Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease (2021)
- Authors:
- USP affiliated authors: PEREIRA, MAURÍCIO DOS SANTOS - FORP ; GUIMARAES, ELAINE APARECIDA DEL BEL BELLUZ - FORP
- Unidade: FORP
- DOI: 10.3389/fphar.2020.617085
- Subjects: ASTROCITOMA; CANABINOIDES; GLUTAMATOS
- Keywords: IL-1β; Astrocyte; Cannabidiol; Dopamine; Glutamate; Microglia; Neuroinflammation; TNF-α
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Pharmacology
- ISSN: 1663-9812
- Volume/Número/Paginação/Ano: v. 11, art. 617085, p. 1-16, 2021
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
PEREIRA, Maurício dos Santos et al. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, v. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2020.617085. Acesso em: 28 dez. 2025. -
APA
Pereira, M. dos S., Abreu, G. H. D., Rocca, J., Hamadat, S., Raisman-Vozari, R., Michel, P. P., & Del Bel, E. A. (2021). Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, 11, 1-16. doi:10.3389/fphar.2020.617085 -
NLM
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2025 dez. 28 ] Available from: https://doi.org/10.3389/fphar.2020.617085 -
Vancouver
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2025 dez. 28 ] Available from: https://doi.org/10.3389/fphar.2020.617085 - Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates
- The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress
- CMT-3 targets different α-synuclein aggregates mitigating their toxic and inflammogenic effects
- Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of Parkinson's disease
- Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption
- Análise de fatores inflamatórios na discinesia induzida por L-DOPA em modelo de camundongos: caracterização da enzima Ciclooxigenase 2
- Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex
- Caffeine improves GABA transport in the striatum of Spontaneously Hypertensive Rats (SHR)
- Doxycycline attenuates l-DOPA-induced dyskinesia through an anti-inflammatory effect in a hemiparkinsonian mouse model
- Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia
Informações sobre o DOI: 10.3389/fphar.2020.617085 (Fonte: oaDOI API)
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