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Artigo de periodico
Integrative mapping of preexisting influenza immune landscapes predicts vaccine response (2025)
Hao, Stephanie ; Tomic, Ivan; Lindsey, Benjamin B; Jagne, Ya Jankey ; Hoschler, Katja; Meijer, Adam ; Quiroz, Juan Manuel Carreño ; Meade, Philip; Sano, Kaori; Peno, Chikondi ; Martins, André Guilherme da Costa ; Bogaert, Debby ; Kampmann, Beate ; Nakaya, Helder Takashi Imoto ; Krammer, Florian ; Silva, Thushan I de; Tomic, Adriana
Source: Journal of Clinical Investigation. Unidades: Interunidades em Biotecnologia, FCF
Subjects: INFLUENZA, VACINAS
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ABNT
HAO, Stephanie et al. Integrative mapping of preexisting influenza immune landscapes predicts vaccine response. Journal of Clinical Investigation, v. 135, n. 18, p. 1-15 art. e189300, 2025Tradução . . Disponível em: https://dx.doi.org/10.1172/JCI189300. Acesso em: 09 fev. 2026.
APA
Hao, S., Tomic, I., Lindsey, B. B., Jagne, Y. J., Hoschler, K., Meijer, A., et al. (2025). Integrative mapping of preexisting influenza immune landscapes predicts vaccine response. Journal of Clinical Investigation, 135( 18), 1-15 art. e189300. doi:10.1172/JCI189300
NLM
Hao S, Tomic I, Lindsey BB, Jagne YJ, Hoschler K, Meijer A, Quiroz JMC, Meade P, Sano K, Peno C, Martins AG da C, Bogaert D, Kampmann B, Nakaya HTI, Krammer F, Silva TI de, Tomic A. Integrative mapping of preexisting influenza immune landscapes predicts vaccine response [Internet]. Journal of Clinical Investigation. 2025 ; 135( 18): 1-15 art. e189300.[citado 2026 fev. 09 ] Available from: https://dx.doi.org/10.1172/JCI189300
Vancouver
Hao S, Tomic I, Lindsey BB, Jagne YJ, Hoschler K, Meijer A, Quiroz JMC, Meade P, Sano K, Peno C, Martins AG da C, Bogaert D, Kampmann B, Nakaya HTI, Krammer F, Silva TI de, Tomic A. Integrative mapping of preexisting influenza immune landscapes predicts vaccine response [Internet]. Journal of Clinical Investigation. 2025 ; 135( 18): 1-15 art. e189300.[citado 2026 fev. 09 ] Available from: https://dx.doi.org/10.1172/JCI189300
Artigo de periodico
C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps (2023)
Silva, Bruna Manuella Souza; Gomes, Giovanni Freitas; Veras, Flávio Protásio ; Cambier, Seppe; Silva, Gabriel Victor Lucena da ; Quadros, Andreza Urba de; Caetité, Diego Brito ; Nascimento, Daniele Carvalho ; Silva, Camilla M.; Silva, Juliana C. ; Damasceno, Samara; Schneider, Ayda Henriques ; Beretta, Fabio; Batah, Sabrina Setembre ; Castro, Ícaro Maia Santos ; Paiva, Isadora Marques ; Rodrigues, Tamara Silva ; Salina, Ana Carolina Guerta ; Martins Júnior, Ronaldo Bragança; Cebinelli, Guilherme Cesar Martelossi ; Bibo, Naira Lopes; Jorge, Daniel M.; Nakaya, Helder Takashi Imoto ; Zamboni, Dario Simões ; Leiria, Luiz Osório Silveira ; Fabro, Alexandre Todorovic ; Alves Filho, José Carlos Farias ; Arruda Neto, Eurico de; Louzada Júnior, Paulo ; Oliveira, Renê Donizete Ribeiro de; Cunha, Larissa Dias da; Mol, Pierre Van; Vanderbeke, Lore; Feys, Simon; Wauters, Els; Brandolini, Laura; Aramini, Andrea; Cunha, Fernando Q.; Köhl, Jörg; Allegretti, Marcello; Lambrechts, Diether; Wauters, Joost; Proost, Paul; Cunha, Thiago Mattar
Source: Journal of Clinical Investigation. Unidades: FCFRP, FMRP
Subjects: COVID-19, PULMÃO, INFLAMAÇÃO, IMUNIDADE
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ABNT
SILVA, Bruna Manuella Souza et al. C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps. Journal of Clinical Investigation, v. 133, n. 12, p. 1-18, 2023Tradução . . Disponível em: https://doi.org/10.1172/JCI163105. Acesso em: 09 fev. 2026.
APA
Silva, B. M. S., Gomes, G. F., Veras, F. P., Cambier, S., Silva, G. V. L. da, Quadros, A. U. de, et al. (2023). C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps. Journal of Clinical Investigation, 133( 12), 1-18. doi:10.1172/JCI163105
NLM
Silva BMS, Gomes GF, Veras FP, Cambier S, Silva GVL da, Quadros AU de, Caetité DB, Nascimento DC, Silva CM, Silva JC, Damasceno S, Schneider AH, Beretta F, Batah SS, Castro ÍMS, Paiva IM, Rodrigues TS, Salina ACG, Martins Júnior RB, Cebinelli GCM, Bibo NL, Jorge DM, Nakaya HTI, Zamboni DS, Leiria LOS, Fabro AT, Alves Filho JCF, Arruda Neto E de, Louzada Júnior P, Oliveira RDR de, Cunha LD da, Mol PV, Vanderbeke L, Feys S, Wauters E, Brandolini L, Aramini A, Cunha FQ, Köhl J, Allegretti M, Lambrechts D, Wauters J, Proost P, Cunha TM. C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps [Internet]. Journal of Clinical Investigation. 2023 ; 133( 12): 1-18.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI163105
Vancouver
Silva BMS, Gomes GF, Veras FP, Cambier S, Silva GVL da, Quadros AU de, Caetité DB, Nascimento DC, Silva CM, Silva JC, Damasceno S, Schneider AH, Beretta F, Batah SS, Castro ÍMS, Paiva IM, Rodrigues TS, Salina ACG, Martins Júnior RB, Cebinelli GCM, Bibo NL, Jorge DM, Nakaya HTI, Zamboni DS, Leiria LOS, Fabro AT, Alves Filho JCF, Arruda Neto E de, Louzada Júnior P, Oliveira RDR de, Cunha LD da, Mol PV, Vanderbeke L, Feys S, Wauters E, Brandolini L, Aramini A, Cunha FQ, Köhl J, Allegretti M, Lambrechts D, Wauters J, Proost P, Cunha TM. C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps [Internet]. Journal of Clinical Investigation. 2023 ; 133( 12): 1-18.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI163105
Artigo de periodico
Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice (2022)
Maganin, Alexandre Gomes de Macedo ; Souza, Guilherme Rabelo de; Fonseca, Miriam D.; Lopes, Alexandre H.; Guimarães, Rafaela Mano ; Dagostin, André; Cecílio, Nerry T.; Mendes, Atlante Silva ; Gonçalves, William Antônio; Silva, Conceição Elidianne Aníbal; Gomes, Francisco Isaac Fernandes ; Marques, Lucas Maciel Mauriz ; Silva, Rangel L.; Arruda, Leticia M.; Santana, Denis A.; Lemos, Henrique; Huang, Lei; Ferreira, Marcela Davoli; Santana-Coelho, Danielle; Sant’Anna, Morena Brasil Martins; Kusuda, Ricardo; Talbot, Jhimmy; Pacholczyk, Gabriela; Buqui, Gabriela A.; Lopes, Norberto Peporine ; Alves Filho, José Carlos Farias ; Leão, Ricardo Maurício Xavier; O’Connor, Jason C.; Cunha, Fernando de Queiroz ; Mellor, Andrew; Cunha, Thiago Mattar
Source: Journal of Clinical Investigation. Unidades: FCFRP, FMRP, FCF
Subjects: CÉLULAS DENDRÍTICAS, METABÓLITOS, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, NERVOS PERIFÉRICOS, DOR CRÔNICA
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ABNT
MAGANIN, Alexandre Gomes de Macedo et al. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, v. 132, n. 23, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1172/JCI153805. Acesso em: 09 fev. 2026.
APA
Maganin, A. G. de M., Souza, G. R. de, Fonseca, M. D., Lopes, A. H., Guimarães, R. M., Dagostin, A., et al. (2022). Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, 132( 23), 1-17. doi:10.1172/JCI153805
NLM
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI153805
Vancouver
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI153805
Artigo de periodico
Lysosomal exocytosis of HSP70 stimulates monocytic BMP6 expression in Sjögren’s syndrome (2022)
Mo, Ying-Qian; Nakamura, Hiroyuki; Tanaka, Tsutomu; Odani, Toshio; Perez, Paola; Ji, Youngmi; French, Benjamin N.; Oliveira, Fabiola Reis; Motta, Ana Carolina Fragoso; Ribeiro-Silva, Alfredo; Rocha, Eduardo Melani
Source: Journal of Clinical Investigation. Unidades: FMRP, FORP
Subjects: PROTEÍNAS MORFOGENÉTICAS ÓSSEAS, CITOCINAS, EXOCITOSE, LISOSSOMOS, SÍNDROME DE SJOGREN
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ABNT
MO, Ying-Qian et al. Lysosomal exocytosis of HSP70 stimulates monocytic BMP6 expression in Sjögren’s syndrome. Journal of Clinical Investigation, v. 132, n. 6, 2022Tradução . . Disponível em: https://doi.org/10.1172/JCI152780. Acesso em: 09 fev. 2026.
APA
Mo, Y. -Q., Nakamura, H., Tanaka, T., Odani, T., Perez, P., Ji, Y., et al. (2022). Lysosomal exocytosis of HSP70 stimulates monocytic BMP6 expression in Sjögren’s syndrome. Journal of Clinical Investigation, 132( 6). doi:10.1172/JCI152780
NLM
Mo Y-Q, Nakamura H, Tanaka T, Odani T, Perez P, Ji Y, French BN, Oliveira FR, Motta ACF, Ribeiro-Silva A, Rocha EM. Lysosomal exocytosis of HSP70 stimulates monocytic BMP6 expression in Sjögren’s syndrome [Internet]. Journal of Clinical Investigation. 2022 ; 132( 6):[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI152780
Vancouver
Mo Y-Q, Nakamura H, Tanaka T, Odani T, Perez P, Ji Y, French BN, Oliveira FR, Motta ACF, Ribeiro-Silva A, Rocha EM. Lysosomal exocytosis of HSP70 stimulates monocytic BMP6 expression in Sjögren’s syndrome [Internet]. Journal of Clinical Investigation. 2022 ; 132( 6):[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI152780
Artigo de periodico
Annexin A1 drives macrophage skewing to accelerate muscle regeneration through AMPK activation (2020)
McArthur, Simon; Juban, Gaëtan; Gobbetti, Thomas; Desgeorges, Thibaut; Theret, Marine; Gondin, Julien; Toller-kawahisa, Juliana Escher; Reutelingsperger, Chris; Chazaud, Bénédicte; Perretti, Mauro; Mounier, Rémi
Source: Journal of Clinical Investigation. Unidade: FMRP
Subjects: PROTEÍNAS QUINASES, ANIMAIS, RATOS, MÚSCULOS, PEPTÍDEOS
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ABNT
MCARTHUR, Simon et al. Annexin A1 drives macrophage skewing to accelerate muscle regeneration through AMPK activation. Journal of Clinical Investigation, v. 130, n. 3, p. 1156-1167, 2020Tradução . . Disponível em: https://doi.org/10.1172/JCI124635. Acesso em: 09 fev. 2026.
APA
McArthur, S., Juban, G., Gobbetti, T., Desgeorges, T., Theret, M., Gondin, J., et al. (2020). Annexin A1 drives macrophage skewing to accelerate muscle regeneration through AMPK activation. Journal of Clinical Investigation, 130( 3), 1156-1167. doi:10.1172/JCI124635
NLM
McArthur S, Juban G, Gobbetti T, Desgeorges T, Theret M, Gondin J, Toller-kawahisa JE, Reutelingsperger C, Chazaud B, Perretti M, Mounier R. Annexin A1 drives macrophage skewing to accelerate muscle regeneration through AMPK activation [Internet]. Journal of Clinical Investigation. 2020 ; 130( 3): 1156-1167.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI124635
Vancouver
McArthur S, Juban G, Gobbetti T, Desgeorges T, Theret M, Gondin J, Toller-kawahisa JE, Reutelingsperger C, Chazaud B, Perretti M, Mounier R. Annexin A1 drives macrophage skewing to accelerate muscle regeneration through AMPK activation [Internet]. Journal of Clinical Investigation. 2020 ; 130( 3): 1156-1167.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI124635
Artigo de periodico
S-nitrosoglutathione reductase-dependent PPARγ denitrosylation participates in MSC-derived adipogenesis and osteogenesis (2015)
Yenong, Cao; Gomes, Samirah Abreu; Rangel, Erika Bevilacqua; Paulino, Ellena Christina; Fonseca, Tatiana Lourdes; Li, Jinliang; Teixeira, Marilia B.; Gouveia, Cecília Helena ; Bianco, Antonio Carlos; Kapiloff, Michael S.; Balkan, Wayne; Hare, Joshua M.
Source: Journal of Clinical Investigation. Unidade: ICB
Subjects: ANATOMIA, OSTEOGÊNESE, ÁCIDOS GRAXOS, CÉLULAS-TRONCO
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ABNT
YENONG, Cao et al. S-nitrosoglutathione reductase-dependent PPARγ denitrosylation participates in MSC-derived adipogenesis and osteogenesis. Journal of Clinical Investigation, v. 125, n. 4, p. 1679-1691, 2015Tradução . . Disponível em: https://doi.org/10.1172/JCI7378. Acesso em: 09 fev. 2026.
APA
Yenong, C., Gomes, S. A., Rangel, E. B., Paulino, E. C., Fonseca, T. L., Li, J., et al. (2015). S-nitrosoglutathione reductase-dependent PPARγ denitrosylation participates in MSC-derived adipogenesis and osteogenesis. Journal of Clinical Investigation, 125( 4), 1679-1691. doi:10.1172/JCI7378
NLM
Yenong C, Gomes SA, Rangel EB, Paulino EC, Fonseca TL, Li J, Teixeira MB, Gouveia CH, Bianco AC, Kapiloff MS, Balkan W, Hare JM. S-nitrosoglutathione reductase-dependent PPARγ denitrosylation participates in MSC-derived adipogenesis and osteogenesis [Internet]. Journal of Clinical Investigation. 2015 ; 125( 4): 1679-1691.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI7378
Vancouver
Yenong C, Gomes SA, Rangel EB, Paulino EC, Fonseca TL, Li J, Teixeira MB, Gouveia CH, Bianco AC, Kapiloff MS, Balkan W, Hare JM. S-nitrosoglutathione reductase-dependent PPARγ denitrosylation participates in MSC-derived adipogenesis and osteogenesis [Internet]. Journal of Clinical Investigation. 2015 ; 125( 4): 1679-1691.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI7378
Artigo de periodico
Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency (2015)
Walter, Jolan E.; Rosen, Lindsey B.; Csomos, Krisztian; Rosenberg, Jacob M.; Mathew, Divij; Keszei, Marton; Ujhazi, Boglarka; Chen, Karin; Lee, Yu Nee; Tirosh, Irit; Dobbs, Kerry; Al-Herz, Waleed; Cowan, Morton J.; Puck, Jennifer; Blessing, Jack J.; Grimley, Michael S.; Malech, Harry; Ravin, Suk See De; Gennery, Andrew R.; Abraham, Roshini S.; Joshi, Avni Y.; Boyce, Thomas G.; Butte, Manish J.; Nadeau, Kari C.; Balboni, Imelda; Sullivan, Kathleen E.; Akhter, Javeed; Adeli, Mehdi; El-Feky, Reem A.; El-Ghoneimy, Dalia H.; Dbaibo, Ghassan; Wakim, Rima; Azzari, Chiara; Palma, Paolo; Cancrini, Caterina; Capuder, Kelly; Condino-Neto, Antonio; Costa-Carvalho, Beatriz Tavares; Oliveira, João Bosco; Roifman, Chaim; Buchbinder, David; Kumanovics, Attila; Franco, Jose Luis; Niehues, Tim; Schuetz, Catharina; Kuijpers, Taco W. ; Yee, Christina; Chou, Janet; Masaad, Michel J.; Geha, Raif; Uzel, Gulbu; Gelman, Rebecca; Holland, Steven M.; Recher, Mike; Utz, Paul J.; Browne, Sarah K.; Notarangelo, Luigi D.
Source: Journal of Clinical Investigation. Unidade: ICB
Subjects: IMUNOLOGIA, ANTICORPOS, CITOCINAS, MUTAÇÃO GENÉTICA, DOENÇAS AUTOIMUNE, DOENÇAS GENÉTICAS
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ABNT
WALTER, Jolan E. et al. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency. Journal of Clinical Investigation, v. 125, n. 11, p. 4135-4148, 2015Tradução . . Disponível em: https://doi.org/10.1172/JCI80477. Acesso em: 09 fev. 2026.
APA
Walter, J. E., Rosen, L. B., Csomos, K., Rosenberg, J. M., Mathew, D., Keszei, M., et al. (2015). Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency. Journal of Clinical Investigation, 125( 11), 4135-4148. doi:10.1172/JCI80477
NLM
Walter JE, Rosen LB, Csomos K, Rosenberg JM, Mathew D, Keszei M, Ujhazi B, Chen K, Lee YN, Tirosh I, Dobbs K, Al-Herz W, Cowan MJ, Puck J, Blessing JJ, Grimley MS, Malech H, Ravin SSD, Gennery AR, Abraham RS, Joshi AY, Boyce TG, Butte MJ, Nadeau KC, Balboni I, Sullivan KE, Akhter J, Adeli M, El-Feky RA, El-Ghoneimy DH, Dbaibo G, Wakim R, Azzari C, Palma P, Cancrini C, Capuder K, Condino-Neto A, Costa-Carvalho BT, Oliveira JB, Roifman C, Buchbinder D, Kumanovics A, Franco JL, Niehues T, Schuetz C, Kuijpers TW, Yee C, Chou J, Masaad MJ, Geha R, Uzel G, Gelman R, Holland SM, Recher M, Utz PJ, Browne SK, Notarangelo LD. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency [Internet]. Journal of Clinical Investigation. 2015 ; 125( 11): 4135-4148.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI80477
Vancouver
Walter JE, Rosen LB, Csomos K, Rosenberg JM, Mathew D, Keszei M, Ujhazi B, Chen K, Lee YN, Tirosh I, Dobbs K, Al-Herz W, Cowan MJ, Puck J, Blessing JJ, Grimley MS, Malech H, Ravin SSD, Gennery AR, Abraham RS, Joshi AY, Boyce TG, Butte MJ, Nadeau KC, Balboni I, Sullivan KE, Akhter J, Adeli M, El-Feky RA, El-Ghoneimy DH, Dbaibo G, Wakim R, Azzari C, Palma P, Cancrini C, Capuder K, Condino-Neto A, Costa-Carvalho BT, Oliveira JB, Roifman C, Buchbinder D, Kumanovics A, Franco JL, Niehues T, Schuetz C, Kuijpers TW, Yee C, Chou J, Masaad MJ, Geha R, Uzel G, Gelman R, Holland SM, Recher M, Utz PJ, Browne SK, Notarangelo LD. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency [Internet]. Journal of Clinical Investigation. 2015 ; 125( 11): 4135-4148.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI80477
Artigo de periodico
Defective telomere elongation and hematopoiesis from telomerase-mutant aplastic anemia iPSCs (2013)
Winkler, Thomas; Hong, So Gun; Decker, Jake E.; Morgan, Mary J.; Wu, Chuafeng; Hughes, William M.; Yang, Yanqin; Wangsa, Danny; Padilla-Nash, Hesed M.; Ried, Thomas; Young, Neal S.; Dunbar, Cynthia E.; Calado, Rodrigo Tocantins
Source: Journal of Clinical Investigation. Unidade: FMRP
Subjects: ANEMIA, DIFERENCIAÇÃO CELULAR, FIBROBLASTOS (CITOLOGIA), CÉLULAS-TRONCO, LEUCÓCITOS
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ABNT
WINKLER, Thomas et al. Defective telomere elongation and hematopoiesis from telomerase-mutant aplastic anemia iPSCs. Journal of Clinical Investigation, v. 123, n. 5, p. 1952-1963, 2013Tradução . . Disponível em: https://doi.org/10.1172/JCI67146. Acesso em: 09 fev. 2026.
APA
Winkler, T., Hong, S. G., Decker, J. E., Morgan, M. J., Wu, C., Hughes, W. M., et al. (2013). Defective telomere elongation and hematopoiesis from telomerase-mutant aplastic anemia iPSCs. Journal of Clinical Investigation, 123( 5), 1952-1963. doi:10.1172/JCI67146
NLM
Winkler T, Hong SG, Decker JE, Morgan MJ, Wu C, Hughes WM, Yang Y, Wangsa D, Padilla-Nash HM, Ried T, Young NS, Dunbar CE, Calado RT. Defective telomere elongation and hematopoiesis from telomerase-mutant aplastic anemia iPSCs [Internet]. Journal of Clinical Investigation. 2013 ; 123( 5): 1952-1963.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI67146
Vancouver
Winkler T, Hong SG, Decker JE, Morgan MJ, Wu C, Hughes WM, Yang Y, Wangsa D, Padilla-Nash HM, Ried T, Young NS, Dunbar CE, Calado RT. Defective telomere elongation and hematopoiesis from telomerase-mutant aplastic anemia iPSCs [Internet]. Journal of Clinical Investigation. 2013 ; 123( 5): 1952-1963.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/JCI67146
Artigo de periodico
Expression of an activating mutation in the gene encoding the KATP channel subunit Kir6.2 in mouse pancreatic ß cells recapitulates neonatal diabetes (2009)
Girard, Christophe A.; Wunderlich, F. Thomas; Shimomura, Kenju; Collins, Stephan; Kaizik, Stephan; Proks, Peter; Abdulkader, Fernando ; Clark, Anne; Ball, Vicky; Zubcevic, Lejla; Bentley, Liz; Clark, Rebecca; Church, Chris; Hugill, Alison; Galvanovskis, Juris; Cox, Roger; Rorsman, Patrik; Brüning, Jens C.; Ashcroft, Frances M.
Source: Journal of Clinical Investigation. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
GIRARD, Christophe A. et al. Expression of an activating mutation in the gene encoding the KATP channel subunit Kir6.2 in mouse pancreatic ß cells recapitulates neonatal diabetes. Journal of Clinical Investigation, v. 119, n. 1, p. 80-90, 2009Tradução . . Disponível em: https://doi.org/10.1172/jci35772. Acesso em: 09 fev. 2026.
APA
Girard, C. A., Wunderlich, F. T., Shimomura, K., Collins, S., Kaizik, S., Proks, P., et al. (2009). Expression of an activating mutation in the gene encoding the KATP channel subunit Kir6.2 in mouse pancreatic ß cells recapitulates neonatal diabetes. Journal of Clinical Investigation, 119( 1), 80-90. doi:10.1172/jci35772
NLM
Girard CA, Wunderlich FT, Shimomura K, Collins S, Kaizik S, Proks P, Abdulkader F, Clark A, Ball V, Zubcevic L, Bentley L, Clark R, Church C, Hugill A, Galvanovskis J, Cox R, Rorsman P, Brüning JC, Ashcroft FM. Expression of an activating mutation in the gene encoding the KATP channel subunit Kir6.2 in mouse pancreatic ß cells recapitulates neonatal diabetes [Internet]. Journal of Clinical Investigation. 2009 ; 119( 1): 80-90.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci35772
Vancouver
Girard CA, Wunderlich FT, Shimomura K, Collins S, Kaizik S, Proks P, Abdulkader F, Clark A, Ball V, Zubcevic L, Bentley L, Clark R, Church C, Hugill A, Galvanovskis J, Cox R, Rorsman P, Brüning JC, Ashcroft FM. Expression of an activating mutation in the gene encoding the KATP channel subunit Kir6.2 in mouse pancreatic ß cells recapitulates neonatal diabetes [Internet]. Journal of Clinical Investigation. 2009 ; 119( 1): 80-90.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci35772
Artigo de periodico
Oral tolerance in the absence of naturally occurring Tregs (2005)
Mucida, Daniel; Kutchukhidze, Nino; Erazo, Agustin; Russo, Momtchilo; Lafaille, Juan J.; Lafaille, Maria A. Curotto de
Source: Journal of Clinical Investigation. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
MUCIDA, Daniel et al. Oral tolerance in the absence of naturally occurring Tregs. Journal of Clinical Investigation, v. 115, n. 7, p. 1923-1933, 2005Tradução . . Disponível em: https://doi.org/10.1172/jci24487. Acesso em: 09 fev. 2026.
APA
Mucida, D., Kutchukhidze, N., Erazo, A., Russo, M., Lafaille, J. J., & Lafaille, M. A. C. de. (2005). Oral tolerance in the absence of naturally occurring Tregs. Journal of Clinical Investigation, 115( 7), 1923-1933. doi:10.1172/jci24487
NLM
Mucida D, Kutchukhidze N, Erazo A, Russo M, Lafaille JJ, Lafaille MAC de. Oral tolerance in the absence of naturally occurring Tregs [Internet]. Journal of Clinical Investigation. 2005 ; 115( 7): 1923-1933.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci24487
Vancouver
Mucida D, Kutchukhidze N, Erazo A, Russo M, Lafaille JJ, Lafaille MAC de. Oral tolerance in the absence of naturally occurring Tregs [Internet]. Journal of Clinical Investigation. 2005 ; 115( 7): 1923-1933.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci24487
Artigo de periodico
A homozygous mutation in HESX1 is associated with evolving hyppopituitarism due to impaired repressor-corepressor interaction (2003)
Carvalho, Luciani R.; Woods, Kathryn S.; Mendonça, Berenice B.; Marcal, Nathalie; Zamparini, Andrea L.; Stifani, Stefano; Brickman, Joshua M.; Arnhold, Ivo J. P.; Dattani, Mehul T.
Source: Journal of Clinical Investigation. Unidade: FM
Assunto: GLÂNDULA PITUITÁRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Luciani R. et al. A homozygous mutation in HESX1 is associated with evolving hyppopituitarism due to impaired repressor-corepressor interaction. Journal of Clinical Investigation, v. 112, n. 8, p. 1192-1201, 2003Tradução . . Disponível em: https://doi.org/10.1172/jci18589. Acesso em: 09 fev. 2026.
APA
Carvalho, L. R., Woods, K. S., Mendonça, B. B., Marcal, N., Zamparini, A. L., Stifani, S., et al. (2003). A homozygous mutation in HESX1 is associated with evolving hyppopituitarism due to impaired repressor-corepressor interaction. Journal of Clinical Investigation, 112( 8), 1192-1201. doi:10.1172/jci18589
NLM
Carvalho LR, Woods KS, Mendonça BB, Marcal N, Zamparini AL, Stifani S, Brickman JM, Arnhold IJP, Dattani MT. A homozygous mutation in HESX1 is associated with evolving hyppopituitarism due to impaired repressor-corepressor interaction [Internet]. Journal of Clinical Investigation. 2003 ; 112( 8): 1192-1201.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci18589
Vancouver
Carvalho LR, Woods KS, Mendonça BB, Marcal N, Zamparini AL, Stifani S, Brickman JM, Arnhold IJP, Dattani MT. A homozygous mutation in HESX1 is associated with evolving hyppopituitarism due to impaired repressor-corepressor interaction [Internet]. Journal of Clinical Investigation. 2003 ; 112( 8): 1192-1201.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci18589
Artigo de periodico
The type 2 iodothyronine deiodinase is essential for adaptative thermogenesis in brown adipose tissue (2001)
Jesus, L A F; Carvalho, S D; Ribeiro, Miriam Oliveira; Schneider, M; Kim , S W; Harney, J W; Larsen, P R; Bianco, Antônio Carlos
Source: Journal of Clinical Investigation. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JESUS, L A F et al. The type 2 iodothyronine deiodinase is essential for adaptative thermogenesis in brown adipose tissue. Journal of Clinical Investigation, v. 108, p. 1379-1385, 2001Tradução . . Disponível em: https://doi.org/10.1172/jci13803. Acesso em: 09 fev. 2026.
APA
Jesus, L. A. F., Carvalho, S. D., Ribeiro, M. O., Schneider, M., Kim , S. W., Harney, J. W., et al. (2001). The type 2 iodothyronine deiodinase is essential for adaptative thermogenesis in brown adipose tissue. Journal of Clinical Investigation, 108, 1379-1385. doi:10.1172/jci13803
NLM
Jesus LAF, Carvalho SD, Ribeiro MO, Schneider M, Kim SW, Harney JW, Larsen PR, Bianco AC. The type 2 iodothyronine deiodinase is essential for adaptative thermogenesis in brown adipose tissue [Internet]. Journal of Clinical Investigation. 2001 ; 108 1379-1385.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci13803
Vancouver
Jesus LAF, Carvalho SD, Ribeiro MO, Schneider M, Kim SW, Harney JW, Larsen PR, Bianco AC. The type 2 iodothyronine deiodinase is essential for adaptative thermogenesis in brown adipose tissue [Internet]. Journal of Clinical Investigation. 2001 ; 108 1379-1385.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci13803
Artigo de periodico
Rho proteins play a critical role in cell migration during the early phase of mucosal restitution (1997)
Santos, Marinilce Fagundes dos ; McCormack, S A; Guo, Z; Okolicany, J; Zheng, Y; Johnson, L R; Tigyi, G
Source: Journal of Clinical Investigation. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Marinilce Fagundes dos et al. Rho proteins play a critical role in cell migration during the early phase of mucosal restitution. Journal of Clinical Investigation, v. 100, n. 1, p. 216-225, 1997Tradução . . Disponível em: https://doi.org/10.1172/jci119515. Acesso em: 09 fev. 2026.
APA
Santos, M. F. dos, McCormack, S. A., Guo, Z., Okolicany, J., Zheng, Y., Johnson, L. R., & Tigyi, G. (1997). Rho proteins play a critical role in cell migration during the early phase of mucosal restitution. Journal of Clinical Investigation, 100( 1), 216-225. doi:10.1172/jci119515
NLM
Santos MF dos, McCormack SA, Guo Z, Okolicany J, Zheng Y, Johnson LR, Tigyi G. Rho proteins play a critical role in cell migration during the early phase of mucosal restitution [Internet]. Journal of Clinical Investigation. 1997 ; 100( 1): 216-225.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci119515
Vancouver
Santos MF dos, McCormack SA, Guo Z, Okolicany J, Zheng Y, Johnson LR, Tigyi G. Rho proteins play a critical role in cell migration during the early phase of mucosal restitution [Internet]. Journal of Clinical Investigation. 1997 ; 100( 1): 216-225.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci119515
Artigo de periodico
Circulating, biologically inactive thyrotropin caused by a mutation in the beta subunit gene (1996)
Medeiros Neto, Geraldo Antônio; Herodotou, D T; Rajan, S; Kommareddi, S; Lacerda, L; Sandrini, R; Boguszewski, Margaret C. S; Hollenberg, A N; Radovick, S; Wondisford, F E
Source: Journal of Clinical Investigation. Unidade: FM
Assunto: MEDICINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MEDEIROS NETO, Geraldo Antônio et al. Circulating, biologically inactive thyrotropin caused by a mutation in the beta subunit gene. Journal of Clinical Investigation, v. 97, n. 5 , p. 1250-6, 1996Tradução . . Disponível em: https://doi.org/10.1172/jci118540. Acesso em: 09 fev. 2026.
APA
Medeiros Neto, G. A., Herodotou, D. T., Rajan, S., Kommareddi, S., Lacerda, L., Sandrini, R., et al. (1996). Circulating, biologically inactive thyrotropin caused by a mutation in the beta subunit gene. Journal of Clinical Investigation, 97( 5 ), 1250-6. doi:10.1172/jci118540
NLM
Medeiros Neto GA, Herodotou DT, Rajan S, Kommareddi S, Lacerda L, Sandrini R, Boguszewski MCS, Hollenberg AN, Radovick S, Wondisford FE. Circulating, biologically inactive thyrotropin caused by a mutation in the beta subunit gene [Internet]. Journal of Clinical Investigation. 1996 ;97( 5 ): 1250-6.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci118540
Vancouver
Medeiros Neto GA, Herodotou DT, Rajan S, Kommareddi S, Lacerda L, Sandrini R, Boguszewski MCS, Hollenberg AN, Radovick S, Wondisford FE. Circulating, biologically inactive thyrotropin caused by a mutation in the beta subunit gene [Internet]. Journal of Clinical Investigation. 1996 ;97( 5 ): 1250-6.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci118540
Artigo de periodico
Cellular mechanism of the conduction abnormalities induced by serum from anti-ro / ssa-positive patients in rabbit hearts (1994)
Garcia, S; Nascimento, J; Bonfá, Eloísa Silva Dutra de Oliveira; Levy, R A; Oliveira, S. F.; Tavares, A V; Carvalho, A. C. C.
Source: Journal of Clinical Investigation. Unidade: FM
Assunto: REUMATOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GARCIA, S et al. Cellular mechanism of the conduction abnormalities induced by serum from anti-ro / ssa-positive patients in rabbit hearts. Journal of Clinical Investigation, v. 93, n. 2 , p. 718-24, 1994Tradução . . Disponível em: https://doi.org/10.1172/jci117025. Acesso em: 09 fev. 2026.
APA
Garcia, S., Nascimento, J., Bonfá, E. S. D. de O., Levy, R. A., Oliveira, S. F., Tavares, A. V., & Carvalho, A. C. C. (1994). Cellular mechanism of the conduction abnormalities induced by serum from anti-ro / ssa-positive patients in rabbit hearts. Journal of Clinical Investigation, 93( 2 ), 718-24. doi:10.1172/jci117025
NLM
Garcia S, Nascimento J, Bonfá ESD de O, Levy RA, Oliveira SF, Tavares AV, Carvalho ACC. Cellular mechanism of the conduction abnormalities induced by serum from anti-ro / ssa-positive patients in rabbit hearts [Internet]. Journal of Clinical Investigation. 1994 ;93( 2 ): 718-24.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci117025
Vancouver
Garcia S, Nascimento J, Bonfá ESD de O, Levy RA, Oliveira SF, Tavares AV, Carvalho ACC. Cellular mechanism of the conduction abnormalities induced by serum from anti-ro / ssa-positive patients in rabbit hearts [Internet]. Journal of Clinical Investigation. 1994 ;93( 2 ): 718-24.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci117025
Artigo de periodico
Peptidase modulation of vasoactive intestinal peptide pulmonary relaxation in tracheal superfused guinea pig lungs (1993)
Lilly, C M; Martins, M. A.; Drazen, J M
Source: Journal of Clinical Investigation. Unidade: FM
Assunto: PATOLOGIA CLÍNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LILLY, C M e MARTINS, M. A. e DRAZEN, J M. Peptidase modulation of vasoactive intestinal peptide pulmonary relaxation in tracheal superfused guinea pig lungs. Journal of Clinical Investigation, v. 91, n. ja 1993, p. 235-43, 1993Tradução . . Disponível em: https://doi.org/10.1172/jci116176. Acesso em: 09 fev. 2026.
APA
Lilly, C. M., Martins, M. A., & Drazen, J. M. (1993). Peptidase modulation of vasoactive intestinal peptide pulmonary relaxation in tracheal superfused guinea pig lungs. Journal of Clinical Investigation, 91( ja 1993), 235-43. doi:10.1172/jci116176
NLM
Lilly CM, Martins MA, Drazen JM. Peptidase modulation of vasoactive intestinal peptide pulmonary relaxation in tracheal superfused guinea pig lungs [Internet]. Journal of Clinical Investigation. 1993 ;91( ja 1993): 235-43.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci116176
Vancouver
Lilly CM, Martins MA, Drazen JM. Peptidase modulation of vasoactive intestinal peptide pulmonary relaxation in tracheal superfused guinea pig lungs [Internet]. Journal of Clinical Investigation. 1993 ;91( ja 1993): 235-43.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci116176
Artigo de periodico
Renal bicarbonate reabsorption in the rat.Iv bicarbonate transport mechanisms in the early and late distal tubule (1993)
Wang, T; Malnic, Gerhard; Giebich, G; Chan, Y L
Source: Journal of Clinical Investigation. Unidade: ICB
Subjects: BIOFÍSICA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WANG, T et al. Renal bicarbonate reabsorption in the rat.Iv bicarbonate transport mechanisms in the early and late distal tubule. Journal of Clinical Investigation, v. 91, p. 2776-84, 1993Tradução . . Disponível em: https://doi.org/10.1172/jci116519. Acesso em: 09 fev. 2026.
APA
Wang, T., Malnic, G., Giebich, G., & Chan, Y. L. (1993). Renal bicarbonate reabsorption in the rat.Iv bicarbonate transport mechanisms in the early and late distal tubule. Journal of Clinical Investigation, 91, 2776-84. doi:10.1172/jci116519
NLM
Wang T, Malnic G, Giebich G, Chan YL. Renal bicarbonate reabsorption in the rat.Iv bicarbonate transport mechanisms in the early and late distal tubule [Internet]. Journal of Clinical Investigation. 1993 ;91 2776-84.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci116519
Vancouver
Wang T, Malnic G, Giebich G, Chan YL. Renal bicarbonate reabsorption in the rat.Iv bicarbonate transport mechanisms in the early and late distal tubule [Internet]. Journal of Clinical Investigation. 1993 ;91 2776-84.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci116519
Artigo de periodico
Renal bicarbonate reabsorption in the rat. Iii. Distal tubule perfusion study of load dependence and bicarbonate permeability (1989)
Chan, Y. L.; Malnic, Gerhard; Giebisch, Gerhard
Source: Journal of Clinical Investigation. Unidade: ICB
Subjects: RATOS, TUBULOS RENAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHAN, Y. L. e MALNIC, Gerhard e GIEBISCH, Gerhard. Renal bicarbonate reabsorption in the rat. Iii. Distal tubule perfusion study of load dependence and bicarbonate permeability. Journal of Clinical Investigation, v. 84, p. 931-938, 1989Tradução . . Disponível em: https://doi.org/10.1172/jci114255. Acesso em: 09 fev. 2026.
APA
Chan, Y. L., Malnic, G., & Giebisch, G. (1989). Renal bicarbonate reabsorption in the rat. Iii. Distal tubule perfusion study of load dependence and bicarbonate permeability. Journal of Clinical Investigation, 84, 931-938. doi:10.1172/jci114255
NLM
Chan YL, Malnic G, Giebisch G. Renal bicarbonate reabsorption in the rat. Iii. Distal tubule perfusion study of load dependence and bicarbonate permeability [Internet]. Journal of Clinical Investigation. 1989 ; 84 931-938.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci114255
Vancouver
Chan YL, Malnic G, Giebisch G. Renal bicarbonate reabsorption in the rat. Iii. Distal tubule perfusion study of load dependence and bicarbonate permeability [Internet]. Journal of Clinical Investigation. 1989 ; 84 931-938.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci114255
Artigo de periodico
Tumor necrosis factor can induced fever in rats without activating protein breakdown in muscle or lipolysis in adipose tissue (1988)
Kettelhut, Isis do Carmo
Source: Journal of Clinical Investigation. Unidade: FMRP
Assunto: BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KETTELHUT, Isis do Carmo. Tumor necrosis factor can induced fever in rats without activating protein breakdown in muscle or lipolysis in adipose tissue. Journal of Clinical Investigation, v. 81, n. 5 , p. 1384-9, 1988Tradução . . Disponível em: https://doi.org/10.1172/jci113467. Acesso em: 09 fev. 2026.
APA
Kettelhut, I. do C. (1988). Tumor necrosis factor can induced fever in rats without activating protein breakdown in muscle or lipolysis in adipose tissue. Journal of Clinical Investigation, 81( 5 ), 1384-9. doi:10.1172/jci113467
NLM
Kettelhut I do C. Tumor necrosis factor can induced fever in rats without activating protein breakdown in muscle or lipolysis in adipose tissue [Internet]. Journal of Clinical Investigation. 1988 ;81( 5 ): 1384-9.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci113467
Vancouver
Kettelhut I do C. Tumor necrosis factor can induced fever in rats without activating protein breakdown in muscle or lipolysis in adipose tissue [Internet]. Journal of Clinical Investigation. 1988 ;81( 5 ): 1384-9.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci113467
Artigo de periodico
Activation of protein breakdown and postageandin - e2 production in rat skeletal muscle in fever is signaled by a macrophage , product distinct from interlenkin-1 or other known monokinase (1988)
Kettelhut, Isis do Carmo
Source: Journal of Clinical Investigation. Unidade: FMRP
Assunto: BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KETTELHUT, Isis do Carmo. Activation of protein breakdown and postageandin - e2 production in rat skeletal muscle in fever is signaled by a macrophage , product distinct from interlenkin-1 or other known monokinase. Journal of Clinical Investigation, v. 81, n. 5 , p. 1378-83, 1988Tradução . . Disponível em: https://doi.org/10.1172/jci113466. Acesso em: 09 fev. 2026.
APA
Kettelhut, I. do C. (1988). Activation of protein breakdown and postageandin - e2 production in rat skeletal muscle in fever is signaled by a macrophage , product distinct from interlenkin-1 or other known monokinase. Journal of Clinical Investigation, 81( 5 ), 1378-83. doi:10.1172/jci113466
NLM
Kettelhut I do C. Activation of protein breakdown and postageandin - e2 production in rat skeletal muscle in fever is signaled by a macrophage , product distinct from interlenkin-1 or other known monokinase [Internet]. Journal of Clinical Investigation. 1988 ;81( 5 ): 1378-83.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci113466
Vancouver
Kettelhut I do C. Activation of protein breakdown and postageandin - e2 production in rat skeletal muscle in fever is signaled by a macrophage , product distinct from interlenkin-1 or other known monokinase [Internet]. Journal of Clinical Investigation. 1988 ;81( 5 ): 1378-83.[citado 2026 fev. 09 ] Available from: https://doi.org/10.1172/jci113466

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