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Artigo de periodico
Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage (2022)
Pinto, Ana Paula ; Rocha, Alisson Luiz da ; Marafon, Bruno Brieda ; Nogueira, Jonatas Evandro ; Branco, Luiz Guilherme de Siqueira ; Pauli, José Rodrigo; Moura, Leandro Pereira de; Cintra, Dennys Esper; Ropelle, Eduardo Rochete; Silva, Adelino Sanchez Ramos da
Fonte: Clinical and Experimental Pharmacology and Physiology. Unidades: EEFERP, FORP, FMRP
Assuntos: DEGRANULAÇÃO CELULAR, INFLAMAÇÃO, INTERLEUCINA 6, EXERCÍCIO FÍSICO
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ABNT
PINTO, Ana Paula et al. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage. Clinical and Experimental Pharmacology and Physiology, v. 49, n. 8, p. 893-902, 2022Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.13677. Acesso em: 25 jun. 2024.
APA
Pinto, A. P., Rocha, A. L. da, Marafon, B. B., Nogueira, J. E., Branco, L. G. de S., Pauli, J. R., et al. (2022). Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage. Clinical and Experimental Pharmacology and Physiology, 49( 8), 893-902. doi:10.1111/1440-1681.13677
NLM
Pinto AP, Rocha AL da, Marafon BB, Nogueira JE, Branco LG de S, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage [Internet]. Clinical and Experimental Pharmacology and Physiology. 2022 ; 49( 8): 893-902.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1111/1440-1681.13677
Vancouver
Pinto AP, Rocha AL da, Marafon BB, Nogueira JE, Branco LG de S, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage [Internet]. Clinical and Experimental Pharmacology and Physiology. 2022 ; 49( 8): 893-902.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1111/1440-1681.13677
Artigo de periodico
Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats (2022)
Amorim, Mateus Ramos ; Jesus, Aline Alves de; Santos Junior, Nilton Nascimento dos ; Rocha, Maria José Alves da ; Nogueira, Jonatas Evandro ; Batalhão, Marcelo Eduardo; Cárnio, Evelin Capellari ; Branco, Luiz Guilherme de Siqueira
Fonte: Inflammation. Unidades: FORP, EEFERP, EERP, FMRP
Assuntos: CECO, CHOQUE SÉPTICO, ENDOTOXINAS, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, ESTRESSE OXIDATIVO
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ABNT
AMORIM, Mateus Ramos et al. Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats. Inflammation, v. 45, n. 3, p. 1239-1253, 2022Tradução . . Disponível em: https://doi.org/10.1007/s10753-021-01617-6. Acesso em: 25 jun. 2024.
APA
Amorim, M. R., Jesus, A. A. de, Santos Junior, N. N. dos, Rocha, M. J. A. da, Nogueira, J. E., Batalhão, M. E., et al. (2022). Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats. Inflammation, 45( 3), 1239-1253. doi:10.1007/s10753-021-01617-6
NLM
Amorim MR, Jesus AA de, Santos Junior NN dos, Rocha MJA da, Nogueira JE, Batalhão ME, Cárnio EC, Branco LG de S. Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats [Internet]. Inflammation. 2022 ; 45( 3): 1239-1253.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1007/s10753-021-01617-6
Vancouver
Amorim MR, Jesus AA de, Santos Junior NN dos, Rocha MJA da, Nogueira JE, Batalhão ME, Cárnio EC, Branco LG de S. Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats [Internet]. Inflammation. 2022 ; 45( 3): 1239-1253.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1007/s10753-021-01617-6
Artigo de periodico
Molecular hydrogen downregulates acute exhaustive exercise-induced skeletal muscle damage (2021)
Nogueira, Jonatas Evandro ; Amorim, Mateus Ramos ; Pinto, Ana Paula ; Rocha, Alisson Luiz da ; Silva, Adelino Sanchez Ramos da ; Branco, Luiz Guilherme de Siqueira
Fonte: Canadian Journal of Physiology and Pharmacology. Unidades: EEFERP, FORP, FMRP
Assuntos: ANIMAIS, ANTI-INFLAMATÓRIOS, ANTIOXIDANTES, INFLAMAÇÃO, ESTRESSE OXIDATIVO, RATOS
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ABNT
NOGUEIRA, Jonatas Evandro et al. Molecular hydrogen downregulates acute exhaustive exercise-induced skeletal muscle damage. Canadian Journal of Physiology and Pharmacology, v. 99, n. 8, p. 812-820, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2020-0297. Acesso em: 25 jun. 2024.
APA
Nogueira, J. E., Amorim, M. R., Pinto, A. P., Rocha, A. L. da, Silva, A. S. R. da, & Branco, L. G. de S. (2021). Molecular hydrogen downregulates acute exhaustive exercise-induced skeletal muscle damage. Canadian Journal of Physiology and Pharmacology, 99( 8), 812-820. doi:10.1139/cjpp-2020-0297
NLM
Nogueira JE, Amorim MR, Pinto AP, Rocha AL da, Silva ASR da, Branco LG de S. Molecular hydrogen downregulates acute exhaustive exercise-induced skeletal muscle damage [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 8): 812-820.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1139/cjpp-2020-0297
Vancouver
Nogueira JE, Amorim MR, Pinto AP, Rocha AL da, Silva ASR da, Branco LG de S. Molecular hydrogen downregulates acute exhaustive exercise-induced skeletal muscle damage [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 8): 812-820.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1139/cjpp-2020-0297
Artigo de periodico
Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss (2020)
Amorim, Mateus Ramos; Moreira, Diego Azevedo; Santos, Bruna Maitan; Ferrari, Gustavo Duarte; Nogueira, Jonatas Evandro ; Deus, Júnia Lara de; Alberici, Luciane Carla; Branco, Luiz Guilherme de Siqueira
Fonte: The Journal of Physiology. Unidades: EEFERP, FCFRP, FORP, FMRP
Assuntos: HIPOTERMIA, LIPOPOLISSACARÍDEOS, PRESSÃO SANGUÍNEA, TEMPERATURA CORPORAL, HIPERTENSÃO, SEPSE
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ABNT
AMORIM, Mateus Ramos et al. Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss. The Journal of Physiology, v. 598, n. 20, p. 4663-4680, 2020Tradução . . Disponível em: https://doi.org/10.1113/JP280321. Acesso em: 25 jun. 2024.
APA
Amorim, M. R., Moreira, D. A., Santos, B. M., Ferrari, G. D., Nogueira, J. E., Deus, J. L. de, et al. (2020). Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss. The Journal of Physiology, 598( 20), 4663-4680. doi:10.1113/JP280321
NLM
Amorim MR, Moreira DA, Santos BM, Ferrari GD, Nogueira JE, Deus JL de, Alberici LC, Branco LG de S. Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss [Internet]. The Journal of Physiology. 2020 ; 598( 20): 4663-4680.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1113/JP280321
Vancouver
Amorim MR, Moreira DA, Santos BM, Ferrari GD, Nogueira JE, Deus JL de, Alberici LC, Branco LG de S. Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss [Internet]. The Journal of Physiology. 2020 ; 598( 20): 4663-4680.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1113/JP280321
Artigo de periodico
Inhaled molecular hydrogen attenuates intense acute exercise-induced hippocampal inflammation in sedentary rats (2020)
Nogueira, Jonatas Evandro ; Deus, Júnia Lara de; Amorim, Mateus Ramos; Batalhão, Marcelo Eduardo; Leão, Ricardo Maurício Xavier; Cárnio, Evelin Capellari ; Branco, Luiz Guilherme de Siqueira
Fonte: Neuroscience Letters. Unidades: EEFERP, EERP, FMRP, FORP
Assuntos: EXERCÍCIO FÍSICO, HIPOCAMPO, CITOCINAS, ESTRESSE OXIDATIVO, MODELOS ANIMAIS DE DOENÇAS
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ABNT
NOGUEIRA, Jonatas Evandro et al. Inhaled molecular hydrogen attenuates intense acute exercise-induced hippocampal inflammation in sedentary rats. Neuroscience Letters, v. 715, p. 1-7, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2019.134577. Acesso em: 25 jun. 2024.
APA
Nogueira, J. E., Deus, J. L. de, Amorim, M. R., Batalhão, M. E., Leão, R. M. X., Cárnio, E. C., & Branco, L. G. de S. (2020). Inhaled molecular hydrogen attenuates intense acute exercise-induced hippocampal inflammation in sedentary rats. Neuroscience Letters, 715, 1-7. doi:10.1016/j.neulet.2019.134577
NLM
Nogueira JE, Deus JL de, Amorim MR, Batalhão ME, Leão RMX, Cárnio EC, Branco LG de S. Inhaled molecular hydrogen attenuates intense acute exercise-induced hippocampal inflammation in sedentary rats [Internet]. Neuroscience Letters. 2020 ; 715 1-7.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1016/j.neulet.2019.134577
Vancouver
Nogueira JE, Deus JL de, Amorim MR, Batalhão ME, Leão RMX, Cárnio EC, Branco LG de S. Inhaled molecular hydrogen attenuates intense acute exercise-induced hippocampal inflammation in sedentary rats [Internet]. Neuroscience Letters. 2020 ; 715 1-7.[citado 2024 jun. 25 ] Available from: https://doi.org/10.1016/j.neulet.2019.134577
Artigo de periodico
Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production (2017)
Nogueira, Jonatas Evandro; Soriano, Renato Nery; Fernández, Rodrigo Alberto Restrepo; Francescato, Heloísa Della Coletta; Saia, Rafael Simone; Coimbra, Terezila Machado; Antunes-Rodrigues, José; Branco, Luiz Guilherme de Siqueira
Fonte: PLOS ONE. Unidades: EEFERP, FMRP, FORP
Assuntos: REGULAÇÃO DA TEMPERATURA CORPORAL, CITOCINAS, FEBRE, CONDICIONAMENTO FÍSICO, ÁREA PREÓPTICA DE ANIMAL
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ABNT
NOGUEIRA, Jonatas Evandro et al. Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production. PLOS ONE, v. 12, n. 1, 2017Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0170468. Acesso em: 25 jun. 2024.
APA
Nogueira, J. E., Soriano, R. N., Fernández, R. A. R., Francescato, H. D. C., Saia, R. S., Coimbra, T. M., et al. (2017). Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production. PLOS ONE, 12( 1). doi:10.1371/journal.pone.0170468
NLM
Nogueira JE, Soriano RN, Fernández RAR, Francescato HDC, Saia RS, Coimbra TM, Antunes-Rodrigues J, Branco LG de S. Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production [Internet]. PLOS ONE. 2017 ; 12( 1):[citado 2024 jun. 25 ] Available from: https://doi.org/10.1371/journal.pone.0170468
Vancouver
Nogueira JE, Soriano RN, Fernández RAR, Francescato HDC, Saia RS, Coimbra TM, Antunes-Rodrigues J, Branco LG de S. Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production [Internet]. PLOS ONE. 2017 ; 12( 1):[citado 2024 jun. 25 ] Available from: https://doi.org/10.1371/journal.pone.0170468

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