ReP USP - Resultado da busca

Artigo de periodico
Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice (2020)
Estrela, Gabriel Rufino ; Wasinski, Frederick ; Gregnani, Marcos Fernandes ; Lima, Leandro Ceotto Freitas ; Arruda, Adriano C.; Morais, Rafael Leite; Malheiros, Denise Maria Avancini Costa; Câmara, Niels Olsen Saraiva ; Pesquero, João Bosco ; Bader, Michael ; Barros, Carlos Castilho ; Araújo, Ronaldo Carvalho
Source: Frontiers in Molecular Biosciences. Unidades: FM, ICB
Subjects: IMUNOLOGIA, ANGIOTENSINAS, INIBIDORES DE ENZIMAS, INFLAMAÇÃO, CAMUNDONGOS, QUIMIOTERÁPICOS, CININAS, RATOS, CREATINA, TÚBULOS RENAIS
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ABNT
ESTRELA, Gabriel Rufino et al. Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice. Frontiers in Molecular Biosciences, v. 7, p. 10 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fmolb.2020.00096. Acesso em: 13 nov. 2024.
APA
Estrela, G. R., Wasinski, F., Gregnani, M. F., Lima, L. C. F., Arruda, A. C., Morais, R. L., et al. (2020). Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice. Frontiers in Molecular Biosciences, 7, 10 . doi:10.3389/fmolb.2020.00096
NLM
Estrela GR, Wasinski F, Gregnani MF, Lima LCF, Arruda AC, Morais RL, Malheiros DMAC, Câmara NOS, Pesquero JB, Bader M, Barros CC, Araújo RC. Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice [Internet]. Frontiers in Molecular Biosciences. 2020 ; 7 10 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fmolb.2020.00096
Vancouver
Estrela GR, Wasinski F, Gregnani MF, Lima LCF, Arruda AC, Morais RL, Malheiros DMAC, Câmara NOS, Pesquero JB, Bader M, Barros CC, Araújo RC. Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice [Internet]. Frontiers in Molecular Biosciences. 2020 ; 7 10 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fmolb.2020.00096
Artigo de periodico
Autosomal-recessive mutations in MESD cause osteogenesis imperfecta (2019)
Moosa, Shahida; Yamamoto, Guilherme L; Garbes, Lutz; Keupp, Katharina; Beleza-Meireles, Ana; Moreno, Carolina Araujo; Valadares, Eugenia Ribeiro; Sousa, Sergio B. de; Maia, Sofia; Kim, Chong Ae
Source: American journal of human genetics. Unidade: FM
Subjects: OSTEOGÊNESE IMPERFEITA, MUTAÇÃO GENÉTICA, PROTEÍNAS MORFOGENÉTICAS ÓSSEAS, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
MOOSA, Shahida et al. Autosomal-recessive mutations in MESD cause osteogenesis imperfecta. American journal of human genetics, v. 105, n. 4, p. 836-843, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ajhg.2019.08.008. Acesso em: 13 nov. 2024.
APA
Moosa, S., Yamamoto, G. L., Garbes, L., Keupp, K., Beleza-Meireles, A., Moreno, C. A., et al. (2019). Autosomal-recessive mutations in MESD cause osteogenesis imperfecta. American journal of human genetics, 105( 4), 836-843. doi:10.1016/j.ajhg.2019.08.008
NLM
Moosa S, Yamamoto GL, Garbes L, Keupp K, Beleza-Meireles A, Moreno CA, Valadares ER, Sousa SB de, Maia S, Kim CA. Autosomal-recessive mutations in MESD cause osteogenesis imperfecta [Internet]. American journal of human genetics. 2019 ; 105( 4): 836-843.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ajhg.2019.08.008
Vancouver
Moosa S, Yamamoto GL, Garbes L, Keupp K, Beleza-Meireles A, Moreno CA, Valadares ER, Sousa SB de, Maia S, Kim CA. Autosomal-recessive mutations in MESD cause osteogenesis imperfecta [Internet]. American journal of human genetics. 2019 ; 105( 4): 836-843.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ajhg.2019.08.008
Artigo de periodico
Dapagliflozin and Cardiovascular Outcomes in Patients With Type 2 Diabetes Mellitus and Previous Myocardial Infarction Subanalysis From the DECLARE-TIMI 58 Trial (2019)
Furtado, Remo H. M; Bonaca, Marc P; Raz, Itamar; Zelniker, Thomas A; Mosenzon, Ofri; Cahn, Avivit; Kuder, Julia; Murphy, Sabina A; Bhatt, Deepak L; Nicolau, José Carlos
Source: Circulation. Unidade: FM
Subjects: DIABETES MELLITUS NÃO INSULINO-DEPENDENTE, INFARTO DO MIOCÁRDIO, ANTIDIABÉTICOS (HIPOGLICEMIANTES), RESULTADO DE TRATAMENTO
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ABNT
FURTADO, Remo H. M et al. Dapagliflozin and Cardiovascular Outcomes in Patients With Type 2 Diabetes Mellitus and Previous Myocardial Infarction Subanalysis From the DECLARE-TIMI 58 Trial. Circulation, v. 139, n. 22, p. 2516-2527, 2019Tradução . . Disponível em: https://doi.org/10.1161/CIRCULATIONAHA.119.039996. Acesso em: 13 nov. 2024.
APA
Furtado, R. H. M., Bonaca, M. P., Raz, I., Zelniker, T. A., Mosenzon, O., Cahn, A., et al. (2019). Dapagliflozin and Cardiovascular Outcomes in Patients With Type 2 Diabetes Mellitus and Previous Myocardial Infarction Subanalysis From the DECLARE-TIMI 58 Trial. Circulation, 139( 22), 2516-2527. doi:10.1161/CIRCULATIONAHA.119.039996
NLM
Furtado RHM, Bonaca MP, Raz I, Zelniker TA, Mosenzon O, Cahn A, Kuder J, Murphy SA, Bhatt DL, Nicolau JC. Dapagliflozin and Cardiovascular Outcomes in Patients With Type 2 Diabetes Mellitus and Previous Myocardial Infarction Subanalysis From the DECLARE-TIMI 58 Trial [Internet]. Circulation. 2019 ; 139( 22): 2516-2527.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1161/CIRCULATIONAHA.119.039996
Vancouver
Furtado RHM, Bonaca MP, Raz I, Zelniker TA, Mosenzon O, Cahn A, Kuder J, Murphy SA, Bhatt DL, Nicolau JC. Dapagliflozin and Cardiovascular Outcomes in Patients With Type 2 Diabetes Mellitus and Previous Myocardial Infarction Subanalysis From the DECLARE-TIMI 58 Trial [Internet]. Circulation. 2019 ; 139( 22): 2516-2527.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1161/CIRCULATIONAHA.119.039996
Artigo de periodico
Locus coeruleus imaging as a biomarker for noradrenergic dysfunction in neurodegenerative diseases (2019)
Betts, Matthew J; Kirilina, Evgeniya; Otaduy, Maria C. G; Ivanov, Dimo; Acosta-cabronero, Julio; Callaghan, Martina F; Lambert, Christian; Cardenas-blanco, Arturo; Pine, Kerrin; Grinberg, Lea Tenenholz
Source: Brain. Unidade: FM
Subjects: BIOMARCADORES, DOENÇAS NEURODEGENERATIVAS, NEURÔNIOS, IMAGEM POR RESSONÂNCIA MAGNÉTICA, NORADRENALINA
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ABNT
BETTS, Matthew J et al. Locus coeruleus imaging as a biomarker for noradrenergic dysfunction in neurodegenerative diseases. Brain, v. 142, p. 2558-2571, 2019Tradução . . Disponível em: https://doi.org/10.1093/brain/awz193. Acesso em: 13 nov. 2024.
APA
Betts, M. J., Kirilina, E., Otaduy, M. C. G., Ivanov, D., Acosta-cabronero, J., Callaghan, M. F., et al. (2019). Locus coeruleus imaging as a biomarker for noradrenergic dysfunction in neurodegenerative diseases. Brain, 142, 2558-2571. doi:10.1093/brain/awz193
NLM
Betts MJ, Kirilina E, Otaduy MCG, Ivanov D, Acosta-cabronero J, Callaghan MF, Lambert C, Cardenas-blanco A, Pine K, Grinberg LT. Locus coeruleus imaging as a biomarker for noradrenergic dysfunction in neurodegenerative diseases [Internet]. Brain. 2019 ; 142 2558-2571.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awz193
Vancouver
Betts MJ, Kirilina E, Otaduy MCG, Ivanov D, Acosta-cabronero J, Callaghan MF, Lambert C, Cardenas-blanco A, Pine K, Grinberg LT. Locus coeruleus imaging as a biomarker for noradrenergic dysfunction in neurodegenerative diseases [Internet]. Brain. 2019 ; 142 2558-2571.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awz193
Artigo de periodico
Non-motor outcomes depend on location of neurostimulation in Parkinson's disease (2019)
Petry-schmelzer, Jan Niklas; Krause, Max; Dembek, Till A; Horn, Andreas; Evans, Julian; Ashkan, Keyoumars; Rizos, Alexandra; Silverdale, Monty; Schumacher, Wibke; Fonoff, Erich Talamoni
Source: Brain. Unidade: FM
Subjects: DOENÇA DE PARKINSON, ESTIMULAÇÃO CEREBRAL, NÚCLEOS TALÂMICOS, ESTUDOS PROSPECTIVOS, RESULTADO DE TRATAMENTO
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ABNT
PETRY-SCHMELZER, Jan Niklas et al. Non-motor outcomes depend on location of neurostimulation in Parkinson's disease. Brain, v. 142, p. 3592-3604, 2019Tradução . . Disponível em: https://doi.org/10.1093/brain/awz285. Acesso em: 13 nov. 2024.
APA
Petry-schmelzer, J. N., Krause, M., Dembek, T. A., Horn, A., Evans, J., Ashkan, K., et al. (2019). Non-motor outcomes depend on location of neurostimulation in Parkinson's disease. Brain, 142, 3592-3604. doi:10.1093/brain/awz285
NLM
Petry-schmelzer JN, Krause M, Dembek TA, Horn A, Evans J, Ashkan K, Rizos A, Silverdale M, Schumacher W, Fonoff ET. Non-motor outcomes depend on location of neurostimulation in Parkinson's disease [Internet]. Brain. 2019 ; 142 3592-3604.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awz285
Vancouver
Petry-schmelzer JN, Krause M, Dembek TA, Horn A, Evans J, Ashkan K, Rizos A, Silverdale M, Schumacher W, Fonoff ET. Non-motor outcomes depend on location of neurostimulation in Parkinson's disease [Internet]. Brain. 2019 ; 142 3592-3604.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awz285
Artigo de periodico
BCL11B mutations in patients affected by a neurodevelopmental disorder with reduced type 2 innate lymphoid cells (2018)
Lessel, Davor; Jorge, Alexander A. L.
Source: Brain. Unidade: FM
Subjects: DESENVOLVIMENTO INTELECTUAL, INFLAMAÇÃO, MUTAÇÃO GENÉTICA
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ABNT
LESSEL, Davor e JORGE, Alexander A. L. BCL11B mutations in patients affected by a neurodevelopmental disorder with reduced type 2 innate lymphoid cells. Brain, v. 141, p. 2299-2311, 2018Tradução . . Disponível em: https://doi.org/10.1093/brain/awy173. Acesso em: 13 nov. 2024.
APA
Lessel, D., & Jorge, A. A. L. (2018). BCL11B mutations in patients affected by a neurodevelopmental disorder with reduced type 2 innate lymphoid cells. Brain, 141, 2299-2311. doi:10.1093/brain/awy173
NLM
Lessel D, Jorge AAL. BCL11B mutations in patients affected by a neurodevelopmental disorder with reduced type 2 innate lymphoid cells [Internet]. Brain. 2018 ; 141 2299-2311.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awy173
Vancouver
Lessel D, Jorge AAL. BCL11B mutations in patients affected by a neurodevelopmental disorder with reduced type 2 innate lymphoid cells [Internet]. Brain. 2018 ; 141 2299-2311.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awy173
Artigo de periodico
Methods for segmentation of rheumatoid arthritis bone erosions in high-resolution peripheral quantitative computed tomography (HR-pQCT) (2018)
Figueiredo, Camille P.; Pereira, Rosa Maria Rodrigues
Source: Seminars in arthritis and rheumatism. Unidade: FM
Subjects: OSSO E OSSOS, ARTRITE REUMATOIDE, TOMOGRAFIA COMPUTADORIZADA DE EMISSÃO
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ABNT
FIGUEIREDO, Camille P. e PEREIRA, Rosa Maria Rodrigues. Methods for segmentation of rheumatoid arthritis bone erosions in high-resolution peripheral quantitative computed tomography (HR-pQCT). Seminars in arthritis and rheumatism, v. 47, n. 5, p. 611-618, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.semarthrit.2017.09.011. Acesso em: 13 nov. 2024.
APA
Figueiredo, C. P., & Pereira, R. M. R. (2018). Methods for segmentation of rheumatoid arthritis bone erosions in high-resolution peripheral quantitative computed tomography (HR-pQCT). Seminars in arthritis and rheumatism, 47( 5), 611-618. doi:10.1016/j.semarthrit.2017.09.011
NLM
Figueiredo CP, Pereira RMR. Methods for segmentation of rheumatoid arthritis bone erosions in high-resolution peripheral quantitative computed tomography (HR-pQCT) [Internet]. Seminars in arthritis and rheumatism. 2018 ; 47( 5): 611-618.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.semarthrit.2017.09.011
Vancouver
Figueiredo CP, Pereira RMR. Methods for segmentation of rheumatoid arthritis bone erosions in high-resolution peripheral quantitative computed tomography (HR-pQCT) [Internet]. Seminars in arthritis and rheumatism. 2018 ; 47( 5): 611-618.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.semarthrit.2017.09.011
Artigo de periodico
A novel complex neurological phenotype due to a homozygous mutation in FDX2 (2018)
Gurgel-giannetti, Juliana; Kok, Fernando
Source: Brain. Unidade: FM
Subjects: FENÓTIPOS, MIOPATIAS CONGÊNITAS ESTRUTURAIS, ATROFIA MUSCULAR, SEQUENCIAMENTO GENÉTICO, IMAGEM POR RESSONÂNCIA MAGNÉTICA
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ABNT
GURGEL-GIANNETTI, Juliana e KOK, Fernando. A novel complex neurological phenotype due to a homozygous mutation in FDX2. Brain, v. 141, p. 2289-2298, 2018Tradução . . Disponível em: https://doi.org/10.1093/brain/awy172. Acesso em: 13 nov. 2024.
APA
Gurgel-giannetti, J., & Kok, F. (2018). A novel complex neurological phenotype due to a homozygous mutation in FDX2. Brain, 141, 2289-2298. doi:10.1093/brain/awy172
NLM
Gurgel-giannetti J, Kok F. A novel complex neurological phenotype due to a homozygous mutation in FDX2 [Internet]. Brain. 2018 ; 141 2289-2298.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awy172
Vancouver
Gurgel-giannetti J, Kok F. A novel complex neurological phenotype due to a homozygous mutation in FDX2 [Internet]. Brain. 2018 ; 141 2289-2298.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/brain/awy172
Artigo de periodico
Cortical abnormalities in bipolar disorder: an MRI analysis of 6503 individuals from the ENIGMA Bipolar Disorder Working Group (2018)
Hibar, D. P; Lafer, B; Busatto, G. F; Chaim, K. T; Gattaz, W. F
Source: Molecular psychiatry. Unidade: FM
Subjects: ANTIPSICÓTICOS, CÓRTEX CEREBRAL, DEPRESSÃO
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ABNT
HIBAR, D. P et al. Cortical abnormalities in bipolar disorder: an MRI analysis of 6503 individuals from the ENIGMA Bipolar Disorder Working Group. Molecular psychiatry, v. 23, n. 4, p. 932-942, 2018Tradução . . Disponível em: https://doi.org/10.1038/mp.2017.73. Acesso em: 13 nov. 2024.
APA
Hibar, D. P., Lafer, B., Busatto, G. F., Chaim, K. T., & Gattaz, W. F. (2018). Cortical abnormalities in bipolar disorder: an MRI analysis of 6503 individuals from the ENIGMA Bipolar Disorder Working Group. Molecular psychiatry, 23( 4), 932-942. doi:10.1038/mp.2017.73
NLM
Hibar DP, Lafer B, Busatto GF, Chaim KT, Gattaz WF. Cortical abnormalities in bipolar disorder: an MRI analysis of 6503 individuals from the ENIGMA Bipolar Disorder Working Group [Internet]. Molecular psychiatry. 2018 ; 23( 4): 932-942.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/mp.2017.73
Vancouver
Hibar DP, Lafer B, Busatto GF, Chaim KT, Gattaz WF. Cortical abnormalities in bipolar disorder: an MRI analysis of 6503 individuals from the ENIGMA Bipolar Disorder Working Group [Internet]. Molecular psychiatry. 2018 ; 23( 4): 932-942.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/mp.2017.73
Artigo de periodico
The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis (2018)
Goncalves-de-albuquerque, Cassiano Felippe; Capelozzi, Vera Luiza
Source: Frontiers in immunology. Unidade: FM
Subjects: INFLAMAÇÃO, LEUCÓCITOS, SEPSE, CHOQUE SÉPTICO, ESTADOS UNIDOS
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ABNT
GONCALVES-DE-ALBUQUERQUE, Cassiano Felippe e CAPELOZZI, Vera Luiza. The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis. Frontiers in immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00901. Acesso em: 13 nov. 2024.
APA
Goncalves-de-albuquerque, C. F., & Capelozzi, V. L. (2018). The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis. Frontiers in immunology, 9. doi:10.3389/fimmu.2018.00901
NLM
Goncalves-de-albuquerque CF, Capelozzi VL. The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis [Internet]. Frontiers in immunology. 2018 ; 9[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2018.00901
Vancouver
Goncalves-de-albuquerque CF, Capelozzi VL. The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis [Internet]. Frontiers in immunology. 2018 ; 9[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2018.00901
Artigo de periodico
Pulmonary arterialhHypertension-related morbidity is prognostic for mortality (2018)
Mclaughlin, Vallerie V.; Souza, Rogerio
Source: Journal of the american college of cardiology. Unidade: FM
Subjects: HIPERTENSÃO PULMONAR, MORBIDADE, PROGNÓSTICO
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ABNT
MCLAUGHLIN, Vallerie V. e SOUZA, Rogerio. Pulmonary arterialhHypertension-related morbidity is prognostic for mortality. Journal of the american college of cardiology, v. 71, n. 7, p. 752-763, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jacc.2017.12.010. Acesso em: 13 nov. 2024.
APA
Mclaughlin, V. V., & Souza, R. (2018). Pulmonary arterialhHypertension-related morbidity is prognostic for mortality. Journal of the american college of cardiology, 71( 7), 752-763. doi:10.1016/j.jacc.2017.12.010
NLM
Mclaughlin VV, Souza R. Pulmonary arterialhHypertension-related morbidity is prognostic for mortality [Internet]. Journal of the american college of cardiology. 2018 ; 71( 7): 752-763.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.jacc.2017.12.010
Vancouver
Mclaughlin VV, Souza R. Pulmonary arterialhHypertension-related morbidity is prognostic for mortality [Internet]. Journal of the american college of cardiology. 2018 ; 71( 7): 752-763.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.jacc.2017.12.010

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