ReP USP - Resultado da busca

Artigo de periodico
Osteoprotective Effects of IL-33/ST2 link to osteoclast Apoptosis (2015)
Lima, Izabella L. A.; Macari, Soraia; Madeira, Mila F. M.; Rodrigues, Letícia F. D.; Colavite, Priscila M.; Garlet, Gustavo Pompermaier; Soriani, Frederico M.; Teixeira, Mauro M.; Fukada, Sandra Y.; Silva, Tarcília A.
Fonte: American Journal of Pathology. Unidade: FOB
Assuntos: APOPTOSE, INTERLEUCINA 1, OSTEOCLASTO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Izabella L. A. et al. Osteoprotective Effects of IL-33/ST2 link to osteoclast Apoptosis. American Journal of Pathology, v. 185, n. 12, p. 3338-3348, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ajpath.2015.08.013. Acesso em: 26 out. 2025.
APA
Lima, I. L. A., Macari, S., Madeira, M. F. M., Rodrigues, L. F. D., Colavite, P. M., Garlet, G. P., et al. (2015). Osteoprotective Effects of IL-33/ST2 link to osteoclast Apoptosis. American Journal of Pathology, 185( 12), 3338-3348. doi:10.1016/j.ajpath.2015.08.013
NLM
Lima ILA, Macari S, Madeira MFM, Rodrigues LFD, Colavite PM, Garlet GP, Soriani FM, Teixeira MM, Fukada SY, Silva TA. Osteoprotective Effects of IL-33/ST2 link to osteoclast Apoptosis [Internet]. American Journal of Pathology. 2015 ; 185( 12): 3338-3348.[citado 2025 out. 26 ] Available from: https://doi.org/10.1016/j.ajpath.2015.08.013
Vancouver
Lima ILA, Macari S, Madeira MFM, Rodrigues LFD, Colavite PM, Garlet GP, Soriani FM, Teixeira MM, Fukada SY, Silva TA. Osteoprotective Effects of IL-33/ST2 link to osteoclast Apoptosis [Internet]. American Journal of Pathology. 2015 ; 185( 12): 3338-3348.[citado 2025 out. 26 ] Available from: https://doi.org/10.1016/j.ajpath.2015.08.013
Artigo de periodico
Inflammasome activation is reactive oxygen species dependent and mediates irinotecan-induced mucositis through IL-1β and IL-18 in mice (2014)
Arifa, Raquel D. N.; Madeira, Mila F. M.; Paula, Talles P. de; Lima, Renata L.; Tavares, Livia D.; Menezes-Garcia, Zélia; Fagundes, Caio T.; Rachid, Milene A.; Ryffel, Bernhard; Zamboni, Dario Simões; Teixeira, Mauro M.; Souza, Danielle G.
Fonte: American Journal of Pathology. Unidade: FMRP
Assuntos: INFLAMAÇÃO (PATOLOGIA), MUCOSA ESPECIALIZADA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARIFA, Raquel D. N. et al. Inflammasome activation is reactive oxygen species dependent and mediates irinotecan-induced mucositis through IL-1β and IL-18 in mice. American Journal of Pathology, v. 184, n. 7, p. 2023-2034, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.ajpath.2014.03.012. Acesso em: 26 out. 2025.
APA
Arifa, R. D. N., Madeira, M. F. M., Paula, T. P. de, Lima, R. L., Tavares, L. D., Menezes-Garcia, Z., et al. (2014). Inflammasome activation is reactive oxygen species dependent and mediates irinotecan-induced mucositis through IL-1β and IL-18 in mice. American Journal of Pathology, 184( 7), 2023-2034. doi:10.1016/j.ajpath.2014.03.012
NLM
Arifa RDN, Madeira MFM, Paula TP de, Lima RL, Tavares LD, Menezes-Garcia Z, Fagundes CT, Rachid MA, Ryffel B, Zamboni DS, Teixeira MM, Souza DG. Inflammasome activation is reactive oxygen species dependent and mediates irinotecan-induced mucositis through IL-1β and IL-18 in mice [Internet]. American Journal of Pathology. 2014 ; 184( 7): 2023-2034.[citado 2025 out. 26 ] Available from: https://doi.org/10.1016/j.ajpath.2014.03.012
Vancouver
Arifa RDN, Madeira MFM, Paula TP de, Lima RL, Tavares LD, Menezes-Garcia Z, Fagundes CT, Rachid MA, Ryffel B, Zamboni DS, Teixeira MM, Souza DG. Inflammasome activation is reactive oxygen species dependent and mediates irinotecan-induced mucositis through IL-1β and IL-18 in mice [Internet]. American Journal of Pathology. 2014 ; 184( 7): 2023-2034.[citado 2025 out. 26 ] Available from: https://doi.org/10.1016/j.ajpath.2014.03.012
Artigo de periodico
The autoimmune regulator (aire), which is defective in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy patients, is expressed in human epidermal and follicular keratinocytes and associates with the intermediate filament protein cytokeratin 17 (2011)
Kumar, Vipul; Pedroza, Luis A.; Mace, Emily M. ; Seeholzer, Steven; Cotsarelis, George; Condino-Neto, Antonio; Payne, Aimee S.; Orange, Jordan S.
Fonte: American Journal of Pathology. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KUMAR, Vipul et al. The autoimmune regulator (aire), which is defective in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy patients, is expressed in human epidermal and follicular keratinocytes and associates with the intermediate filament protein cytokeratin 17. American Journal of Pathology, v. 178, n. 3, p. 983-988, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.ajpath.2010.12.007. Acesso em: 26 out. 2025.
APA
Kumar, V., Pedroza, L. A., Mace, E. M., Seeholzer, S., Cotsarelis, G., Condino-Neto, A., et al. (2011). The autoimmune regulator (aire), which is defective in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy patients, is expressed in human epidermal and follicular keratinocytes and associates with the intermediate filament protein cytokeratin 17. American Journal of Pathology, 178( 3), 983-988. doi:10.1016/j.ajpath.2010.12.007
NLM
Kumar V, Pedroza LA, Mace EM, Seeholzer S, Cotsarelis G, Condino-Neto A, Payne AS, Orange JS. The autoimmune regulator (aire), which is defective in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy patients, is expressed in human epidermal and follicular keratinocytes and associates with the intermediate filament protein cytokeratin 17 [Internet]. American Journal of Pathology. 2011 ; 178( 3): 983-988.[citado 2025 out. 26 ] Available from: https://doi.org/10.1016/j.ajpath.2010.12.007
Vancouver
Kumar V, Pedroza LA, Mace EM, Seeholzer S, Cotsarelis G, Condino-Neto A, Payne AS, Orange JS. The autoimmune regulator (aire), which is defective in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy patients, is expressed in human epidermal and follicular keratinocytes and associates with the intermediate filament protein cytokeratin 17 [Internet]. American Journal of Pathology. 2011 ; 178( 3): 983-988.[citado 2025 out. 26 ] Available from: https://doi.org/10.1016/j.ajpath.2010.12.007
Artigo de periodico
Therapeutic administration of K'M POT.+' lectin protects mice against Paracoccidioides brasiliensis infection via interleukin-12 production in a toll-like receptor 2-dependent mechanism (2008)
Coltri, Kely C.; Oliveira, Leandro L.; Pinzan, Camila F.; Vendrusculo, Patrícia E.; Martinez, Roberto; Goldman, Maria Helena; Panunto-Castelo, Ademilson; Roque-Barreira, Maria Cristina
Fonte: American Journal of Pathology. Unidades: FMRP, FFCLRP, EERP
Assuntos: PARACOCCIDIOIDES BRASILIENSIS, GRANULOMA, DOENÇAS INFECCIOSAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COLTRI, Kely C. et al. Therapeutic administration of K'M POT.+' lectin protects mice against Paracoccidioides brasiliensis infection via interleukin-12 production in a toll-like receptor 2-dependent mechanism. American Journal of Pathology, v. 173, n. 2, p. 423-432, 2008Tradução . . Acesso em: 26 out. 2025.
APA
Coltri, K. C., Oliveira, L. L., Pinzan, C. F., Vendrusculo, P. E., Martinez, R., Goldman, M. H., et al. (2008). Therapeutic administration of K'M POT.+' lectin protects mice against Paracoccidioides brasiliensis infection via interleukin-12 production in a toll-like receptor 2-dependent mechanism. American Journal of Pathology, 173( 2), 423-432.
NLM
Coltri KC, Oliveira LL, Pinzan CF, Vendrusculo PE, Martinez R, Goldman MH, Panunto-Castelo A, Roque-Barreira MC. Therapeutic administration of K'M POT.+' lectin protects mice against Paracoccidioides brasiliensis infection via interleukin-12 production in a toll-like receptor 2-dependent mechanism. American Journal of Pathology. 2008 ; 173( 2): 423-432.[citado 2025 out. 26 ]
Vancouver
Coltri KC, Oliveira LL, Pinzan CF, Vendrusculo PE, Martinez R, Goldman MH, Panunto-Castelo A, Roque-Barreira MC. Therapeutic administration of K'M POT.+' lectin protects mice against Paracoccidioides brasiliensis infection via interleukin-12 production in a toll-like receptor 2-dependent mechanism. American Journal of Pathology. 2008 ; 173( 2): 423-432.[citado 2025 out. 26 ]
Artigo de periodico
Biliary and Pancreatic Dysgenesis in Mice Harboring a Mutation in Pkhd1 (2008)
Gallagher,Anna-Rachel; Esquivel, Ernie L.; Briere, Tiffany S.; Tian, Xin; Mitobe, Michihiro; Menezes, Luis F.; Markowitz, Glen S.; Jain, Dhanpat; Onuchic, Luiz F.; Somlo, Stefan
Fonte: American Journal of Pathology. Unidade: FM
Assuntos: CISTOS, RIM (PATOLOGIA, DOENÇAS HEREDITÁRIAS (GENÉTICA), MUTAÇÃO GENÉTICA, MODELOS ANIMAIS, CAMUNDONGOS, FIBROSE CÍSTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GALLAGHER, Anna-Rachel et al. Biliary and Pancreatic Dysgenesis in Mice Harboring a Mutation in Pkhd1. American Journal of Pathology, v. 172, n. 2, p. 417-429, 2008Tradução . . Disponível em: https://doi.org/10.2353/ajpath.2008.070381. Acesso em: 26 out. 2025.
APA
Gallagher, A. -R., Esquivel, E. L., Briere, T. S., Tian, X., Mitobe, M., Menezes, L. F., et al. (2008). Biliary and Pancreatic Dysgenesis in Mice Harboring a Mutation in Pkhd1. American Journal of Pathology, 172( 2), 417-429. doi:10.2353/ajpath.2008.070381
NLM
Gallagher A-R, Esquivel EL, Briere TS, Tian X, Mitobe M, Menezes LF, Markowitz GS, Jain D, Onuchic LF, Somlo S. Biliary and Pancreatic Dysgenesis in Mice Harboring a Mutation in Pkhd1 [Internet]. American Journal of Pathology. 2008 ; 172( 2): 417-429.[citado 2025 out. 26 ] Available from: https://doi.org/10.2353/ajpath.2008.070381
Vancouver
Gallagher A-R, Esquivel EL, Briere TS, Tian X, Mitobe M, Menezes LF, Markowitz GS, Jain D, Onuchic LF, Somlo S. Biliary and Pancreatic Dysgenesis in Mice Harboring a Mutation in Pkhd1 [Internet]. American Journal of Pathology. 2008 ; 172( 2): 417-429.[citado 2025 out. 26 ] Available from: https://doi.org/10.2353/ajpath.2008.070381
Artigo de periodico
CCR2 receptor is essential to activate microbicidal mechanisms to control Toxoplasma gondii infection in the central nervous system (2008)
Benevides, Luciana; Milanezi, Cristiane Maria; Yamauchi, Lucy Megumi; Benjanmim, Cláudia Farias; Silva, João Santana da; Silva, Neide Maria
Fonte: American Journal of Pathology. Unidade: FMRP
Assuntos: TOXOPLASMA GONDII, SISTEMA NERVOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BENEVIDES, Luciana et al. CCR2 receptor is essential to activate microbicidal mechanisms to control Toxoplasma gondii infection in the central nervous system. American Journal of Pathology, v. 173, n. 3, p. 741-751, 2008Tradução . . Acesso em: 26 out. 2025.
APA
Benevides, L., Milanezi, C. M., Yamauchi, L. M., Benjanmim, C. F., Silva, J. S. da, & Silva, N. M. (2008). CCR2 receptor is essential to activate microbicidal mechanisms to control Toxoplasma gondii infection in the central nervous system. American Journal of Pathology, 173( 3), 741-751.
NLM
Benevides L, Milanezi CM, Yamauchi LM, Benjanmim CF, Silva JS da, Silva NM. CCR2 receptor is essential to activate microbicidal mechanisms to control Toxoplasma gondii infection in the central nervous system. American Journal of Pathology. 2008 ; 173( 3): 741-751.[citado 2025 out. 26 ]
Vancouver
Benevides L, Milanezi CM, Yamauchi LM, Benjanmim CF, Silva JS da, Silva NM. CCR2 receptor is essential to activate microbicidal mechanisms to control Toxoplasma gondii infection in the central nervous system. American Journal of Pathology. 2008 ; 173( 3): 741-751.[citado 2025 out. 26 ]
Artigo de periodico
Macrophage migration inhibitory factor is up-regulated in human first-trimester placenta stimulated by soluble antigen of Toxoplasma gondii, resulting in increased monocyte adhesion on villous explants (2008)
Ferro, Eloisa Amália Vieira; Mineo, José Roberto; Letta, Francesca; Bechi, Nicoletta; Romagnoli, Roberta; Silva, Deise Aparecida Oliveira; Sorda, Giuseppina; Bevilacqua, Estela Maris Andrade Forell ; Paulesu, Luana Ricci
Fonte: American Journal of Pathology. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERRO, Eloisa Amália Vieira et al. Macrophage migration inhibitory factor is up-regulated in human first-trimester placenta stimulated by soluble antigen of Toxoplasma gondii, resulting in increased monocyte adhesion on villous explants. American Journal of Pathology, v. 172, n. 1, p. 50-58, 2008Tradução . . Acesso em: 26 out. 2025.
APA
Ferro, E. A. V., Mineo, J. R., Letta, F., Bechi, N., Romagnoli, R., Silva, D. A. O., et al. (2008). Macrophage migration inhibitory factor is up-regulated in human first-trimester placenta stimulated by soluble antigen of Toxoplasma gondii, resulting in increased monocyte adhesion on villous explants. American Journal of Pathology, 172( 1), 50-58.
NLM
Ferro EAV, Mineo JR, Letta F, Bechi N, Romagnoli R, Silva DAO, Sorda G, Bevilacqua EMAF, Paulesu LR. Macrophage migration inhibitory factor is up-regulated in human first-trimester placenta stimulated by soluble antigen of Toxoplasma gondii, resulting in increased monocyte adhesion on villous explants. American Journal of Pathology. 2008 ; 172( 1): 50-58.[citado 2025 out. 26 ]
Vancouver
Ferro EAV, Mineo JR, Letta F, Bechi N, Romagnoli R, Silva DAO, Sorda G, Bevilacqua EMAF, Paulesu LR. Macrophage migration inhibitory factor is up-regulated in human first-trimester placenta stimulated by soluble antigen of Toxoplasma gondii, resulting in increased monocyte adhesion on villous explants. American Journal of Pathology. 2008 ; 172( 1): 50-58.[citado 2025 out. 26 ]
Artigo de periodico
SIKVAV, a Laminin 'alfa'1-derived peptide, interacts with integrins and increases protease activity of a human salivary gland adenoid cystic carcinoma cell line through the ERK 1/2 signaling pathway (2007)
Freitas, Vanessa Morais ; Vilas-Boas, Vanessa Fonseca; Pimenta, Daniel C.; Loureiro, Vania; Juliano, Maria A.; Carvalho, Márcia R.; Pinheiro, João J. V.; Camargo, Antonio C. M.; Moriscot, Anselmo Sigari ; Hoffman, Matthew P.; Jaeger, Ruy Gastaldoni
Fonte: American Journal of Pathology. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FREITAS, Vanessa Morais et al. SIKVAV, a Laminin 'alfa'1-derived peptide, interacts with integrins and increases protease activity of a human salivary gland adenoid cystic carcinoma cell line through the ERK 1/2 signaling pathway. American Journal of Pathology, v. 171, n. 1, p. 124-138, 2007Tradução . . Acesso em: 26 out. 2025.
APA
Freitas, V. M., Vilas-Boas, V. F., Pimenta, D. C., Loureiro, V., Juliano, M. A., Carvalho, M. R., et al. (2007). SIKVAV, a Laminin 'alfa'1-derived peptide, interacts with integrins and increases protease activity of a human salivary gland adenoid cystic carcinoma cell line through the ERK 1/2 signaling pathway. American Journal of Pathology, 171( 1), 124-138.
NLM
Freitas VM, Vilas-Boas VF, Pimenta DC, Loureiro V, Juliano MA, Carvalho MR, Pinheiro JJV, Camargo ACM, Moriscot AS, Hoffman MP, Jaeger RG. SIKVAV, a Laminin 'alfa'1-derived peptide, interacts with integrins and increases protease activity of a human salivary gland adenoid cystic carcinoma cell line through the ERK 1/2 signaling pathway. American Journal of Pathology. 2007 ; 171( 1): 124-138.[citado 2025 out. 26 ]
Vancouver
Freitas VM, Vilas-Boas VF, Pimenta DC, Loureiro V, Juliano MA, Carvalho MR, Pinheiro JJV, Camargo ACM, Moriscot AS, Hoffman MP, Jaeger RG. SIKVAV, a Laminin 'alfa'1-derived peptide, interacts with integrins and increases protease activity of a human salivary gland adenoid cystic carcinoma cell line through the ERK 1/2 signaling pathway. American Journal of Pathology. 2007 ; 171( 1): 124-138.[citado 2025 out. 26 ]
Artigo de periodico
Toxoplasma gondii infection reveals a novel regulatory role for galectin-3 inthe interface of innate and adaptive immunity (2006)
Bernardes, Emerson Soares; Silva, Neide M.; Ruas, Luciana Pereira; Mineo, José Roberto; Loyola, Adriano Motta; Hsu, Daniel K.; Liu, Fu-Tong; Chammas, Roger; Roque-Barreira, Maria Cristina
Fonte: American Journal of Pathology. Unidades: FM, FMRP
Assuntos: TOXOPLASMA GONDII, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BERNARDES, Emerson Soares et al. Toxoplasma gondii infection reveals a novel regulatory role for galectin-3 inthe interface of innate and adaptive immunity. American Journal of Pathology, v. 168, n. 6, p. 1910-1920, 2006Tradução . . Acesso em: 26 out. 2025.
APA
Bernardes, E. S., Silva, N. M., Ruas, L. P., Mineo, J. R., Loyola, A. M., Hsu, D. K., et al. (2006). Toxoplasma gondii infection reveals a novel regulatory role for galectin-3 inthe interface of innate and adaptive immunity. American Journal of Pathology, 168( 6), 1910-1920.
NLM
Bernardes ES, Silva NM, Ruas LP, Mineo JR, Loyola AM, Hsu DK, Liu F-T, Chammas R, Roque-Barreira MC. Toxoplasma gondii infection reveals a novel regulatory role for galectin-3 inthe interface of innate and adaptive immunity. American Journal of Pathology. 2006 ; 168( 6): 1910-1920.[citado 2025 out. 26 ]
Vancouver
Bernardes ES, Silva NM, Ruas LP, Mineo JR, Loyola AM, Hsu DK, Liu F-T, Chammas R, Roque-Barreira MC. Toxoplasma gondii infection reveals a novel regulatory role for galectin-3 inthe interface of innate and adaptive immunity. American Journal of Pathology. 2006 ; 168( 6): 1910-1920.[citado 2025 out. 26 ]
Artigo de periodico
Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas's disease cardiomyopathy (2005)
Cunha Neto, Edecio; Dzau, Victor J.; Allen, Paulo D.; Stamatiou, Dimitri; Benvenutti, Luiz; Higuchi, M. Lourdes; Koyama, Natalia S.; Silva, João Santana da; Kalil Filho, Jorge Elias; Liew, Choong-Chin
Fonte: American Journal of Pathology. Unidades: FMRP, FM
Assuntos: DOENÇA DE CHAGAS (IMUNOLOGIA), CARDIOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA NETO, Edecio et al. Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas's disease cardiomyopathy. American Journal of Pathology, v. 167, p. 305-313, 2005Tradução . . Acesso em: 26 out. 2025.
APA
Cunha Neto, E., Dzau, V. J., Allen, P. D., Stamatiou, D., Benvenutti, L., Higuchi, M. L., et al. (2005). Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas's disease cardiomyopathy. American Journal of Pathology, 167, 305-313.
NLM
Cunha Neto E, Dzau VJ, Allen PD, Stamatiou D, Benvenutti L, Higuchi ML, Koyama NS, Silva JS da, Kalil Filho JE, Liew C-C. Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas's disease cardiomyopathy. American Journal of Pathology. 2005 ; 167 305-313.[citado 2025 out. 26 ]
Vancouver
Cunha Neto E, Dzau VJ, Allen PD, Stamatiou D, Benvenutti L, Higuchi ML, Koyama NS, Silva JS da, Kalil Filho JE, Liew C-C. Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas's disease cardiomyopathy. American Journal of Pathology. 2005 ; 167 305-313.[citado 2025 out. 26 ]
Artigo de periodico
Interferon-y-induced nitric oxide causes intrinsic intestinal denervation in Trypanosoma cruzi infected mice (2004)
Arantes, Rosa M. E.; Marche, Homero H. F.; Bahia, Maria T.; Cunha, Fernando Queiróz; Rossi, Marco Antônio; Silva, João Santana da
Fonte: American Journal of Pathology. Unidade: FMRP
Assuntos: ÓXIDO NÍTRICO, TRYPANOSOMA CRUZI
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARANTES, Rosa M. E. et al. Interferon-y-induced nitric oxide causes intrinsic intestinal denervation in Trypanosoma cruzi infected mice. American Journal of Pathology, v. 164, n. 4, p. 1361-1368, 2004Tradução . . Acesso em: 26 out. 2025.
APA
Arantes, R. M. E., Marche, H. H. F., Bahia, M. T., Cunha, F. Q., Rossi, M. A., & Silva, J. S. da. (2004). Interferon-y-induced nitric oxide causes intrinsic intestinal denervation in Trypanosoma cruzi infected mice. American Journal of Pathology, 164( 4), 1361-1368.
NLM
Arantes RME, Marche HHF, Bahia MT, Cunha FQ, Rossi MA, Silva JS da. Interferon-y-induced nitric oxide causes intrinsic intestinal denervation in Trypanosoma cruzi infected mice. American Journal of Pathology. 2004 ; 164( 4): 1361-1368.[citado 2025 out. 26 ]
Vancouver
Arantes RME, Marche HHF, Bahia MT, Cunha FQ, Rossi MA, Silva JS da. Interferon-y-induced nitric oxide causes intrinsic intestinal denervation in Trypanosoma cruzi infected mice. American Journal of Pathology. 2004 ; 164( 4): 1361-1368.[citado 2025 out. 26 ]
Artigo de periodico
Rheumatic heart disease: proinflammatory cytokines play a role in the progression and maintenance of valvular lesions (2004)
Guilherme, Luiza; Cury, Patricia; Dermachi, Lea M. F.; Coelho, Veronica; Abel, Lúcia; Lopez, Ana P.; Oshiro, Sandra Emiko; Aliotti, Selma; Cunha-Neto, Edécio; Pomerantzeff, Pablo M. A.; Tanaka, Ana C.; Kalil, Jorge
Fonte: American Journal of Pathology. Unidade: FM
Assuntos: FEBRE REUMÁTICA, DOENÇAS REUMÁTICAS, CARDIOPATIAS, VALVAS CARDÍACAS, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUILHERME, Luiza et al. Rheumatic heart disease: proinflammatory cytokines play a role in the progression and maintenance of valvular lesions. American Journal of Pathology, v. 165, n. 5, p. 1583-1591, 2004Tradução . . Acesso em: 26 out. 2025.
APA
Guilherme, L., Cury, P., Dermachi, L. M. F., Coelho, V., Abel, L., Lopez, A. P., et al. (2004). Rheumatic heart disease: proinflammatory cytokines play a role in the progression and maintenance of valvular lesions. American Journal of Pathology, 165( 5), 1583-1591.
NLM
Guilherme L, Cury P, Dermachi LMF, Coelho V, Abel L, Lopez AP, Oshiro SE, Aliotti S, Cunha-Neto E, Pomerantzeff PMA, Tanaka AC, Kalil J. Rheumatic heart disease: proinflammatory cytokines play a role in the progression and maintenance of valvular lesions. American Journal of Pathology. 2004 ; 165( 5): 1583-1591.[citado 2025 out. 26 ]
Vancouver
Guilherme L, Cury P, Dermachi LMF, Coelho V, Abel L, Lopez AP, Oshiro SE, Aliotti S, Cunha-Neto E, Pomerantzeff PMA, Tanaka AC, Kalil J. Rheumatic heart disease: proinflammatory cytokines play a role in the progression and maintenance of valvular lesions. American Journal of Pathology. 2004 ; 165( 5): 1583-1591.[citado 2025 out. 26 ]
Artigo de periodico
Chemokine production and leukocyte recruitment to the lungs of Paracoccidioides brasiliensis-infected mice is modulated by interferon-'gama' (2003)
Souto, Janeusa Trindade de; Aliberti, Júlio C.; Campanelli, Ana Paula; Livonesi, Márcia Cristina; Maffei, Cláudia Maria Leite; Ferreira, Beatriz Rossetti ; Travassos, Luiz R.; Martinez, Roberto; Rossi, Marcos Antonio; Silva, João Santana da
Fonte: American Journal of Pathology. Unidade: FMRP
Assuntos: PARACOCCIDIOIDES BRASILIENSIS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUTO, Janeusa Trindade de et al. Chemokine production and leukocyte recruitment to the lungs of Paracoccidioides brasiliensis-infected mice is modulated by interferon-'gama'. American Journal of Pathology, v. 163, n. 2, p. 583-590, 2003Tradução . . Acesso em: 26 out. 2025.
APA
Souto, J. T. de, Aliberti, J. C., Campanelli, A. P., Livonesi, M. C., Maffei, C. M. L., Ferreira, B. R., et al. (2003). Chemokine production and leukocyte recruitment to the lungs of Paracoccidioides brasiliensis-infected mice is modulated by interferon-'gama'. American Journal of Pathology, 163( 2), 583-590.
NLM
Souto JT de, Aliberti JC, Campanelli AP, Livonesi MC, Maffei CML, Ferreira BR, Travassos LR, Martinez R, Rossi MA, Silva JS da. Chemokine production and leukocyte recruitment to the lungs of Paracoccidioides brasiliensis-infected mice is modulated by interferon-'gama'. American Journal of Pathology. 2003 ; 163( 2): 583-590.[citado 2025 out. 26 ]
Vancouver
Souto JT de, Aliberti JC, Campanelli AP, Livonesi MC, Maffei CML, Ferreira BR, Travassos LR, Martinez R, Rossi MA, Silva JS da. Chemokine production and leukocyte recruitment to the lungs of Paracoccidioides brasiliensis-infected mice is modulated by interferon-'gama'. American Journal of Pathology. 2003 ; 163( 2): 583-590.[citado 2025 out. 26 ]
Artigo de periodico
Modulation of chemokine production and inflammatory responses in interferon-'gama'- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection (2001)
Aliberti, Júlio César Soares; Souto, Janeusa Trindade; Marino, Ana P. M. P.; Lannes-Vieira, Joseli; Teixeira, Mauro M.; Farber, Joshua; Gazzinelli, Ricardo T.; Silva, João Santana da
Fonte: American Journal of Pathology. Unidade: FMRP
Assuntos: TRYPANOSOMA CRUZI, DOENÇA DE CHAGAS, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALIBERTI, Júlio César Soares et al. Modulation of chemokine production and inflammatory responses in interferon-'gama'- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection. American Journal of Pathology, v. 158, n. 4, p. 1433-1140, 2001Tradução . . Acesso em: 26 out. 2025.
APA
Aliberti, J. C. S., Souto, J. T., Marino, A. P. M. P., Lannes-Vieira, J., Teixeira, M. M., Farber, J., et al. (2001). Modulation of chemokine production and inflammatory responses in interferon-'gama'- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection. American Journal of Pathology, 158( 4), 1433-1140.
NLM
Aliberti JCS, Souto JT, Marino APMP, Lannes-Vieira J, Teixeira MM, Farber J, Gazzinelli RT, Silva JS da. Modulation of chemokine production and inflammatory responses in interferon-'gama'- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection. American Journal of Pathology. 2001 ; 158( 4): 1433-1140.[citado 2025 out. 26 ]
Vancouver
Aliberti JCS, Souto JT, Marino APMP, Lannes-Vieira J, Teixeira MM, Farber J, Gazzinelli RT, Silva JS da. Modulation of chemokine production and inflammatory responses in interferon-'gama'- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection. American Journal of Pathology. 2001 ; 158( 4): 1433-1140.[citado 2025 out. 26 ]
Artigo de periodico
Interferon-'gama' and tumor necrosis factor-'alfa' determine resistance to paracoccidioides brasiliensis infection in Mice (2000)
Souto, Janeusa Trindade de; Figueiredo, Florêncio; Furlanetto, Alexxandra; Pfeffer, Klaus; Rossi, Marcos Antonio; Silva, João Santana da
Fonte: American Journal of Pathology. Unidade: FMRP
Assuntos: IMUNOPARASITOLOGIA, IMUNOLOGIA, TUBERCULOSE (EPIDEMIOLOGIA), DOENÇAS INFECCIOSAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUTO, Janeusa Trindade de et al. Interferon-'gama' and tumor necrosis factor-'alfa' determine resistance to paracoccidioides brasiliensis infection in Mice. American Journal of Pathology, v. 156, n. 5, p. 1811-20, 2000Tradução . . Acesso em: 26 out. 2025.
APA
Souto, J. T. de, Figueiredo, F., Furlanetto, A., Pfeffer, K., Rossi, M. A., & Silva, J. S. da. (2000). Interferon-'gama' and tumor necrosis factor-'alfa' determine resistance to paracoccidioides brasiliensis infection in Mice. American Journal of Pathology, 156( 5), 1811-20.
NLM
Souto JT de, Figueiredo F, Furlanetto A, Pfeffer K, Rossi MA, Silva JS da. Interferon-'gama' and tumor necrosis factor-'alfa' determine resistance to paracoccidioides brasiliensis infection in Mice. American Journal of Pathology. 2000 ; 156( 5): 1811-20.[citado 2025 out. 26 ]
Vancouver
Souto JT de, Figueiredo F, Furlanetto A, Pfeffer K, Rossi MA, Silva JS da. Interferon-'gama' and tumor necrosis factor-'alfa' determine resistance to paracoccidioides brasiliensis infection in Mice. American Journal of Pathology. 2000 ; 156( 5): 1811-20.[citado 2025 out. 26 ]
Artigo de periodico
Detection of tgf-b1 in dermal lesions of different clinical forms of leprosy (1996)
Goulart, I M B; Figueiredo, Florêncio; Coimbra, Terezila Machado; Foss, Norma Tiraboschi
Fonte: American Journal of Pathology. Unidade: FMRP
Assunto: HANSENÍASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOULART, I M B et al. Detection of tgf-b1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology, v. 148, n. 3 , p. 911-7, 1996Tradução . . Acesso em: 26 out. 2025.
APA
Goulart, I. M. B., Figueiredo, F., Coimbra, T. M., & Foss, N. T. (1996). Detection of tgf-b1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology, 148( 3 ), 911-7.
NLM
Goulart IMB, Figueiredo F, Coimbra TM, Foss NT. Detection of tgf-b1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology. 1996 ;148( 3 ): 911-7.[citado 2025 out. 26 ]
Vancouver
Goulart IMB, Figueiredo F, Coimbra TM, Foss NT. Detection of tgf-b1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology. 1996 ;148( 3 ): 911-7.[citado 2025 out. 26 ]
Artigo de periodico
Detection of transforming growth factor-'beta'1 in dermal lesions of different clinical forms of leprosy (1996)
Goulart, Isabela Maria Bernardes; Figueiredo, Florêncio; Coimbra, Terezila Machado; Foss, Norma Tiraboschi
Fonte: American Journal of Pathology. Unidade: FMRP
Assunto: HANSENÍASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOULART, Isabela Maria Bernardes et al. Detection of transforming growth factor-'beta'1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology, v. 148, n. 3, p. 911-917, 1996Tradução . . Acesso em: 26 out. 2025.
APA
Goulart, I. M. B., Figueiredo, F., Coimbra, T. M., & Foss, N. T. (1996). Detection of transforming growth factor-'beta'1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology, 148( 3), 911-917.
NLM
Goulart IMB, Figueiredo F, Coimbra TM, Foss NT. Detection of transforming growth factor-'beta'1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology. 1996 ; 148( 3): 911-917.[citado 2025 out. 26 ]
Vancouver
Goulart IMB, Figueiredo F, Coimbra TM, Foss NT. Detection of transforming growth factor-'beta'1 in dermal lesions of different clinical forms of leprosy. American Journal of Pathology. 1996 ; 148( 3): 911-917.[citado 2025 out. 26 ]
Artigo de periodico
Aids - related lymphoma in Brazil: histopathology, immunophenotype, and association with epstein-barr virus (1996)
Bacchi, C. E.; Bacchi, M M; Rabenhorst, S H; Soares, Fernando Augusto; Fonseca Junior, L E; Barbosa, H S; Weiss, L M; Gown, A M
Fonte: American Journal of Pathology. Unidade: FMRP
Assuntos: LINFOMA, SÍNDROME DE IMUNODEFICIÊNCIA ADQUIRIDA, HEMATOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BACCHI, C. E. et al. Aids - related lymphoma in Brazil: histopathology, immunophenotype, and association with epstein-barr virus. American Journal of Pathology, v. 105, n. 2 , p. 230-7, 1996Tradução . . Acesso em: 26 out. 2025.
APA
Bacchi, C. E., Bacchi, M. M., Rabenhorst, S. H., Soares, F. A., Fonseca Junior, L. E., Barbosa, H. S., et al. (1996). Aids - related lymphoma in Brazil: histopathology, immunophenotype, and association with epstein-barr virus. American Journal of Pathology, 105( 2 ), 230-7.
NLM
Bacchi CE, Bacchi MM, Rabenhorst SH, Soares FA, Fonseca Junior LE, Barbosa HS, Weiss LM, Gown AM. Aids - related lymphoma in Brazil: histopathology, immunophenotype, and association with epstein-barr virus. American Journal of Pathology. 1996 ;105( 2 ): 230-7.[citado 2025 out. 26 ]
Vancouver
Bacchi CE, Bacchi MM, Rabenhorst SH, Soares FA, Fonseca Junior LE, Barbosa HS, Weiss LM, Gown AM. Aids - related lymphoma in Brazil: histopathology, immunophenotype, and association with epstein-barr virus. American Journal of Pathology. 1996 ;105( 2 ): 230-7.[citado 2025 out. 26 ]
Artigo de periodico
Up- regulation of t helper 2 and down - regulation of t helper 1 cytokines during murine retrovirus - induced immunodeficiency syndrome in hances susceptibility of a resistant mouse strain to l. Amazonensis (1995)
Barral Neto, M; Silva, João Santana da; Barral, A; Grabstein, K; Reed, S G
Fonte: American Journal of Pathology. Unidade: FMRP
Assunto: SÍNDROME DE IMUNODEFICIÊNCIA ADQUIRIDA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BARRAL NETO, M et al. Up- regulation of t helper 2 and down - regulation of t helper 1 cytokines during murine retrovirus - induced immunodeficiency syndrome in hances susceptibility of a resistant mouse strain to l. Amazonensis. American Journal of Pathology, v. 146, p. 135-42, 1995Tradução . . Acesso em: 26 out. 2025.
APA
Barral Neto, M., Silva, J. S. da, Barral, A., Grabstein, K., & Reed, S. G. (1995). Up- regulation of t helper 2 and down - regulation of t helper 1 cytokines during murine retrovirus - induced immunodeficiency syndrome in hances susceptibility of a resistant mouse strain to l. Amazonensis. American Journal of Pathology, 146, 135-42.
NLM
Barral Neto M, Silva JS da, Barral A, Grabstein K, Reed SG. Up- regulation of t helper 2 and down - regulation of t helper 1 cytokines during murine retrovirus - induced immunodeficiency syndrome in hances susceptibility of a resistant mouse strain to l. Amazonensis. American Journal of Pathology. 1995 ;146 135-42.[citado 2025 out. 26 ]
Vancouver
Barral Neto M, Silva JS da, Barral A, Grabstein K, Reed SG. Up- regulation of t helper 2 and down - regulation of t helper 1 cytokines during murine retrovirus - induced immunodeficiency syndrome in hances susceptibility of a resistant mouse strain to l. Amazonensis. American Journal of Pathology. 1995 ;146 135-42.[citado 2025 out. 26 ]
Artigo de periodico
Mice with homozygous disruption of the mdr2 p-glycoprotein gene: a novel animal model for studies of nonsuppurative inflammatory cholangitis and hepatocarcinogenesis (1994)
Mauad, T H; Nieuwkerk, C M J V; Dingemans, K P; Smit, J J M; Schinkel, A H; Notenboom, R G E; Weerman, M A V D B; Verkruisen, R P; Groen, A K; Elferink, R P J O; Valk, M A V D
Fonte: American Journal of Pathology. Unidade: HU
Assuntos: PATOLOGIA, ANATOMIA PATOLÓGICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAUAD, T H et al. Mice with homozygous disruption of the mdr2 p-glycoprotein gene: a novel animal model for studies of nonsuppurative inflammatory cholangitis and hepatocarcinogenesis. American Journal of Pathology, v. no 1994, n. 5 , p. 1237-45, 1994Tradução . . Acesso em: 26 out. 2025.
APA
Mauad, T. H., Nieuwkerk, C. M. J. V., Dingemans, K. P., Smit, J. J. M., Schinkel, A. H., Notenboom, R. G. E., et al. (1994). Mice with homozygous disruption of the mdr2 p-glycoprotein gene: a novel animal model for studies of nonsuppurative inflammatory cholangitis and hepatocarcinogenesis. American Journal of Pathology, no 1994( 5 ), 1237-45.
NLM
Mauad TH, Nieuwkerk CMJV, Dingemans KP, Smit JJM, Schinkel AH, Notenboom RGE, Weerman MAVDB, Verkruisen RP, Groen AK, Elferink RPJO, Valk MAVD. Mice with homozygous disruption of the mdr2 p-glycoprotein gene: a novel animal model for studies of nonsuppurative inflammatory cholangitis and hepatocarcinogenesis. American Journal of Pathology. 1994 ; no 1994( 5 ): 1237-45.[citado 2025 out. 26 ]
Vancouver
Mauad TH, Nieuwkerk CMJV, Dingemans KP, Smit JJM, Schinkel AH, Notenboom RGE, Weerman MAVDB, Verkruisen RP, Groen AK, Elferink RPJO, Valk MAVD. Mice with homozygous disruption of the mdr2 p-glycoprotein gene: a novel animal model for studies of nonsuppurative inflammatory cholangitis and hepatocarcinogenesis. American Journal of Pathology. 1994 ; no 1994( 5 ): 1237-45.[citado 2025 out. 26 ]

Unidades USP

ReP

Filtros

Unidades USP

Departamento

Autores

Autores USP

Ano de publicação

Assuntos

Indexado em

Afiliação dos autores externos normalizada

Afiliação dos autores externos não normalizada