; 218( 7): 1110-1118.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiy273
Vancouver
Ntumngia FB, Thomson-Luque R, Galusic S, Frato G, Frischmann S, Peabody DS, Chackerian B, Ferreira MU, King CL, Adams JH. Identification and immunological characterization of the ligand domain of Plasmodium vivax reticulocyte binding protein 1a [Internet]. Journal of Infectious Diseases. 2018 ; 218( 7): 1110-1118.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiy273
Artigo de periodico
Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity (2017)
Schimke, Lena F; Hibbard, James; Martinez-Barricarte, Ruben; Khan, Taj Ali; Cavalcante, Ricardo de Souza; Oliveira-Junior, Edgar Borges de; França, Tábata Takahashi; Iqbal, Asif; Yamamoto, Guilherme; Arslanian, Christina; Feriotti, Claudia; Costa, Tânia Alves; Bustamante, Jacinta; Boisson-Dupuis, Stéphanie; Casanova, Jean-Laurent; Barbuto, José Alexandre Marzagão ; Zatz, Mayana ; Mendes, Rinaldo Poncio; Calich, Vera Lúcia Garcia; Ochs, Hans D; Torgerson, Troy R; Cabral-Marques, Otavio; Condino Neto, Antonio
Fonte: Journal of Infectious Diseases. Unidades: ICB, IB
Assuntos: PARACOCCIDIOIDOMICOSE, IMUNOLOGIA, MUTAÇÃO GENÉTICA, PREDISPOSIÇÃO GENÉTICA PARA DOENÇA, INFECÇÕES BACTERIANAS E MICOSES
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SCHIMKE, Lena F et al. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, v. 216, n. 12, p. 1623-1634, 2017Tradução . . Disponível em: https://doi.org/10.1093/infdis/jix522. Acesso em: 29 jun. 2025.
APA
Schimke, L. F., Hibbard, J., Martinez-Barricarte, R., Khan, T. A., Cavalcante, R. de S., Oliveira-Junior, E. B. de, et al. (2017). Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, 216( 12), 1623-1634. doi:10.1093/infdis/jix522
NLM
Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jix522
Vancouver
Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jix522
Artigo de periodico
Specific biomarkers associated with neurological complications and congenital central nervous system abnormalities from Zika virus-infected patients in Brazil (2017)
Kam, Yiu-Wing; Leite, Juliana Almeida; Lum, Fok-Moon; Tan, Jeslin J. L.; Lee, Bernett; Judice, Carla C.; Teixeira, Daniel Augusto de Toledo; Andreata-Santos, Robert; Vinolo, Marco A.; Angerami, Rodrigo; Resende, Mariangela Ribeiro; Freitas, Andre Ricardo Ribas; Amaral, Eliana; Passini Junior, Renato; Costa, Maria Laura; Guida, José Paulo; Arns, Clarice Weis; Ferreira, Luís Carlos de Souza ; Rénia, Laurent; Proença-Modena, Jose Luiz; Ng, Lisa F. P.; Costa, Fabio T. M.
Fonte: Journal of Infectious Diseases. Unidade: ICB
Assuntos: MICROBIOLOGIA, ZIKA VÍRUS, SISTEMA NERVOSO CENTRAL, BIOMARCADORES
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KAM, Yiu-Wing et al. Specific biomarkers associated with neurological complications and congenital central nervous system abnormalities from Zika virus-infected patients in Brazil. Journal of Infectious Diseases, v. 16, n. 2, p. 172-181, 2017Tradução . . Disponível em: https://doi.org/10.1093/infdis/jix261. Acesso em: 29 jun. 2025.
APA
Kam, Y. -W., Leite, J. A., Lum, F. -M., Tan, J. J. L., Lee, B., Judice, C. C., et al. (2017). Specific biomarkers associated with neurological complications and congenital central nervous system abnormalities from Zika virus-infected patients in Brazil. Journal of Infectious Diseases, 16( 2), 172-181. doi:10.1093/infdis/jix261
NLM
Kam Y-W, Leite JA, Lum F-M, Tan JJL, Lee B, Judice CC, Teixeira DA de T, Andreata-Santos R, Vinolo MA, Angerami R, Resende MR, Freitas ARR, Amaral E, Passini Junior R, Costa ML, Guida JP, Arns CW, Ferreira LC de S, Rénia L, Proença-Modena JL, Ng LFP, Costa FTM. Specific biomarkers associated with neurological complications and congenital central nervous system abnormalities from Zika virus-infected patients in Brazil [Internet]. Journal of Infectious Diseases. 2017 ; 16( 2): 172-181.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jix261
Vancouver
Kam Y-W, Leite JA, Lum F-M, Tan JJL, Lee B, Judice CC, Teixeira DA de T, Andreata-Santos R, Vinolo MA, Angerami R, Resende MR, Freitas ARR, Amaral E, Passini Junior R, Costa ML, Guida JP, Arns CW, Ferreira LC de S, Rénia L, Proença-Modena JL, Ng LFP, Costa FTM. Specific biomarkers associated with neurological complications and congenital central nervous system abnormalities from Zika virus-infected patients in Brazil [Internet]. Journal of Infectious Diseases. 2017 ; 16( 2): 172-181.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jix261
Artigo de periodico
Persistent HIV Type 1 Seronegative Status Is Associated With Lower CD8+ T-Cell Activation (2016)
Kuebler, Peter J.; Mehrotra, Megha L.; Shaw, Brian I.; Leadabrand, Kaitlyn S.; Milush, Jeffrey M.; York, Vanessa A.; Defechereux, Patricia; Grant, Robert M.; Kallás, Esper G.; Nixon, Douglas F.
Fonte: Journal of Infectious Diseases. Unidade: FM
Assuntos: HIV, SISTEMA IMUNE, TRANSMISSÃO DE DOENÇAS
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KUEBLER, Peter J. et al. Persistent HIV Type 1 Seronegative Status Is Associated With Lower CD8+ T-Cell Activation. Journal of Infectious Diseases, v. 213, n. 4, p. 569-573, 2016Tradução . . Disponível em: http://jid.oxfordjournals.org/content/213/4/569.full.pdf+html. Acesso em: 29 jun. 2025.
APA
Kuebler, P. J., Mehrotra, M. L., Shaw, B. I., Leadabrand, K. S., Milush, J. M., York, V. A., et al. (2016). Persistent HIV Type 1 Seronegative Status Is Associated With Lower CD8+ T-Cell Activation. Journal of Infectious Diseases, 213( 4), 569-573. doi:10.1093/infdis/jiv425
NLM
Kuebler PJ, Mehrotra ML, Shaw BI, Leadabrand KS, Milush JM, York VA, Defechereux P, Grant RM, Kallás EG, Nixon DF. Persistent HIV Type 1 Seronegative Status Is Associated With Lower CD8+ T-Cell Activation [Internet]. Journal of Infectious Diseases. 2016 ; 213( 4): 569-573.[citado 2025 jun. 29 ] Available from: http://jid.oxfordjournals.org/content/213/4/569.full.pdf+html
Vancouver
Kuebler PJ, Mehrotra ML, Shaw BI, Leadabrand KS, Milush JM, York VA, Defechereux P, Grant RM, Kallás EG, Nixon DF. Persistent HIV Type 1 Seronegative Status Is Associated With Lower CD8+ T-Cell Activation [Internet]. Journal of Infectious Diseases. 2016 ; 213( 4): 569-573.[citado 2025 jun. 29 ] Available from: http://jid.oxfordjournals.org/content/213/4/569.full.pdf+html
Artigo de periodico
Naturally acquired binding-inhibitory antibodies to Plasmodium vivax duffy binding protein and clinical immunity to malaria in rural amazonians (2016)
Nicolete, Vanessa C.; Frischmann, Sarah; Barbosa, Susana; King, Christopher L.; Ferreira, Marcelo Urbano
Fonte: Journal of Infectious Diseases. Unidade: ICB
Assuntos: PARASITOLOGIA, PLASMODIUM, MALÁRIA, ANTICORPOS, PROTEÍNAS, IMUNIDADE, ANTÍGENOS, QUIMIOCINAS
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NICOLETE, Vanessa C. et al. Naturally acquired binding-inhibitory antibodies to Plasmodium vivax duffy binding protein and clinical immunity to malaria in rural amazonians. Journal of Infectious Diseases, v. 214, n. 10, p. 1539-1546, 2016Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiw407. Acesso em: 29 jun. 2025.
APA
Nicolete, V. C., Frischmann, S., Barbosa, S., King, C. L., & Ferreira, M. U. (2016). Naturally acquired binding-inhibitory antibodies to Plasmodium vivax duffy binding protein and clinical immunity to malaria in rural amazonians. Journal of Infectious Diseases, 214( 10), 1539-1546. doi:10.1093/infdis/jiw407
NLM
Nicolete VC, Frischmann S, Barbosa S, King CL, Ferreira MU. Naturally acquired binding-inhibitory antibodies to Plasmodium vivax duffy binding protein and clinical immunity to malaria in rural amazonians [Internet]. Journal of Infectious Diseases. 2016 ; 214( 10): 1539-1546.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiw407
Vancouver
Nicolete VC, Frischmann S, Barbosa S, King CL, Ferreira MU. Naturally acquired binding-inhibitory antibodies to Plasmodium vivax duffy binding protein and clinical immunity to malaria in rural amazonians [Internet]. Journal of Infectious Diseases. 2016 ; 214( 10): 1539-1546.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiw407
Artigo de periodico
Transfusion-Transmitted Dengue and Associated Clinical Symptoms During the 2012 Epidemic in Brazil (2016)
Sabino, Ester Cerdeira ; Loureiro, Paula; Lopes, Maria Esther; Capuani, Ligia; McClure, Christopher; Chowdhury, Dhuly; Di-Lorenzo-Oliveira, Claudia; Oliveira, Lea C.; Linnen, Jeffrey M.; Lee, Tzong-Hae; Gonçalez, Thelma; Brambilla, Donald; Kleinman, Steve; Busch, Michael P.; Custer, Brian
Fonte: Journal of Infectious Diseases. Unidade: FM
Assuntos: DENGUE, SINAIS E SINTOMAS, TRANSMISSÃO DE DOENÇAS
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SABINO, Ester Cerdeira et al. Transfusion-Transmitted Dengue and Associated Clinical Symptoms During the 2012 Epidemic in Brazil. Journal of Infectious Diseases, v. 213, n. 5, p. 694-702, 2016Tradução . . Disponível em: http://jid.oxfordjournals.org/content/213/5/694.full.pdf+html. Acesso em: 29 jun. 2025.
APA
Sabino, E. C., Loureiro, P., Lopes, M. E., Capuani, L., McClure, C., Chowdhury, D., et al. (2016). Transfusion-Transmitted Dengue and Associated Clinical Symptoms During the 2012 Epidemic in Brazil. Journal of Infectious Diseases, 213( 5), 694-702. doi:10.1093/infdis/jiv326
NLM
Sabino EC, Loureiro P, Lopes ME, Capuani L, McClure C, Chowdhury D, Di-Lorenzo-Oliveira C, Oliveira LC, Linnen JM, Lee T-H, Gonçalez T, Brambilla D, Kleinman S, Busch MP, Custer B. Transfusion-Transmitted Dengue and Associated Clinical Symptoms During the 2012 Epidemic in Brazil [Internet]. Journal of Infectious Diseases. 2016 ; 213( 5): 694-702.[citado 2025 jun. 29 ] Available from: http://jid.oxfordjournals.org/content/213/5/694.full.pdf+html
Vancouver
Sabino EC, Loureiro P, Lopes ME, Capuani L, McClure C, Chowdhury D, Di-Lorenzo-Oliveira C, Oliveira LC, Linnen JM, Lee T-H, Gonçalez T, Brambilla D, Kleinman S, Busch MP, Custer B. Transfusion-Transmitted Dengue and Associated Clinical Symptoms During the 2012 Epidemic in Brazil [Internet]. Journal of Infectious Diseases. 2016 ; 213( 5): 694-702.[citado 2025 jun. 29 ] Available from: http://jid.oxfordjournals.org/content/213/5/694.full.pdf+html
Artigo de periodico
Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection (2015)
Nascimento, Manuela Sales Lima; Carregaro, Vanessa; Lima Júnior, Djalma Souza; Costa, Diego Luís; Ryffel, Bernhard; Duthie, Malcolm S.; Jesus, Amélia de; Almeida, Roque Pacheco de; Silva, João Santana da
Fonte: Journal of Infectious Diseases. Unidade: FMRP
Assuntos: IMUNOLOGIA CELULAR, INTERFERONS, INTERLEUCINAS, LEISHMANIOSE VISCERAL, LINFÓCITOS T
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NASCIMENTO, Manuela Sales Lima et al. Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection. Journal of Infectious Diseases, v. 211, n. 6, p. 1015-1025, 2015Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu531. Acesso em: 29 jun. 2025.
APA
Nascimento, M. S. L., Carregaro, V., Lima Júnior, D. S., Costa, D. L., Ryffel, B., Duthie, M. S., et al. (2015). Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection. Journal of Infectious Diseases, 211( 6), 1015-1025. doi:10.1093/infdis/jiu531
NLM
Nascimento MSL, Carregaro V, Lima Júnior DS, Costa DL, Ryffel B, Duthie MS, Jesus A de, Almeida RP de, Silva JS da. Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection [Internet]. Journal of Infectious Diseases. 2015 ; 211( 6): 1015-1025.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu531
Vancouver
Nascimento MSL, Carregaro V, Lima Júnior DS, Costa DL, Ryffel B, Duthie MS, Jesus A de, Almeida RP de, Silva JS da. Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection [Internet]. Journal of Infectious Diseases. 2015 ; 211( 6): 1015-1025.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu531
Artigo de periodico
Interleukin 1 receptor–driven neutrophil recruitment accounts to MyD88–dependent pulmonary clearance of Legionella pneumophila infection in vivo (2015)
Mascarenhas, Danielle Pini Alves; Pereira, Marcelo S. F.; Manin, Graziele Zenaro; Hori, Juliana Issa; Zamboni, Dario Simões
Fonte: Journal of Infectious Diseases. Unidade: FMRP
Assuntos: NEUTRÓFILOS, INTERLEUCINA 1, RECEPTORES, LEGIONELLA PNEUMOPHILA (IMUNOLOGIA), MODELOS ANIMAIS DE DOENÇAS
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ABNT
MASCARENHAS, Danielle Pini Alves et al. Interleukin 1 receptor–driven neutrophil recruitment accounts to MyD88–dependent pulmonary clearance of Legionella pneumophila infection in vivo. Journal of Infectious Diseases, v. 211, n. 2, p. 322-330, 2015Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu430. Acesso em: 29 jun. 2025.
APA
Mascarenhas, D. P. A., Pereira, M. S. F., Manin, G. Z., Hori, J. I., & Zamboni, D. S. (2015). Interleukin 1 receptor–driven neutrophil recruitment accounts to MyD88–dependent pulmonary clearance of Legionella pneumophila infection in vivo. Journal of Infectious Diseases, 211( 2), 322-330. doi:10.1093/infdis/jiu430
NLM
Mascarenhas DPA, Pereira MSF, Manin GZ, Hori JI, Zamboni DS. Interleukin 1 receptor–driven neutrophil recruitment accounts to MyD88–dependent pulmonary clearance of Legionella pneumophila infection in vivo [Internet]. Journal of Infectious Diseases. 2015 ; 211( 2): 322-330.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu430
Vancouver
Mascarenhas DPA, Pereira MSF, Manin GZ, Hori JI, Zamboni DS. Interleukin 1 receptor–driven neutrophil recruitment accounts to MyD88–dependent pulmonary clearance of Legionella pneumophila infection in vivo [Internet]. Journal of Infectious Diseases. 2015 ; 211( 2): 322-330.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu430
Artigo de periodico
Induction of inhibitory receptors on T cells during Plasmodium vivax malaria impairs cytokine production (2015)
Costa, Pedro A. C; Leoratti, Fabiana M. S; Figueiredo, Maria M; Tada, Mauro Shugiro; Pereira, Dhelio Batista; Junqueira, Caroline; Soares, Irene da Silva ; Barber, Daniel L; Gazzinelli, Ricardo T; Antonelli, Lis Ribeiro do Valle
Fonte: Journal of Infectious Diseases. Unidade: FCF
Assuntos: PLASMODIUM, MALÁRIA, RECEPTORES
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ABNT
COSTA, Pedro A. C et al. Induction of inhibitory receptors on T cells during Plasmodium vivax malaria impairs cytokine production. Journal of Infectious Diseases, p. 1-12, 2015Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiv306. Acesso em: 29 jun. 2025.
APA
Costa, P. A. C., Leoratti, F. M. S., Figueiredo, M. M., Tada, M. S., Pereira, D. B., Junqueira, C., et al. (2015). Induction of inhibitory receptors on T cells during Plasmodium vivax malaria impairs cytokine production. Journal of Infectious Diseases, 1-12. doi:10.1093/infdis/jiv306
NLM
Costa PAC, Leoratti FMS, Figueiredo MM, Tada MS, Pereira DB, Junqueira C, Soares I da S, Barber DL, Gazzinelli RT, Antonelli LR do V. Induction of inhibitory receptors on T cells during Plasmodium vivax malaria impairs cytokine production [Internet]. Journal of Infectious Diseases. 2015 ; 1-12.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiv306
Vancouver
Costa PAC, Leoratti FMS, Figueiredo MM, Tada MS, Pereira DB, Junqueira C, Soares I da S, Barber DL, Gazzinelli RT, Antonelli LR do V. Induction of inhibitory receptors on T cells during Plasmodium vivax malaria impairs cytokine production [Internet]. Journal of Infectious Diseases. 2015 ; 1-12.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiv306
Artigo de periodico
Tr-1-like CD4+CD25-CD127-/lowFOXP3-cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis (2015)
Costa, Diego L.; Cardoso, Tiago M.; Queiroz, Adriano; Milanezi, Cristiane Maria; Bacellar, Olívia; Carvalho, Edgar M.; Silva, João Santana da
Fonte: Journal of Infectious Diseases. Unidade: FMRP
Assuntos: INTERFERONS, LINFÓCITOS T, INTERLEUCINAS, LEISHMANIOSE CUTÂNEA, LEISHMANIA BRASILIENSIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Diego L. et al. Tr-1-like CD4+CD25-CD127-/lowFOXP3-cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis. Journal of Infectious Diseases, v. 211, n. 5, p. 708-718, 2015Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu406. Acesso em: 29 jun. 2025.
APA
Costa, D. L., Cardoso, T. M., Queiroz, A., Milanezi, C. M., Bacellar, O., Carvalho, E. M., & Silva, J. S. da. (2015). Tr-1-like CD4+CD25-CD127-/lowFOXP3-cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis. Journal of Infectious Diseases, 211( 5), 708-718. doi:10.1093/infdis/jiu406
NLM
Costa DL, Cardoso TM, Queiroz A, Milanezi CM, Bacellar O, Carvalho EM, Silva JS da. Tr-1-like CD4+CD25-CD127-/lowFOXP3-cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis [Internet]. Journal of Infectious Diseases. 2015 ; 211( 5): 708-718.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu406
Vancouver
Costa DL, Cardoso TM, Queiroz A, Milanezi CM, Bacellar O, Carvalho EM, Silva JS da. Tr-1-like CD4+CD25-CD127-/lowFOXP3-cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis [Internet]. Journal of Infectious Diseases. 2015 ; 211( 5): 708-718.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu406
Artigo de periodico
The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins (2015)
Abreu, Afonso G.; Fraga, Tatiana Rodrigues; Martínez, Adriana Patricia Granados; Kondo, Marcia Y.; Juliano, Maria A.; Juliano, Luiz; Navarro-Garcia, Fernando; Isaac, Lourdes ; Barbosa, Angela Silva; Elias, Waldir Pereira
Fonte: Journal of Infectious Diseases. Unidade: ICB
Assuntos: ESCHEICHIA COLI, IMUNOPROTEINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ABREU, Afonso G. et al. The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins. Journal of Infectious Diseases, v. 212, n. 1, p. 106-115, 2015Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiv013. Acesso em: 29 jun. 2025.
APA
Abreu, A. G., Fraga, T. R., Martínez, A. P. G., Kondo, M. Y., Juliano, M. A., Juliano, L., et al. (2015). The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins. Journal of Infectious Diseases, 212( 1), 106-115. doi:10.1093/infdis/jiv013
NLM
Abreu AG, Fraga TR, Martínez APG, Kondo MY, Juliano MA, Juliano L, Navarro-Garcia F, Isaac L, Barbosa AS, Elias WP. The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins [Internet]. Journal of Infectious Diseases. 2015 ; 212( 1): 106-115.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiv013
Vancouver
Abreu AG, Fraga TR, Martínez APG, Kondo MY, Juliano MA, Juliano L, Navarro-Garcia F, Isaac L, Barbosa AS, Elias WP. The serine protease pic from enteroaggregative Escherichia coli mediates immune evasion by the direct cleavage of complement proteins [Internet]. Journal of Infectious Diseases. 2015 ; 212( 1): 106-115.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiv013
Artigo de periodico
Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis (2014)
Loures, Flávio V.; Araújo, Eliseu Frank de; Feriotti, Claudia; Bazan, Silvia B.; Costa, Tânia Alves da; Brown, Gordon D.; Calich, Vera Lucia Garcia
Fonte: Journal of Infectious Diseases. Unidade: ICB
Assuntos: IMUNOLOGIA, MACRÓFAGOS, PARACOCCIDIOIDOMICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio V. et al. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, v. 210, n. 5, p. 762-773, 2014Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu136. Acesso em: 29 jun. 2025.
APA
Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., Costa, T. A. da, Brown, G. D., & Calich, V. L. G. (2014). Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, 210( 5), 762-773. doi:10.1093/infdis/jiu136
NLM
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu136
Vancouver
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jiu136
Artigo de periodico
Immune evasion by pathogenic Leptospira strains: the secretion of proteases that directly cleave complement proteins (2014)
Fraga, Tatiana Rodrigues; Courrol, Daniella dos Santos; Castiblanco-Valencia, Mónica Marcela; Hirata, Izaura Yoshico; Vasconcellos, Silvio Arruda ; Juliano, Luiz; Barbosa, Angela Silva; Isaac, Lourdes
Fonte: Journal of Infectious Diseases. Unidades: FMVZ, ICB
Assuntos: ENZIMAS PROTEOLÍTICAS, IMUNIDADE, LEPTOSPIROSE
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ABNT
FRAGA, Tatiana Rodrigues et al. Immune evasion by pathogenic Leptospira strains: the secretion of proteases that directly cleave complement proteins. Journal of Infectious Diseases, v. 209, n. 6, p. 876-886, 2014Tradução . . Disponível em: https://doi.org/10.1093/infdis/jit569. Acesso em: 29 jun. 2025.
APA
Fraga, T. R., Courrol, D. dos S., Castiblanco-Valencia, M. M., Hirata, I. Y., Vasconcellos, S. A., Juliano, L., et al. (2014). Immune evasion by pathogenic Leptospira strains: the secretion of proteases that directly cleave complement proteins. Journal of Infectious Diseases, 209( 6), 876-886. doi:10.1093/infdis/jit569
NLM
Fraga TR, Courrol D dos S, Castiblanco-Valencia MM, Hirata IY, Vasconcellos SA, Juliano L, Barbosa AS, Isaac L. Immune evasion by pathogenic Leptospira strains: the secretion of proteases that directly cleave complement proteins [Internet]. Journal of Infectious Diseases. 2014 ; 209( 6): 876-886.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jit569
Vancouver
Fraga TR, Courrol D dos S, Castiblanco-Valencia MM, Hirata IY, Vasconcellos SA, Juliano L, Barbosa AS, Isaac L. Immune evasion by pathogenic Leptospira strains: the secretion of proteases that directly cleave complement proteins [Internet]. Journal of Infectious Diseases. 2014 ; 209( 6): 876-886.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jit569
Artigo de periodico
Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP (2012)
Castiblanco-Valencia, Mónica Marcela; Fraga, Tatiana Rodrigues; Silva, Ludmila Bezerra da; Monaris, Denize; Abreu, Patrícia Antônia Estima; Strobel, Stefanie; Józsi, Mihály; Isaac, Lourdes ; Barbosa, Angela Silva
Fonte: Journal of Infectious Diseases. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
CASTIBLANCO-VALENCIA, Mónica Marcela et al. Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP. Journal of Infectious Diseases, v. 205, n. 6, p. 995-1004, 2012Tradução . . Disponível em: https://doi.org/10.1093/infdis/jir875. Acesso em: 29 jun. 2025.
APA
Castiblanco-Valencia, M. M., Fraga, T. R., Silva, L. B. da, Monaris, D., Abreu, P. A. E., Strobel, S., et al. (2012). Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP. Journal of Infectious Diseases, 205( 6), 995-1004. doi:10.1093/infdis/jir875
NLM
Castiblanco-Valencia MM, Fraga TR, Silva LB da, Monaris D, Abreu PAE, Strobel S, Józsi M, Isaac L, Barbosa AS. Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP [Internet]. Journal of Infectious Diseases. 2012 ; 205( 6): 995-1004.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jir875
Vancouver
Castiblanco-Valencia MM, Fraga TR, Silva LB da, Monaris D, Abreu PAE, Strobel S, Józsi M, Isaac L, Barbosa AS. Leptospiral immunoglobulin-like proteins interact with human complement regulators Factor H, FHL-1, FHR-1, and C4BP [Internet]. Journal of Infectious Diseases. 2012 ; 205( 6): 995-1004.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jir875
Artigo de periodico
Genetic and functional evidence implicating DLL1 as the gene that influences susceptibility to visceral Leishmaniasis at chromosome 6q27 (2011)
Fakiola, Michaela; Miller, E. Nancy; Fadl, Manal; Mohamed, Hiba S.; Jamieson, Sarra E.; Francis, Richard W.; Cordell, Heather J.; Peacock, Christopher S.; Raju, Madhuri; Khalil, Etahir A.; Elhassan, Ahmed; Musa, Ahmed M.; Silveira, Fernando; Shaw, Jeffrey Jon; Sundar, Shyam; Jeronimo, Selma M. B.; Ibrahim, Muntaser E.; Blackwell, Jenefer M.
Fonte: Journal of Infectious Diseases. Unidade: ICB
Assunto: PARASITOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAKIOLA, Michaela et al. Genetic and functional evidence implicating DLL1 as the gene that influences susceptibility to visceral Leishmaniasis at chromosome 6q27. Journal of Infectious Diseases, v. 204, n. 3, p. 467-477, 2011Tradução . . Disponível em: https://doi.org/10.1093/infdis/jir284. Acesso em: 29 jun. 2025.
APA
Fakiola, M., Miller, E. N., Fadl, M., Mohamed, H. S., Jamieson, S. E., Francis, R. W., et al. (2011). Genetic and functional evidence implicating DLL1 as the gene that influences susceptibility to visceral Leishmaniasis at chromosome 6q27. Journal of Infectious Diseases, 204( 3), 467-477. doi:10.1093/infdis/jir284
NLM
Fakiola M, Miller EN, Fadl M, Mohamed HS, Jamieson SE, Francis RW, Cordell HJ, Peacock CS, Raju M, Khalil EA, Elhassan A, Musa AM, Silveira F, Shaw JJ, Sundar S, Jeronimo SMB, Ibrahim ME, Blackwell JM. Genetic and functional evidence implicating DLL1 as the gene that influences susceptibility to visceral Leishmaniasis at chromosome 6q27 [Internet]. Journal of Infectious Diseases. 2011 ; 204( 3): 467-477.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jir284
Vancouver
Fakiola M, Miller EN, Fadl M, Mohamed HS, Jamieson SE, Francis RW, Cordell HJ, Peacock CS, Raju M, Khalil EA, Elhassan A, Musa AM, Silveira F, Shaw JJ, Sundar S, Jeronimo SMB, Ibrahim ME, Blackwell JM. Genetic and functional evidence implicating DLL1 as the gene that influences susceptibility to visceral Leishmaniasis at chromosome 6q27 [Internet]. Journal of Infectious Diseases. 2011 ; 204( 3): 467-477.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1093/infdis/jir284
Artigo de periodico
On the cytoadhesion of Plasmodium vivax-infected erythrocytes (2010)
Carvalho, Bruna Oliveira e; Lopes, Stefanie Costa Pinto; Nogueira, Paulo Afonso; Orlandi, Patricia Puccinelli; Bargieri, Daniel Youssef ; Blanco, Yara Carollo; Mamoni, Ronei Luciano; Leite, Juliana Alves; Rodrigues, Mauricio Martins; Soares, Irene da Silva ; Oliveira, Tatiane Rodrigues; Wunderlich, Gerhard ; Lacerda, Marcus V. G; Portillo, Hernando Antonio del; Araujo, Maria O. G; Russell, Bruce; Suwanarusk, Rossarin; Snounou, Georges; Rénia, Laurent; Costa, Fábio Trindade Maranhão
Fonte: Journal of Infectious Diseases. Unidades: FCF, ICB
Assuntos: PLASMODIUM, MALÁRIA, PARASITOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Bruna Oliveira e et al. On the cytoadhesion of Plasmodium vivax-infected erythrocytes. Journal of Infectious Diseases, v. 202, n. 4, p. 638-647, 2010Tradução . . Disponível em: https://doi.org/10.1086/654815. Acesso em: 29 jun. 2025.
APA
Carvalho, B. O. e, Lopes, S. C. P., Nogueira, P. A., Orlandi, P. P., Bargieri, D. Y., Blanco, Y. C., et al. (2010). On the cytoadhesion of Plasmodium vivax-infected erythrocytes. Journal of Infectious Diseases, 202( 4), 638-647. doi:10.1086/654815
NLM
Carvalho BO e, Lopes SCP, Nogueira PA, Orlandi PP, Bargieri DY, Blanco YC, Mamoni RL, Leite JA, Rodrigues MM, Soares I da S, Oliveira TR, Wunderlich G, Lacerda MVG, Portillo HA del, Araujo MOG, Russell B, Suwanarusk R, Snounou G, Rénia L, Costa FTM. On the cytoadhesion of Plasmodium vivax-infected erythrocytes [Internet]. Journal of Infectious Diseases. 2010 ; 202( 4): 638-647.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1086/654815
Vancouver
Carvalho BO e, Lopes SCP, Nogueira PA, Orlandi PP, Bargieri DY, Blanco YC, Mamoni RL, Leite JA, Rodrigues MM, Soares I da S, Oliveira TR, Wunderlich G, Lacerda MVG, Portillo HA del, Araujo MOG, Russell B, Suwanarusk R, Snounou G, Rénia L, Costa FTM. On the cytoadhesion of Plasmodium vivax-infected erythrocytes [Internet]. Journal of Infectious Diseases. 2010 ; 202( 4): 638-647.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1086/654815

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