Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage (2022)
- Authors:
- USP affiliated authors: NOGUEIRA, JONATAS EVANDRO - EEFERP ; BRANCO, LUIZ GUILHERME DE SIQUEIRA - FORP ; SILVA, ADELINO SANCHEZ RAMOS DA - EEFERP ; PINTO, ANA PAULA - EEFERP ; ROCHA, ALISSON LUIZ DA - FMRP ; MARAFON, BRUNO BRIEDA - EEFERP
- Unidades: EEFERP; FORP; FMRP
- DOI: 10.1111/1440-1681.13677
- Subjects: DEGRANULAÇÃO CELULAR; INFLAMAÇÃO; INTERLEUCINA 6; EXERCÍCIO FÍSICO
- Keywords: Autophagy; Exercise; Liver; mTORC1; Rapamycin
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Clinical and Experimental Pharmacology and Physiology
- ISSN: 0305-1870
- Volume/Número/Paginação/Ano: v. 49, n. 8, p. 893-902, 2022
- Status:
- Nenhuma versão em acesso aberto identificada
-
ABNT
PINTO, Ana Paula et al. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage. Clinical and Experimental Pharmacology and Physiology, v. 49, n. 8, p. 893-902, 2022Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.13677. Acesso em: 01 abr. 2026. -
APA
Pinto, A. P., Rocha, A. L. da, Marafon, B. B., Nogueira, J. E., Branco, L. G. de S., Pauli, J. R., et al. (2022). Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage. Clinical and Experimental Pharmacology and Physiology, 49( 8), 893-902. doi:10.1111/1440-1681.13677 -
NLM
Pinto AP, Rocha AL da, Marafon BB, Nogueira JE, Branco LG de S, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage [Internet]. Clinical and Experimental Pharmacology and Physiology. 2022 ; 49( 8): 893-902.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1111/1440-1681.13677 -
Vancouver
Pinto AP, Rocha AL da, Marafon BB, Nogueira JE, Branco LG de S, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage [Internet]. Clinical and Experimental Pharmacology and Physiology. 2022 ; 49( 8): 893-902.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1111/1440-1681.13677 - Molecular hydrogen downregulates acute exhaustive exercise-induced skeletal muscle damage
- Muscle endoplasmic reticulum stress in exercise
- Hepatic LC3 II/I ratio is not modulated in exercised mice
- The combination of fasting, acute resistance exercise, and protein ingestion led to different responses of autophagy markers in gastrocnemius and liver samples
- Moderate, but not excessive, training attenuates autophagy machinery in metabolic tissues
- Impact of different physical exercises on the expression of autophagy markers in mice
- One bout of aerobic exercise can enhance the expression of Nr1d1 in oxidative skeletal muscle samples
- Recent advances in molecular hydrogen research reducing exercise-induced oxidative stress and inflammation
- The proinflammatory effects of chronic excessive exercise
- Role of TLR4 in physical exercise and cardiovascular diseases
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