ReP USP - Resultado da busca

Artigo de periodico
4′-fluorocannabidiol associated with capsazepine restrains L-DOPA-induced dyskinesia in hemiparkinsonian mice: contribution of anti-inflammatory and anti-glutamatergic mechanisms (2024)
Pereira, Maurício dos Santos ; Abreu, Gabriel Henrique Dias de; Vanderlei, Leonardo Calaça Arruda ; Raisman-Vozari, Rita; Guimarães, Francisco Silveira ; Lu, Hui-Chen; Michel, Patrick Pierre; Del Bel, Elaine Aparecida
Source: Neuropharmacology. Unidades: FMRP, FORP
Subjects: ANTI-INFLAMATÓRIOS, CANABINOIDES, ANIMAIS DE LABORATÓRIO, DOENÇA DE PARKINSON, GLUTAMATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos et al. 4′-fluorocannabidiol associated with capsazepine restrains L-DOPA-induced dyskinesia in hemiparkinsonian mice: contribution of anti-inflammatory and anti-glutamatergic mechanisms. Neuropharmacology, v. 251, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2024.109926. Acesso em: 30 abr. 2026.
APA
Pereira, M. dos S., Abreu, G. H. D. de, Vanderlei, L. C. A., Raisman-Vozari, R., Guimarães, F. S., Lu, H. -C., et al. (2024). 4′-fluorocannabidiol associated with capsazepine restrains L-DOPA-induced dyskinesia in hemiparkinsonian mice: contribution of anti-inflammatory and anti-glutamatergic mechanisms. Neuropharmacology, 251. doi:10.1016/j.neuropharm.2024.109926
NLM
Pereira M dos S, Abreu GHD de, Vanderlei LCA, Raisman-Vozari R, Guimarães FS, Lu H-C, Michel PP, Del Bel EA. 4′-fluorocannabidiol associated with capsazepine restrains L-DOPA-induced dyskinesia in hemiparkinsonian mice: contribution of anti-inflammatory and anti-glutamatergic mechanisms [Internet]. Neuropharmacology. 2024 ; 251[citado 2026 abr. 30 ] Available from: https://doi.org/10.1016/j.neuropharm.2024.109926
Vancouver
Pereira M dos S, Abreu GHD de, Vanderlei LCA, Raisman-Vozari R, Guimarães FS, Lu H-C, Michel PP, Del Bel EA. 4′-fluorocannabidiol associated with capsazepine restrains L-DOPA-induced dyskinesia in hemiparkinsonian mice: contribution of anti-inflammatory and anti-glutamatergic mechanisms [Internet]. Neuropharmacology. 2024 ; 251[citado 2026 abr. 30 ] Available from: https://doi.org/10.1016/j.neuropharm.2024.109926
Artigo de periodico
The two synthetic cannabinoid compounds 4′-F-CBD and HU-910 efficiently restrain inflammatory responses of brain microglia and astrocytes (2024)
Pereira, Maurício dos Santos ; Santos, Bruna Maitan ; Gimenez, Rocio; Guimarães, Francisco Silveira ; Raisman-Vozari, Rita; Del Bel, Elaine Aparecida ; Michel, Patrick Pierre
Source: Glia. Unidades: FMRP, FORP, EERP
Subjects: ASTRÓCITOS, CÉREBRO, CANABINOIDES, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, ANIMAIS DE LABORATÓRIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos et al. The two synthetic cannabinoid compounds 4′-F-CBD and HU-910 efficiently restrain inflammatory responses of brain microglia and astrocytes. Glia, v. 72, n. 3, p. 529-545, 2024Tradução . . Disponível em: https://doi.org/10.1002/glia.24489. Acesso em: 30 abr. 2026.
APA
Pereira, M. dos S., Santos, B. M., Gimenez, R., Guimarães, F. S., Raisman-Vozari, R., Del Bel, E. A., & Michel, P. P. (2024). The two synthetic cannabinoid compounds 4′-F-CBD and HU-910 efficiently restrain inflammatory responses of brain microglia and astrocytes. Glia, 72( 3), 529-545. doi:10.1002/glia.24489
NLM
Pereira M dos S, Santos BM, Gimenez R, Guimarães FS, Raisman-Vozari R, Del Bel EA, Michel PP. The two synthetic cannabinoid compounds 4′-F-CBD and HU-910 efficiently restrain inflammatory responses of brain microglia and astrocytes [Internet]. Glia. 2024 ; 72( 3): 529-545.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1002/glia.24489
Vancouver
Pereira M dos S, Santos BM, Gimenez R, Guimarães FS, Raisman-Vozari R, Del Bel EA, Michel PP. The two synthetic cannabinoid compounds 4′-F-CBD and HU-910 efficiently restrain inflammatory responses of brain microglia and astrocytes [Internet]. Glia. 2024 ; 72( 3): 529-545.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1002/glia.24489
Artigo de periodico
Cannabidiol and it fluorinate analog PECS-101 reduces hyperalgesia and allodynia in trigeminal neuralgia via TRPV1 receptors (2024)
Escobar-Espinal, Daniela Maria ; Estela, Airam Nicole Vivanco ; Silva, Núbia Barros Pereira da ; Pereira, Maurício dos Santos ; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida ; Nascimento, Glauce Crivelaro do
Source: Progress in Neuro-Psychopharmacology and Biological Psychiatry. Unidades: FMRP, FORP
Subjects: CANABINOIDES, DOR FACIAL, NERVO TRIGÊMEO, QUALIDADE DE VIDA, FISIOPATOLOGIA, SISTEMA NERVOSO CENTRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ESCOBAR-ESPINAL, Daniela Maria et al. Cannabidiol and it fluorinate analog PECS-101 reduces hyperalgesia and allodynia in trigeminal neuralgia via TRPV1 receptors. Progress in Neuro-Psychopharmacology and Biological Psychiatry, v. 132, p. 1-12, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.pnpbp.2024.110996. Acesso em: 30 abr. 2026.
APA
Escobar-Espinal, D. M., Estela, A. N. V., Silva, N. B. P. da, Pereira, M. dos S., Guimarães, F. S., Del Bel, E. A., & Nascimento, G. C. do. (2024). Cannabidiol and it fluorinate analog PECS-101 reduces hyperalgesia and allodynia in trigeminal neuralgia via TRPV1 receptors. Progress in Neuro-Psychopharmacology and Biological Psychiatry, 132, 1-12. doi:10.1016/j.pnpbp.2024.110996
NLM
Escobar-Espinal DM, Estela ANV, Silva NBP da, Pereira M dos S, Guimarães FS, Del Bel EA, Nascimento GC do. Cannabidiol and it fluorinate analog PECS-101 reduces hyperalgesia and allodynia in trigeminal neuralgia via TRPV1 receptors [Internet]. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 2024 ; 132 1-12.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1016/j.pnpbp.2024.110996
Vancouver
Escobar-Espinal DM, Estela ANV, Silva NBP da, Pereira M dos S, Guimarães FS, Del Bel EA, Nascimento GC do. Cannabidiol and it fluorinate analog PECS-101 reduces hyperalgesia and allodynia in trigeminal neuralgia via TRPV1 receptors [Internet]. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 2024 ; 132 1-12.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1016/j.pnpbp.2024.110996
Artigo de periodico
Doxycycline attenuates l-DOPA-induced dyskinesia through an anti-inflammatory effect in a hemiparkinsonian mouse model (2022)
Pereira, Maurício dos Santos ; Nascimento, Glauce Crivelaro do ; Bortolanza, Mariza ; Michel, Patrick Pierre; Raisman-Vozari, Rita; Del Bel, Elaine Aparecida
Source: Frontiers in Pharmacology. Unidades: FMRP, FORP
Subjects: NEUROLOGIA, INFLAMAÇÃO, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos et al. Doxycycline attenuates l-DOPA-induced dyskinesia through an anti-inflammatory effect in a hemiparkinsonian mouse model. Frontiers in Pharmacology, v. 13, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphar.2022.1045465. Acesso em: 30 abr. 2026.
APA
Pereira, M. dos S., Nascimento, G. C. do, Bortolanza, M., Michel, P. P., Raisman-Vozari, R., & Del Bel, E. A. (2022). Doxycycline attenuates l-DOPA-induced dyskinesia through an anti-inflammatory effect in a hemiparkinsonian mouse model. Frontiers in Pharmacology, 13. doi:10.3389/fphar.2022.1045465
NLM
Pereira M dos S, Nascimento GC do, Bortolanza M, Michel PP, Raisman-Vozari R, Del Bel EA. Doxycycline attenuates l-DOPA-induced dyskinesia through an anti-inflammatory effect in a hemiparkinsonian mouse model [Internet]. Frontiers in Pharmacology. 2022 ; 13[citado 2026 abr. 30 ] Available from: https://doi.org/10.3389/fphar.2022.1045465
Vancouver
Pereira M dos S, Nascimento GC do, Bortolanza M, Michel PP, Raisman-Vozari R, Del Bel EA. Doxycycline attenuates l-DOPA-induced dyskinesia through an anti-inflammatory effect in a hemiparkinsonian mouse model [Internet]. Frontiers in Pharmacology. 2022 ; 13[citado 2026 abr. 30 ] Available from: https://doi.org/10.3389/fphar.2022.1045465
Artigo de periodico
Cannabidiol has therapeutic potential for myofascial pain in female and male parkinsonian rats (2021)
Estela, Airam Nicole Vivanco; Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida ; Nascimento, Glauce Crivelaro do
Source: Neuropharmacology. Unidades: FORP, FMRP
Subjects: CANABINOIDES, ANALGÉSICOS, ANTICONVULSIVANTES, DOR FACIAL, NOCICEPTIVIDADE, DOENÇA DE PARKINSON
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ESTELA, Airam Nicole Vivanco et al. Cannabidiol has therapeutic potential for myofascial pain in female and male parkinsonian rats. Neuropharmacology, v. 196, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2021.108700. Acesso em: 30 abr. 2026.
APA
Estela, A. N. V., Pereira, M. dos S., Guimarães, F. S., Del Bel, E. A., & Nascimento, G. C. do. (2021). Cannabidiol has therapeutic potential for myofascial pain in female and male parkinsonian rats. Neuropharmacology, 196. doi:10.1016/j.neuropharm.2021.108700
NLM
Estela ANV, Pereira M dos S, Guimarães FS, Del Bel EA, Nascimento GC do. Cannabidiol has therapeutic potential for myofascial pain in female and male parkinsonian rats [Internet]. Neuropharmacology. 2021 ; 196[citado 2026 abr. 30 ] Available from: https://doi.org/10.1016/j.neuropharm.2021.108700
Vancouver
Estela ANV, Pereira M dos S, Guimarães FS, Del Bel EA, Nascimento GC do. Cannabidiol has therapeutic potential for myofascial pain in female and male parkinsonian rats [Internet]. Neuropharmacology. 2021 ; 196[citado 2026 abr. 30 ] Available from: https://doi.org/10.1016/j.neuropharm.2021.108700
Artigo de periodico
The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress (2021)
Ferreira Junior, Nilson Carlos; Pereira, Maurício dos Santos ; Francis, Nour; Ramirez, Paola; Martorell, Paula; González-Lizarraga, Florencia; Figadère, Bruno; Chehin, Rosana; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick Pierre
Source: Cells. Unidade: FORP
Subjects: ESTRESSE OXIDATIVO, GLICOSE, METABOLISMO, ANTIBIÓTICOS, AGENTES ANTIMICROBIANOS, FARMACOLOGIA EXPERIMENTAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA JUNIOR, Nilson Carlos et al. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, v. 10, n. 8, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10082163. Acesso em: 30 abr. 2026.
APA
Ferreira Junior, N. C., Pereira, M. dos S., Francis, N., Ramirez, P., Martorell, P., González-Lizarraga, F., et al. (2021). The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, 10( 8), 1-16. doi:10.3390/cells10082163
NLM
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2026 abr. 30 ] Available from: https://doi.org/10.3390/cells10082163
Vancouver
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2026 abr. 30 ] Available from: https://doi.org/10.3390/cells10082163
Artigo de periodico
Caffeine improves GABA transport in the striatum of Spontaneously Hypertensive Rats (SHR) (2021)
Kubrusly, Regina Célia Cussa; Valli, Thais da Rosa; Ferreira, Mariana Nunes Marinho Ritter; Moura, Pamella de; Borges-Martins, Vladimir Pedro Peralva; Martins, Robertta Silva; Ferreira, Danielle Dias Pinto; Sathler, Matheus Figueiredo; Reis, Ricardo Augusto de Melo; Ferreira, Gustavo Costa; Manhães, Alex Christian; Pereira, Maurício dos Santos
Source: Neurotoxicity Research. Unidade: FORP
Subjects: GABA, RECEPTORES, NEUROTRANSMISSORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KUBRUSLY, Regina Célia Cussa et al. Caffeine improves GABA transport in the striatum of Spontaneously Hypertensive Rats (SHR). Neurotoxicity Research, v. 39, n. 6, p. 1946-1958, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12640-021-00423-0. Acesso em: 30 abr. 2026.
APA
Kubrusly, R. C. C., Valli, T. da R., Ferreira, M. N. M. R., Moura, P. de, Borges-Martins, V. P. P., Martins, R. S., et al. (2021). Caffeine improves GABA transport in the striatum of Spontaneously Hypertensive Rats (SHR). Neurotoxicity Research, 39( 6), 1946-1958. doi:10.1007/s12640-021-00423-0
NLM
Kubrusly RCC, Valli T da R, Ferreira MNMR, Moura P de, Borges-Martins VPP, Martins RS, Ferreira DDP, Sathler MF, Reis RA de M, Ferreira GC, Manhães AC, Pereira M dos S. Caffeine improves GABA transport in the striatum of Spontaneously Hypertensive Rats (SHR) [Internet]. Neurotoxicity Research. 2021 ; 39( 6): 1946-1958.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1007/s12640-021-00423-0
Vancouver
Kubrusly RCC, Valli T da R, Ferreira MNMR, Moura P de, Borges-Martins VPP, Martins RS, Ferreira DDP, Sathler MF, Reis RA de M, Ferreira GC, Manhães AC, Pereira M dos S. Caffeine improves GABA transport in the striatum of Spontaneously Hypertensive Rats (SHR) [Internet]. Neurotoxicity Research. 2021 ; 39( 6): 1946-1958.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1007/s12640-021-00423-0
Artigo de periodico
Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease (2021)
Pereira, Maurício dos Santos ; Abreu, Gabriel Henrique Dias; Rocca, Jeremy; Hamadat, Sabah; Raisman-Vozari, Rita; Michel, Patrick Pierre; Guimarães, Elaine Aparecida Del Bel Belluz
Source: Frontiers in Pharmacology. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, DISCINESIAS, LESÃO CEREBRAL CRÔNICA, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos et al. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease. Frontiers in Pharmacology, v. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2020.617085. Acesso em: 30 abr. 2026.
APA
Pereira, M. dos S., Abreu, G. H. D., Rocca, J., Hamadat, S., Raisman-Vozari, R., Michel, P. P., & Guimarães, E. A. D. B. B. (2021). Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease. Frontiers in Pharmacology, 11, 1-16. doi:10.3389/fphar.2020.617085
NLM
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Guimarães EADBB. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2026 abr. 30 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Vancouver
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Guimarães EADBB. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2026 abr. 30 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Artigo de periodico
Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease (2021)
Pereira, Maurício dos Santos ; Abreu, Gabriel Henrique Dias; Rocca, Jeremy; Hamadat, Sabah; Raisman-Vozari, Rita; Michel, Patrick Pierre; Del Bel, Elaine Aparecida
Source: Frontiers in Pharmacology. Unidade: FORP
Subjects: ASTROCITOMA, CANABINOIDES, GLUTAMATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos et al. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, v. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2020.617085. Acesso em: 30 abr. 2026.
APA
Pereira, M. dos S., Abreu, G. H. D., Rocca, J., Hamadat, S., Raisman-Vozari, R., Michel, P. P., & Del Bel, E. A. (2021). Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, 11, 1-16. doi:10.3389/fphar.2020.617085
NLM
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2026 abr. 30 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Vancouver
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2026 abr. 30 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Artigo de periodico
Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex (2020)
Kubrusly, Regina Célia Cussa; Martins, Robertta Silva; Souza, Lisiane de Santana; Carvalho, Mariana Pinheiro de; Borges-Martins, Vladimir Pedro Peralva; Sathler, Matheus Figueiredo; Ferreira, Danielle Dias Pinto; Pereira, Maurício dos Santos; Peccinalli, Ney Ronner; Pandolfo, Pablo; Reis, Ricardo Augusto de Melo; Ferreira, Gustavo Costa; Manhães, Alex Christian
Source: Neurotoxicity Research. Unidade: FORP
Subjects: COCAÍNA, MODELOS ANIMAIS, NEUROTRANSMISSORES, NEUROLOGIA, GABA, RECEPTORES, ATIVIDADE MOTORA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KUBRUSLY, Regina Célia Cussa et al. Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex. Neurotoxicity Research, v. 38, n. 3, p. 824-832, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-020-00259-0. Acesso em: 30 abr. 2026.
APA
Kubrusly, R. C. C., Martins, R. S., Souza, L. de S., Carvalho, M. P. de, Borges-Martins, V. P. P., Sathler, M. F., et al. (2020). Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex. Neurotoxicity Research, 38( 3), 824-832. doi:10.1007/s12640-020-00259-0
NLM
Kubrusly RCC, Martins RS, Souza L de S, Carvalho MP de, Borges-Martins VPP, Sathler MF, Ferreira DDP, Pereira M dos S, Peccinalli NR, Pandolfo P, Reis RA de M, Ferreira GC, Manhães AC. Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex [Internet]. Neurotoxicity Research. 2020 ; 38( 3): 824-832.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1007/s12640-020-00259-0
Vancouver
Kubrusly RCC, Martins RS, Souza L de S, Carvalho MP de, Borges-Martins VPP, Sathler MF, Ferreira DDP, Pereira M dos S, Peccinalli NR, Pandolfo P, Reis RA de M, Ferreira GC, Manhães AC. Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex [Internet]. Neurotoxicity Research. 2020 ; 38( 3): 824-832.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1007/s12640-020-00259-0
Artigo de periodico
Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption (2020)
Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick P.
Source: Glia. Unidades: FMRP, FORP
Subjects: CANABINOIDES, GLICOSE, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos et al. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, v. 68, n. 3, p. 561-573, 2020Tradução . . Disponível em: https://doi.org/10.1002/glia.23738. Acesso em: 30 abr. 2026.
APA
Pereira, M. dos S., Guimarães, F. S., Del Bel, E. A., Raisman-Vozari, R., & Michel, P. P. (2020). Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, 68( 3), 561-573. doi:10.1002/glia.23738
NLM
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1002/glia.23738
Vancouver
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1002/glia.23738
Artigo de periodico
Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia (2020)
Ferreira Junior, Nilson Carlos ; Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida
Source: Neurotoxicity Research. Unidades: FMRP, FORP
Subjects: RECEPTORES, CANABINOIDES, INFLAMAÇÃO, DOENÇA DE PARKINSON
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA JUNIOR, Nilson Carlos et al. Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia. Neurotoxicity Research, v. 37, n. 1, p. 12-29, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-019-00109-8. Acesso em: 30 abr. 2026.
APA
Ferreira Junior, N. C., Pereira, M. dos S., Guimarães, F. S., & Del Bel, E. A. (2020). Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia. Neurotoxicity Research, 37( 1), 12-29. doi:10.1007/s12640-019-00109-8
NLM
Ferreira Junior NC, Pereira M dos S, Guimarães FS, Del Bel EA. Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia [Internet]. Neurotoxicity Research. 2020 ; 37( 1): 12-29.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1007/s12640-019-00109-8
Vancouver
Ferreira Junior NC, Pereira M dos S, Guimarães FS, Del Bel EA. Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia [Internet]. Neurotoxicity Research. 2020 ; 37( 1): 12-29.[citado 2026 abr. 30 ] Available from: https://doi.org/10.1007/s12640-019-00109-8
Artigo de periodico
CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects (2020)
González-Lizárraga, Florencia; Ploper, Diego; Ávila, César L.; Socías, Sergio B.; Pereira, Mauricio dos Santos; Machín, Belén; Del Bel, Elaine Aparecida ; Michel, Patrick Pierre; Pietrasanta, Lía I.; Raisman-Vozari, Rita; Chehín, Rosana
Source: Scientific Reports. Unidade: FORP
Subjects: REPOSICIONAMENTO DE FÁRMACOS, INFLAMAÇÃO, DOENÇA DE PARKINSON, FARMACOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZÁLEZ-LIZÁRRAGA, Florencia et al. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects. Scientific Reports, v. 10, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-76927-0. Acesso em: 30 abr. 2026.
APA
González-Lizárraga, F., Ploper, D., Ávila, C. L., Socías, S. B., Pereira, M. dos S., Machín, B., et al. (2020). CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects. Scientific Reports, 10( 1). doi:10.1038/s41598-020-76927-0
NLM
González-Lizárraga F, Ploper D, Ávila CL, Socías SB, Pereira M dos S, Machín B, Del Bel EA, Michel PP, Pietrasanta LI, Raisman-Vozari R, Chehín R. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2026 abr. 30 ] Available from: https://doi.org/10.1038/s41598-020-76927-0
Vancouver
González-Lizárraga F, Ploper D, Ávila CL, Socías SB, Pereira M dos S, Machín B, Del Bel EA, Michel PP, Pietrasanta LI, Raisman-Vozari R, Chehín R. CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects [Internet]. Scientific Reports. 2020 ; 10( 1):[citado 2026 abr. 30 ] Available from: https://doi.org/10.1038/s41598-020-76927-0
Artigo de periodico
Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates (2019)
Acuña, Leonardo; Hamadat, Sabah; Corbalán, Natalia S.; González-Lizárraga, Florencia; Pereira, Maurício dos Santos ; Rocca, Jérémy; Díaz, Julia Sepúlveda; Del-Bel, Elaine Aparecida; Papy-García, Dulce; Chehín, Rosana N.; Michel, Patrick P.; Raisman-Vozari, Rita
Source: Cells. Unidade: FORP
Subjects: DOENÇA DE PARKINSON, INFLAMAÇÃO, CITOCINAS, NEURÔNIOS, ANTIBIÓTICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ACUÑA, Leonardo et al. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates. Cells, v. 8, n. 8, p. [17] , 2019Tradução . . Disponível em: https://doi.org/10.3390/cells8080776. Acesso em: 30 abr. 2026.
APA
Acuña, L., Hamadat, S., Corbalán, N. S., González-Lizárraga, F., Pereira, M. dos S., Rocca, J., et al. (2019). Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates. Cells, 8( 8), [17] . doi:10.3390/cells8080776
NLM
Acuña L, Hamadat S, Corbalán NS, González-Lizárraga F, Pereira M dos S, Rocca J, Díaz JS, Del-Bel EA, Papy-García D, Chehín RN, Michel PP, Raisman-Vozari R. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates [Internet]. Cells. 2019 ; 8( 8): [17] .[citado 2026 abr. 30 ] Available from: https://doi.org/10.3390/cells8080776
Vancouver
Acuña L, Hamadat S, Corbalán NS, González-Lizárraga F, Pereira M dos S, Rocca J, Díaz JS, Del-Bel EA, Papy-García D, Chehín RN, Michel PP, Raisman-Vozari R. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates [Internet]. Cells. 2019 ; 8( 8): [17] .[citado 2026 abr. 30 ] Available from: https://doi.org/10.3390/cells8080776
Tese (Doutorado)
Análise de fatores inflamatórios na discinesia induzida por L-DOPA em modelo de camundongos: caracterização da enzima Ciclooxigenase 2 (2017)
Pereira, Maurício dos Santos; Guimarães, Elaine Aparecida Del Bel Belluz (Orientador)
Unidade: FMRP
Subjects: DISCINESIAS, DOENÇA DE PARKINSON, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Maurício dos Santos. Análise de fatores inflamatórios na discinesia induzida por L-DOPA em modelo de camundongos: caracterização da enzima Ciclooxigenase 2. 2017. Tese (Doutorado) – Universidade de São Paulo, Ribeirão Preto, 2017. Disponível em: https://teses.usp.br/teses/disponiveis/17/17134/tde-24042018-181827/. Acesso em: 30 abr. 2026.
APA
Pereira, M. dos S. (2017). Análise de fatores inflamatórios na discinesia induzida por L-DOPA em modelo de camundongos: caracterização da enzima Ciclooxigenase 2 (Tese (Doutorado). Universidade de São Paulo, Ribeirão Preto. Recuperado de https://teses.usp.br/teses/disponiveis/17/17134/tde-24042018-181827/
NLM
Pereira M dos S. Análise de fatores inflamatórios na discinesia induzida por L-DOPA em modelo de camundongos: caracterização da enzima Ciclooxigenase 2 [Internet]. 2017 ;[citado 2026 abr. 30 ] Available from: https://teses.usp.br/teses/disponiveis/17/17134/tde-24042018-181827/
Vancouver
Pereira M dos S. Análise de fatores inflamatórios na discinesia induzida por L-DOPA em modelo de camundongos: caracterização da enzima Ciclooxigenase 2 [Internet]. 2017 ;[citado 2026 abr. 30 ] Available from: https://teses.usp.br/teses/disponiveis/17/17134/tde-24042018-181827/

USP Schools

ReP

Filtros

Authors

USP affiliated authors

Date published

Subjects

Source title

Funding Agencies

Indexado em

Non normalized affiliation